ライフサイエンスコーパス: growth arrest

1 other subpopulation undergoes senescence and growth arrest.

2 ine acetylation during growth transition and growth arrest.

3 ding to inhibition of mTORC1 and cancer cell growth arrest.

4 or alpha (TNF-alpha) that induced tumor cell growth arrest.

5 thway to regulate genes involved in cellular growth arrest.

6 induce membrane permeabilization and fungal growth arrest.

7 significantly exceed those required for cell growth arrest.

8 tized cells to proteasome inhibitor-mediated growth arrest.

9 t deletion of floxed CRK3, resulting in G2/M growth arrest.

10 of the DNA damage response and a G1/S-phase growth arrest.

11 ifesting as apoptosis highly correlated with growth arrest.

12 rring on a single day, the simultaneous silk growth arrest.

13 FKBP12, specifically inhibits TORC1, causing growth arrest.

14 e in Huh7.5 cells during HCV (JFH-1)-induced growth arrest.

15 d in the loss of HER3 and AKT activation and growth arrest.

16 while nevi harboring the same mutations have growth arrest.

17 ession, leading to persistent DNA damage and growth arrest.

18 ial cells, thus promoting cell apoptosis and growth arrest.

19 eads to elevated levels of p53, resulting in growth arrest.

20 d NF-kappaB signaling, ultimately leading to growth arrest.

21 armacological inhibitors of PKDs resulted in growth arrest.

22 ependent cytostatic Smad signaling to induce growth arrest.

23 prenylation, without affecting the senescent growth arrest.

24 l conditions applied simultaneously led to a growth arrest.

25 ls internalized enough RG7787 to only induce growth arrest.

26 stress, triggering thylakoid senescence and growth arrest.

27 curs despite relatively uniform induction of growth arrest.

28 r signaling to the MAP kinase p38 to mediate growth arrest.

29 hosphorylation and suppressed DspA/E-induced growth arrest.

30 n containing WT mazEF resulted in reversible growth arrest.

31 ring their activity over several days during growth arrest.

32 e cellular states acquired after the initial growth arrest.

33 remature senescence, a state of irreversible growth arrest.

34 a coli's stress response and is activated on growth arrest.

35 f neighboring cells, and competence-mediated growth arrest.

36 in a subset of cells triggers organism-wide growth arrest.

37 RhoA activation in dendrites coincident with growth arrest.

38 eting these oncogenic pathways induced tumor growth arrest.

39 hase, thereby linking pulcherriminic acid to growth arrest.

40 downstream effectors mediates BAI1-dependent growth arrest.

41 ry SASP independent of senescence-associated growth arrest.

42 U, lon, and clpXP to promote survival during growth arrest.

43 Ai promotes radiation-induced senescence and growth arrest.

44 using a plasmid system prevented the initial growth arrest.

45 In support of this inference, heat and growth arrest act synergistically to kill cells, and pro

46 Senescent cells have undergone permanent growth arrest, adopt an altered secretory phenotype, and

47 f PKA targets upon sulfate-induced exit from growth arrest after sulfur starvation.

48 damage response, apoptosis and p53-mediated growth-arrest, all of which are under the control of the

49 atrol (10 microM, 48 hr) induces both a cell growth arrest and a metabolic reprogramming in colon can

50 C/EBPbeta axis controls the senescence-like growth arrest and a PAK4 phosphorylation residue (RELB-S

51 r, Atg7-deficient MCs entered into premature growth arrest and accumulated reactive oxygen species (R

52 ontransformed cells undergo a TP53-dependent growth arrest and activate interferon signaling in respo

53 tment of tumor cells with induces both cells growth arrest and alter the spectral lines in a dose-dep

54 ermore, immune dormancy promotes cancer cell growth arrest and angiogenic control.

55 Sabutoclax induced growth arrest and apoptosis in pancreatic cancer cells a

56 ates expression of these targets and induces growth arrest and apoptosis in persister cells, at doses

57 Notch activation even promoted growth arrest and apoptosis of colorectal carcinoma cell

58 y RNA interference has been shown to promote growth arrest and apoptosis of tumor cells.

59 MCs showed enhanced DSB repair and decreased growth arrest and apoptosis, whereas SM22alpha-(DeltaC)N

60 However, excess ceramide is toxic, causing growth arrest and apoptosis.

61 more resistant to BET-inhibitor-induced cell growth arrest and apoptosis.

62 ablished mechanism of action is induction of growth arrest and apoptosis.

