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1 s the most biologically potent polyphenol of green tea.
2 cals in nutraceutical products obtained from green tea.
3 ed on polypropylene added with catechins and green tea.
4 tioxidants to study in coffee, black tea and green tea.
5 re the main contributors to the taste of RTD green tea.
6 urine after single bolus intake of black or green tea.
7 the nutraceutical potential of a beverage as green tea.
8 e and antisecretory benefits of red wine and green tea.
9 cups/d, and we observed no association with green tea.
10 tion to two food products, i.e. red wine and green tea.
11 tine analysis of multi-pesticide residues in green tea.
12 coctions of Limonium algarvense flowers, and green tea.
13 -3-gallate (EGCG) is the major polyphenol in green tea.
14 with seaweed, but inconclusive for black and green teas.
15 pigallocatechin gallate (EGCG) were found in green teas.
16 of the black teas were as efficacious as the green teas.
17 nsities of bitterness and astringency in RTD green teas.
18 tioxidant capacity of twenty-four commercial green teas.
19 found that treatment of A/J mice with either green tea (0.6% in water) or a defined green tea catechi
22 he green-Mediterranean dieters also consumed green tea (3-4 cups/d) and a Wolffia globosa (Mankai str
23 es of three beverages, coffee, black tea and green tea, along with their major components, were inves
26 ers and drinks per week), beverages (coffee, green tea and milk) and foods (yoghurt, cheese, natto, t
31 s were detected after 3 months of storage in green tea and soy products, while 6 months were necessar
33 chin gallate (EGCG), the major polyphenol in green tea and the main bioactive compound responsible fo
36 roxyl radical-scavenging activity, higher on green tea, and iron chelating potential, higher on L. al
37 tea varieties, respectively, known for good green tea, and they were used as reference standards.
38 xidant and anti-inflammatory properties than green tea, and thus, may be useful for alleviating sympt
40 he well-known pleiotropic health benefits of green tea are mainly attributed to epigallocatechin-3-ga
42 ed classes (Argentinean green tea; Brazilian green tea; Argentinean black tea; Brazilian black tea; a
43 Evidence is accumulating that catechins in green tea as well as theaflavins and thearubigins from b
44 ion of the five studied classes (Argentinean green tea; Brazilian green tea; Argentinean black tea; B
49 o antioxidant and antifibrotic properties of green tea (Camellia sinensis, Theaceae) were investigate
53 ither green tea (0.6% in water) or a defined green tea catechin extract (polyphenon E; 2.0 g/kg in di
56 ular targets in cardiac muscle for the major green tea catechin, (-)-epigallocatechin-3-gallate (EGCG
57 The most abundant and biologically active green tea catechin, (-)-epigallocatechin-3-gallate or (-
58 alysis of the interactions between the major green tea catechin, epigallo-catechin 3-gallate (EGCG),
60 scribed as the most potently chemopreventive green tea catechin; however, the low bioavailability of
61 is observed over time under the influence of green tea catechins (GTC), which are suggested to offer
68 ns between the milk fat globule membrane and green tea catechins provided useful information about th
70 ocatechin gallate (EGCG) is one of the major green tea catechins that is suggested to have a role as
72 droxyl groups are important in mediating the green tea catechins' inhibitory effect towards HGF/Met s
74 and laccase, both from Agaricus bisporus, on green tea catechins, the oxidation process was directed
75 comprehensive characterization of commercial green tea (CGT) with the employment of independent varia
78 tically applied to chemical structures of 75 green tea components, resulting in a virtual library of
