ライフサイエンスコーパス: granulation

1 c myelocytes that showed abnormal basophilic granulation.

2 the surface of the Sun-a phenomenon known as granulation.

3 unds which provide wound drainage and tissue granulation.

4 poor re-epithelialization, angiogenesis and granulation.

5 s of key parameters such as inflammation and granulation.

6 location and topographical correspondence of granulations.

7 enting the mouse equivalent of the arachnoid granulations.

8 y for lactic acid production from OFIC(350) (granulation 350 um) hydrolysate and OFIFP extract withou

9 Arachnoid granulations (AG) are poorly investigated.

10 bone, which arise as the result of arachnoid granulations' (AG) protruding and causing the resorption

11 model of CSF outflow across human arachnoid granulations (AGs) as an approximation of in vivo condit

12 tion (2-3 weeks after insertion to allow for granulation and fixation of viscera) and formation of th

13 nd on the type of formulating activity, with granulation and mixing being most prone to API losses.

14 ewly grown microvessels resembled vessels of granulation and neoplastic tissue in many aspects.

15 thelialization, contraction and formation of granulation and scar tissue.

16 Intimal granulations and lipid wall bubbles were also significan

17 samples obtained by traditional moulding and granulation, and also via a novel spray-drying powdering

18 ng dispersal, phototaxis, biofilm formation, granulation, and symbiosis, is explored.

19 conditions and influent composition for CaP granulation are still unknown.

20 gradation and dissolution loss of LISs after granulation, as well as the low Li(+) concentration in s

21 ipid profile, oxidative stress, carotid IMT, granulations at the IM layer and near-wall low density b

22 The pattern and amount of collagen-rich granulation bed tissue, manufactured by fibroblasts, was

23 0.05) in the SM+BP group (as for the intimal granulation/bubbles) with minimal variations in the comp

24 on, interstitial fibrosis, desquamation, and granulation by an experienced pulmonary pathologist.

25 e particles enhanced reepithelialization and granulation, by 2- and 3-fold respectively, when compare

26 ated with respect to their botanical origin, granulation, colour and sensory properties.

27 Granulation, colour, and sensory properties of honey wer

28 se in wound healing (reepithelialization and granulation) compared to the wild-type control.

29 as longer in subjects with lesser degrees of granulation/connective tissue deposition (fibroblastic f

30 res of IPF, i.e., fibrosis, cellularity, the granulation/connective tissue deposition, and the total

31 Secretion occurs through de-granulation during post-infective development, and the p

32 aphy, we also directly observe dilatancy and granulation effects, which lead to fracture above a crit

33 a model using human arachnoid membrane with granulations for the study of conditions such as Alzheim

34 nflammation, epidermis re-epithelialization, granulation formation, and proper wound healing in mice.

35 ions on skull radiographs, seen as arachnoid granulations fovea in CT.

36 Fibroblasts of healthy and granulation gingiva are phenotypically heterogeneous wit

37 ferences may affect activities of normal and granulation gingiva.

38 uch as cell proliferation, angiogenesis, and granulation growth were investigated.

39 in ANAMMOX-mediated systems, focusing on (i) granulation; (ii) biofilm formation on carrier materials

40 defies typically postulated requirements for granulation in biotechnology, i.e., the need for hydrody

41 ements can be used to probe the processes of granulation in concentrated colloidal suspensions.

42 o the mouse Ren1d-null background, restoring granulation in juxtaglomerular cells.

43 er in the arachnoid membrane adjacent to the granulations, in addition to the flow through the AGs.

44 Notably, we also identified arachnoid granulations involved in cerebrospinal fluid resorption,

45 promotes quench granulation, suggesting that granulation is modulated by heterogeneous stress fields

46 The brain's arachnoid membrane with granulations is an important biological barrier whose re

47 tion, and that it reduces vascularization of granulation issue, probably through disabling of the sho

48 inhibiting cell proliferation, angiogenesis, granulation maturation, collagen deposition, and muscula

49 te granulocytic differentiation with profuse granulation, mature, clumped chromatin, and myeloperoxid

50 Anterior lamellar repair was by granulation (n = 2), local skin flaps (n = 3), or skin g

51 red by vacuum drying, freeze drying or spray granulation of aqueous mixtures of omega-3 oil and beta-

52 The Fourier power of granulation on a star's surface correlates physically wi

53 decreased CSF reabsorption via the arachnoid granulations or other pathways.