63 may be pivotal to sensitizing CTCL cells to growth arrest and apoptosis.

64 SMCs showed reduced DSB repair and increased growth arrest and apoptosis.

65 egrin beta7 expression and function prior to growth arrest and apoptosis.

66 , the bipartite PrpS-PrsS module can trigger growth arrest and cell death in vegetative cells.

67 he inter-relationships among autophagy, cell growth arrest and cell death induced by these ligands re

68 in trophoblast cell line HTR8/SVneo induced growth arrest and cellular senescence via activation of

69 pollen-expressed EXO70 isoforms resulted in growth arrest and characteristic phenotypic deviations o

70 ccur more rapidly in protease mutants during growth arrest and correlates with the onset of cell deat

71 aurin/PKC412 are sensitive to GO-203-induced growth arrest and death.

72 ous ASM or ceramide enhanced epithelial cell growth arrest and death.

73 r long term physiological responses, such as growth arrest and differentiation.

74 K-eEF2K axis) causes tumour cells to undergo growth arrest and differentiation.

75 at the DNA damage and stress response genes, Growth arrest and DNA damage (GADD45) and Apoptosis sign

76 We have identified a member of the growth arrest and DNA damage (Gadd45) protein family, Ga

77 as mediated through cell-cycle regulation by Growth Arrest and DNA Damage 45 gamma (Gadd45gamma).

78 in in vivo We also demonstrate that Gadd45g (growth arrest and DNA damage inducible gamma) is a direc

79 riptional targets, glucose-6-phosphatase and growth arrest and DNA damage protein 45 alpha (Gadd45alp

80 Additionally, FIH-1 regulates growth arrest and DNA damage-45 alpha (GADD45alpha), a n

81 Growth arrest and DNA damage-inducible beta (GADD45b) pl

82 Following relief of the stress, the growth arrest and DNA damage-inducible protein GADD34 as

83 e mRNA coding for the stress response factor growth arrest and DNA-damage inducible 45 (GADD45) can a

84 ut genome-wide cDNA screen, we identify here growth arrest and DNA-damage-inducible protein 45 gamma

85 loss of DNA methylation requires the Gadd45 (Growth arrest and DNA-damage-inducible) gene family, ver

86 rain-derived neurotrophic factor (Bdnf), and growth arrest and DNA-damage-inducible, beta (Gadd45b)]

87 Ras-transformed cells override a131-induced growth arrest and enter mitosis where a131's ability to

88 ene expression and controls TGF-beta-induced growth arrest and epithelial-to-mesenchymal transition (

89 ermidine, and spermine resulting in cellular growth arrest and eventual death.

90 Whereas a high pheromone dose induces growth arrest and formation of a shmoo-like morphology i

91 f mating pheromone, the yeast cell undergoes growth arrest and forms a shmoo-like morphology; however

92 splay an accelerated senescence sustained by growth arrest and increased senescence-associated beta-g

93 suppressors of MDM33 overexpression-induced growth arrest and isolated binding partners by immunopre

94 (INK4a) and p19(ARF), which are required for growth arrest and myeloid differentiation following Hhex

95 ked T antigen knockdown by inducing MCC cell growth arrest and neuron-like differentiation.

96 cellular response characterized by a stable growth arrest and other phenotypic alterations that incl

97 reduced ER stress or Xbp1 splicing promotes growth arrest and premature senescence through hyperacti

98 of the MYC -428 and -525 ESE eRNA caused LCL growth arrest and reduced cell growth.

99 ition of TOR by inducible RNAi, led to plant growth arrest and reduced expression of BR-responsive ge

100 kers characteristic of HGPS cells, including growth arrest and senescence-associated beta-gal (SA-bet

101 istant cells, an effect associated with both growth arrest and senescence.

102 These alterations induce p15/16 growth arrest and senescence.

103 Cells committed to growth arrest and shmoo formation exhibited sustained Fu

104 d H3K9 methylation levels in cells and cause growth arrest and substantial apoptosis in LNCaP prostat

105 isms demonstrates that a reversible cellular growth arrest and the transcription factor, nuclear fact

106 hypothesis of oncogene-induced senescence in growth arrest and tumor suppression in melanocytic nevi

107 he HIV protease inhibitor ritonavir elicited growth arrest and/or apoptosis in multiple myeloma.

108 rawal of the single oncogenic event leads to growth arrest and/or cancer regression.

109 entiated cancer cell-derived adipocytes were growth-arrested and lost their cellular plasticity.

