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1 nd Th2 cells participate in the ovum-induced granulomatous inflammation.
2 14-amino acid cyclic peptide that regulates granulomatous inflammation.
3 and necroses, intravascular coagulation, and granulomatous inflammation.
4 e or after the onset of experimental chronic granulomatous inflammation.
5 m albumin reflected mainly lung cellular and granulomatous inflammation.
6 h fibrosis, crypt abscesses, crypt loss, and granulomatous inflammation.
7 microbial killing, recurrent infections, and granulomatous inflammation.
8 mation, and IL-32 emerged as a key driver of granulomatous inflammation.
9 th protein abundance varying by proximity to granulomatous inflammation.
10 haracterized by the presence of noncaseating granulomatous inflammation.
11 but these may include carcinogenic change or granulomatous inflammation.
12 intravenously into eight mice bearing local granulomatous inflammation.
13 nocytes, and macrophages and produce typical granulomatous inflammation.
14 necrotizing vasculitis, and eosinophil-rich granulomatous inflammation.
15 d by degradation of interstitial crystals by granulomatous inflammation.
16 attenuated P. acnes-induced ear swelling and granulomatous inflammation.
17 relieving P. acnes-induced ear swelling and granulomatous inflammation.
18 lium-specific CD4(+) T cells in the lung and granulomatous inflammation.
19 ng anti-IL-17 mAb markedly inhibited hepatic granulomatous inflammation.
20 also resulted in significant exacerbation of granulomatous inflammation.
21 aggregates within poorly organized areas of granulomatous inflammation.
22 ics of T cell influx and mobilization during granulomatous inflammation.
23 4 days but were associated with considerable granulomatous inflammation.
24 o multiple facets of type-2 hypersensitivity granulomatous inflammation.
25 odulate T cell recruitment and activation in granulomatous inflammation.
26 the predominant finding (42.2%), followed by granulomatous inflammation (29%), steatosis/steatohepati
28 i develop severe CD4 T cell-mediated hepatic granulomatous inflammation against parasite eggs associa
29 e displaying diminished type 2 responses and granulomatous inflammation, also do not fully default to
30 embedded tissue specimens studied, 57 showed granulomatous inflammation and 53 had been cultured for
31 nonhematopoietic cells may partly facilitate granulomatous inflammation and bacterial dissemination.
32 a from the tissues, leading to local chronic granulomatous inflammation and compensatory adaptive imm
33 icited a late xenograft reaction, leading to granulomatous inflammation and dissolution of the membra
34 humans is attributed to parasite egg-induced granulomatous inflammation and fibrosis in the host live
35 ansoni, parasite eggs cause hepatointestinal granulomatous inflammation and fibrosis mediated by CD4
36 ed signaling cues in macrophages to restrain granulomatous inflammation and highlight the therapeutic
37 thermore, a significant reduction of hepatic granulomatous inflammation and IL-17 production in inter
40 Crohn disease (CD) are both characterized by granulomatous inflammation and related to nucleotide oli
41 yllium (Be) exposure and is characterized by granulomatous inflammation and the accumulation of Be-re
42 occupational lung disorder characterized by granulomatous inflammation and the accumulation of beryl
43 ure in the workplace and is characterized by granulomatous inflammation and the accumulation of beryl
44 beryllium disease (CBD) is characterized by granulomatous inflammation and the accumulation of CD4(+
45 crotizing vasculitis that is associated with granulomatous inflammation and the presence of anti-neut
46 at while a reduction in IL-4/IL-13-dependent granulomatous inflammation and tissue eosinophilia was o
47 Aspiration biopsy of the vertebra revealed granulomatous inflammation and yeast forms; culture yiel
48 ignals emerged as important regulators of GA granulomatous inflammation, and IL-32 emerged as a key d
49 crotizing and crescentic glomerulonephritis, granulomatous inflammation, and systemic necrotizing vas
50 tant role for CCR5, MIP-1 alpha, and CCR2 in granulomatous inflammation, and that CCR5 and MIP-1 alph
51 nsure that any further complications such as granulomatous inflammation are managed and documented.
53 osure to antigen, WT mice developed a marked granulomatous inflammation associated with an increase i
56 llmark of Mycobacterium-induced pathology is granulomatous inflammation at the site of infection.
