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1 ilm in a viable but antibiotic-tolerant slow-growth state.
2  influences tumor progression to an invasive growth state.
3 red to reflect a switch back to a vegetative growth state.
4 rowth and entry into a stationary phase-like growth state.
5 ulent at the skin, regardless of its initial growth state.
6 a coli cells from an aerobic to an anaerobic growth state.
7 lation of CAD were measured as a function of growth state.
8 is enzyme is required, according to cellular growth state.
9 growth factor and not mediated by changes in growth state.
10 ransition from a stationary to proliferative growth state.
11 nsition from a stationary to a proliferative growth state.
12 ooth muscle cells into a quiescent G(0)-like growth state.
13 in metabolism in these organisms between the growth states.
14 ctions between species in steady exponential growth states.
15 olonged period of suppressed budget and reef growth states.
16 served in these pathogens in their different growth states.
17 oes not significantly differ between the two growth states.
18 h its formulation and use to compute optimal growth states.
19 to assess transcription during each of these growth states.
20 rcoil barriers than wild-type strains in all growth states.
21 stoma and WI38 fibroblast cells in different growth states.
22 uits to switch cells to a high synthesis-low growth state after first growing to a large population c
23 ires reactivating injured neurons' intrinsic growth state and enabling growth in an inhibitory enviro
24 nglion cells (RGCs) to switch into an active growth state and extend lengthy axons down the nerve.
25 cript abundances are influenced by bacterial growth state and plasmid carriage.
26 esults suggest that the response may be cell growth state and protein kinase C-dependent.
27 en nuclei and mitochondria, depending on the growth state and stress environment of the cell.
28 visual input, the eyes revert to a "default" growth state and that the similarities between the effec
29 fully transit from a biofilm to a planktonic growth state and to spawn novel communities in new local
30 o transit from the biofilm to the planktonic growth state and to spawn novel communities in new local
31 ding nutrient availability, and adjust their growth state and virulence functions accordingly.
32 es MnSOD expression in response to different growth states and radiation.
33 hen E. coli cells transition to an anaerobic growth state, and that the expression of 712 (49%) of th
34  expression profile and switch into a strong growth state; and (2) whether inactivating RhoA, a conve
35            When neurons are triggered into a growth state, as in the conditioning lesion paradigm, H4
36                      This altered epithelial growth state associates with persistent features of rege
37 BP mRNA, and its expression was regulated by growth state, being most prominent in quiescent endothel
38  motor neurons to stimulate their endogenous growth state, bone marrow stromal cell grafts in lesion
39  Typhimurium, speed up the return to a rapid growth state by preventing the proteolysis of functional
40 orphogenesis to a hyphal form, a filamentous growth state, C. albicans FRE8 mRNA is induced, which le
41     To elucidate the factors responsible for growth state-dependent regulation of pyrimidine biosynth
42           For the first time, we demonstrate growth-state-dependent changes in the mass signatures of
43                     These data show that the growth-state-dependent tyrosine phosphorylation of p27 m
44 y and noninhibitory, due to its differential-growth-state-dependent tyrosine phosphorylation.
45 indicating that the bacteria were in a rapid growth state despite specific nutrient limitations.
46 bet hedging results from multiple epigenetic growth states determined by a combination of stochastic
47 eradicates cells grown in numerous different growth states (e.g. planktonic cultures and highly robus
48  the existence of bacteria in two additional growth states, filamentous and biofilm encased.
49 IGF-1R was observed during the transition of growth states from exponential to quiescent.
50          Maintenance of the stationary phase growth state has been proposed to be critical for the vi
51                                         Such growth states have been recently defined as dormant or p
52 t the new pole attractor; and (5) the robust growth state identified by Wang et al. corresponds to ou
53 ring transitions into, and out of, the rapid growth state in glucose, while TORC1 is important for th
54 ricyte shape, contractility, and endothelial growth state in microvascular cell co-cultures.
55 ling states: (i) a Gtr1/2 on, Pib2 on, rapid growth state in nutrient replete conditions; (ii) a Gtr1
56 ) a Gtr1/2 inhibited, Pib2 on, adaptive/slow growth state in poor-quality growth medium; and (iii) a
57 ransition from the biofilm to the planktonic growth state in response to various cues.
58 for maintenance of the anchorage-independent growth state in RK3E cells transformed by beta-catenin,
59 y, but rather is expressed during a range of growth states in a dynamic environment.
60  amounts and fluctuates considerably between growth states in response to physiological changes.
61  so strongly that they are forced out of the growth state into stressed, nongrowing states.