110 sses such as differentiation, proliferation, growth arrest, and apoptosis.

111 A single- and double-strand break formation, growth arrest, and apoptosis.

112 n fail to redifferentiate, undergo premature growth arrest, and become atrophic.

113 stimulation of p53 transcriptional activity, growth arrest, and cellular senescence.

114 8 in PC3, DU145 and LNCaP cells induced cell growth arrest, and decreased tumour volumes and mortalit

115 eristic of young animals, reduces markers of growth arrest, and increases keratinocyte and fibroblast

116 in translation, massive protein aggregation, growth arrest, and lethality.

117 al flg22 responses including gene induction, growth arrest, and plasma membrane depolarization.

118 teins increased AML cell maturation, induced growth arrest, and prolonged survival in an AML mouse mo

119 major driver of aging and cell death during growth arrest, and that coordinated activity of the heat

120 in was refractory to myristic acid-dependent growth arrest, and unlike the wild-type strain, was susc

121 sive mechanism characterized by irreversible growth arrest, apoptosis resistance, production of a sen

122 expression of both transformability and the growth arrest are bet-hedging adaptations that improve f

123 ogression to melanoma, but only after stable growth arrest as nevi.

124 ssion of the AapA1 toxin in H. pylori causes growth arrest associated with rapid morphological transf

125 growth in LNCaP by rescuing LNCaP cells from growth arrest at G1 phase due to the lack of androgen.

126 ant led to a visible virescent phenotype and growth arrest at low temperature.

127 tivation of CCN5 in TNBC cells promotes cell growth arrest at the G0/G1 phase, reduces cell prolifera

128 P1 to trigger DNA damage response and induce growth arrest at the G2/M phase, to induce senescence, a

129 27 and Wee1 to trigger DNA damage and induce growth arrest at the G2/M phase.

130 OC included chronic osteomyelitis, growth arrest, avascular necrosis, chronic dislocation,

131 lomerular epithelial cells that are normally growth-arrested because of the expression of cyclin-depe

132 lone and propranolol shortened the period of growth arrest by 84 days (P = 0.0125 vs control) and inc

133 ate distinct cellular outputs: p38-dependent growth arrest, cAMP response element-binding protein-dep

134 Means by which growth arrest can be overcome and how melanocytic nevi r

135 A growth arrest caused by the TbCAE knockdown was rescued

136 The growth arrest caused by TOR inactivation was partially r

137 eased fitness can result from a catastrophic growth arrest caused by unexpected darkness in a small s

138 and 4t) potently inhibited cell survival and growth, arrested cell cycle, and blocked angiogenesis an

139 At the plasma membranes of growth-arrested cells, AJUBA LIM proteins do not inhibit

140 motility, decreases adhesion, and induces a growth arrest, changes associated with a complete EMT th

141 escape lethal DNA damage effects by inducing growth arrest commonly referred to as cellular dormancy.

142 broblasts with proliferating and transiently growth arrested controls using a combination of differen

143 The intratumor bleeding and tumor growth arrest could be reverted by depletion of Ly6G(+)

144 riments show that HBI1 inhibits PAMP-induced growth arrest, defense gene expression, reactive oxygen

145 by inhibition of Exportin-1 (XPO1) promoted growth arrest, demonstrating that the biological effects

146 estigated the expression and function of the growth arrest DNA damage (Gadd45) family during aging an

147 eline and learning-induced expression of the growth arrest DNA damage (Gadd45) gamma is selectively i

148 tion in primary endothelial cells with KSHV, growth arrest DNA damage-inducible gene 45 beta (GADD45B

149 enrichment of biological pathways related to growth arrest, DNA damage response and the lysosomal pat

150 Restoration of NOTCH signaling caused growth arrest due to activation of the NOTCH effector HE

151 abA5e displayed delayed seed germination and growth arrest during oxidative stress.

152 o maintain growth in favorable conditions or growth arrest during stress.

153 L-1 inhibited cell proliferation by inducing growth arrest during the G1/S phase transition, promoted

154 d alkaline pH, suggesting that heat, pH, and growth arrest effectively impose a similar type of prote

155 ance in exponentially growing and stationary growth-arrested epimastigote parasites.

156 yl-tRNA hydrolase counteracts TacT-dependent growth arrest, explaining how bacterial persisters can r

157 bules recovering from AKI undergo pathologic growth arrest, fail to redifferentiate, and become atrop

158 Loss of C/EBPbeta expression prevents growth arrest following androgen deprivation or anti-and

159 testosterone, and reduced strength alongside growth arrest for up to 2 years after injury.