79 Unexpectedly, we discovered that various green tea constituents, in particular (-)-epigallocatech
80 tion of metabolites over a 24-h period after green tea consumption corresponded to 28.5% of the inges
81 cts are required before a recommendation for green tea consumption for cancer prevention should be ma
87 late (EGCG), a flavanoid polyphenol found in green tea; curcumin, found in tumeric; and resveratrol (
89 (EGCG), the main polyphenolic constituent of green tea, decreased beta-amyloid (Abeta) levels and pla
91 tudy that epigallocatechin gallate (EGCG), a green tea-derived catechin, acts as a potent suppressor
94 tion of AuNPs capped with gallic acid (GA, a green-tea-derived polyphenol) using differential pulse v
95 dant capacity (TEAC) of red wine, coffee and green tea determined using this method were: (1.20 +/- 0
96 e compounds present in new broccoli-enriched green tea drinks and their potential antitumoral activit
99 ously shown that two of the polyphenols from green tea (epigallocatechin gallate (EGCG) and epicatech
100 on was to determine whether an ingredient of green tea, epigallocatechin gallate (EGCG) could attenua
101 gate the effects of two main constituents of green tea, (-)-epigallocatechin-3-gallate (EGCG) and caf
104 allate (EGCG), the major polyphenol found in green tea, exerts antiproliferative and proapoptotic eff
107 e randomly allocated to 1350 mg encapsulated green tea extract (540 mg GTC) with 50 mg vitamin C or p
109 extracts of ulam raja (UREX) and commercial green tea extract (GTE) added individually at 200 and 50
110 type I collagen synthesis and the effects of green tea extract (GTE) and its major catechin, (-)-epig
111 of lettuce extract (LE) with quercetin (QC), green tea extract (GTE) or grape seed extract (GSE) was
112 dy was to examine the efficacy and impact of green tea extract (GTE) supplementation high in epigallo
114 extract (PRE), cherry stem extract (CSE) and green tea extract (GTE) were added to sour cherry juice
115 anufactured containing free catechin or free green tea extract (GTE), and liposomal encapsulated cate
116 f alpha-tocopherol (TOH) in combination with green tea extract (GTE), the green tea polyphenol (-)-ep
117 ct (CSE), pomegranate rind extract (PRE) and green tea extract (GTE)] on anthocyanin content, colour,
120 al trials have shown an inhibitory effect of green tea extract against the progression of prostate pr
121 was to test the chemopreventive potential of green tea extract and EGCG after tannase-mediated hydrol
122 almost 50% of the total catechin content in green tea extract and has very potent antioxidant effect
125 incubated with epigallocatechin gallate and green tea extract at 62 or 37 degrees C for 180 min.
128 cence quenching measurements showed that the green tea extract contained components that interacted w
129 after 1 month of chronic administration of a green tea extract containing epigallocatequin-3-gallate
132 n inhibitory role of oral supplementation of green tea extract on a precancerous lesion of oral cavit
133 Our study found that oral administration of green tea extract prevented CCl(4)-induced hepatic fibro
134 illus subtilis were found to be sensitive to green tea extract via disc diffusion assay (zone of inhi
135 -catechin, (-)-epigallocatechin gallate, and green tea extract were added to washed MFGs to examine p
137 red the interactions and reactivities of the green tea extract, (-)-epigallocatechin-3-gallate [(2R,3
138 implicated agents include anabolic steroids, green tea extract, and multi-ingredient nutritional supp
139 tor inhibitors, cyclooxygenase-2 inhibitors, green tea extract, and peroxisome proliferator activated
140 phenols (vanillin, epigallocatechin gallate, green tea extract, and protocatechualdehyde) at inhibiti
