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1 estrogen-replaced nonhuman primates (African green monkeys).
2 ces were obtained for cynomolgus macaque and green monkey.
3 rimate species: marmoset, rhesus macaque and green monkey.
4 otype 3 (SFVagm-3), isolated from an African green monkey.
5 g natural SIV variants isolated from African green monkeys.
6 model of lymphatic obstruction using African green monkeys.
7 -3-fold higher in cynomolgus monkeys than in green monkeys.
8 sterol levels were 50% lower than was fed to green monkeys.
9 CAT2 mRNA nor protein was diet-responsive in green monkeys.
10 ary cholesterol, and less responsive African green monkeys.
11 simian immunodeficiency viruses from African green monkeys.
12 tralization titers in RSV-preexposed African green monkeys.
13 vet, grivet, and tantalus species of African green monkeys.
14 nted in the striatum of MPTP-treated African green monkeys.
15 n the dorsal and ventral striatum of African green monkeys.
16 candidates after a single passage in African green monkeys.
17 SARS-CoV-2 using SARS-CoV-2 infected African green monkeys.
18 d suPAR levels and glomerulopathy in African green monkeys.
19 acaques, rhesus macaques, and vervet African green monkeys.
20 immunodeficiency virus infections of African Green Monkeys.
21 both LDL receptor-deficient mice and African green monkeys.
24 uman A3C (hA3C), human A3DE (hA3DE), African green monkey A3F (agmA3F), and rhesus macaque A3F (rhA3F
25 V) Vif was shown to bind and degrade African green monkey A3G (agmA3G) and, unexpectedly, human A3C.
26 re, we report the DNA sequence of an African green monkey AAV integration site isolated from CV-1 cel
27 t rhesus macaque APOBEC3G (rhA3G) or African green monkey (AGM) APOBEC3G (agmA3G) because of a failur
28 for studying HeV infection, with the African green monkey (AGM) appearing to most faithfully reproduc
33 mined the pathogenesis of HeV in the African green monkey (AGM) following intratracheal inoculation.
34 inical studies were conducted in the African green monkey (AGM) inhalational model of pneumonic plagu
36 his issue, we established a neonatal African green monkey (AGM) nonhuman primate model that could be
38 sely, the Vif protein encoded by the African green monkey (agm) simian immunodeficiency virus (SIV) c
39 the A3G-Vif interaction within four African green monkey (AGM) subspecies, which are each naturally
41 ved by comparing a natural SIV host, African green monkey (AGM) to an AIDS susceptible species, rhesu
42 n, we isolated cDNA clones of human, African green monkey (AGM), and NIH/Swiss mouse CCR5s, and we qu
44 b from its natural host, the sabaeus African green monkey (AGM), to a new host, the pigtailed macaque
45 investigate replication in primates, African green monkeys (AGM) and rhesus macaques (n = 4) were ino
52 were assessed in naturally infected African green monkeys (AGM) of the vervet subspecies (Chlorocebu
54 genic differences between strains, 4 African green monkeys (AGM) were exposed to NiVM and 4 AGMs were
55 ssive infection (SIVagm infection of African green monkeys (AGM)), and transient, controlled infectio
63 iral pathogens in two populations of African green monkeys (AGMs) (Chlorocebus sabaeus) from Africa a
68 geographically dispersed species of African green monkeys (AGMs) are all infected with highly divers
75 roximately 98-99% identical) CCR5 of African green monkeys (AGMs) avidly binds beta-chemokines and fu
76 est the hypothesis that SIV-infected African green monkeys (AGMs) avoid AIDS due to virus replication
77 th m102.4 for the ability to protect African green monkeys (AGMs) from a stringent NiV challenge.
80 report two cases of ARDS in two aged African green monkeys (AGMs) infected with SARS-CoV-2 that had p
81 fection in sooty mangabeys (SMs) and African green monkeys (AGMs) is associated with low levels of im
82 of rVSV-DeltaG-NiVBG, we vaccinated African green monkeys (AGMs) one year before challenge with an u
84 s of chronically SIV-infected sabeus African green monkeys (AGMs) revealed that gastrointestinal memo
86 d from pigtailed macaques (PTMs) and African green monkeys (AGMs) that experience different SIVsab in
88 iV delivered by the aerosol route in African green monkeys (AGMs) used the Malaysia strain (NiVM), wh
89 p of 10 rhesus macaques (RMs) and 10 African green monkeys (AGMs) was exposed to aerosolized B. pseud
91 ans from chronically SIVagm-infected African green monkeys (AGMs) were frequently CXCR5(+) and entere
99 eficiency virus (SIV) hosts, such as African green monkeys (AGMs), sustain nonpathogenic SIV infectio
110 caques [PTMs]) and nonpathogenic (in African green monkeys [AGMs]) SIVsab infections to assess the si
112 lizing antibody titers obtained from African green monkeys and after human vaccination and natural in
113 strate that nonhuman primates (NHPs; African green monkeys and cynomolgus macaques) harbor serosal B
114 tified multiple SAMHD1 haplotypes in African Green Monkeys and find that the vpr gene from different
115 hosts (for example, sooty mangabeys, African green monkeys and mandrills) share many features of HIV
117 ts and cholesterol to nonpathogenic (African green monkeys) and pathogenic (pigtailed macaques) SIV h
118 utcomes of SIVsab infection in PTMs, African green monkeys, and rhesus macaques are different, the us
119 ciently in the respiratory tracts of African green monkeys, and the infected animals developed a high
120 TRIM5alpha B30.2 domain v1 region of African green monkeys are also associated with broader antiretro
121 ic infections in natural hosts, such African green monkeys, are characterized by a lack of gut microb
123 administered separately to groups of African green monkeys by the intranasal/intratracheal route.
