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1 ished against capsaicin-induced cough in the guinea pig.
2 small animal model for JUNV infection is the guinea pig.
3 protective efficacy against EBOV and SUDV in guinea pigs.
4 lamycin, resulting in reduced ototoxicity in guinea pigs.
5 tial for establishing bacterial infection in guinea pigs.
6 athetic ganglia (SSG) from latently infected guinea pigs.
7 he induction of NAbs and T cell responses in guinea pigs.
8 ently HSV-1-infected mice and HSV-2-infected guinea pigs.
9 ), which was normalized in metformin-treated guinea pigs.
10 ditory neurons of the inferior colliculus in guinea pigs.
11 induced sudden sensorineural hearing loss in guinea pigs.
12 rvallo termed Car(91) that was attenuated in guinea pigs.
13 n normal hearing, tinnitus, and non-tinnitus guinea pigs.
14 ous than the parental EA viruses in mice and guinea pigs.
15 e faithfully maintained upon transmission to guinea pigs.
16 2)--to subdivide IC GABAergic cells in adult guinea pigs.
17 d Ag preparations were analyzed in immunized guinea pigs.
18 displayed marked attenuation in the lungs of guinea pigs.
19 of rodent models such as mice, rabbits, and guinea pigs.
20 10 New Zealand White rabbits and 14 Hartley guinea pigs.
21 d latent infection of dorsal root ganglia in guinea pigs.
22 ariant (SUDV-GA) that is uniformly lethal to guinea pigs.
23 viously seen in MARV-infected mice or inbred guinea pigs.
24 DMT in a controlled feeding experiment with guinea pigs.
25 and attenuated phenotypes, respectively, in guinea pigs.
26 mapped to the outer domain of gp120 for some guinea pigs.
27 lication and transmission of bovine FLUDV in guinea pigs.
28 fferentiates these intercollicular nuclei in guinea pigs.
29 operties of cell culture-derived D123-HMW in guinea pigs.
30 expression may be shared between humans and guinea pigs.
31 man bladder mucosa presented similarities to guinea pigs.
32 ratios (SNRs) in anesthetized male or female guinea pigs.
33 munogenic when administered intradermally to guinea pigs.
34 t cross-react in vaccinated or in uninfected guinea pigs.
35 iated with disease, which was more severe in guinea pigs.
36 1, JR32, Lp01, and AA100 to cause disease in guinea pigs.
37 cacy than ADI-15878 alone in EBOV-challenged guinea pigs.
38 that project bilaterally to the MGs in adult guinea pigs.
40 enic, we determined if Car(91) could protect guinea pigs against Guanarito virus (GTOV), a distantly
42 Here we show TRPV4-induced activation of guinea pig airway-specific primary nodose ganglion cells
44 paring pharmacokinetics from carbon-14 dosed guinea pigs analyzed by both CRDS and accelerator mass s
45 ith enhanced virulence in selected cellular, guinea pig and C3HeB/FeJ mouse infection models, the lat
47 o urban air pollution leads to activation of guinea pig and human sensory nerves, which are responsib
48 e cleaned particles evoked depolarization of guinea pig and human vagus, and this was inhibited by a
49 ss neurochemical diversity than reported for guinea pig and other species but receive input from nerv
50 oss multiple species in the family Caviidae (guinea pigs and cavies), consistent with a potential fun
51 dy responses, we sequentially infected mice, guinea pigs and ferrets with divergent H1N1 or H3N2 subt
52 species but decreased across development in guinea pigs and members of the Mus genus, animals that n
54 rod increased motility in inflamed colons of guinea pigs and mice, whereas administration of GR113808
56 estigate viral pathogenesis (mouse, hamster, guinea-pig and ferret) are naturally resistant to MERS-C
58 However, rodent models, including the mouse, guinea pig, and hamster, also exist for Ebola, Marburg,
60 c solutions caused depolarization of murine, guinea pig, and human vagus and firing of Adelta-fibers
61 and competition binding assays at the human, guinea pig, and mouse H(2) receptors (co-)expressed in H
66 tains motility in healthy colons of mice and guinea pigs, and reduces inflammation in colons of mice
67 Together, this study indicates that pregnant guinea pigs are an appropriate animal model to study rep
70 Our findings shed light on JUNV infection in guinea pigs as a model for human disease and suggest tha
72 OVA) in mice and by house dust mite (HDM) in guinea pigs, as well as investigating the action of IRL2
73 itive to either ITDs or ILDs in anesthetized guinea pig, before, during, and following recovery from
74 l entorhinal cortex of the in vitro isolated guinea pig brain during focal interictal and ictal disch
75 e and female mice and in the isolated female guinea pig brain preparation during perfusion with 4-AP.
