ライフサイエンスコーパス: grey

1 c and brainstem pathways (eg, periaqueductal grey).

2 cyte stem cells prevents stress-induced hair greying.

3 s rely on regions such as the periaqueductal grey(4,5).

4 ternary Holocene) grey-brown alluvium and Q4 grey alluvium that had a relatively low soil bulk densit

5 to the left anterior insula, periaqueductal grey and hypothalamus among other areas.

6 s show that spinal cord lesions involve both grey and white matter from the early multiple sclerosis

7 and high doses, and showed normalization of grey and white matter imaging parameters at the higher d

8 We used 7 T MRI to: (i) characterize the grey and white matter pathology in the cervical spinal c

9 C-PBB3 binding capacities in the neocortical grey and white matter segments than healthy control subj

10 Previous reports of altered grey and white matter structure in Major Depressive Diso

11 althy young adults (n = 797), using regional grey and white matter volume, fractional anisotropy, and

12 An earlier meta-analysis showed reduced grey and white matter volumes in individuals with 22q11.

13 Lesion fractions within the spinal cord grey and white matter were related to the lesion fractio

14 In the brain (both grey and white matter), glutamate was also extremely hig

15 inal cord imaging at 7 T was used to segment grey and white matter, including lesions therein.

16 t solute speed was faster in CSF compared to grey and white matter.

17 rates of brain atrophy were seen in both the grey and white matter.

18 e MacTel area; and (4) less frequent retinal greying and more frequent hyperpigmentations compared wi

19 bioaccessibility of lithium from black, Earl Grey, and green teas were evaluated by inductively coupl

20 ese two different age groups and diseases, a grey area exists with regard to hepatocellular carcinoma

21 ta about the topic, some conclusions rely on grey areas and are reason of debate.

22 bour as well as non-reassuring fetal status, grey areas potentially affected by subjective clinical a

23 By generalizing Planck's theory to any grey body and deriving the completely general nonlocal f

24 n soils formed from Q4 (Quaternary Holocene) grey-brown alluvium and Q4 grey alluvium that had a rela

25 We studied stopover behaviour of Grey-cheeked Thrush (Catharus minimus) at a site in nort

26 rid zone between all-black carrion crows and grey-coated hooded crows, we decipher the interplay betw

27 red onto filter membranes and analyzed using grey color intensity analysis.

28 irectly obtain the radiation pattern of this grey exciton that deviates from that of the bright excit

29 forbidden out-of-plane, hence referred to as grey exciton.

30 n experiment on radio-tracking of individual grey field slugs in an arable field and associated data

31 has associated stress with accelerated hair greying (formation of unpigmented hairs)(1,2), but so fa

32 line, Embase, LILACS, Cochrane Library, Open Grey, Google Scholar, and Research Gate, as complemented

33 s of zoonotic diseases; and two species, the grey-headed flying-fox (Pteropus poliocephalus) and the

34 ars can be used to monitor a large colony of grey-headed flying-foxes near Melbourne.

35 sses (black-browed Thalassarche melanophris, grey-headed T. chrysostoma, and light-mantled albatrosse

36 ka (4.6 +/- 0.3 x 10(-5) mg.mL(-1).min(-1)), grey ironbark (E. paniculate; 3.4 +/- 0.2 x 10(-5) mg.mL

37 Canities (or hair greying) is an age-linked loss of the natural pigment ca

38 ular mechanisms of the responses of maize to grey leaf spot (GLS) disease caused by Cercospora zeina,

39 Texture features of grey level co-occurrence matrix (GLCM) of moisture predi

40 n; Gender and Muscle Soreness; Short Run Low Grey Level Emphasis and Headaches, Inverse Difference Mo

41 ical feature descriptor on spatial domain or grey level, and single-function prediction algorithm's l

42 A grey-level co-occurrence matrix (GLCM) computed from the

43 Grey lines indicate topics reviewed elsewhere.

44 We included papers that were published or in grey literature and had no language restrictions.

45 , supplemented by hand searching of relevant grey literature and snowballing references from the sele

46 of this study include that we did not review grey literature and that we were unable to conduct moder

47 EDLINE, Embase, and PsycINFO), internet, and grey literature databases, and disseminated data request

48 mbase, PsycINFO, Scopus, Web of Science, and grey literature for publications between Jan 1, 1993, an

49 mprehensively reviewed the peer-reviewed and grey literature for rigorously evaluated programmes that

50 Grey literature included national and international poli

51 tic review we searched for peer-reviewed and grey literature publications reporting associations betw

52 ) by searching MEDLINE, Embase, CENTRAL, and grey literature published between Jan 1, 2005, and Sept

53 restricted to clinical studies and relevant grey literature published in English.