127 le directed secretion of both Ags in African green monkey cells and functioned as an adjuvant for MHC
128 pr proteins are capable of arresting African green monkey cells but are completely inactive in human
132 IgA did not inhibit HAV infection of African green monkey cells, suggesting that the IgA and the viru
133 n after entry into rhesus macaque or African green monkey cells, where, paradoxically, the interactio
138 s musculus), hamster (Mesocricetus auratus), green monkey (Ceropithecus aethiops) and human (Homo sap
139 Herein we report that two species of African green monkeys (Chlorocebus sabaeus and C. pygerythrus) i
140 cyte-depleting antibodies to sabaeus African green monkeys (Chlorocebus sabaeus) before challenge wit
141 herpesviruses recently identified in African green monkeys, Chlorocebus rhadinovirus types 1 and 2 (C
143 d a range of restriction in mice and African green monkeys comparable with that of two attenuated RSV
146 nd in vivo studies in cynomolgus and African Green monkeys demonstrated a >10-fold higher lung tissue
147 rom chimpanzees] and SIVagmSab [from African green monkeys]) discordantly in different regions of the
148 nts from humans, rhesus monkeys, and African green monkeys displayed different but overlapping restri
149 e show that many CD4(+) T cells from African green monkeys downregulate CD4 in vivo as they enter the
150 We have collected the brains of African green monkeys during multiple Nipah virus, Bangladesh st
151 ery atherosclerosis were examined in African green monkeys fed diets containing cholesterol and 35% o
154 y shown that intranasal SV protected African green monkeys from challenge with the related human para
156 ruses (family Arteriviridae) in wild African green monkeys from Zambia (malbroucks [Chlorocebus cynos
162 uated the immunological responses of African green monkeys immunized with multiple F and G protein-ba
165 The lesions seen in the brain of African green monkeys infected with Nipah virus, Bangladesh were
166 d/nonprogressive infection) and from African green monkeys infected with SIVsab9315BR (nonpathogenic
169 set of clinical anthrax disease, the African green monkey is a suitable animal model exhibiting a dis
171 enuated viruses adapted to growth in African green monkey kidney (AGMK) and MRC-5 cells, respectively
175 ed from a cDNA expression library of African green monkey kidney (AGMK) cells by using protective mon
176 Hepatitis A virus (HAV) infects African green monkey kidney (AGMK) cells via the HAV cellular re
177 ar receptor 1 (havcr-1) and protects African green monkey kidney (AGMK) clone GL37 cells (GL37 cells)
179 sackievirus B6 replication in living Buffalo green monkey kidney (BGMK) cells via Tat peptide deliver
180 To characterize interactions between African green monkey kidney (BS-C-1) cell proteins and the predi
181 M175/P16, enhance growth in cultured African green monkey kidney (BS-C-1) cells but not in fetal rhes
183 and establishing stably transfected African green monkey kidney (CV1) cell lines expressing reporter
185 , and produced very small plaques on African green monkey kidney (Vero) cells that were similar in si
186 ding porcine kidney (PK15) cells and African green monkey kidney (Vero) cells, was inhibited by noncy
