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1 hrough MyoII assembly via regulation of Rap1-guanosine triphosphate.
2  guanosine diphosphate-bound complex to eIF2-guanosine triphosphate.
3 ubunit to exchange guanosine diphosphate for guanosine triphosphate.
4 tated by binding to guanosine diphosphate or guanosine triphosphate.
5 osine monophosphate (AMP) in the presence of guanosine triphosphate.
6 eled, biotinylated, or cross-linker-modified guanosine triphosphates.
7 uanosine triphosphate (8-oxo-dGTP) and 8-oxo-guanosine triphosphate (8-oxo-GTP) from the nucleotide p
8              The loss of PI3K binding to Ras-guanosine triphosphate abolishes this PI3K activation, w
9 gnaling by converting active Ras is bound to guanosine triphosphate, activating Ras into inactive Ras
10 ll polarity in the context of elevated Cdc42-guanosine triphosphate activity, similar to nonmalignant
11                                              Guanosine triphosphate analog competition assays and mut
12 P, and ribo-GTP as well as the thymidine and guanosine triphosphate analogs ddTTP, ddGTP, and dITP.
13  the corneas were permeabilized to introduce guanosine triphosphate analogs into the corneal epitheli
14 an electrode containing the non-hydrolysable guanosine triphosphate analogue, guanosine 5'-thio-triph
15 App, and an associated depletion of cellular guanosine triphosphate and adenosine triphosphate pools,
16 30) catalyzes the reversible condensation of guanosine triphosphate and beta-l-fucose-1-phosphate to
17  the nucleotide-sugar GDP-beta-l-fucose from guanosine triphosphate and beta-l-fucose-1-phosphate.
18 G in a conformation intermediate between the guanosine triphosphate and guanosine diphosphate forms.
19 , and a new 1.45 A structure in complex with guanosine triphosphate and RNA cap analog.
20 e VPS15 pseudokinase domain binds tightly to guanosine triphosphate and stabilizes a web of interacti
21 entry site RNA, elongation factor eEF2, GTP (guanosine triphosphate), and sordarin, hygromycin B pref
22 s reaction, but also adenosine triphosphate, guanosine triphosphate, and cytidine triphosphate were s
23                           Surprisingly, ADP, guanosine triphosphate, and the nonhydrolyzable ATP anal
24                                              Guanosine triphosphates are the prevalent oxidized nucle
25  = 0.0027], and mutations located within the guanosine triphosphate-ase region (odds ratio = 2.29, 95
26 utative Rap1 effector, colocalizes with Rap1-guanosine triphosphate at the leading edge and is requir
27  initiating NTP concentrations [adenosine or guanosine triphosphate (ATP or GTP), depending on the rr
28  (Gsalpha) of the stimulatory heterotrimeric guanosine triphosphate binding protein (G protein) Gs ac
29   The Vam2/6p complex then binds to Ypt7p, a guanosine triphosphate binding protein of the Rab family
30 ng or mutation of the SR-[Formula: see text] guanosine triphosphate binding site cause an N-glycosyla
31                        Septins are conserved guanosine triphosphate-binding cytoskeletal proteins inv
32                             A heterotrimeric guanosine triphosphate-binding protein (G protein)-coupl
33 utively activated stimulatory heterotrimeric guanosine triphosphate-binding protein alpha subunit (Ga
34   Mammalian nucleostemin (NS) is a nucleolar guanosine triphosphate-binding protein implicated in cel
35 ated protein kinases ERK-1 and ERK-2 and the guanosine triphosphate-binding protein p21ras were found
36                            Activation of the guanosine triphosphate-binding protein Rac1, which was d
37 n activated nucleotide exchange on the small guanosine triphosphate-binding protein RhoA in seconds.
38                                    NRAS is a guanosine triphosphate-binding protein whose most well-c
39                                      A novel guanosine triphosphate-binding protein, chronic renal fa
40                                 The ras-like guanosine triphosphate-binding protein, rho, has recentl
41 predicted a protein with homology to several guanosine triphosphate-binding protein-coupled seven-tra
42 nd appeared to be mediated by heterotrimeric guanosine triphosphate-binding proteins (G proteins).
