ライフサイエンスコーパス: growth stimulation

1 can be a determinant of tumor E2 content and growth stimulation.

2 region, designated yfe, essential for SAB11 growth stimulation.

3 SAPK pathway that correlated with a 150-190% growth stimulation.

4 ompounds counteracted exogenous ET-1-induced growth stimulation.

5 e normal control of E2F2 expression during a growth stimulation.

6 S may be an effector of Ras in cAMP-mediated growth stimulation.

7 was expressed at a constant level following growth stimulation.

8 d, plant-to-plant signalling for defence and growth stimulation.

9 n essential component for DNA synthesis upon growth stimulation.

10 crimination of aberrant and normal levels of growth stimulation.

11 tion, elevated BfeA receptor production, and growth stimulation.

12 nd eventually neutralise carbon gains due to growth stimulation.

13 s of atmospheric CO(2), possibly due to tree growth stimulation.

14 content or stability of UAP56 mRNA following growth stimulation.

15 of rp mRNA do not change significantly after growth stimulation.

16 le the signal transduction pathways for cell growth stimulation.

17 y the response of these livers to additional growth stimulation.

18 ls and partially mediated the PGE(2)-induced growth stimulation.

19 ivation of the E2F3a promoter in response to growth stimulation.

20 wild-type Smad4 abolished the BMP-2-mediated growth stimulation.

21 define the molecular mechanism of carcinoma growth stimulation, a three-dimensional co-culture model

22 a cells, which are responsive to TGFbeta1 by growth stimulation, a truncating mutation at Gln-311 was

23 On the other hand, growth stimulations also lead to PKM2 phosphorylation, w

24 RNA and protein are increased in response to growth stimulation, although the precise regulatory mech

25 Plant growth stimulation and alterations of fungal community c

26 umber of cellular phenotypes as divergent as growth stimulation and growth inhibition.

27 to monitor heparin's potentiation of LEDGF's growth stimulation and heparin's role in the translocati

28 structural factor controlling both microbial growth stimulation and iron uptake.

29 tes to RET kinase activation, signaling, and growth stimulation and may therefore be an attractive th

30 inked to cell division, as it was induced by growth stimulation and repressed by growth inhibition.

31 Remarkably, cell growth stimulation and suppression of apoptosis by endog

32 X in order to form heme that is required for growth stimulation and survival in vivo Consequently, B.

33 C1 were hypersensitive to estrogen-dependent growth stimulation and that DLC1 had an accelerating eff

34 te the signal transduction pathways for cell growth stimulation and therapeutic resistance.

35 chemically diverse fatty acids and cAMP for growth stimulation and whilst some selective stimulatory

36 'Nipponbare') seedlings had a slow onset of growth stimulation, and barley (Hordeum vulgare) had a t

37 in embryogenesis, tissue regeneration, cell growth stimulation, and cell proliferation.

38 ndance of eEF-2 kinase have been observed on growth stimulation, and increased enzyme activity is cha

39 ation rates and mitotic index 24 hours after growth stimulation, and the ability of RPE cells to repo

40 Growth stimulation appears to be due to a reduction in t

41 tivate mitogenesis, while in the presence of growth stimulation, as is frequently seen with vasculopa

42 ular myocytes, including evidence indicating growth stimulation at concentrations in the same range a

43 M proteins on axons and SCs indicated direct growth stimulation beyond SC-mediated guidance.

44 vitro or entry into the cell cycle following growth stimulation, but it markedly reduced the growth o

45 ay of several factors, including the loss of growth stimulation by 5-LO products, the induction of PP

46 not bovine serum albumin (0.15%) suppressed growth stimulation by ATRA.

47 small interfering RNA against wnt-1 blunted growth stimulation by core gene, whereas transfection of

48 Growth stimulation by IFN-alpha/beta is independent of o

49 metastatic melanoma cells did not respond to growth stimulation by IGF-1 because of a constitutive ac

50 lationship between eicosanoid metabolism and growth stimulation by IL-6 and HGF, two important biliar

51 bdate, and tungstate competitively inhibited growth stimulation by molybdate.