54 eb of Science, clinical trials registers and grey literature sources from inception to December 2017

55 explosions at munitions sites, although the grey literature suggests that AXO is a substantial probl

56 Grey literature was not included in the search.

57 ematic review of published peer-reviewed and grey literature was undertaken, along with in-depth poli

58 ches, reference checking and searches of the grey literature were also undertaken.

59 Qualitative studies and grey literature were excluded.

60 re mixed-methods studies, and four [7%] were grey literature).

61 nic databases, including Embase and MEDLINE, grey literature, and reference lists for primary researc

62 1 and 2019, and searches of dictionaries and grey literature, as well as hand-searching were conducte

63 ing Medline, Embase, Web of Science, and the grey literature, current to July 8, 2020, for randomized

64 wer of floristic studies, often available in grey literature, for understanding long-term biotic chan

65 We also searched the grey literature, such as government reports, conference

66 through searches in MEDLINE, PubMed, and the grey literature, through references from relevant articl

67 EDLINE, EMBASE, Cochrane and sources for the grey literature.

68 takeholders were also contacted for relevant grey literature.

69 ffects, so we extended the search to include grey literature.

70 by hand-searching and internet searches for grey literature.

71 c databases were searched for published and 'grey' literature, with 151 hits.

72 23)Na-MRI and calculated the TSC in cortical grey matter (CGM), deep grey matter, normal-appearing wh

73 r: total brain (g = -0.96; p < 0.001); total grey matter (g = -0.81, p < 0.001); and total white matt

74 d fatigue (SF) associates more strongly with grey matter (GM) changes than reversible fatigue (RF).

75 gesting increased transmission time, whereas grey matter (GM) in auditory cortex partially mediates a

76 namely (1) higher K(i) in healthy individual grey matter (GM) versus white matter (WM), (2) GM/WM cer

77 Our aim was to investigate the impact of grey matter (GM) volume alterations in lobules VI to VII

78 Voxel-based analyses were used to assess grey matter (GM) volume, white matter (WM) microstructur

79 Grey matter (GM), white matter (WM), density, and GM/WM

80 partate was reduced in white (p = 0.030) and grey matter (p = 0.038) after HI.

81 athologies to test the prediction that: deep grey matter abnormalities frequently seen in preterm inf

82 uclei on advanced MRI sequences such as fast grey matter acquisition T1 inversion recovery, quantitat

83 re, while combining brain-predicted age with grey matter and cerebrospinal fluid volumes (themselves

84 ed a negative correlation between prefrontal grey matter and striatal dopamine synthesis capacity, bu

85 shared common associations with whole-brain grey matter and the Monetary Incentive Delay fMRI task,

86 lar risk is associated with reduced cerebral grey matter and white matter integrity within a fronto-p

87 es overlap with cytoarchitecturally distinct grey matter areas and may serve as the structural basis

88 ignificantly decreased cerebral volume in 14 grey matter areas at baseline magnetic resonance imaging

89 The necrotic lesions were seen in the grey matter at 100 Gy and in white matter at 60 Gy.

90 neuropsychological test scores and regional grey matter atrophy (including longitudinal data in a su

91 resent study was therefore to assess whether grey matter atrophy and amyloid pathology contribute to

92 vels were not associated with progression of grey matter atrophy and did not predict survival.

93 Stroked SHRs displayed grey matter atrophy and had a two-fold increase in apopt

94 sociated with grey/white matter volume loss; grey matter atrophy in cognitively unimpaired was specif

95 alysis aims to assess patterns of cerebellar grey matter atrophy in seven neurodegenerative condition

96 inal patterns of flortaucipir PET uptake and grey matter atrophy in the atypical phenotypes, demonstr

97 rks contribute to cognitive dysfunction, and grey matter atrophy is an early sign of potential future

98 Grey matter atrophy is associated with the degree of dis

99 Grey matter atrophy was not predicted by diffuse axonal

100 alysis identified a single component for the grey matter atrophy, while two components were found for

101 tion (R(2)=0.36) test scores, independent of grey matter atrophy.