160 Our previous studies showed: (i) that growth-arrested G0/G1 rat mesangial cells stimulated to

161 onsequently enhanced cisplatin-induced tumor growth arrest in a patient-derived xenograft model.

162 progression by inducing senescence-mediated growth arrest in aberrantly proliferating Nf1-/- SC.

163 Importantly, PAK4 prevents senescence-like growth arrest in breast cancer cells in vitro, in vivo a

164 TR-CD4) potently induced IFN-gamma-dependent growth arrest in cancer cells.

165 Cellular senescence is a permanent growth arrest in cells with damage or stress that could

166 rescue experiments reversed miR-203-induced growth arrest in cSCC, which highlights the importance o

167 Indeed, the growth arrest in early infected cells could be rescued b

168 lating the balance between proliferation and growth arrest in hematopoietic progenitors, myeloid line

169 p53-mediated fibrotic gene reprogramming and growth arrest in HK-2 cells.

170 ides effective immunity but can also lead to growth arrest in infected cells, necessitating a means t

171 ts depletion robustly induces cell death and growth arrest in MTC cell lines in culture and in mouse

172 PIP4Ks inhibition by a131 causes reversible growth arrest in normal cells by transcriptionally upreg

173 lone and propranolol attenuates burn-induced growth arrest in pediatric burn patients.

174 After bud growth arrest in phyB-1, expression of dormancy-associat

175 TP53BP1 and USP28, but not TRIM37, prevented growth arrest in response to prolonged mitotic duration.

176 In these conditions, Csm6 causes growth arrest in the host and prevents further plasmid r

177 ioception mechanism is a key step leading to growth arrest in the whole sepal in response to its own

178 promote radiation-induced TAZ inhibition and growth arrest in these tumor cells.

179 Here we show that BTIC growth arrest in vitro and in vivo is accomplished via c

180 Absence of HtrA led to growth arrest in vitro that was partially restored by an

181 amount equivalent to the amount that induced growth arrest in vitro.

182 Here, rat mesangial cells (RMCs) were growth-arrested in the G(0)/G(1) phase of cell division,

183 The tested analogues overcame growth arrest induced by a 72 h treatment with alpha-dif

184 -14-mediated Sp1 protein stabilization, G2/M growth arrest induction, and anchorage-independent growt

185 llen tubes to protect microtubules and avoid growth arrest involved in sexual incompatibility reactio

186 by the addition of pyruvate suggesting that growth arrest is due to a pH-dependent checkpoint on met

187 ticular, we focus on the mechanisms by which growth arrest is established after BRAF(V600E) mutation.

188 BAI1-mediated growth arrest is independent of its Rac1-dependent synap

189 Instead, a reversible growth arrest is induced that can be overcome by reoxyge

190 We suggest that nevus growth arrest is more likely related to the cell interac

191 a wild-type shoot; however, the mechanism of growth arrest is not understood.

192 suggest that IR-induced p16(INK4a) dependent growth arrest is reversible in mice and that sustained p

193 ntal effects on plant development, including growth arrest, leaf necrosis, and reduced seed productio

194 of this repression system leads to a strong growth arrest, likely due to overly rapid galactose cata

195 ing stationary phase and in conjunction with growth arrest linked to carbon starvation.

196 Cellular senescence is a process of cellular growth arrest linked with aging and inflammation.

197 Braf(V600E) induces benign, growth-arrested melanocytic nevus development, but also

198 etal ciliopathies suffer from premature bone growth arrest, mirroring skeletal features associated wi

199 ociated with a dose- and time-dependent cell growth arrest, mitochondrial damage, and apoptosis induc

200 Growth-arrested Mtb is resuscitated by the addition of p

201 Here we show that growth arrest of AREG-silenced keratinocytes occurs in G

202 tivation through Stk11 (Lkb1) loss abrogates growth arrest of Braf(V600E) melanocytic nevi, but is in

203 rate that the G-quadruplex ligand 20A causes growth arrest of cancer cells in culture and in a HeLa c

204 ynthetic activity results in an irreversible growth arrest of CRC.