141 hin-3-gallate (EGCG), a major constituent of green tea extract, exhibits effects of anti-inflammation
142 untargeted LC-MS(n) (n </= 3) data set of a green tea extract, generated on an LC-LTQ/Orbitrap hybri
143 ioxidants were added to the prepared matrix: green tea extract, rosemary extract, a mix of tocopherol
147 ration Solutions and 3255 Camellia sinensis (Green Tea) Extract with certified concentrations of cate
148 onsistent results of an inhibitory effect of green tea extracts and tea polyphenols on the developmen
152 tures of the natural oil body emulsions with green tea extracts, aiming to serve as a base for functi
155 ds of natural origin (black, white, red, and green tea extracts, phytic acid) to inhibit TMAO-demethy
156 r the reduction of MbFe(IV)O by catechin and green tea extracts, though possible confounding reaction
157 the mechanism of thearubigin formation from green tea flavan-3-ols, a model system, based on electro
158 inetics of binding between alpha-amylase and green tea flavonoids were investigated by fluorescence q
160 keloids and imply a therapeutic potential of green tea for the intervention and prevention of keloids
161 llate (EGCG), a redox-active polyphenol from green tea, for 32 consecutive days with step-wise increa
162 gallate (EGCG), the main catechin present in green tea, forms complexes with the casein micelles and
164 evelop a thermo-reversible sustained-release green tea gel and to study its clinical effects on patie
165 ve local drug therapy with thermo-reversible green tea gel has shown to reduce pockets and inflammati
166 the range of 0.375-12mg in totally 1.75g of green tea, green coffee and mixed herbal tea by using FT
167 the first time, detection of sibutramine in green tea, green coffee and mixed herbal tea using ATR-F
168 current study investigated the potential of green tea (GT) to improve uncoupling of endothelial nitr
169 properties of pure plant extracts (PPEs) of green tea (GT), black tea (BT) and soybean individually
175 Black tea, although not as well studied as green tea, has also shown cancer-preventive effects in l
176 techin-3-gallate (EGCG), a catechin found in green tea, has been recognized as a potential therapeuti
177 (EGCG), the principal polyphenol present in green tea, has been shown to be effective at preventing
178 -gallate (EGCG), the principal polyphenol in green tea, has been shown to inhibit the growth of many
179 in-3-O-gallate (EGCG), a major ingredient of green tea, has been shown, for example, to possess antic
180 atechin-3-gallate (EGCG), a main catechin of green tea, has been suggested to inhibit hepatic glucone
181 late (EGCG), a major polyphenol component of green tea, has recently been identified as an inhibitor
188 radation of catechins and other phenolics in green tea infusions were monitored using fast HPLC/MS se
191 , a major biologically active constituent of green tea, inhibits activation of the epidermal growth f
192 GCG), an anti-inflammatory compound found in green tea, inhibits IL-1beta-induced IL-6 production and
196 evidence in support of a protective role of green tea intake in the development of oral-digestive tr
198 ng white women, whereas caffeinated soda and green tea intakes were associated with increased estradi
201 in-3-gallate (EGCG), the major polyphenol in green tea, is a direct antagonist of androgen action.
202 -gallate (EGCG), a polyphenol extracted from green tea, is an antioxidant with chemopreventive and ch
203 of coffee and potentially black tea, but not green tea, is associated with lower risk of type 2 diabe
204 atechin-3-O-gallate, the main flavan-3-ol in green tea, is unclear because it appears unmetabolized i
205 activity and the basic mechanism of aqueous green tea leaf extract on selected bacterial strains.