54 In addition, granulation phase resolution is delayed, with persistent

55 ibrations can be used to control jamming and granulation, resulting in a flowable fluid.

56 s prevents PRD formation and promotes quench granulation, suggesting that granulation is modulated by

57 variations on timescales of hours arise from granulation, then such variations should correlate with

58 ocardium, acutely cryoinjured myocardium, or granulation tissue (6 days after injury).

59 sis, were occluded with mucus (syngeneic) or granulation tissue (allogeneic).

60 -4.13) and with risk of developing laryngeal granulation tissue (p = 0.02).

61 e assessment of wound area and percentage of granulation tissue (PGT) are important for optimizing wo

62 ounds curetted on day 5 were 23% filled with granulation tissue 1 day later and 99% filled 3 days lat

63 Fbln5 is upregulated in the granulation tissue 14 days after full-thickness wounding

64 We identified wound granulation tissue 3 days post-CD in both strains, albei

65 afts demonstrated significantly less lumenal granulation tissue 35.3%+/-32 than the allograft implant

66 roup demonstrated significantly less lumenal granulation tissue 48.3%+/-23.7 when compared with the i

67 kinking managed intraoperatively (0% vs 7%), granulation tissue after 12 weeks (1% vs 11%), and sutur

68 histologic response (three of four with >95% granulation tissue and <5% necrosis, one of four with 95

69 the number of myofibroblasts present in the granulation tissue and accelerates wound closure and con

70 inases are rapidly induced in the dermis and granulation tissue and at the leading edge of the epider

71 alse-positive findings due to enhancement of granulation tissue and benign breast tissue remain limit

72 , cryopyrin, and caspase-1, localized to the granulation tissue and cardiomyocytes bordering the infa

73 reases in blood flow and permeability in rat granulation tissue and corresponding vascular changes in

74 n preventing the anisotropic organization of granulation tissue and delaying wound healing.

75 wiss mice resulted in a large stimulation of granulation tissue and fibrosis at the site of injection

76 of wound-site macrophages to fibroblasts in granulation tissue and impairment of such response in di

77 ontrol), especially in cells re-epithelizing granulation tissue and in mucosa in proximity to the ulc

78 Additionally, cells in the granulation tissue and keratinocytes at wound edges show

79 luid collections are surrounded by a wall of granulation tissue and may contain necrotic debris.

80 resorption, and extensive inflammation with granulation tissue and polymorphonuclear leukocytes.

81 serve as a "barrier," limiting expansion of granulation tissue and protecting the noninfarcted myoca

82 ration, and vessel formation to form a thick granulation tissue and re-epithelialization of the wound

83 9.5 were expressed by fibroblasts during the granulation tissue and remodeling phases wound healing.

84 ed transgene expression in myofibroblasts in granulation tissue and responsiveness to transforming gr

85 es of cutaneous wounding resulted in reduced granulation tissue and scarring.

86 Diode lasers can remove granulation tissue and submucosal bacteria, and if it ac

87 dified papilla preservation technique, where granulation tissue and visible calculus were removed wit

88 significantly and time-dependently affected granulation tissue appearance (P = 0.004).

89 ed wound closure, decreased inflammation and granulation tissue area, and normalized mechanical prope

90 Granulation tissue area, vascularity, and IGF1 and EGF r

91 rkable increase in the vascular component in granulation tissue as compared to Ad-LacZ controls.

92 chondrification centers and persisted within granulation tissue at the expanding soft callus front.

93 On postoperative day 7, the thickness of granulation tissue at the graft-wound bed interface was

94 r-BB protein (n = 2) resulted in only modest granulation tissue at the margin, but no significant dif

95 lized in the preliminary matrix organized in granulation tissue before trabecular bone formation in t

96 ross-linked collagenous matrix is formed and granulation tissue cells become apoptotic.