76 oth tangential brain slices and the isolated guinea pig brain with the potassium channel blocker 4-am
78 y untreated exhaust ward air, and another 90 guinea pigs breathed only air from the same six-bed tube
83 brane fusion activity and viral virulence in guinea pigs, but it did not significantly affect cell en
84 mmune cells was depleted from HSV-2-infected guinea pigs by injection of an anti-CD4 monoclonal antib
85 region induce QTc prolongation in immunized guinea-pigs by targeting the HERG channel independently
87 data suggest that CO induces arrhythmias in guinea pig cardiac myocytes via the ONOO(-)-mediated inh
88 GC synaptic connections in the retina of the guinea pig (Cavia porcellus) by recording inhibitory cur
89 GC synaptic connections in the retina of the guinea pig (Cavia porcellus) by recording inhibitory cur
90 mic contractile indices in detergent-skinned guinea pig (Cavia porcellus) cardiac muscle fibers in th
91 or colliculus (IC, auditory midbrain) of the guinea pig (Cavia porcellus, male and female), we show t
92 for the initial introduction of domesticated guinea pigs (Cavia porcellus) beyond South America into
93 ibed circumstantial evidence suggesting that guinea pigs (Cavia porcellus) lack CD59, at least on ery
95 an express a protein of the expected size in guinea pig cells in vitro, and (iii) the expressed prote
96 (i) enRep-M9l mRNA is broadly transcribed in guinea pig cells, (ii) the cloned enRep-M9l transcript c
99 uch animal models-the rhesus macaque CMV and guinea pig CMV-are characterized by congenital infection
100 asured the travelling wave in regions of the guinea pig cochlea that respond to low frequencies (<2 k
101 -linear processing of sound performed by the guinea pig cochlea varies substantially between the coch
102 vivo to study the low-frequency part of the guinea pig cochlea, and found that sound stimulation cau
105 utively expressing GFP (H9 Cre-LoxP) in deaf guinea pig cochleae that were pre-conditioned to reduce
107 (125)I-neurotrophin-3 was injected into guinea pig cochleae using a sealed injection technique c
111 e bioassay, in vivo electrophysiology, and a guinea pig conscious cough model to investigate a role f
112 activation within the inferior colliculus of guinea pigs, corresponding to the targeted, modelled and
115 ediate and largely irreversible, whereas, in guinea pigs, counts of immunostained synaptic puncta can
116 cycle (DISC) viral vaccine strain based on a guinea pig cytomegalovirus (GPCMV) capsid mutant was eva
118 a novel vaccine strategy against congenital guinea pig cytomegalovirus (GPCMV) infection was develop
122 spite exhibiting no clinical signs, infected guinea pigs developed seroconversion and the viral antig
123 human and in a panel of animal models, i.e. guinea pig, dog, cat, rat, rabbit, ferret, mouse, hamste
124 utput, in vivo recordings were made from the guinea pig dorsal cochlear nucleus, a cerebellar-like br
128 ifted strains and show that both chicken and guinea pig erythrocytes contain complex sialylated N-gly
129 termined the glycome profiles of chicken and guinea pig erythrocytes to gain insights into reduced ag
130 by 3 weeks postinoculation, 75% of pregnant guinea pigs experienced stillbirths or spontaneous abort
131 ve efficacy of the DeltaBCG TK was tested in guinea pigs experimentally infected with M. bovis by aer
132 controlled feeding experiment with 36 adult guinea pigs, fed exclusively with three different natura
134 cinated with the local trivalent vaccine and guinea pig FMDV antiserum, which is routinely used as tr
136 coprotein rabbit antiserum protected Hartley guinea pigs from lethal intraperitoneal infection with J
137 nt 46 complete mitogenomes of archaeological guinea pigs from sites in Peru, Bolivia, Colombia, the C