102 pathology was associated with the extent of grey matter atrophy.

103 gions of interest were limited to 0.5 cm3 of grey matter centred around sites that had been identifie

104 For grey matter cortical analysis, free water fraction was i

105 Test and cortical thickness and spinal cord grey matter cross-sectional area of the Timed 25-Foot Wa

106 Intrathecal inflammation correlates with the grey matter damage since the early stages of multiple sc

107 okines, previously found to be associated to grey matter damage, and the disease activity, among 99 p

108 f amyloid-beta, tau, glucose metabolism, and grey matter degeneration in 15 cognitively normal mutati

109 We derived measures of grey matter degeneration in a priori NbM and the entorhi

110 Relationships between grey matter densities and cognitive scores were analysed

111 In conclusion, reduced grey matter density and elevated NFL concentrations in C

112 obiological underpinning and correlated with grey matter density in prefrontal and parietal cortex, a

113 individuals with stroke demonstrated higher grey matter density in secondary motor areas ipsilateral

114 significant association between the reduced grey matter density in the AD-vulnerable regions and inc

115 alidated the association between CSF NFL and grey matter density in the parietotemporal cortex, entor

116 s/posterior cingulate cortex and hippocampal grey matter density were significantly associated with i

117 Rat forebrain grey matter extracts contain a similar activity, and the

118 igate the clinical validity of the different grey matter features, we evaluated whether grey matter v

119 ures and two other common clinical measures (grey matter fraction and MMSE), obtained an accuracy of

120 les of supratentorial and cerebellar damage (grey matter fraction, T2 lesion volume, metrics of cereb

121 correlations between fine motor impairment, grey matter hypoperfusion, and white matter volume loss.

122 esonance imaging (MRI) measures of white and grey matter in a large population-derived cohort to inve

123 in the white matter was greater than in the grey matter in all phenotypes (P < 0.001); however when

124 on volumes were comparable between white and grey matter in primary progressive patients.

125 e DMN within DLPFC, as well as less cortical grey matter in regions sensitive to these external task

126 ciated abnormalities of structure (decreased grey matter in right dorsolateral prefrontal cortex and

127 and right inferior temporal gyrus; increased grey matter in right insula, right putamen, left tempora

128 which is mediated by progressive atrophy of grey matter indicative of increased Alzheimer's disease

129 outcome was defined as death or significant grey matter injury on MRI according to a previously vali

130 Infants with significant grey matter injury on MRI require less active cooling to

131 de improved contrast for better detection of grey matter lesions.

132 requent bilateral, large, brainstem and deep grey matter lesions.

133 , in order to discriminate between white and grey matter location of contacts.

134 Here, we examine the effect of education on grey matter loss over time.

135 echanisms, including, but not restricted to, grey matter loss.

136 by-voxel morphometry revealed no significant grey matter loss.

137 a family history of dementia and obesity, on grey matter macro- and microstructure across the whole b

138 Cortical gyrification represents a novel grey matter metric distinctive from grey matter thicknes

139 .9), we test for signatures of loneliness in grey matter morphology, intrinsic functional coupling, a

140 tia, age range 56-88 years), we investigated grey matter organization and volume differences in agein

141 ttainment was associated with slower loss of grey matter over time in mutation carriers.

142 16% of white and 14% of mixed cortical and grey matter patient regions showed FA decreases greater

143 morphometric networks and in the whole-brain grey matter pattern characterized individuals with cogni

144 We also derived a measure of whole-brain grey matter pattern organization by correlating grey mat

145 Preservation of the whole-brain grey matter pattern was also related to lower risk of de

146 sed an expected increase in frontal cortical grey matter perfusion but unexpected perfusion decreases

147 activity, and (iii) a module to compute the Grey Matter Proximity Index, i.e. the distance of each c

148 78, p=0.0087; whole-brain r=0.602, p<0.0001; grey matter r=0.518, p<0.0001; white matter r=0.588, p<0

149 RI scanning in seven children revealed large grey matter reductions over the left temporoparietal reg

150 Using an inferior cerebellar grey matter reference, 80-100-min 18F-flortaucipir PET s

151 aplacian Principal Component Analysis on 112 grey matter region-of-interest volumes was used to summa