43 to identify alpha-subunits of heterotrimeric guanosine triphosphate-binding proteins in lens cell pop
44               The rho-subfamily of monomeric guanosine triphosphate-binding proteins is implicated in
45            We have shown previously that the guanosine triphosphate-binding proteins Rac and Rho link
46            Mammalian septins are a family of guanosine triphosphate-binding proteins thought to play
47 f at least four large families of regulatory guanosine triphosphate-binding proteins, including the A
48  septins, a conserved family of polymerizing guanosine triphosphate-binding proteins, localized to th
49 ion have been characterized, including small guanosine triphosphate-binding proteins, soluble N-ethyl
50 rotein is a member of the Ras superfamily of guanosine triphosphate-binding proteins.
51 a,l]P-induced hyperplasia, but the levels of guanosine triphosphate-bound (active) H-ras protein and
52 wnstream of Ras and cycle between the active guanosine triphosphate-bound and inactive guanosine diph
53 ec3 that are critical for its binding to the guanosine triphosphate-bound form of Cdc42.
54 ically interacting and coassembling with the guanosine triphosphate-bound form of Dnm1.
55  that EVI5 preferentially interacts with the guanosine triphosphate-bound form of Rab11, and in a GAP
56 process that depended on the presence of the guanosine triphosphate-bound form of the small guanosine
57 ed spatially and MglA only binds MreB in the guanosine triphosphate-bound form, the motility complexe
58                                              Guanosine triphosphate-bound Ran, but not guanosine diph
59 y been defined as an interaction surface for guanosine triphosphate-bound Ras, single amino acid subs
60 l-length Trio, led to increased abundance of guanosine triphosphate-bound RhoA (RhoA.GTP) in human ce
61 the small GTPases RalA and RalB to an active guanosine triphosphate-bound state.
62 uanosine monophosphate (cGMP) synthesis from guanosine triphosphate by RetGC1 in the presence of E6S/
63 -binding family of proteins that bind to the guanosine triphosphate cap at growing microtubule plus e
64 ap structures and found that an unmethylated guanosine triphosphate cap led to the lowest bias and hi
65 otein synthesis via assembly of the 7-methyl-guanosine triphosphate cap-dependent translation complex
66  properties, including the requirement for a guanosine triphosphate cofactor and the generation of lo
67        Additionally, we infer that the bound guanosine triphosphate cofactor interacts with the termi
68 yed a significantly increased stimulation by guanosine triphosphate compared with the E23/S1369 haplo
69       Crystal structures of a CTA1:ARF6-GTP (guanosine triphosphate) complex reveal that binding of t
70 ial cells (HBMECs) induces the expression of guanosine triphosphate cyclohydrolase (GCH1), the rate-l
71 ase, and no abnormality in the gene encoding guanosine triphosphate cyclohydrolase 1.
72                                              Guanosine triphosphate cyclohydrolase I (GTPCHI) is a cr
73                    BH(4) and the activity of guanosine triphosphate cyclohydrolase I decreased in ihM
74                      ATII increased vascular guanosine triphosphate cyclohydrolase I expression and b
75 nflammatory cell-dependent increase of iNOS, guanosine triphosphate cyclohydrolase I, tetrahydrobiopt
76 rate-limiting enzyme for BH(4) production is guanosine triphosphate cyclohydrolase-1 (GTPCH-1).