138 cross-reactive mAb that completely protected guinea pigs from the lethal challenge with heterologous
139 g (GP83), determined via a newly established guinea pig gamma interferon enzyme-linked immunosorbent
141 ches to investigate a fusion gene carried by guinea pigs (genus Cavia) that is partially derived from
142 doparvovirus) that was incorporated into the guinea pig germ line between approximately 22 and 35 mil
145 ssible reassortant genotypes demonstrated in guinea pigs (Gt2, Gt3, Gt7, Gt10 and Gt13) were also the
146 stent glucose intolerance, metformin-treated guinea pigs had a 2.8-fold reduction in lung lesion burd
148 this inadequacy, mitochondrial function from guinea pig hearts was quantified using several well-esta
153 athogenic NWAs, enhancing viral infection in guinea pigs.IMPORTANCE JUNV is one of five known NWAs th
159 more, recordings of neural activity from the guinea pig inferior colliculus have shown that individua
160 lving mammalian intracochlear anatomy, fixed guinea pig inner ears were imaged as whole temporal bone
166 tes that the natural and cultural history of guinea pigs is more complex than previously known and ha
171 We applied this to experimentally infected guinea pig lung sections and were able to distinguish bo
172 mes in the persistence of M. tuberculosis in guinea pig lungs and underscore the applicability of thi
173 In vitro, we show that infection of primary guinea pig macrophages results in greater JUNV replicati
174 igh-frequency, subthreshold resonance in the guinea pig medial superior olive (MSO) was recently link
180 of conditioned neutrophils in a neutropenic guinea pig model increased bacterial clearance of Shigel
187 T cells in control of virus shedding using a guinea pig model of genital HSV-2 infection that recapit
191 rticles called "aerosolized fomites." In the guinea pig model of influenza virus transmission, we sho
193 munogenicity and protective efficacy, in the guinea pig model of recurrent genital herpes, of subunit
194 the protective therapeutic efficacy, in the guinea pig model of recurrent genital herpes, of subunit
202 he transition to heart failure, we studied a guinea-pig model of angiotensin II infusion (400 ng kg(-
205 Strain-associated virulence was studied in guinea pig, monocyte-derived macrophage, and lysozyme re
206 d histiocytic arteritis and periarteritis in guinea pigs more than 2 months after recovery from acute
208 age-clamp recordings were made from isolated guinea pig myocytes as well as from human embryonic kidn
209 mine the basis for CO-induced arrhythmias in guinea pig myocytes in which action potentials (APs) mor
210 ssion of these TS channels in cultured adult guinea pig myocytes, combined with a quantitative ventri
213 ents done on the altricial rat and precocial guinea pig neonate demonstrated that the effect of vasop
214 ation in auditory neurons of chinchillas and guinea pigs of both sexes, and show how heterogeneous tu
216 with FP8v2 to boost the breadth elicited in guinea pigs of FP-directed responses induced by immunoge
223 itionally, a CRISPR/Cas9 strategy identified guinea pig platelet-derived growth factor receptor alpha
224 ted against EqCXCL16 or by pretreatment with guinea pig polyclonal antibody against EqCXCL16 protein
227 ere, we show that transgenic mice expressing guinea pig prion protein are fully susceptible to vCJD a
229 experimental challenge of immunocompromised guinea pigs, r129 induced significant weight loss, longe
230 the cervix of four placental mammals, mouse, guinea pig, rabbit and armadillo, and one marsupial, opo
231 KA phosphorylation sites conserved in human, guinea pig, rabbit, rat, and mouse alpha1C subunits.
232 different inducers; untreated male monkeys, guinea pigs, rabbits, and hamsters; and female dogs.