97 film infection in wounds results in impaired granulation tissue collagen leading to compromised wound

98 and platelet releasate were 14% filled with granulation tissue compared with less than 4% granulatio

99 concept that IL-1Ra modulates MCL-localized granulation tissue components and cytokine production to

100 ing by enhancing basement membrane proteins, granulation tissue components, and angiogenic factors.

101 mulating the M2 macrophages and altering the granulation tissue components.

102 he fibrin inside the chambers is replaced by granulation tissue consisting of new blood vessels, macr

103 examination revealed a characteristic cap of granulation tissue covering tortuous nondysplastic crypt

104 is a significant reduction in the extent of granulation tissue deposition and the subsequent formati

105 ing agent, enhancing reepithelialization and granulation tissue deposition by 64+/-5 and 83+/-12% ove

106 in response to injury, resulting in delayed granulation tissue deposition in PKCalpha-/- wounds.

107 analysis showed that epithelium ingrowth and granulation tissue deposition were significantly impaire

108 ithelialization, angiogenesis, inflammation, granulation tissue deposition, and enhanced collagen org

109 , malformed vasculature followed by abundant granulation tissue deposition.

110 thrombus deposition and acute inflammation, granulation tissue development, and ultimately smooth mu

111 repair a 3-day lag occurs between injury and granulation tissue development.

112 on, synthesize growth factors, and stimulate granulation tissue development.

113 Granulation tissue did not form in day 3 wounds, which h

114 exhibited impaired development of functional granulation tissue due to severely reduced differentiati

115 in vessels that developed in sponge-induced granulation tissue during 1 month derived from circulati

116 e is highly induced in dermal fibroblasts of granulation tissue during cutaneous wound repair.

117 the organization and vascularization of the granulation tissue during healing, possibly by modulatin

118 tly stimulates neovascularization within the granulation tissue during the first week of treatment, f

119 y and function in specific cell types in the granulation tissue during the healing process is unknown

120 Routinely discarded periodontal granulation tissue exhibits epithelial characteristics d

121 fter receiving the lower viral dose, cardiac granulation tissue expressed MyoD mRNA and protein, but

122 CTX also reduced dermal granulation tissue fibroblast population increases induc

123 Once granulation tissue filled the wound and invasive angioge

124 The granulation tissue filling the wound during healing also

125 increased keratinocyte migration (7.5-fold), granulation tissue formation (2.8-fold), cell proliferat

126 ology are currently limited by stent-induced granulation tissue formation adjacent to the stent.

127 using AuNP-coated SEMS successfully treated granulation tissue formation after stent placement in th

128 nsgenic mice and was associated with reduced granulation tissue formation and highly diminished wound

129 imulatory effect of overexpressed activin on granulation tissue formation and reepithelialization of

130 This work directly relates to both granulation tissue formation and regression during wound

131 d cardiomyocytes, while mediating aspects of granulation tissue formation and remodeling.

132 d results in accelerated healing and reduced granulation tissue formation and scarring.

133 into the fibrin-laden wound is critical for granulation tissue formation and subsequent healing.

134 into the fibrin-laden wound is critical for granulation tissue formation and subsequent healing.

135 e, or response to infection, but it promoted granulation tissue formation and suppressed leukocyte ne

136 analysis of skin wounds demonstrated delayed granulation tissue formation and vascularization during

137 cellular/ultrastructural studies and in vivo granulation tissue formation assays combined with transc

138 synthesis or action reduces TGF-beta-induced granulation tissue formation by inhibiting both collagen

139 GA-LL37 NP-treated wounds displayed advanced granulation tissue formation by significant higher colla

140 We initiated local granulation tissue formation either by implanting small

141 to the fibrin matrix significantly increased granulation tissue formation in a dose-dependent manner.

142 sient, role during invasive angiogenesis and granulation tissue formation in a healing wound.

143 on of miR-21 inhibited epithelialization and granulation tissue formation in a rat wound model.

144 onists of alphaVbeta3 specifically inhibited granulation tissue formation in a transient manner durin

145 pendent increase in epithelial migration and granulation tissue formation in both murine and porcine

146 ic blocking of alphavbeta3 function inhibits granulation tissue formation in cutaneous wounds.