77 rahydrobiopterin (BH4) biosynthetic enzymes (guanosine triphosphate cyclohydrolase-1 and dihydrofolat
78 lease by either agonist, indicating that the guanosine triphosphate-dependent actions of VIP and carb
79 e-miRNAs directly and specifically, in a Ran guanosine triphosphate-dependent manner, but interacts o
80 ith and dissociate from donor membranes in a guanosine triphosphate-dependent manner, can also active
81 RNAs) are exported to the cytoplasm in a Ran.guanosine triphosphate-dependent manner.
82 gest that CRFG may be involved in regulating guanosine triphosphate-dependent nuclear events that are
83 c proteins including a guanosine diphosphate/guanosine triphosphate exchange factor for Sar1p have be
84             Type 2 nodes with protein Blt1p, guanosine triphosphate exchange factor Gef2p, and kinesi
85 te that Rab-activating guanosine diphosphate/guanosine triphosphate exchange factors (GEFs) display t
86 ), eukaryotic initiation factor (eIF) 2, and guanosine triphosphate form a ternary complex (TC).
87              (125)I-CCK-8 binding and [(35)S]guanosine triphosphate gamma S (GTP gamma S) binding stu
88 ver under both basal and agonist stimulated (guanosine triphosphate gamma S and forskolin) conditions
89 form of the Rho-associated protein (Rac) and guanosine triphosphate (GTP) (RacGTP) was immunolabeled
90                 Ca2+ mobilization induced by guanosine triphosphate (GTP) analog guanosine 5'-0-(3 th
91 m extorquens MeaB bound to a nonhydrolyzable guanosine triphosphate (GTP) analog guanosine-5'-[(beta,
92 dels of the dynamin helical polymer bound to guanosine triphosphate (GTP) analogs define earlier stag
93 ger, cGAMP, is generated using intracellular guanosine triphosphate (GTP) and adenosine triphosphate
94 resence of 32P-alpha-adenosine triphosphate, guanosine triphosphate (GTP) and either carbachol or end
95                       The interconversion of guanosine triphosphate (GTP) and guanosine diphosphate (
96                                              Guanosine triphosphate (GTP) and its non-hydrolyzable an
97 s have been hampered by the high affinity of guanosine triphosphate (GTP) and lack of allosteric regu
98 ps26/29/35 trimer specifically binds to Rab7-guanosine triphosphate (GTP) and localizes to Rab7-conta
99 ongation factor Tu (EF-Tu), which hydrolyzes guanosine triphosphate (GTP) and releases tRNA in respon
100                   P-element transposase uses guanosine triphosphate (GTP) as a cofactor for transposi
101     Dynamin superfamily molecular motors use guanosine triphosphate (GTP) as a source of energy for m
102 ted by cell-surface receptors, which promote guanosine triphosphate (GTP) binding and dissociation of
103 nd maturing phagosomes, whereas inactivating guanosine triphosphate (GTP) binding blocks the dissocia
104 show that the causative mutation lies in the guanosine triphosphate (GTP) binding pocket of alpha-1 t
105 own as 1,3-beta-glucan synthase), requires a guanosine triphosphate (GTP) binding protein for activit
106 f CXCR4 and Rac-1 in lipid rafts facilitated guanosine triphosphate (GTP) binding/activation of Rac-1
107  guanosine triphosphatase Ran is loaded with guanosine triphosphate (GTP) by the chromatin-bound guan
108 seconds) and extensive (30% to 40% of total) guanosine triphosphate (GTP) charging of endogenous plat
109 tic chromosomes were surrounded by steep Ran guanosine triphosphate (GTP) concentration gradients, in
110                                              Guanosine triphosphate (GTP) cyclohydrolase I (GCH1) cat
111 of 6BH(4) de novo synthesis is controlled by guanosine triphosphate (GTP) cyclohydrolase I (GTPCHI) a
112           Vav, a guanosine diphosphate (GDP)-guanosine triphosphate (GTP) exchange factor for Rac tha
113 eotide-releasing factor C3G, which catalyzes guanosine triphosphate (GTP) exchange on Rap1.