62 gest that protein preservation during a slow-growth state is a conserved microbial strategy that faci
63      Adjusting endothelial metabolism to the growth state is central to normal vessel growth and func
64 ics in response to nutrient availability and growth state is poorly understood, yet essential for the
65 plasmic molecular crowding and morphological growth states is unclear.
66 e with cells that accumulate during abnormal growth states, like prostate cancer.
67 mounting for the importance of the intrinsic growth state of a neuron as a crucial determinant of its
68 scriptional switch that promotes an enhanced growth state of adult sensory neurons.
69            Based on microscopic imaging, the growth state of an independent culture developing in the
70 (FI, B-50 or neuromodulin) in regulating the growth state of axon terminals.
71 cate that this is achieved through a boosted growth state of dorsal column projecting sensory neurons
72                                The intrinsic growth state of injured neurons is, therefore, a key det
73 rve regeneration by increasing the intrinsic growth state of injured neurons.
74 tion through the mature optic nerve when the growth state of neurons is activated.
75 ong regeneration even without activating the growth state of neurons.
76                                          The growth state of P. aeruginosa in CF airways is probably
77 nvestigated whether increasing the intrinsic growth state of primary sensory neurons by a conditionin
78 nts that synergistically alter the intrinsic growth state of RGCs produce unprecedented levels of axo
79 tion of retraction was dependent on both the growth state of the axon and the activation state of the
80 , modified backbone, probe concentration and growth state of the bacteria were investigated.
81 not known whether the metabolic pathways and growth state of the bacterium influence synthesis and se
82 ption, tightly coupled to the cell cycle and growth state of the cell, is a key process for understan
83 operons, regulons, and stimulons to suit the growth state of the cell.
84 ed the effect of critical parameters such as growth state of the cells and availability of intracellu
85 to grow within D. discoideum depended on the growth state of the cells.
86 ally identical counterparts, identifying the growth state of the culture based on single-organism spe
87 ct seems to be primarily associated with the growth state of the epithelium.
88 hibition was conditional, dependent upon the growth state of the inhibitory cell and the pili express
89 ontact with agar ingredients rather than the growth state of the microorganisms determined the infect
90 ion of effectors that increase the intrinsic growth state of the neurons.
91 we have found to be expressed inversely with growth state of the SMC.
92 um-derived NO controls the contractility and growth state of the underlying vascular smooth muscle ce
93      We determined the influence of cellular growth state on DEN type 2 (DEN2) replication in mosquit
94 al strategy that facilitates the return to a growth state once nutrients become available.
95 stresses impact a cell's decision to enter a growth state or a quiescent state.
96 by ATP-dependent proteases while in the slow-growth state or stationary phase.
97 owth, which we propose to call 'oligotrophic growth state', provides an alternative strategy for B. s
98 e-entry into the cell cycle from sub-optimal growth states rather than promoting or controlling the p
99 s cerevisiae exhibits alternative vegetative growth states referred to as the yeast form and the fila
100 ted mRNA was in the polysome fraction in all growth states regardless of labeling time, indicating th
101 and beta-lactams, giving rise to an abnormal growth state termed persistence.
102 ect and glutamine addiction as features of a growth state that provides resistance to metabolic stres
103 oietic stem cells (HSCs) can convert between growth states that have marked differences in bioenerget
104  efficiency may be partly due to the special growth states that mycobacteria enter to avoid being kil
105 effects are largely driven by differences in growth states that persist into the perturbation experim
106 al microtubules and was dependent on polymer growth state; the microtubule-related fluorescence dissi
107 ironmental stresses and transitions from one growth state to another, the transcriptional coupling th
108 ady-state cellular pre-16S rRNA pools during growth state transitions in Escherichia coli.
109 during metabolic adjustments associated with growth state transitions.
110 mooth muscle cells (SMCs) are in a quiescent growth state under normal physiological conditions, but
111 of heterogeneous cultures depending on their growth state using the destruction-free optical method o
112                                This enhanced growth state was accompanied by an increase in the expre
113  both regions, transitions from quiescent to growth states were accompanied by reorganization of MTs
114 al context of the organisms as well as their growth state - whether they are growing, non-growing or
115 ely other factors) defines two distinct axon growth states, which are critical for proper circuit for
116 r TG storage in cells in response to varying growth states, which may have broad implications for thi
117 the hypothesis that manipulation of cellular growth state will facilitate identification of viral and
118 s) are plastic cells that can switch between growth states with different bioenergetic and biosynthet

 
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