147 complete re- epithelialization and profound granulation tissue formation in excisional and incisiona

148 Granulation tissue formation in punch wounds of juvenile

149 Lovastatin (5 microM, 8 d) reduced granulation tissue formation in the wound chambers by 64

150 the myocardial scar, suggesting expansion of granulation tissue formation into the noninfarcted terri

151 Granulation tissue formation is a critical step in infar

152 Granulation tissue formation is an example of new tissue

153 During early granulation tissue formation of wound repair, new capill

154 Thus, granulation tissue formation resumed promptly and indepe

155 ial cell chemotaxis, vascular sprouting, and granulation tissue formation upon skin injury, these act

156 ared to their wild-type littermates although granulation tissue formation was nonhomogeneous and ther

157 In addition, improved granulation tissue formation was observed along with hig

158 During granulation tissue formation, alphaVbeta3 was expressed

159 g was induced by rapid re-epithelialization, granulation tissue formation, and accompanied by angioge

160 uced, and careful analysis of wound closure, granulation tissue formation, and angiogenesis revealed

161 with compromised wound closure, insufficient granulation tissue formation, and blunted induction of M

162 fibroblast activation is a limiting step of granulation tissue formation, and continued cell stimula

163 t mice show suppressed inflammation, delayed granulation tissue formation, and markedly reduced colla

164 g delayed contraction, decreased and delayed granulation tissue formation, and reduced new blood vess

165 Wound closure, reepithelialization, granulation tissue formation, and remodeling were delaye

166 ed to accelerate wound re-epithelialization, granulation tissue formation, and synergistically improv

167 telets and macrophages, is not important for granulation tissue formation, and that it reduces vascul

168 are known to be associated with significant granulation tissue formation, and this property provides

169 l-thickness, cutaneous wounds, with enhanced granulation tissue formation, angiogenesis, cell prolife

170 The rate of granulation tissue formation, based on the time to 76-10

171 PO and observed dose-dependent inhibition of granulation tissue formation, consistent with an importa

172 ccelerated reepithelialization and increased granulation tissue formation, fibroblast migration, and

173 with reepithelialization and is followed by granulation tissue formation, including neutrophil and m

174 hese studies show that re-epithelialization, granulation tissue formation, including the establishmen

175 Granulation tissue formation-related variables were sign

176 Granulation tissue formation-related variables were sign

177 mal and steroid-impaired pigs, CRT increased granulation tissue formation.

178 ssociated with a proangiogenic effect during granulation tissue formation.

179 ridge formation, decreased inflammation, and granulation tissue formation.

180 ctivation might be the rate-limiting step in granulation tissue formation.

181 ayed wound re-epithelialization and impaired granulation tissue formation.

182 nduced in peri-infarct cardiomyocytes during granulation tissue formation.

183 cantly accelerated, resulting in the limited granulation tissue formation.

184 is study provided histological evidence that granulation tissue forming under clinically exposed and

185 In 14 and 21 d incised wounds and in chronic granulation tissue from nonhealing ulcers there was stro

186 sions, nail dystrophy and exuberant vascular granulation tissue in certain epithelia, especially conj

187 ranulation tissue compared with less than 4% granulation tissue in control wounds.

188 Furthermore, granulation tissue in fibrinogen-deficient mice failed t

189 at in wild-type mice, the early formation of granulation tissue in fibrinogen-deficient mice was edem

190 roximately 1600 mum within wound (neodermis)/granulation tissue in lesions made on the skin of mice.

191 by topical application of VEGF and FGF-2 to granulation tissue in skin chambers, and 2) suramin, a c

192 is caused by expansion of microvascular-rich granulation tissue in some locations and collagen-rich s

193 backs of Sprague-Dawley rats and 1 wk later, granulation tissue in the chamber was exposed twice dail

194 aryngeal injury (i.e., mucosal ulceration or granulation tissue in the larynx) was present in 57 pati

195 Some patients developed granulation tissue in the larynx, urethra, lacrimal duct

196 wounds, which corresponds with the increased granulation tissue in these wounds.