210 il samples increased in the fallowing order: green tea<yellow tea<blackberry<BHT<cranberry<lemon<oil
212 results of antioxidant activity of black and green tea microsuspensions with the results of the analy
213 in gallate (EGCG), a bioactive polyphenol in green tea, mimics metabolic actions of insulin to inhibi
214 tumors and green tea-treated tumors (either green tea or polyphenon E) were compared to determine th
218 laboratories around the world suggests that green tea, particularly its major polyphenolic constitue
219 to study the antioxidant activity of banana, green tea, pink guava, and honeydew and the results were
220 ombination with green tea extract (GTE), the green tea polyphenol (-)-epicatechin (EC) or the isomeri
225 less invasive phenotype, the effects of the green tea polyphenol epigallocatechin-3 gallate (EGCG) o
227 Previously, we showed that the bioactive green tea polyphenol epigallocatechin-3-gallate (EGCG) i
229 tory effects of Polyphenon E [a standardized green tea polyphenol preparation containing 65% (-)-epig
230 Activation of hINV promoter activity by the green tea polyphenol, (-)-epigellocathecin-3-gallate, is
231 ary chemopreventive agents, sulforaphane and green tea polyphenol, and that this reduction involves u
233 e that PKC-delta is required for calcium and green tea polyphenol-dependent regulation of end respons
238 cells, we show that 2-day pretreatment with green tea polyphenols (GTPP) and their active ingredient
239 cluding epigallocatechin-3-gallate (EGCG) in green tea polyphenols (GTPs) and sulforaphane (SFN) in b
241 Recently we demonstrated that a subset of green tea polyphenols are potent inhibitors of glutamate
242 itro studies reveal the possible benefits of green tea polyphenols as cancer therapeutic agents to in
245 Taken together, our results indicate that green tea polyphenols may have the potential to negate t
247 mized intervention trials on the efficacy of green tea polyphenols or extracts are required before a
249 rammonemia forms of GDH are inhibited by the green tea polyphenols, epigallocatechin gallate and epic
250 late (EGCG), the major polyphenol present in green tea, possesses potent anti-inflammatory and antipr
253 icacy of BZM and suggest that consumption of green tea products may be contraindicated during cancer
254 s determination of 400 pesticide residues in green tea products using ultra performance liquid and ga
255 GCG), a catechin-base flavonoid derived from green tea protects retina neurones in situ from ischemia
256 echin-3-gallate (EGCG), a major component of green tea, protects against certain types of cancers, al
257 Clones K-purple and TRFK 91/1 showed high green tea quality indices with the latter doubling with
261 ination of soy phytochemical concentrate and green tea reduced serum insulin-like growth factor-I con
263 3-gallate (EGCG), a polyphenolic compound of green tea, results in activation of p53 and induction of
264 f gene expressions that both offer clues for green tea's potential mechanisms of action and provide a
265 gallate (EGCG) and caffeine in 29 commercial green tea samples available in a Saudi Arabian local mar
266 Analysis (PCA), which grouped the black and green tea samples into 3 different clusters, respectivel
267 tile compounds in seven ready-to-drink (RTD) green tea samples were analysed and quantified using liq
272 EGCG), the major polyphenolic constituent of green tea, sensitizes TRAIL-resistant LNCaP cells to TRA
273 r amount of polyphenols compared to standard green tea shoot (GL) while epigallocatechin gallate (EGC
274 No melatonin could be detected in black and green tea, sour cherry, sour cherry concentrate, kefir (
275 ceutical products based on natural extracts (green tea, soy, royal jelly and grapes) observing the ap
277 e therapeutic and toxicological potential of green tea supplementation is evaluated and may provide a
278 s incubated with the mixture of broccoli and green tea than on cells exposed to control infusions.
279 chin gallate (EGCG) is a major polyphenol in green tea that has beneficial effects in the prevention
282 gallocatechin gallate (EGCG), a component of green tea, to be considered in the context for neuroprot
284 e those TUM genes whose expression levels in green tea-treated tumors returned to levels seen in norm
289 never drank tea, regular tea intake (mostly green tea) was associated with reduced risk of all diges
290 20 micromol/L (-)-epigallocatechin gallate (green tea) was restored back to wild-type levels in ATPa
291 his work, a much simpler mixture, HWE buried green tea, was investigated by Nuclear Magnetic Resonanc
292 GCG), the principal polyphenol isolated from green tea, was recently shown to inhibit Hsp90; however,
295 bility of lithium from black, Earl Grey, and green teas were evaluated by inductively coupled plasma
296 nium and Brugmansia Arborea seeds (pasta and green tea), whereas coca leaf tea was directly analysed.
297 ggested TEAC(red wine) > TEAC(coffee) > TEAC(green tea), which is the same as DPPH, spectrophotometri
298 rotocol was tested with 11 black teas and 11 green teas, which can be easily distinguished by their m
299 ns between intakes of coffee, black tea, and green tea with the risk of type 2 diabetes in Singaporea
300 epigallocatechin-3-gallate (EGCG), found in green tea, with Abeta polypeptides, using a combination