57 administration was associated with enhanced granulomatous inflammation, but did not prevent infectio
58 ught to elucidate the role of osteopontin in granulomatous inflammation by characterizing its express
59 Exserohilum provokes robust neutrophilic and granulomatous inflammation capable of thwarting fungal g
60 understanding of the pathways underlying the granulomatous inflammation characteristic of sarcoidosis
61 ysis of all 3 cases revealed elastolysis and granulomatous inflammation characterized by multinucleat
64 osin-stained sections revealed non-caseating granulomatous inflammation consistent with skeletal sarc
65 n is controlled through either neutrophil or granulomatous inflammation, depending on the fungal spec
66 t studies support the concept that pulmonary granulomatous inflammation directed by interferon (IFN)-
67 ains had similar degrees of interstitial and granulomatous inflammation during the first 12 weeks of
69 exclude infectious and autoimmune causes of granulomatous inflammation, followed by dermatologic wor
70 racterized by pronounced hepatic egg-induced granulomatous inflammation in a proinflammatory cytokine
75 tory for macrophages and induced less severe granulomatous inflammation in mice, demonstrating that t
76 linical presentation, finding nonnecrotizing granulomatous inflammation in one or more tissue samples
78 hogenic antigens relevant to immune-mediated granulomatous inflammation in sarcoidosis, we used a lim
83 with schistosomiasis mansoni, where chronic granulomatous inflammation in the liver causes portal hy
84 ritical for the development of Th17-mediated granulomatous inflammation in the lung in response to S.
85 dritic cell-mediated development of a type 1 granulomatous inflammation in the lung in response to SC
91 characterized by an increase in the area of granulomatous inflammation, increased numbers of resisti
92 type 1 (TH1) lymphocytes, attenuates chronic granulomatous inflammation induced by bacterial cell wal
93 responses are required for the generation of granulomatous inflammation induced by inhaled S. chartar
97 such as periprosthetic osteolysis, in which granulomatous inflammation is initiated by particle phag
100 plays an important role in controlling acute granulomatous inflammation, it plays no essential role i
101 lation of silica into adult rats resulted in granulomatous inflammation leading to fibrosis and alveo
103 of monocyte chemotactic protein-1 (MCP-1) to granulomatous inflammation mediated by Th1- and Th2-rela
105 d suppression in Th2 cytokine expression and granulomatous inflammation observed in egg/CpG-sensitize
107 ase of ROS, as occurs in the immune-specific granulomatous inflammation of chronic beryllium disease
109 pportunistic pathogenic bacterium, developed granulomatous inflammation of the ileum, characterized b
110 nfiltrative cardiomyopathy that results from granulomatous inflammation of the myocardium and may pre
111 se resulting from lymphocytic and frequently granulomatous inflammation of the peripheral airways, al
112 ial granulomatosis (OFG) is characterized by granulomatous inflammation of the soft tissues of maxill
113 medium and large arteries, characterized by granulomatous inflammation of the three-layered vessel w
114 is (GPA; or Wegener's granulomatosis) is the granulomatous inflammation of the upper respiratory trac
116 hese mice in exacerbated hepatic egg-induced granulomatous inflammation or in the levels of IL-17 ind
117 is syndrome had chronic active colitis, with granulomatous inflammation present in 7 of 11 patients (
120 iency with recurrent pyogenic infections and granulomatous inflammation, results from loss of phagocy
121 ciation with bioaerosols and the presence of granulomatous inflammation support consideration of myco
123 in 2 patients and demonstrated noncaseating granulomatous inflammation surrounding tattoo ink in the
124 ith schistosome helminths is associated with granulomatous inflammation that forms around parasite eg
125 Sarcoidosis is characterized by noncaseating granulomatous inflammation that involves the lungs or ly
126 asite eggs elicit a Th cell-mediated hepatic granulomatous inflammation that leads to scarring, porta
127 While a non-degradable UPS polymer induces granulomatous inflammation that persists over months at
128 . parvum in sensitized rats caused pulmonary granulomatous inflammation that was histologically simil
129 Although one-third of patients progress from granulomatous inflammation to severe lung damage, the mo
130 s granulomatous lesions are detected, severe granulomatous inflammation was detected in only one of t
132 Since both responses usually correlate with granulomatous inflammation, we tested six prominently ex
133 mma/TNF-dominant) and 2 (IL-4/IL-5-dominant) granulomatous inflammation were analyzed in mice with kn
136 though the soluble and cellular mediators of granulomatous inflammation were qualitatively similar in
137 Regions of interest (ROIs) with and without granulomatous inflammation were selected to examine prot
138 y beryllium exposure and is characterized by granulomatous inflammation with accumulation of CD4+ T c
139 Pulmonary tuberculosis is characterized by granulomatous inflammation with an extensive infiltratio
140 Pulmonary tuberculosis is characterized by granulomatous inflammation with an extensive infiltratio
141 C57BL/6 mice, but not DBA/2 mice, developed granulomatous inflammation with an increase in lung inde
142 and C was C > S > W and ranged from intense granulomatous inflammation with caseous necrosis for inf
144 The pathology of human coccidioidomycosis is granulomatous inflammation with many neutrophils surroun
145 nge with 10(6) S. chartarum spores developed granulomatous inflammation with multinucleate giant cell
146 and challenged with 10(6) SC spores develop granulomatous inflammation with multinucleate giant cell
147 e inflammation early progressing to chronic, granulomatous inflammation with necrosis later that spre
148 opathological examination revealed extensive granulomatous inflammation within the dermis and subcuta
149 worms produce ova that incite focal Th2-type granulomatous inflammation within the liver and intestin
150 ry infection prolonged survival and enhanced granulomatous inflammation without reducing lung CFU.