197 p-regulated in newly formed blood vessels of granulation tissue in vivo.

198 The prerequisites for granulation tissue induction are not known but hypotheti

199 ests angiogenesis with transformation of the granulation tissue into a scar.

200 ocal angiogenesis, and limiting expansion of granulation tissue into the noninfarcted area.

201 Granulation tissue is routinely discarded in periodontal

202 accelerated replacement of cardiomyocytes by granulation tissue leading to a thin mature scar at 14 d

203 processes including re-epithelialization and granulation tissue matrix deposition.

204 Vbeta3 showed little or no expression in the granulation tissue microvasculature.

205 Granulation tissue myofibroblasts and infiltrating macro

206 e recipient airway demonstrated less lumenal granulation tissue obstruction and better preservation o

207 Four patients with granulation tissue occluding the airway were treated wit

208 dy, we show that CCN3 is highly expressed in granulation tissue of cutaneous wounds 5-7 days after in

209 ntron was expressed in myofibroblasts within granulation tissue of cutaneous wounds in a pattern that

210 showed that, similar to TSP1-null mice, the granulation tissue of double-null mice was not excessive

211 margins of human keloid samples, and in the granulation tissue of newly deposited ECM in a mouse mod

212 ransgene expression in myofibroblasts within granulation tissue of skin wounds.

213 round the healing margins and throughout the granulation tissue of superficial ulcerative wounds.

214 cells of adjacent hair follicles, and to the granulation tissue of the wounds.

215 in processes involved in development of the granulation tissue of wounds, but little is known about

216 re relatively frequent (n = 55, 68%), mainly granulation tissue or local erythema.

217 onia without histological evidence of either granulation tissue or pulmonary fibrosis.

218 PDGF B-chain did not decrease the extent of granulation tissue or vascular lesion formation, and tha

219 e healing (24days) and exhibited accelerated granulation tissue production, epithelial maturation, an

220 iferation, in vivo engraftment, experimental granulation tissue reconstitution, and tissue vascularit

221 retained specifically within the presumptive granulation tissue region of the wound at a higher densi

222 Following the granulation tissue removal, intrabony defect was filled

223 ptozotocin-induced diabetic rats to elicit a granulation tissue response and to collect acute wound f

224 ll surgical sponges to elicit a foreign body granulation tissue response, or by ligating the left com

225 hol sponges were implanted to elicit a naive granulation tissue response, removed at defined time poi

226 N-terminal domain is a key regulator of the granulation tissue response, with important implications

227 pyogenic granuloma is an exuberant growth of granulation tissue secondary to irritation.

228 ponge implants in Flk-1(LacZ) knock-in mice, granulation tissue showed many more LacZ-positive cells

229 ells/blood vessel lumen, M2 macrophages, and granulation tissue size without compromising the mechani

230 Hypoxia peaked in the granulation tissue stage at day 4 and correlated with in

231 In the tuberculous granulation tissue surrounding caseous and liquefied pul

232 ment of wound closure, increased blood flow, granulation tissue thickness (GTT), and CD31 that correl

233 db/db mice markedly increased angiogenesis, granulation tissue thickness, and wound closure rates, w

234 terized by decreased contraction and reduced granulation tissue thickness.

235 human vs human comparisons of wound area and granulation tissue tracings.

236 preparation, a palatal mini-flap was raised, granulation tissue was eliminated by means of ultrasonic

237 e angiogenesis and maturation of provisional granulation tissue was enhanced in response to genetic d

238 tion, generation of thick, well-vascularized granulation tissue was enhanced, in parallel with increa

239 hire pigs and harvested at various times, no granulation tissue was observed before day 4.

240 In contrast, granulation tissue was observed in wounds receiving fibr

241 analysis of esophageal biopsies demonstrated granulation tissue with acute and chronic inflammation.

242 nt of inflammatory macrophages, formation of granulation tissue with angiogenesis, and finally tissue

243 The subacute response exhibited granulation tissue with early fibrous encapsulation (pan

244 crypt destruction, erosions, ulcerations, or granulation tissue) and clinical endpoints at the end of

245 unctional enzyme expression by repair cells (granulation tissue) growing into the gap.