52 term quiescence and down-regulated following growth stimulation by serum.

53 ultured mouse PA-SMCs, TERT was expressed on growth stimulation by serum.

54 the AKAP12alpha CArG boxes are shielded from growth stimulation by the absence of a binding site for

55 Moreover, growth stimulation depended on overall GSK3 activity, co

56 esis, apoptosis, and either growth arrest or growth stimulation depending on the cellular context.

57 having distinct biological effects, such as growth stimulation, differentiation, invasiveness, and m

58 ide with distinct biological effects such as growth stimulation, differentiation, invasiveness, and m

59 hat van der Sleen's conclusions of a lack of growth stimulation do not hold.

60 1 impairs ciliary dynamics and abolishes the growth stimulation effects of sonic hedgehog (SHH) treat

61 iferation, and LIF is able to counteract the growth stimulation elicited by EGF.

62 The CO(2) growth stimulation exceeds that of most other woody spec

63 However, growth stimulation following sunlight exposure of DOM ca

64 re dependent on exogenous, receptor-mediated growth stimulation for cell cycle entry and progression,

65 mes are emerging as significant mediators of growth stimulation for epithelial cells.

66 tion is involved with growth factor-mediated growth stimulation for lung cancer cell lines.

67 logical effects on cultured cells, including growth stimulation, growth inhibition, and induction of

68 an cells to this family of ligands including growth stimulation, growth inhibition, apoptosis and ind

69 t the physiological basis of light-activated growth stimulation has not yet been determined.

70 xide enrichment, although causing short-term growth stimulation in a range of European tree species,

71 tibody significantly inhibited the T47D cell growth stimulation in coculture with primary fibroblasts

72 ntration of dihydrotestosterone required for growth stimulation in CWR-R1 and LNCaP-C4-2 cells was fo

73 ormones is therefore necessary for autocrine growth stimulation in lung tumors and possibly in the ea

74 receptor as a mediator of MMP-2 release and growth stimulation in response to type I collagen.

75 Growth stimulation in rice correlated with a decrease in

76 VEGF treatment led to extensive growth stimulation in six of seven pancreatic cancer cel

77 ative response of neoblasts to amputation or growth stimulation in Smed-CHD4(RNAi) animals was dimini

78 alter enterobactin transport, but eliminated growth stimulation in the absence of a siderophore.

79 o confer a defect in norepinephrine-mediated growth stimulation in the presence of transferrin.

80 kinase pathway, thereby leading to excessive growth stimulation in this malignancy.

81 Fibroblast-dependent carcinoma cell growth stimulation in three-dimensional coculture was ab

82 After growth stimulation in vivo by 70% partial hepatectomy (P

83 rought years and were partially mitigated by growth stimulation in wet years.

84 hat HXK1 is involved in sucrose-induced leaf growth stimulation independent of GPT2.

85 , and E2F3a genes are similarly regulated by growth stimulation, involving a combination of E2F-depen

86 rogen-responsive element (ARE) indicate that growth stimulation is accompanied by androgen receptor (

87 001 which is of particular interest for bone growth stimulation is achievable by this assembly.

88 reases in intrinsic water use efficiency, no growth stimulation is observed.

89 tin 1b for M. hydrocarbonoclasticus-specific growth stimulation may be a poor strategy for oil-spill

90 ution of Ca2+ appears to be a consequence of growth stimulation, not a critical step in the early pha

91 cells, synchronized in late G1 and following growth stimulation, now progressed into S-phase identica

92 s, suggesting a potential autocrine-mediated growth stimulation of a subset of cancers by VEGF.

93 tivities similar to those of IL-2, including growth stimulation of activated T cells, induction of cy

94 D1 may be a target gene for leptin mediated growth stimulation of breast cancer cells and molecular

95 increased expression of ER target genes and growth stimulation of breast cancer cells.

