ライフサイエンスコーパス: gonococcus

1 being previously thought to be absent in the gonococcus.

2 may also play a role in the survival of the gonococcus.

3 etence, and epithelial cell adherence of the gonococcus.

4 actually influence DNA transformation of the gonococcus.

5 porA in some meningococci was absent in the gonococcus.

6 Gonococcus, a bacterium resistant to most antibiotics, c

7 i functioned for the counterselection in the gonococcus, albeit with low efficiency.

8 the recombination-dependent processes of the gonococcus and further define the pathways of homologous

9 d contributed to the emergence of PCD in the gonococcus and meningococcus and potentially to the appe

10 orA was acquired by a common ancestor of the gonococcus and meningococcus but inactivated in the gono

11 p island, which is largely restricted to the gonococcus and meningococcus.

12 e fusion with pedestal formation between the gonococcus and the epithelial cell, gonococci within vac

13 irst major discriminating factor between the gonococcus and the other Neisseria and carries genes for

14 arbored by commensals and transferred to the gonococcus, and discuss the impact of contemporary selec

15 biotic resistance, or when such pathogens as gonococcus are being considered.

16 We show that actin footprints are induced in gonococcus-challenged primary urethral epithelium.

17 The nap island in the gonococcus encodes 3 cationic, bacteriocin-like peptides

18 ercome several subversive mechanisms whereby gonococcus evades complement, including binding to C4b-b

19 ABC transport systems are functional in the gonococcus for the acquisition of iron.

20 DE and FarAB-MtrE efflux systems protect the gonococcus from hydrophobic antimicrobial substances tha

21 redundant defense mechanisms may protect the gonococcus from reactive oxygen species during infection

22 The pilus of Neisseria gonorrhoeae (the gonococcus Gc), the causative agent of gonorrhoea, promo

23 chnique to create tandem duplications in the gonococcus (Gc) genome.

24 stigated the effect of Neisseria gonorrheae (gonococcus [GC]) exposure on HIV replication in primary

25 The Neisseria gonorrhoeae (the gonococcus [Gc]) opacity-associated (Opa) proteins media

26 ea (Neisseria gonorrhoeae, also known as the gonococcus [GC]), and chlamydia (Chlamydia trachomatis).

27 Neisseria gonorrhoeae (gonococcus [GC]), is highly adapted to the human host, t

28 oaches for confirming Neisseria gonorrhoeae (gonococcus [GC])-positive nucleic acid amplification tes

29 block of the pilus of Neisseria gonorrhoeae (gonococcus [GC]).

30 Phagocytosis of Opa+ Neisseria gonorrhoeae (gonococcus, GC) by neutrophils is in part dependent on t

31 enic variation in Neisseria gonorrhoeae (the gonococcus, Gc) is regulated by iron availability.

32 Neisseria gonorrhoeae (the gonococcus, Gc) is the causative agent of gonorrhoea, an

33 Neisseria gonorrhoeae (the gonococcus, Gc) triggers a potent inflammatory response

34 mutate the genome of Neisseria gonorrhoeae (gonococcus; Gc).

35 urvive this interaction, suggesting that the gonococcus has evolved many defenses against PMN killing

36 cell-sorted to identify antibodies that kill gonococcus in a bactericidal assay.

37 in the level of inhibitory activity between gonococcus-infected and noninfected patients in either c

38 were not elevated in genital secretions from gonococcus-infected compared with uninfected patients.

39 ristic of IgA1 protease activity was seen in gonococcus-infected or control patients.

40 The level of serum IL-6 was higher in gonococcus-infected than in uninfected patients at recru

41 The subject with sialylated gonococcus infection had an extended incubation period,

42 dies provided protection in a mouse model of gonococcus infection.

43 n response to Neisseria gonorrhoeae (GC, for gonococcus) infection and that conditioned medium from G

44 This raises the question of how the gonococcus infects men if N-acetyllactosamine residues a

45 data indicate that PLD augments CR3-mediated gonococcus invasion of and survival within cervical epit

46 Despite the fact that the gonococcus is not normally exposed to UV irradiation or

47 Neisseria gonorrhoeae (the gonococcus) is an obligate human pathogen and the causat

48 Neisseria gonorrhoeae (the gonococcus) is the causative agent of the sexually trans

49 specific pathogen Neisseria gonorrhoeae (the gonococcus), is characterized by the influx of polymorph

50 chocystis sp. and, like those organisms, the gonococcus lacks a norC homolog.

51 tion to the genitourinary niche, making them gonococcus-like and distinguishing them from other N. me

52 e data suggest that biofilm formation by the gonococcus may represent a response that is linked to th

53 cus and meningococcus but inactivated in the gonococcus on speciation.

54 al the extent of plasmid-mediated AMR in the gonococcus, particularly in less wealthy countries, wher

55 ed to share genetic characteristics with the gonococcus, raising the question of the extent to which

56 aim is to articulate a shared agenda across gonococcus-related fields from microbiology to epidemiol

57 o slow the increasing incidence of resistant gonococcus require insight into the factors that contrib

58 Lack of PorA expression in the gonococcus resulted from mutations in the promoter regio

59 identified a locus that, when mutated in the gonococcus, results in a significant increase in toxicit

60 tance to reinfection, with the generation of gonococcus-specific circulating immunoglobulin G and vag

61 he human pathogen Neisseria gonorrhoeae (the gonococcus), the pathways of homologous recombination ar

62 in the recombination-based processes of the gonococcus, these genes were inactivated in the N. gonor

63 nd benefit associated with resistance of the gonococcus to a clinically relevant antibiotic.

64 subsequently mutagenized and returned to the gonococcus to generate a Kat- strain that was phenotypic

65 roposed to function in the adaptation of the gonococcus to host environments.

66 res a regulatory response on the part of the gonococcus to respond to this significant change in envi

67 nt of gonorrhoea, promotes attachment of the gonococcus to the host epithelium and is essential for t

68 endent ABC transport system that enables the gonococcus to transport iron into the cell subsequent to

69 unique features of ihf transcription in the gonococcus which questions whether certain aspects if ih

70 ignificant effect in determining whether the gonococcus will be able to successfully establish an inf

71 ous work has identified several genes in the gonococcus with significant identity to the pilin invers