205 MPDH2 activity triggers nucleolar stress and growth arrest of glioblastoma cells even in the absence

206 ew conceptual paradigm for understanding the growth arrest of melanocytic nevi in vivo termed stable

207 ule-mediated inhibition of HUWE1 resulted in growth arrest of MM cell lines without significantly eff

208 Previously, we found that cAMP-induced growth arrest of PC12 and NS-1 cells requires Epac2-depe

209 c effects of aristolochic acid are linked to growth arrest of proximal tubular epithelial cells; howe

210 lar environments, some of which may favour a growth arrest of Salmonella facilitating immune evasion

211 neural progenitor cells (hNPCs) and leads to growth arrest of these cells, which are critical for bra

212 Unexpectedly, the rapamycin hypersensitive growth arrest of vip1-1 cells was dependent on the prese

213 ar senescence, a stress-induced irreversible growth arrest often characterized by expression of p16(I

214 high cell population imposes an irreversible growth arrest on CRCs.

215 ilization/activation, and p53-dependent cell growth arrest or apoptosis upon DNA damage.

216 ization and activation of p53 and subsequent growth arrest or apoptosis.

217 daptive gene expression programme leading to growth arrest or cell death.

218 with CW remodeling and biosynthesis to avoid growth arrest or integrity loss.

219 When NR2F1 is blocked in vivo, growth arrest or survival of dormant DTCs is interrupted

220 em cells can reside in a state of reversible growth arrest, or quiescence, for prolonged periods of t

221 an enhanced Cu-dependent phenotype involving growth arrest, oxidative stress, floral bud abortion, an

222 Intriguingly, mortalin depletion induced growth arrest partly via the MEK/ERK pathway, whereas it

223 port a model in which miR167-mediated anther growth arrest permits anther dehiscence.

224 racterized by reduced cell division rates or growth arrest, persistence, or lysis, concomitant with I

225 opic introduction of SPRY4 recapitulated the growth arrest phenotype of dual BRAF(V600E)/NRAS(Q61) ex

226 pc2, was constructed that exhibited a severe growth-arrest phenotype.

227 kout strain fails to exhibit the long-lived, growth-arrested phenotype, suggesting that altered regul

228 Analysis of the compounds cellular growth arrest phenotypes and microtubule dynamics sugges

229 D2 attenuates the transcriptional output and growth arrest phenotypes downstream of IFN signaling in

230 ) that triggers cell wall alkalinization and growth arrest, possibly through the inhibition of plasma

231 This growth arrest prevents long-term outgrowth of the majori

232 l rat ECFCs isolated from hyperoxic alveolar growth-arrested rat lungs mimicking bronchopulmonary dys

233 Senescence, a durable form of growth arrest, represents a primary response to numerous

234 ys (l-ascorbate utilization and metabolism), growth arrest response, heat shock response, DNA recombi

235 MazF-mediated growth arrest resulted in an increase in survival of bac

236 mes of cellular growth, including a phase of growth arrest resulting from toxicity of the metabolic p

237 of YjhX (85 amino acid residues) causes cell-growth arrest resulting in cell death, while YjhQ (181 r

238 oncogenic stress to normal cells, leading to growth arrest (senescence) or apoptosis.

239 MYCN loss led to growth arrest, sensitizing cells for apoptosis following

240 It also activated apoptotic and cell growth arrest signaling.

241 Here, we studied the role of growth arrest specific 6 (GAS6), a ligand of the TYRO3-A

242 xl, one of the tyrosine kinase receptors for growth arrest specific 6 (Gas6).

243 The TAM receptor ligand, growth arrest specific 6, re-establishes the normal FM r

244 enib-resistant Huh-7 cells, inhibiting TYRO3/growth arrest specific 6-mediated cancer cell migration

245 horts was located in the long non-coding RNA growth arrest specific five gene (GAS5) (p < 10(-24)).

246 jections by Shh requires its binding partner growth arrest specific gene 1 (Gas1) and its signaling c

247 was originally identified in fibroblasts as growth arrest specific gene 3 (gas3) and is expressed br

248 Growth arrest specific gene two (GAS2) is a highly conse

249 proteins related to hepatogenic lineage like growth arrest specific protein 6, oncostatin M, hepatocy

250 The down-regulation of lncRNA growth arrest specific transcript 5 (GAS5) has been repo

251 GF signaling by activating the expression of growth arrest-specific 1 (Gas1), a novel WT1 target gene

252 ding Fas-associated phosphatase 1 (Fap1) and growth arrest-specific 2 (Gas2) and activates genes enco

253 viously shown that a member of the family of growth arrest-specific 2-like proteins, GAS2-like 1 (G2L

254 Here, we demonstrate that the lncRNA growth arrest-specific 5 (GAS5) suppresses TGF-beta/Smad

255 Based on recent evidence that (1) growth arrest-specific 6 (Gas6) regulates the expression

256 The AXL receptor and its activating ligand, growth arrest-specific 6 (GAS6), are important drivers o

257 E2 stimulated LCs to produce growth arrest-specific 6 (GAS6), which mediates phagocyt

258 tic cancer cells treated with the AXL ligand growth arrest-specific 6 (GAS6).