152 terms of links (correlations) between nodes (grey matter regions) and to extract information out of t

153 33.9]; p=0.003) and atrophy rate in several grey matter regions, but not with change in Frontotempor

154 logical changes in the spinal cord white and grey matter resulting from injury can be observed with M

155 nce regarding the impact of APOE-epsilon4 on grey matter structure in asymptomatic individuals remain

156 the PRS in the relative volumes of four deep grey matter structures (caudate nucleus, thalamus, subth

157 NIH10), we investigated such differences for grey matter thickness (GM(th)), grey matter volume (GM(v

158 a novel grey matter metric distinctive from grey matter thickness or volume and detects differences

159 ght hemisphere white matter and post-therapy grey matter tissue density changes in bilateral temporal

160 Total grey matter tissue samples were obtained from medial OFC

161 ferences for grey matter thickness (GM(th)), grey matter volume (GM(vol)) and white matter surface ar

162 In responders across 9 datasets grey matter volume (GMV) was significantly higher in the

163 f-interest volumes was used to summarise the grey matter volume (GMV).

164 dy, we tested for differences in subcortical grey matter volume (n = 1157) and white matter integrity

165 ce of a mutation was associated with a lower grey matter volume (P = 0.002), even in presymptomatic s

166 ope of the correlation between education and grey matter volume (P = 0.007).

167 Differences in white matter integrity, grey matter volume and free water fraction between C9+ a

168 to test the relationship between prefrontal grey matter volume and striatal K(i)(cer).

169 rain atrophy in patients with MS in terms of grey matter volume and white matter volume as well as lo

170 trials to prevent its detrimental effect on grey matter volume and, eventually, cognition.

171 isk subjects had better cognition and higher grey matter volume at baseline; moreover, higher educati

172 First, we obtained a composite measure of grey matter volume by graph-Laplacian principal componen

173 Grey matter volume correlated negatively with illness du

174 Additional analyses using adjusted grey matter volume demonstrated a sequential pattern of

175 TMEM106B genotype did not influence grey matter volume directly on its own but in mutation c

176 -related neuropathologies on cross-sectional grey matter volume in a cohort of non-FTLD subjects.

177 .e. TMEM106B polymorphism, rs1990622 T/C) on grey matter volume in a large cohort of presymptomatic s

178 delling strategy that tests whether baseline grey matter volume in a seed region accounts for longitu

179 y matter pattern organization by correlating grey matter volume in all networks across all participan

180 Education directly affected grey matter volume in all the samples (P = 0.02) with lo

181 characteristic analyses to evaluate how well grey matter volume in each network and whole-brain patte

182 derived morphometric networks and extracted grey matter volume in each network.

183 ole striatal dopamine synthesis capacity and grey matter volume in left (pFWE corr. = 0.017) and righ

184 ted with neurocognitive impairments; smaller grey matter volume in limbic regions such as the amygdal

185 have, however, reported reduced hippocampal grey matter volume in MDD and reduced white matter integ

186 used meta-analytic coordinates of decreased grey matter volume in migraineurs as seed regions to gen

187 rphometry showed higher anterior hippocampal grey matter volume in mixed, compared to single supply.

188 Significant changes were found only in grey matter volume in MS subjects.

189 er's disease contributed additive effects on grey matter volume in nearly all networks, except fronta

190 We found that local grey matter volume in the left anterior superior tempora

191 l dopamine synthesis capacity and prefrontal grey matter volume in treatment-responsive patients with

192 Patients had grey matter volume increases in the temporal and limbic

193 presumed to be healthy in our sample and its grey matter volume is positively correlated with one's l

194 sed network mapping, we show that regions of grey matter volume loss in migraineurs localize to a com

195 er's disease exhibited different patterns of grey matter volume loss, with more extensive temporopari

196 clinical disease severity measures, NfL and grey matter volume of the frontal, temporal and parietal

197 Test (r(s) =-0.32, p=0.002) and with smaller grey matter volume of the parahippocampal gyri (r(s) =-0

198 t grey matter features, we evaluated whether grey matter volume or whole-brain pattern was related to

199 Significant grey matter volume reductions appeared in OSA throughout

200 ubjects show extensive regionally-demarcated grey matter volume reductions in areas that control cogn

201 s more consistent loneliness associations in grey matter volume than other cortical brain networks.