114 naling), appears to function by facilitating guanosine triphosphate (GTP) exchange on the heterotrime
115 ation factor G (EF-G) hydrolyzes energy-rich guanosine triphosphate (GTP) for every amino acid incorp
116 anism is critical for spatial control of Ran-guanosine triphosphate (GTP) gradients that guide mitoti
117 Direct contacts between the RNA and CRM1/Ran-guanosine triphosphate (GTP) highlight the critical role
118                   RAS proteins are conserved guanosine triphosphate (GTP) hydrolases (GTPases) that a
119 opsin (R*) and inactivation of transducin by guanosine triphosphate (GTP) hydrolysis are the leading
120                               Finally, using guanosine triphosphate (GTP) hydrolysis assays, we demon
121 actions and by accelerating reactions of the guanosine triphosphate (GTP) hydrolysis cycle.
122 -bound, state of the oncoprotein and require guanosine triphosphate (GTP) hydrolysis for inhibition.
123  requires a functional Sar1 NH2 terminus and guanosine triphosphate (GTP) hydrolysis.
124 a defined catalytic residue for carrying out guanosine triphosphate (GTP) hydrolysis.
125 ,(7) a site that changes conformation during guanosine triphosphate (GTP) hydrolysis.(8) Consequently
126 n signaling 9 (RGS9)-catalyzed hydrolysis of guanosine triphosphate (GTP) in G protein-phosphodiester
127 embrane-associated rab3D significantly bound guanosine triphosphate (GTP) in overlay assays.
128  tyrosine kinase (RTK) ligands increase RhoA-guanosine triphosphate (GTP) in untransformed and transf
129    Addition of cytosol and the hydrolysis of guanosine triphosphate (GTP) induced caveolar fission.
130                Specifically, initiation with guanosine triphosphate (GTP) is required for efficient p
131 e ability of MLE to bind to ssRNA, ssDNA and guanosine triphosphate (GTP) less severely.
132                               Contrastingly, guanosine triphosphate (GTP) levels always greatly excee
133  from Cdc42 and elevates intracellular Cdc42-guanosine triphosphate (GTP) levels in cells with inacti
134 nd controls rat sarcoma viral oncogene (RAS)-guanosine triphosphate (GTP) levels.
135 f rat hepatocytes with TDCA and TCA promoted guanosine triphosphate (GTP) loading of G(i1alpha), G(i2
136 binding to the integrin alphaIIbbeta3 and by guanosine triphosphate (GTP) loading of Galpha13.
137 s transiently transfected with empty vector, guanosine triphosphate (GTP) locked dominant active Rab4
138 es; beginning with the N(7) methylation of a guanosine triphosphate (GTP) molecule, followed by the c
139 ctly associates with the alpha-tubulin-bound guanosine triphosphate (GTP) molecule, impairing the int
140 n between Crm1 and Gcn4 requires neither Ran-guanosine triphosphate (GTP) nor the nuclear export sequ
141 les were attributed to the hydrolysis of the guanosine triphosphate (GTP) nucleotide bound to the bet
142 hosphorylation reduces intracellular GDP and guanosine triphosphate (GTP) pools and decreases mitogen
143 rtical motor complexes that depends on a Ran-guanosine triphosphate (GTP) signal [12], which is suffi
144 enase (IMPDH) is the rate-limiting enzyme in guanosine triphosphate (GTP) synthesis and assembles int
145  for guanosine diphosphate (GDP) relative to guanosine triphosphate (GTP) that is consistent with a 3
146            Here, we find that the binding of guanosine triphosphate (GTP) to one subunit inhibits the
147                                              Guanosine triphosphate (GTP) turnover, guanosine diphosp
148 expected allosteric activation of SOS by Ras-guanosine triphosphate (GTP) was conspicuously absent in
149 ent in vitro-selected RNA aptamers that bind guanosine triphosphate (GTP) with K(d)s ranging from 8 m
150 polymerization is coupled with hydrolysis of guanosine triphosphate (GTP) within beta-tubulin.
