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1 n-like Ca(2+)-binding proteins termed GCAPs (guanylate cyclase-activating proteins).
3 three Ca(2+)-binding proteins, recoverin and guanylate cyclase activating proteins 1 (GCAP1) and GCAP
6 sites (EF-hands) of the GUCA1A gene encoding guanylate cyclase-activating protein 1 (GCAP1) cause slo
10 ack on the cyclase together with its paralog guanylate cyclase-activating protein 1, which has the sa
12 outer segments of GC double knock-out mice, guanylate cyclase-activating proteins 1 and 2, and cycli
17 stem is composed of two parts: Ca(2+) sensor guanylate cyclase activating protein (GCAP) and ROS-GC.
18 and transgenic retinal rods with and without guanylate cyclase activating protein (GCAP) to disrupt C
19 addition, two of the proteins, recoverin and guanylate cyclase activating protein (GCAP), appear to b
20 ons were made to rods expressing mutant Y99C guanylate cyclase activating protein (GCAP)-1, to unders
22 ack loop was disrupted in mouse rods lacking guanylate cyclase activating proteins GCAP1 and GCAP2 (G
23 cyclase activator 1A (GUCA1A), the gene for guanylate cyclase activating protein (GCAP1), in a famil
26 hrough the calcium binding proteins: through guanylate cyclase activating proteins (GCAPs) in the cas
28 racteristic of light-adapted photoreceptors, guanylate cyclase-activating protein (GCAPs) activate Re
34 oteins found in retinal photoreceptor cells (guanylate cyclase activating protein, protein kinase A,
35 is coexpressed with its specific modulator, guanylate cyclase activating protein type 1 (GCAP1), in
36 1 coexisted with its other Ca(2+) modulator, guanylate cyclase activating protein type 1 (GCAP1).
37 ies with the Ca(2+) binding proteins, GCAPs (guanylate cyclase activating proteins), which stimulate