202 Grey matter volume was assessed globally.

203 the relationships between TDP-43 stages and grey matter volume while controlling for other pathologi

204 (i.e. premorbid intelligence and whole brain grey matter volume) had a positive relationship with the

205 These patterns were accompanied by increased grey matter volume, higher mean diffusivity, and enhance

206 nce of neuronal injury, measured as regional grey matter volume, in 16 OSA children (8 male, 8.1 +/-

207 e best model for SDMT showed that lower deep grey matter volume, reduced efficiency and male gender w

208 We assessed grey matter volume, white matter (WM) microstructural me

209 Impulsivity was not associated with grey matter volume.

210 o tissue myelin content, as well as quantify grey matter volume.

211 gher education has been related to increased grey matter volume.

212 l attainment; and (iii) TMEM106B genotype on grey matter volume.

213 en cognition and both 18F-AV-1451 uptake and grey matter volume.

214 eveloping cognitive impairment, more so than grey matter volume.

215 reward and threat expectancies and regional grey matter volumes (rGMV).

216 imer's disease from ageing, heterogeneity in grey matter volumes across morphometric networks and in

217 Grey matter volumes adjusted for pathological and demogr

218 ptake and rates of accumulation and baseline grey matter volumes and rates of atrophy across phenotyp

219 ep habits are associated with regional brain grey matter volumes and school grade average in early ad

220 For each subject, cortical and subcortical grey matter volumes were generated using a parcellation

221 Mapping toolbox to further control for local grey matter volumes, 11C-PiB uptake, or both.

222 Regional grey matter volumes, and regional binding of 18F-AV-1451

223 ehavior and suggests that alterations in the grey matter volumes, i.e., brain morphology, of specific

224 Subtype 1 showed widespread lower grey matter volumes, most prominent in thalamus, nucleus

225 inhibition, which manifests through reduced grey matter volumes, this region is presumed to be healt

226 be associated with cingulate and prefrontal grey matter volumes.

227 amyloid burden, but are in part mediated by grey matter volumes.

228 frontal lobe networks, where differences in grey matter were more specific to ageing.

229 MRI detects focal lesions in the white and grey matter with high sensitivity (with significantly le

230 tion (14/14), present in meninges, white and grey matter, associated with variable tissue destruction

231 sponders show different alterations in brain grey matter, but the findings are inconsistent.

232 the TSC in cortical grey matter (CGM), deep grey matter, normal-appearing white matter (WM) and WM l

233 Between white and grey matter, the absolute lesion volume in the white mat

234 al dips in WM tracts compared with activated grey matter, thus calling for significant changes to cur

235 Regional volumetric measures of grey matter, white matter, and CSF were used to identify

236 litis mouse model of CNS inflammation and in grey matter-containing multiple sclerosis lesions.

237 Mediation analyses revealed both direct and grey matter-mediated effects of 18F-AV-1451 uptake on co

238 he contours of strain and strain rate at the grey matter-white matter boundary were mapped.

239 rain injury patients were also mapped at the grey matter-white matter boundary.

240 scored for cortical lesion types I-IV (mixed grey matter/white matter, intracortical, subpial and cor

241 nd mean diffusivity (MD) in 25 bilateral (10 grey matter; 15 white matter) regions-of-interest (ROIs)

242 ether cerebrovascular function (measured via grey-matter cerebral blood flow (gmCBF)) is altered in y

243 ised by both common and distinct patterns of grey-matter volume changes.

244 Higher grey-matter volumes in the caudate nucleus and the left

245 mparisons indicated that findings of smaller grey-matter volumes relative to controls in the right do

246 Disruption of SEP4 in the plant grey mould fungus Botrytis cinerea completely blocked IF

247 rate that Jar1/KDM5 in Botrytis cinerea, the grey mould fungus, plays a crucial role in these process

248 Basal ganglia are subcortical grey nuclei that play essential roles in controlling vol

249 in individual columns of the periaqueductal grey (PAG) during breathlessness and its conditioned ant

250 The midbrain periaqueductal grey (PAG) lies at the heart of the defence-arousal syst

251 ns by over 300 million years of evolution: a Grey parrot (Psittacus erithacus).