151 s), such as adenosine triphosphate (ATP) and guanosine triphosphate (GTP), are signaling and bioenerg
152 ne diphosphate ribosylation factor 1 (Arf-1)-guanosine triphosphate (GTP), cargo sorting signals, and
153 d the availability of ATP, which regenerates guanosine triphosphate (GTP), powers ribosomes, and prom
154 heir reduced intrinsic rate of hydrolysis of guanosine triphosphate (GTP), which results in their acc
155 ctivation by adenosine triphosphate (ATP) or guanosine triphosphate (GTP), with ATP favoring the inco
156  phagocytosis, we focused on the role of Rho-guanosine triphosphate (GTP)-ases.
157 olecules to the cleft between the N-terminal guanosine triphosphate (GTP)-binding and the C-terminal
158 and contains an adenosine triphosphate (ATP)/guanosine triphosphate (GTP)-binding motif that has homo
159 ning as a nucleotide exchange factor for the guanosine triphosphate (GTP)-binding protein Arf1p, is r
160                  Here, the role of the small guanosine triphosphate (GTP)-binding protein CDC42Hs in
161 Raf-1 protein kinase is coupled to the small guanosine triphosphate (GTP)-binding protein Ras.
162 (MT) stabilization is regulated by the small guanosine triphosphate (GTP)-binding protein Rho and its
163 rm stress fibers and activation of the small guanosine triphosphate (GTP)-binding protein Rho.
164 diverse receptor subtypes includes the small guanosine triphosphate (GTP)-binding protein, p21ras.
165            We identified an HU-induced small guanosine triphosphate (GTP)-binding protein, secretion-
166                                              Guanosine triphosphate (GTP)-binding proteins are involv
167 in-treated cells showed translocation of ras guanosine triphosphate (GTP)-binding proteins from membr
168                   The Rho subfamily of small guanosine triphosphate (GTP)-binding proteins, through t
169             CIITA contains motifs similar to guanosine triphosphate (GTP)-binding proteins.
170 he authors demonstrate that TBSV co-opts the guanosine triphosphate (GTP)-bound active form of the en
171           Ras proteins cycle between active, guanosine triphosphate (GTP)-bound and inactive, guanosi
172 ng podosome formation increased the level of guanosine triphosphate (GTP)-bound ARF1.
173  diphosphate (GDP)-bound form (RanGDP) and a guanosine triphosphate (GTP)-bound form (RanGTP) and pla
174 ant proteins, we show that Ypt31/32 in their guanosine triphosphate (GTP)-bound form interact directl
175 C2[E62K] displayed characteristics of active guanosine triphosphate (GTP)-bound RAC2 including enhanc
176                  Quantification of activated guanosine triphosphate (GTP)-bound Ras protein and elect
177 n is initiated by the interaction of active, guanosine triphosphate (GTP)-bound Ras-related protein 1
178 0beta-Rab5 association maintains Rab5 in its guanosine triphosphate (GTP)-bound state and enhances th
179 leles encode proteins that accumulate in the guanosine triphosphate (GTP)-bound state.
180                         Dominantly inherited guanosine triphosphate (GTP)-cyclohydrolase deficiency,
181 rnesyl-dependent, but neither palmitoyl- nor guanosine triphosphate (GTP)-dependent, fashion.
182 es are ultimately due to FtsZ's capacity for guanosine triphosphate (GTP)-dependent, reversible polym
183  activated and deactivated for assembly by a guanosine triphosphate (GTP)-driven reaction cycle, and
184 atrunculin A primed platelets for Ca(2+)- or guanosine triphosphate (GTP)-gamma-S-induced alpha-granu
185 aused lethality and perturbed binding to Ran guanosine triphosphate (GTP)-importin-beta, accumulation
186 aT) resides within a domain that undergoes a guanosine triphosphate (GTP)-induced conformational chan
187 , of VASH1/SVBP is much greater on mimics of guanosine triphosphate (GTP)-MTs than on guanosine dipho
188 nding protein-related protein 1L (ORP1L) are guanosine triphosphate (GTP)-Rab7 effectors that instiga
189 the density of elongated tubulin dimers like guanosine triphosphate (GTP)-tubulin.
190 lation of both substrates is opposed by Ypt7-guanosine triphosphate (GTP).
