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1 turation, leading to the nanomelic long bone growth disorder.
2  mechanism of how JAK2 mutation induces cell growth disorder.
3 nd determinants of biological aging in these growth disorders.
4 gs useful for the treatment of various human growth disorders.
5  underscored by its association with several growth disorders.
6 s are known as the genetic basis of skeletal growth disorders.
7 tations are associated with several dominant growth disorders.
8 r identifying complications, including fetal growth disorders.
9 nd proportions and the aetiology of skeletal growth disorders.
10 so arise in isolation, causing developmental growth disorders.
11 r benefits to select patients with rare bone growth disorders, acute promyelocytic leukemia, and some
12 ing genes associated with both developmental growth disorders and hematological malignancies.
13  overlap genes that are mutated in monogenic growth disorders and highlight new biological candidates
14 ew knowledge about the physical basis behind growth disorders and highlight the fact that a single RT
15 erlying principles of hair development, hair growth disorders, and skin cancers.
16                                              Growth disorders are common in pediatric patients with i
17 vide useful therapeutic tools for human hair growth disorders based on premature or retarded catagen
18 hylation of this gene is associated with the growth disorders Beckwith-Wiedemann and Silver-Russell s
19  IGF2-H19 expression and the two contrasting growth disorders, Beckwith-Wiedemann syndrome (BWS) and
20  in the long-term treatment of children with growth disorders, but there is considerable treatment re
21              Primary microcephaly is a brain growth disorder characterized by a severe reduction of b
22 yndrome is an autosomal-recessive primordial growth disorder characterized by significant intrauterin
23 sts may be useful for the management of hair growth disorders characterized by premature entry into c
24 dopsis (Arabidopsis thaliana) shows retarded growth, disordered chloroplast development, and delayed
25  an open study of consecutive referrals to a growth disorder clinic from which 16 short children (hei
26 ppropriate detection and management of Fetal Growth Disorders (FGD).
27                           Common sequelae of growth disorders include neurodevelopmental defects, som
28 ith sporadic Wilms tumor without features of growth disorders, including 12% of bilateral cases (P =
29 ormation on the placental phenotype in fetal growth disorders--information which might contribute to
30                   3-M syndrome, a primordial growth disorder, is associated with mutations in CUL7 an
31 transcription factor in the brain in certain growth disorders may contribute to neurocognitive defici
32 one (hGH) in vivo is linked to metabolic and growth disorders such as cancer, diabetes, and acromegal
33 onsidered a candidate gene for the imprinted growth disorder, the Silver-Russell syndrome (SRS), but
34 cling abnormalities underlie many human hair growth disorders, the accurate classification of individ
35  alpha (DNMT3A) are commonly associated with growth disorders, whereas somatic mutations are often as
36 asia type I (TDI) is a lethal human skeletal growth disorder with a prevalence of 1 in 20,000 to 1 in