187 N2, H5N1 and H7N9 vaccine viruses in African green monkey kidney and Madin-Darby canine kidney cells.
189 s and deletions were monitored in an African green monkey kidney cell culture system (COS-7 cells) as
190 reliminary studies indicated that an African green monkey kidney cell line (Vero) is a suitable syste
194 within a few cycles of infection in African green monkey kidney cell lines CV-1, CV-1P, TC-7, MA-134
195 generates H2O2, was introduced into African green monkey kidney cells (CV-1 cells) under the control
196 ell homeostasis were investigated in African green monkey kidney cells (CV-1) by assessing the appear
197 1.3, was heterologously expressed in African Green Monkey kidney cells (CV-1) using a vaccinia virus/
198 n U937 cells (histiocytic lymphoma), African green monkey kidney cells (MARC-145 and Vero), primary m
199 y of HSV-1 derived from immortalized African green monkey kidney cells (Vero), immortalized human ker
200 e microsurgically removed from BSC-1 African green monkey kidney cells before the completion of S pha
202 Hepatitis A virus (HAV) infects African green monkey kidney cells via HAV cellular receptor 1 (h
203 2D cell culture infection model with Buffalo Green Monkey kidney cells was employed and infection wit
206 lanoma cell line but not to the CV-1 African green monkey kidney cells, which express CD44 at low lev
212 eir respective intracellular niches, African green monkey kidney epithelial (Vero) cells, A/J mouse b
213 tion was detected in only six: three African green monkey kidney epithelial cell lines (Vero, Vero E6
214 0 DNA replication in infected BSC-1 (African green monkey kidney epithelial) cells, albeit at a great
215 ve in 293T (embryonic kidney), Vero (African-green monkey kidney epithelial), 3T12 (mouse fibroblast)
216 interact with CD66a-transfected COS (African green monkey kidney) and CHO (Chinese hamster ovary) cel
219 o cells, which were derived from the African green monkey kidney, represent one of the few mammalian
223 ferent species of naturally infected African green monkeys living in different regions across Africa.
224 , complete suppression of macaque or African green monkey Lv1 was achieved by the additive effect of
226 natural SIV hosts (sooty mangabeys, African green monkeys, mandrills, sun-tailed monkeys, and chimpa
228 ogether, these data suggest that the African green monkey model exhibits important similarities to hu
230 ed the use of RSV (Memphis 37) in an African green monkey model of intranasal infection and identifie
231 IV5/G) protein in the cotton rat and African green monkey models for their replication, immunogenicit
232 pliced exon 10 were identical between human, green monkey, mouse, rat, and pig, while 207 consecutive
234 mbinant viruses were administered to African green monkeys (NDV-BC and NDV-LS) and rhesus monkeys (ND
235 The infecting SFV originated from an African green monkey (one person) and baboons (three people).
245 tion and deletion analysis in BSC-1 (African green monkey, renal epithelial) cells revealed that the
250 0 mg/kg IV administration of 1 in an African Green monkey RSV model demonstrated a >2-log(10) reducti
251 ; the human and, to a low level, the African green monkey sequences bound soluble HCV E2 (sE2) and in
252 eavage-efficient mutant, R-R-R-R, in African green monkeys showed that there was no detectable change
254 We now report a novel function of African green monkey simian immunodeficiency virus (SIVagm) Vif
255 only human Apo3G (hApo3G), whereas, African green monkey simian immunodeficiency virus (SIVagm) Vif
256 ian immunodeficiency virus SIV(SM)), African green monkey (SIV(AGM)), and Sykes' monkey (SIV(SYK)) is
257 munodeficiency viruses isolated from African green monkeys (SIVagm) contain a single accessory gene h
258 odeficiency virus (SIV) that infects African green monkeys (SIVagm) contains a vpr homologue, which e
259 Simian immunodeficiency virus from African green monkeys (SIVagm) results in asymptomatic infection
260 onpathogenic infection with SIV from African green monkeys (SIVagm), follicles remain generally virus
261 d simian immunodeficiency virus from African green monkeys (SIVagm), in one round of viral replicatio
265 (simian immunodeficiency virus from African green monkeys [SIVagm] and Rhesus macaques [SIVmac]), th
267 apped mangabey (SIVrcm), the sabaeus African green monkey (SIVagmSAB), and the chimpanzee (SIVcpz) an
268 odeficiency virus (SIV) that infects African green monkeys (SIVagmTAN), unlike human Apobec3DE, which
271 developed to measure SIVagm from two African green monkey subspecies demonstrated high levels of SIV
272 ed with that previously observed for African green monkeys, suggesting that the HAE model has potenti
274 D4(+) T cells of young mandrills and African green monkeys than on those of adults, we propose that l
279 or NiV infection, we exposed 6 adult African green monkeys to a large-particle (approximately 12 mum)
281 e compared the plasma virome of West African green monkeys to that in their descendants after importa
285 east two different retroviruses, and African green monkey TRIM5alpha was able to inhibit infection by
290 s in one eye of juvenile rhesus macaques and green monkeys, we combined cDNA subtractions, microarray
291 It was found that all vaccinated African green monkeys were completely protected against subseque
293 er respiratory tract of RSV-infected African green monkeys when administered once daily via intratrac
294 otective efficacy in cotton rats and African green monkeys, which are among the best available animal
295 HAV) was originally isolated from an African green monkey with hepatitis and appears to represent a t
296 4 tissue samples from a NiV-infected African green monkey with viral loads as low as 52 genome copies
297 ore natural NHP model, we challenged African green monkeys with the Bangladesh strain of NiV by the i
298 We infected 35 Asian macaques and African green monkeys with viruses that do or do not express Vpx