191  catalyzed by elongation factor G (EF-G) and guanosine triphosphate (GTP).
192  or without adenosine triphosphate (ATP) and guanosine triphosphate (GTP).
193 ed for ATP biosynthesis via ADSS in place of guanosine triphosphate (GTP).
194 embranes and the percentage of Rab7 bound to guanosine triphosphate (GTP).
195 dition, we demonstrated that OA induces Rho1 guanosine triphosphate (GTP)ase activation in the follic
196 GFP)-tagged dynamin, a large mechanochemical guanosine triphosphate (GTP)ase implicated in the libera
197                                The mammalian guanosine triphosphate (GTP)ase-activating protein RanGA
198 on of force by phenylephrine, endothelin and guanosine triphosphate (GTP)gammaS, but did not inhibit
199 a unique complex in which hydrolyses of both guanosine triphosphates (GTP) are activated in a shared
200 hen Anillin is knocked down, active Rho (Rho-guanosine triphosphate [GTP]), F-actin, and myosin II ar
201 plicated in chromatin-stimulated nucleation, guanosine triphosphate(GTP)-bound Ran and its effector,
202 r 1 (ARF1) is proposed to be involved in the guanosine triphosphate- (GTP-) dependent, reversible ass
203 The growth and shortening of microtubules in guanosine triphosphate-(GTP-) mediated dynamic instabili
204             NF1 encodes neurofibromin, a Ras guanosine triphosphate (GTPase) activating protein that
205       This spatiotemporal regulation of Rab7 guanosine triphosphate/guanosine diphosphate cycling occ
206                          The role of dynamin guanosine triphosphate hydrolase (GTPase) activity in co
207 tal organization and activity of Rho and Rac guanosine triphosphate hydrolase (GTPase) and Yes-associ
208 omain of transducin seems to move toward the guanosine triphosphate hydrolase (GTPase) domain.
209  biologic functions partly through the small guanosine triphosphate hydrolase (GTPase) Rac1 (ras-rela
210 ired for TLR-induced activation of the small guanosine triphosphate hydrolase (GTPase) Rac1 (ras-rela
211            Mutations in the small Rho-family guanosine triphosphate hydrolase RAC2, critical for acti
212 to the plasma membrane is regulated by small guanosine triphosphate hydrolases (GTPases) and is essen
213 sing number of assembly factors, among which guanosine triphosphate hydrolases (GTPases) are the most
214 ns associated with the Ras homolog family of guanosine triphosphate hydrolases (Rho GTPases), and cel
215       DOCK8 and DOCK11 activate CDC42, a Rho-guanosine triphosphate hydrolases involved in actin cyto
216 domain of PLC-beta3 subsequently accelerates guanosine triphosphate hydrolysis by Galpha(q), causing
217 ng of guanosine-5'-O-(3-thiotriphosphate) or guanosine triphosphate hydrolysis by the G protein.
218 egulates actin (dis-)assembly, and catalytic guanosine triphosphate hydrolysis is found in tubulin (d
219 ified as AS250) that directly stimulates the guanosine triphosphate hydrolysis of RalA.
220 th of immature myeloid cells by accelerating guanosine triphosphate hydrolysis on Ras proteins.
221 utes and remained stable for hours, required guanosine triphosphate hydrolysis, and recruited four GB
222        Dynamin-related protein 1 (Drp1) is a guanosine triphosphate-hydrolyzing mechanoenzyme importa
223 ore insertion required the generation of Ran guanosine triphosphate in the nuclear and cytoplasmic co
224 MP) in vitro from adenosine triphosphate and guanosine triphosphate in the presence of DNA but not RN
225  was performed using (35)S-GTPgammaS (GTP is guanosine triphosphate) in primate brains.