246 ialization, presence or absence of swelling, granulation tissue, and bleeding on gentle palpation wer

247 latelet and fibrin deposition, inflammation, granulation tissue, and finally, fibrous encapsulation.

248 mages correlated with liquefaction necrosis, granulation tissue, and tumor.

249 g wound re-epithelialization, formulation of granulation tissue, and vascularization.

250 Day 4 wounds were 3% filled with granulation tissue, day 5 wounds 48% filled, and day 7 w

251 of TGF beta leading to enhanced formation of granulation tissue, even in the absence of obvious infec

252 istology showed channel remnants composed of granulation tissue, fibrosis, and new vessels (NV).

253 e re-surfaced before the formation of normal granulation tissue, leading to a defective epidermal arc

254 ion (loss of respiratory epithelium, luminal granulation tissue, lymphocytic tracheitis) with increas

255 The rates of granulation tissue, otalgia, and facial palsy were 90.9%

256 is, early wound provisional matrix, and late granulation tissue, respectively.

257 monocyte infiltration/giant cell formation (granulation tissue, the intimal and subintimal synovial

258 alpha-smooth muscle actin and are present in granulation tissue, where they are responsible for wound

259 ar density of MRL-MSC-generated experimental granulation tissue.

260 lar weight S100A7 in human wound exudate and granulation tissue.

261 activation of alpha-SMA in myofibroblasts in granulation tissue.

262 d replacement of injured cardiomyocytes with granulation tissue.

263 of alpha-SMA expression in myofibroblasts in granulation tissue.

264 d replacement of injured cardiomyocytes with granulation tissue.

265 in myofibroblasts and smooth muscle cells of granulation tissue.

266 tion of necrotic tissue and replacement with granulation tissue.

267 nificantly inhibited in vivo angiogenesis in granulation tissue.

268 f inducible nitric oxide synthase protein in granulation tissue.

269 ermal wound margin and over fibronectin-rich granulation tissue.

270 skin wounds and impaired angiogenesis in the granulation tissue.

271 gnificantly reduced vascularity of the wound granulation tissue.

272 growth factor, is highly expressed in dermal granulation tissue.

273 emonstrated peripheral flow at US because of granulation tissue.

274 rregularly organized and highly vascularized granulation tissue.

275 th the abnormal collagen organization in the granulation tissue.

276 d associated with the formation of abdominal granulation tissue.

277 placement by a thin layer of highly vascular granulation tissue.

278 tion of fibroblasts, and angiogenesis in the granulation tissue.

279 necrotic areas, connective tissue stroma or granulation tissue.

280 e the site of injury with newly vascularized granulation tissue.

281 service independently traced wound area and granulation tissue.

282 h increased risk of aspiration and laryngeal granulation tissue.

283 ce re-epithelialization and the formation of granulation tissue.

284 ient has chronic refractory otorrhea and ear granulation tissue.

285 epithelialization and increased formation of granulation tissue.

286 cterized by more inflammatory, necrotic, and granulation tissue.

287 ement of an intrauterine device, and vaginal granulation tissue.

288 g the formation of new arterioles within the granulation tissue.

289 r, increasingly in antigen-negative areas of granulation tissue.

290 d regulates their cytokine production in the granulation tissue.

291 elimination of tumor cells and formation of granulation tissue.

292 tor (VEGF), Flk1, and VE-cadherin in ECs and granulation tissues (GTs) of full-thickness wounds.

293 ts show that Cyr61 is inducibly expressed in granulation tissues after wounding and that Cyr61 activa

294 of active wounds of living T1D subjects, and granulation tissues from mice with streptozotocin-induce

295 r, specifically with the transition from the granulation to the remodeling phases of the wound healin

296 Spray granulation was found to be the superior drying method f

297 heric velocities are dominated by convective granulation (which has been considered before for spicul

298 c rises in viscosity, leading to jamming and granulation, with increasing shear rate.

299 genic determination gene, MyoD, into cardiac granulation (wound repair) tissue.

300 Granulation (wound) tissue is routinely removed during g