259 In contrast, MerTK and its known ligands, growth arrest-specific 6 and Protein S, were downregulat

260 Of these variants, two protein S/growth arrest-specific 6 chimeras, with either the whole

261 ernative model for SRC and MET activation by growth arrest-specific 6 in ccRCC and identify AXL as a

262 , substituted by the corresponding domain in growth arrest-specific 6, were unable to enhance TFPI.

263 verexpression of TAMs and their major ligand Growth arrest-specific factor 6 (Gas6) is associated wit

264 by endogenous ligands, protein S (PROS1) and growth arrest-specific gene 6 (GAS6).

265 oduces a stop codon in amino acid 308 of the growth arrest-specific protein 2-like 2 (GAS2L2).

266 egulation of larval diapause, the long-lived growth arrest state called dauer arrest, in Caenorhabdit

267 ular senescence defines an irreversible cell growth arrest state linked to loss of tissue function an

268 to be important for cellular longevity in a growth-arrested state.

269 Although the multiplicative and growth-arrested states play key roles in Leishmania deve

270 One of these, the differentiation of growth-arrested stumpy forms in the mammalian blood into

271 hock protease FtsH is generally required for growth arrest survival of Pseudomonas aeruginosa, and th

272 ssion of a second activating oncogene led to growth arrest ("synthetic suppression") in a subset of c

273 amphiregulin (AREG), results in keratinocyte growth arrest that cannot be rescued by soluble extracel

274 common molecular mechanisms that enforce the growth arrest that characterizes the phenotype, the impa

275 Cellular senescence entails an irreversible growth arrest that evolved in part to prevent cancer.

276 Cellular senescence is a stable cell growth arrest that is characterized by the silencing of

277 Cellular senescence is a stable growth arrest that is implicated in tissue ageing and ca

278 renovated ancient programs of stress-induced growth arrest that were already present in ferns, bryoph

279 d SMAD3-mediated fibrotic gene induction and growth arrest that were reversed by ectopic PPM1A expres

280 t phenotype and its functional output beyond growth arrest: the senescence-associated secretory pheno

281 lgae is a stress response tightly coupled to growth arrest, thereby imposing a major limitation on pr

282 or, and GEF substrate of, Epac2 in mediating growth arrest through p38 activation in NS-1 cells.

283 ck the DNA damage response (DDR) and ensuing growth arrest through suppression of ATM-FOXO3a associat

284 nd effective strategy for inducing cancerous growth arrest through the direct epigenetic regulation o

285 ition is required for apoptosis, but not for growth arrest, through a mechanism involving the derepre

286 s absence switches response to TGF-beta from growth arrest to EMT.

287 uring the early stage, cells transition from growth-arrested to growing.

288 Cellular senescence, an irreversible growth arrest triggered by a variety of stressors, plays

289 Here macrophages, growth-arrested tubular epithelial cells, the endotheliu

290 splanted RAS mutant organoids confirmed this growth arrest upon pan-HER/MEK combination therapy.

291 iated Bax activation and that increased cell growth arrest was associated with elevated expression of

292 wn-regulation using shRNA, which caused cell growth arrest, was accompanied by increased H3K27me3 at

293 ion of the Kras(ex3op) allele, which induced growth arrest when oncogenic and exhibited tumor-suppres

294 Inhibiting PGE2 production led to growth arrest, whereas addition of MSC-derived PGE2 rest

295 ectopic CELF1 overexpression caused G1-phase growth arrest, whereas CELF1 silencing promoted cell pro

296 Depletion of YAP resulted in growth arrest, whereas its overexpression promoted cell

297 -catenin in the liver induces senescence and growth arrest, which is overcome by combined CAR activat

298 Noise can drive entry of single cells into growth arrest while a fast-growing majority sustains the

299 the proteases function hierarchically during growth arrest, with FtsH and ClpXP having primary, nonre

300 e fit by any simple cell-autonomous model of growth arrest, yet were easily fit by models based on co