226  we show that FtsY-SecY complex formation is guanosine triphosphate independent but requires a phosph
227 ion of the rate constant for dissociation of guanosine triphosphate indicated that at pH 7.5 the rele
228 ite inhibited eukaryotic initiation factor 2-guanosine triphosphate-initiator methionyl transfer RNA
229 A Rag mutant that is constitutively bound to guanosine triphosphate interacted strongly with mTORC1,
230                            Knockdown of Rap1-guanosine triphosphate-interacting adaptor molecule (RIA
231 cells have suggested a critical role of Rap1-guanosine triphosphate-interacting adaptor molecule (RIA
232 te cyclase (sGC) catalyzes the conversion of guanosine triphosphate into cyclic guanosine-3',5'-monop
233 served large GTPases (enzymes that hydrolyze guanosine triphosphate) involved in endocytosis and vesi
234 olves a complex rearrangement in which C8 of guanosine triphosphate is inserted between C2' and C3' o
235 alyzed exchange of guanosine diphosphate for guanosine triphosphate is proposed.
236 hate indicated that at pH 7.5 the release of guanosine triphosphate is rate-limiting.
237  small guanosine triphosphatases, binding to guanosine triphosphate leads to interaction with downstr
238  convenient synthetic routes to the oxidized guanosine triphosphate lesions spiroiminodihydantoin-2'-
239 evels correlating with heightened basal Rac1-guanosine triphosphate levels and increased reactivity.
240 ve Src alone was sufficient to stimulate Rit-guanosine triphosphate levels.
241 echanism of action may be related to altered guanosine triphosphate loading.
242  of hairpin proteins linked by hydrolysis of guanosine triphosphate nucleotides.
243 ng the exchange of guanosine diphosphate for guanosine triphosphate on Ras.
244 g, the enzyme exhibits strong preference for guanosine triphosphate over adenosine triphosphate as th
245      Mechanistically, we identified cellular guanosine triphosphate pool depletion as a key mediator
246 oteins normally using adenosine triphosphate/guanosine triphosphate, probably explains the disease.
247 BB, which are associated with CDC42, a small guanosine triphosphate protein linked to T-cell activati
248 m conversion of guanosine diphosphate-Rac to guanosine triphosphate-Rac following ITAM stimulation.
249 plasmic complex, an association regulated by guanosine triphosphate rac1 ([GTP]rac1) but not by [GTP]
250 s sustained by karyopherins (Kaps) and a Ran guanosine triphosphate (RanGTP) gradient that imports nu
251 d, and escort NLS-NCs through NPCs while Ran guanosine triphosphate (RanGTP) promotes their release f
252 or importinbeta1), to the cytoplasm in a Ran guanosine triphosphate (RanGTP)-mediated manner.
253 dle assembly by generating a gradient of Ran guanosine triphosphate (RanGTP).
254 n phosphoinositide-3 kinase (PI3K) and small guanosine triphosphate Ras signaling networks commonly d
255 rotein synthesis via tRNA aminoacylation and guanosine triphosphate regeneration.
256 ion of translation factor eIF-4E by 7-methyl guanosine triphosphate-Sepharose.
257 nteract with effector proteins when bound to guanosine triphosphate, stimulating downstream signaling
258                  The NiRAN selectively binds guanosine triphosphate, strengthening proposals for the
259 t retain the intrinsic capacity to hydrolyze guanosine triphosphate, suggesting that the mechanism of
260    Resting and activated sGC enzyme converts guanosine triphosphate to an important second messenger
261 -A catalyzes the intracellular conversion of guanosine triphosphate to cGMP (cyclic 3',5'-guanosine m
262 1 is capable of catalyzing the conversion of guanosine triphosphate to cGMP.
263  the conformational change of EF-Tu from the guanosine triphosphate to guanine diphosphate conformati
264 by accelerating the hydrolysis of active Ras-guanosine triphosphate to inactive Ras-guanosine diphosp
265         Amino acids stimulate the binding of guanosine triphosphate to RagA and RagB but the factors
266 ency of spontaneous rescue unless patches of guanosine triphosphate tubulin are artificially embedded
267 at microtubules elongate by addition of bent guanosine triphosphate tubulin to the tips of curving pr

 
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