234 receiving the monovalent vaccines, sera from guinea pigs receiving the trivalent vaccine bound and ne
235 d secondary transmissions of BSE and vCJD in guinea pigs result in long incubation periods of approxi
236 from OFF delta retinal ganglion cells in the guinea pig retina and monitored synaptic currents that w
242 armacokinetic and pharmacodynamic studies in guinea pigs showed that a single 5-min application of a
243 ophils to the lung, cytokine release, and in guinea pig skin, inhibits allergen-induced vascular perm
244 the ENS has been extensively studied in the guinea pig small intestine, but less is known about colo
248 tic markers on cochlear inner hair cells, in guinea pigs surviving from 1 day to 6 months after a syn
250 sed a greater number of coughs in CS-exposed guinea pigs than in control animals; similar increased r
251 We performed a genomic screen in pregnant guinea pigs that led to the identification of 201 lister
252 t of virus infections of cell culture and of guinea pigs that the SSP plays an essential role in medi
253 implanted in the cochlea of an anesthetized guinea pig, the in vivo voltage response from the PIAT w
256 dy indicate that a novel gene has evolved in guinea pigs through fusion of host and virus genes.
257 There is a need to translate findings in guinea pig to mouse, a species increasingly used in ente
258 e ability of cells in the auditory system of guinea pigs to compare interaural level differences (ILD
259 delling with experiments in heart cells from guinea pigs to determine how cellular electrical activit
261 ally, we identify a modern reintroduction of guinea pigs to Puerto Rico, where local inhabitants use
262 demonstrate marked susceptibility of Hartley guinea pigs to uniformly lethal disease when challenged
263 as the probable source of the earliest known guinea pigs transported, as part of the exotic pet trade
270 mmune response to Mtb infection in untreated guinea pigs was associated with a marked increase in ene
271 FLUDV replicated in the respiratory tract of guinea pigs was similar to observations described previo
276 oculation would induce reproductive disease, guinea pigs were infected at mid-gestation and monitored
277 roSprayer aerosolizer, BL/6 mice and Hartley guinea pigs were infected intratracheally with C. burnet
283 n-cut lung slices (PCLS) of allergen-exposed guinea pigs were used to assess the effects of butyrate
284 ccine efficacy during pregnancy, nonpregnant guinea pigs were vaccinated with S19, 16MDeltavjbR + Qui
286 of allergen-specific blocking IgG in outbred guinea pigs which had been immunized with recombinant bi
287 We assessed hearing recovery in LPS-applied guinea pigs, which were either left untreated or were sy
288 bacteremia in mice and abortion in pregnant guinea pigs, while complementation of capsule expression
289 s exceed those from feeding experiments with guinea pigs who received plants with different phytolith
290 ly, we find that an uninfected, virus-immune guinea pig whose body is contaminated with influenza vir
294 t al. (1959) reported that treating pregnant guinea pigs with testosterone had enduring effects on th
295 ate a trivalent vaccine, and immunization of guinea pigs with V2-SET vaccines resulted in increased b
296 e also demonstrate that vaccinating mice and guinea pigs with WCV prior to LPA challenge is capable o
298 e tested the hypothesis that immunization of guinea-pigs with a peptide corresponding to the E-pore r
300 ntratracheal (IT) inoculation of nonpregnant guinea pigs would replicate features of clinical disease