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コーパス検索結果 (１語後でソート)

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1     GMF-null mice developed normally without gross abnormality.
2  mice were viable and fertile and lacked any gross abnormality.
3 t expected Mendelian ratios and exhibited no gross abnormalities.
4     NKCC2A-/- mice were viable and showed no gross abnormalities.
5 thologs, respectively, did not result in any gross abnormalities.
6 ase expression, develop normally and show no gross abnormalities.
7 evere reductions in brain size without other gross abnormalities.
8  mild ear development phenotype but no other gross abnormalities.
9 TPalpha(-/-)) mice are viable and display no gross abnormalities.
10  The PS1 cKO mouse is viable and exhibits no gross abnormalities.
11 iable, with moderate hemolytic anemia but no gross abnormalities.
12  knock-in mice, which are viable and have no gross abnormalities.
13  and fertile and do not exhibit any apparent gross abnormalities.
14                        Consistent with these gross abnormalities, Acbp is highly expressed in the pil
15 n these transductants does not introduce any gross abnormalities at the provirus-host DNA junctions.
16 omozygous Dusp16tp/tp mice developed without gross abnormalities but died perinatally.
17  misregulation of this pathway may result in gross abnormalities in cell number and function that may
18 activation of either or both genes causes no gross abnormalities in early spinal cord neurogenesis; h
19  of the postnatal TR4-/- cerebellum revealed gross abnormalities in foliation; specifically, lobule V
20           Rigorous characterization found no gross abnormalities in Mac(TRAP) mice and confirmed tran
21 ress, deletion of AK1 did not translate into gross abnormalities in nucleotide levels, gamma-ATP turn
22 roblasts derived from these mice do not have gross abnormalities in survival, proliferation, or clath
23                  Electron micrographs showed gross abnormalities in the cuticle of RNAi animals.
24 ch binding ability or specificity results in gross abnormalities in the function of the enzyme.
25  as a mechanism for describing and detecting gross abnormalities in the genome for many decades.
26 active protease coding sequence displayed no gross abnormalities in the lens, while mice with the act
27 , the lens opacities appeared to result from gross abnormalities in the shape and organization of cel
28 The CD93(-/-) mice were viable and showed no gross abnormalities in their development.
29 argeted ablation of the VEGF gene results in gross abnormalities in vascular patterning.
30 sed of normal 4R/3R-tau ratios indicating no gross abnormality in tau splicing.
31      By specific histochemistry, we detected gross abnormality in the distribution of small intestina
32 n OGS cell lines, however, karyotyping shows gross abnormalities involving chromosome 2 (location of
33 by the fact that mice lacking MARCKS exhibit gross abnormalities of central nervous system developmen
34 shaped body primordium but did not result in gross abnormalities of the trajectories of unicolumnar n
35                                           No gross abnormality of NK or platelet function was observe
36 photophobia and cannot be fully explained by gross abnormality of ocular anatomy or differences in ge
37                                Patients with gross abnormalities on macroscopic examination had a sig
38 the larval neuromuscular junction reveals no gross abnormalities, superresolution stimulated emission
39  vivo magnetic resonance spectroscopy (MRS); gross abnormalities using magnetic resonance imaging (MR
40 s of IFN-gamma than do controls, although no gross abnormalities were detected in lymphocyte populati
41 ions in the development of T and B cells, no gross abnormalities were detected in peripheral lymphocy
42                                  Although no gross abnormalities were detected in the Rp2(null) mice,
43                                     No other gross abnormalities were identified.
44                              In contrast, no gross abnormalities were observed in the posterior midgu