252 ter videos systems, we show modifications in grey reef shark (Carcharhinus amblyrhynchos) occurrence

253 e and most abundant predator at Palmyra, the grey reef shark (Carcharhinus amblyrhynchos).

254 (above 150 km2) can protect the behavior of grey reef sharks found in the wilderness.

255 imated a density of 21.3 (95% CI 17.8, 24.7) grey reef sharks/km(2), which is an order of magnitude l

256 Instead, hair greying results from activation of the sympathetic nerve

257 alculated for each group after measuring the grey scale LMTK2 signal intensity of each individual neu

258 vesselness algorithm, in conjunction with a grey scale morphology filter and the removal of cell bod

259 Thyroid grey-scale and power Doppler US were performed, and a Wi

260 Grey-scale face classification is experimentally demonst

261 tom-up synthetic biology approach can detect grey-scale images and patterns of light moving across th

262 We studied the relationship between grey seal (Halichoerus grypus) behaviour and environment

263 S equipped with thermal sensors, imaging two grey seal (Halichoerus grypus) breeding colonies in east

264 or energy expenditure in 52 known individual grey seal (Halichoerus grypus) mothers, repeatedly acros

265 We provide evidence that grey seal mothers typically exhibit energy compensation

266 igh-resolution oceanographic, fisheries, and grey seal movement data, our study provides a scientific

267 ore conclude that these porpoises survived a grey seal predatory attack, with the bite lesions repres

268 al drivers in a capital breeding phocid, the grey seal.

269 Grey seals (Halichoerus grypus) are typical capital bree

270 Milk was collected from Atlantic grey seals (Halichoerus grypus) periodically from birth

271 ng top-down control by their main predators (grey seals and saithe).

272 Skipping breeding in female grey seals appears to be an individual mass-dependent co

273 to quantify changes in movement behaviour of grey seals while at sea.

274 mes of five mother and pup pairs of Atlantic grey seals, Halichoerus grypus, from birth to weaning.

275 ns had occurred due to traumatic injury from grey seals.

276 sons we measured resting HRV of 57 lactating grey seals.

277 chromosome inactivation in a marsupial, the grey short-tailed opossum (Monodelphis domestica).

278 indicate the appearance of bright, dark and grey solitons dwelling in the vicinity of the boundary o

279 rt that, in mice, acute stress leads to hair greying through the fast depletion of melanocyte stem ce

280 nd/or duration, with the color changing from grey to red with time of exposure at high temperature (4

281 was significant difference between the mean grey values of CNT vs. AD and neocortical LBD vs. AD.

282 Mean grey values were calculated for each group after measuri

283 distinct lesion characteristics in cortical grey versus subcortical white matter and neurodegenerati

284 hin-group analyses, increased periaqueductal grey volume was associated with role limitations due to

285 ed the cerebellum significantly changing its grey (volume increase of left VIIIa, relative decrease o

286 organic pollutants from sewage or so-called grey water.

287 , while slightly more hyper-reflective ROIs (grey) were detected with the PLEX Elite, more likely to

288 mb of internal capsule), level of brainstem, grey- white matters on levels of centrum semiovale (CS),

289 Our results indicate that the evolution of grey/white matter injury and blood-brain barrier disrupt

290 showed that NfL levels were associated with grey/white matter volume loss; grey matter atrophy in co

291 ation between plasma NfL and MRI measures of grey/white matter volumes in the Alzheimer's Disease Neu

292 The grey wolf is among the few Holarctic large carnivores th

293 itical debate underlying the recovery of the grey wolf throughout Europe.

294 s breeder turnover in cooperatively breeding grey wolves (Canis lupus Linnaeus 1758).

295 ong-term radiotelemetry and census data from grey wolves Canis lupus in the Upper Peninsula of Michig

296 or and lowest where coyotes co-occurred with grey wolves Canis lupus.

297 s exemplified by the recently re-established grey wolves of the Pacific Northwest states of Washingto

298 (mDNA) in 4% normal size (NS: < 44 CGGs), 6% grey zone (CGG 45-54) and 12% premutation (CGG 54-199) a

299 une-tolerant (IT), immune-inactive (IC), and grey zone (GZ), respectively, showed active antiviral cy

300 value at 4 ng/ml and the crucial diagnosis 'grey zone' of 4-10 ng/ml of PSA in serum for an accurate