96 se results demonstrate that retinoid-induced growth stimulation of Calu-1 cells is associated with en

97 the fibroblast surface, leading to paracrine growth stimulation of carcinoma cells by Sdc1 ectodomain

98 We now demonstrate that TGFbeta1 growth stimulation of colon carcinoma cells is Ras-depen

99 plasia (OSSN) may result from the continuous growth stimulation of corneal epithelial progenitor cell

100 ient and begins to be turned off 3-6 h after growth stimulation of cultured cells.

101 Thus, additional growth stimulation of diseased uPA-expressing liver indu

102 VciB, but not VciA, was required for growth stimulation of E. coli and Shigella flexneri stra

103 or growth inhibition of epithelial cells and growth stimulation of fibroblasts.

104 he predominant IGF involved in the autocrine growth stimulation of human lung and breast epithelial t

105 d SAPK/JNK signaling pathway, which leads to growth stimulation of MCF-7 breast tumor cells.

106 tivity blocks the induction of S phase after growth stimulation of normal mouse embryo fibroblasts, i

107 tent ERK1/2 activation and serum-independent growth stimulation of only uPAR-rich cells.

108 Growth stimulation of prostate cancer cells with 5alpha-

109 ponse to high glucose concentration required growth stimulation of RMCs by serum and activation of pr

110 extracellular osmolality, and they required growth stimulation of SMCs by serum.

111 Growth stimulation of some colon cancer cells as well as

112 ther, our results suggest that auxin-induced growth stimulation of tobacco leaf strips results primar

113 ion to functioning as a positive effector of growth, stimulation of the MEK/MAPK pathway can result i

114 Meanwhile, loss of scrA led to growth stimulation on fructooligosaccharides, due in lar

115 lls, but is stabilized immediately following growth stimulation or inhibition of protein synthesis.

116 rences in functional v-src expression, or by growth stimulation or suppression via paracrine mechanis

117 oduced by Fusarium moniliforme, resulting in growth stimulation or toxicity.

118 ytes were examined, there was no evidence of growth stimulation over a wide range of PADMA 28 concent

119 fically blocked Sdc1-mediated carcinoma cell growth stimulation, pointing toward MMPs as critical enz

120 In the absence of UCP2, endothelial growth stimulation provoked mitochondrial network fragme

121 Activation of HsOrc1 transcription following growth stimulation requires G1 cyclin-dependent kinase a

122 e to elevated TACE activity, complements the growth stimulation resulting from increased release of T

123 mopexin, Hepa cells become refractory to the growth stimulation seen with 0.1-0.75 microM heme-hemope

124 ee-ring data contain evidence for historical growth stimulation that was concealed due to failing reg

125 of Na/K-ATPase and converts ouabain-induced growth stimulation to growth inhibition in LLC-PK1 cells

126 fects in hematopoietic cells that range from growth stimulation to induction and prevention from apop

127 bolic dose-response growth curve, indicating growth stimulation until the chimera begins to compete w

128 carcinogenesis, including auto- or paracrine growth stimulation, upregulation of angiogenesis, and st

129 eased activation of TGF-beta1) and augmented growth stimulation (via decreased degradation of the IGF

130 enterobactin at approximately 10 microM, but growth stimulation was abolished when an omega (Omega) c

131 We observed that growth stimulation was associated with stimulation of ER

132 In culture, the growth stimulation was evident when senescent cells comp

133 e CoMFA models for receptor binding and cell growth stimulation were optimized through the use of var

134 Vibrio casei had the strongest growth stimulation when exposed to all fungi.

135 of 5-HETE to breast cancer cells resulted in growth stimulation, whereas selective biochemical inhibi

136 Extant models predict that this growth stimulation will continue to cause a net carbon u

137 Thus, current tree growth stimulation will, inevitably, result in a lagged

138 Moreover, growth stimulation with exogenous, purified apo-Ybt prov

139 Nicotiana sylvestris resulted in substantial growth stimulation, with a 3-fold increase in height in