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1 ) and depletion of Foxp3 Treg abrogates skin-graft tolerance.
2 TGF)-beta within islets to achieve long-term graft tolerance.
3 ritical for the induction and maintenance of graft tolerance.
4  plus anti-CD40L antibodies does not disrupt graft tolerance.
5 ty could be used to promote the induction of graft tolerance.
6 peutically to synergize in the generation of graft tolerance.
7 instill long-lived allogeneic and xenogeneic graft tolerance.
8 be in induction of a chimeric state to allow graft tolerance.
9 nological responses and aid the induction of graft tolerance.
10 , which may contribute to the maintenance of graft tolerance.
11  of each treatment in achieving maximal skin graft tolerance.
12 blood of patients that spontaneously develop graft tolerance.
13 ale-reactive T-cell population and permanent graft tolerance.
14 sorders, including cancer, autoimmunity, and graft tolerance.
15 us BMTx improves graft survival and promotes graft tolerance.
16 latory functions that contribute to specific graft tolerance.
17 D8(+) T cells critical to the development of graft tolerance.
18 mopoietic cells to promote induction of skin graft tolerance across full MHC barriers.
19 or cell infusions can be used to induce skin graft tolerance across MHC barriers, accompanied by spec
20 ological malignancies and promoting lifelong graft tolerance after solid organ transplantation.
21 venous injection of soluble beta-gal-induced graft tolerance and a lack of detectable beta-gal-specif
22 l of CD4 reconstitution correlated with skin graft tolerance and an absence of induced anti-donor xen
23  associated with optimal donor-specific skin graft tolerance and avoidance of the anti-donor xenoanti
24         Highly disparate xenogeneic pig skin graft tolerance and efficient repopulation of mouse CD4+
25  been proposed that this chimerism may imply graft tolerance and permit withdrawal of immunosuppressi
26 gs with anti-CD25 antibodies prevented islet graft tolerance and resulted in rejection.
27         How these subsets also contribute to graft tolerance and the protection of chronically immuno
28 lineage mixed chimerism; donor-specific skin-graft tolerance; and in vitro tolerance were observed in
29 nse was associated with, but did not insure, graft tolerance, as the inopportune timing of B7 blockad
30 rved in LTRs withdrawn from SRL and if blood/graft tolerance biomarkers were predictive of successful
31 erved in LTR withdrawn from SRL and if blood/graft tolerance biomarkers were predictive of successful
32         Highly disparate xenogeneic pig skin graft tolerance can be achieved by grafting FP THY alone
33 nd allowed 60% of treated animals to develop graft tolerance (>120 days), when donor sMHC were combin
34  treatment in this model to achieve pig skin graft tolerance have not previously been defined.
35 n of mixed chimerism and donor-specific skin graft tolerance in 3 Gy-irradiated mice receiving fully
36 ion facilitates macrochimerism induction and graft tolerance in a mouse skin transplantation model.
37 e early alloimmune response) leads to robust graft tolerance in a purely alloimmune setting and prolo
38 3 Gy TBI is not essential for donor pig skin graft tolerance induction.
39 apoptosis is required for the development of graft tolerance, induction strategies that use IL-2-inde
40 rt in the presence of competent T(reg), then graft tolerance is lost.
41               In a model of T(reg)-dependent graft tolerance, it is shown that GZB- deficient mice ar
42  of "split tolerance" characterized by local graft tolerance mediated by donor regulatory T cells (Tr
43 ecific microchimerism in graft acceptance or graft tolerance remains to be elucidated.
44                          Donor-specific skin graft tolerance was evaluated, and CD4 reconstitution an
45                         However, spontaneous graft tolerance was restored by parking the irradiated L
46      Using a CD4(+) TCR transgenic model for graft tolerance, we have unveiled the independent contri
47                     Mixed chimerism and skin graft tolerance were achieved in NOD mice receiving anti
48 erism induces life-long donor-specific organ graft tolerance while obviating the need for chronic imm
49 hat can provide immunomodulation, supporting graft tolerance, while minimizing the need for immunosup