ライフサイエンスコーパス: gut-associated

1 ir their ability to effectively colonize the gut-associated and internal lymphoid tissues.

2 ic organs including spleen, lymph nodes, and gut-associated and mucosal lymphoid tissues; third, it i

3 7-dependent immune and autoimmune responses, gut-associated as well as systemic, including inflammato

4 emonstrated, it remains unclear in models of gut-associated B cell lymphopoiesis, such as that of the

5 Application of our pipeline to 2,815 human-gut associated bacteria showed high correlation between

6 days (P = 0.003), driven by the presence of gut-associated bacteria (e.g., species of the Lachnospir

7 te that the lung microbiome is enriched with gut-associated bacteria in sepsis and ARDS, potentially

8 In this study, we characterize gut-associated bacteria in their ability to decarboxylat

9 Our results suggest that H. zea gut-associated bacteria indirectly mediate plant-insect

10 Detection of gut-associated bacteria was also associated with the pre

11 obiome (bacterial burden and enrichment with gut-associated bacteria) predict outcomes in critically

12 explore the diversification of two groups of gut-associated bacteria, Gilliamella and Snodgrassella,

13 s, lung communities were dominated by viable gut-associated bacteria.

14 rs of the Bacteroidetes, one of two dominant gut-associated bacterial phyla, process complex glycans

15 re widespread in B. fragilis and other human gut-associated Bacteroidales, strictly requires the H(+)

16 Infant gut-associated bifidobacteria has a metabolic pathway th

17 animals and was associated with depletion of gut-associated CD4(+) lymphocytes, none of the animals m

18 roduction within several compartments of the gut-associated cell population.

19 n alpha4beta7 and CCR9, the receptor for the gut-associated chemokine TECK/CCL25.

20 Gut-associated Citrobacter koseri genomes harbored 47 po

21 ay contribute to the etiology or severity of gut-associated conditions such as Crohn's Disease.

22 Gut-associated DC from MyD88(-/-) mice, which lack most

23 Moreover, gut-associated DC from TLR2(-/-) mice, or from mice in w

24 le of RA in the functional specialization of gut-associated DC in cell cultures and mice.

25 Importantly, RA regulated the ability of gut-associated DC to produce RA, induce T cells to local

26 dynamic migratory reprogramming by skin- and gut-associated DC.

27 Gut-associated dendritic cells (DC) metabolize vitamin A

28 Gut-associated dendritic cells (DC) synthesize all-trans

29 ysis of vitamin A into retinoic acid (RA) in gut-associated dendritic cells (DCs) enhances the transf

30 ma cruzi often results in chronic heart- and gut-associated disease known as Chagas disease.

31 ve historically not shared environments with gut-associated E. coli.

32 intact signaling pathways, but not T cells, gut-associated flora, or environmental pathogens.

33 w for the first time that a natural mosquito gut-associated fungus can alter Ae. aegypti physiology i

34 These gut-associated germinal centres can support targeted ant

35 nes from single cells, we find that 5-10% of gut-associated germinal centres from specific-pathogen-f

36 The frequency of highly selected gut-associated germinal centres is markedly higher in ge

37 on of B cells can take place in steady-state gut-associated germinal centres, at a rate that is tunab

38 ty of TGF-beta1 to coordinately induce other gut-associated homing pathways.

39 all, our results identify NPF and RYamide as gut-associated hormones in A. aegypti that link host att

40 ances addressing the diverse implications of gut-associated IDO1 expression are herein reviewed.

41 ce the bulk of natural serum IgM and much of gut-associated IgA, are an important component of the ea

42 levels of most circulating Ig subclasses and gut-associated IgA, which are elicited in response to ch

43 (AREG) is a dominant functional signature of gut-associated ILC2s.

44 Nfil3 is critical for normal development of gut-associated ILC3s in a cell-intrinsic manner.

45 vivo and in vitro indexes of peripheral and gut-associated immune response are tested.

46 epithelial expression of IL17 in the larval gut-associated immune response.

47 r pair involved in T cell development and in gut-associated immune responses.

48 ein (pFv)), a human sialoprotein involved in gut-associated immunity, have both recently been shown t

49 Gut-associated inflammation plays a crucial role in the

50 iate targets for therapeutic intervention in gut-associated inflammation, we tested the ability of ra

51 LPS levels, suggesting a possible repair of gut-associated junctions and decreased microbial translo

52 particular, integrating MAGs nearly doubled gut-associated K. pneumoniae phylogenetic diversity, and

53 opulation structure and genomic landscape of gut-associated lineages.

54 -related decline in lung-associated, but not gut-associated, LN immune function linked to the accumul

55 d LNs with age, but not in the corresponding gut-associated LNs.

56 s possibility, we cultured murine splenic or gut-associated lymph node cDCs with scrapie-infected who

57 T cell recognition of cross-presented Ag in gut-associated lymph nodes was tolerogenic.

58 le for the rapid depletion of CD4 T cells in gut-associated lymphatic tissue (GALT), spleen, and lymp

59 immunodeficiency virus-1 (HIV-1) infections, gut-associated lymphatic tissue (GALT), the largest comp

60 y massive depletion of CD4+ T lymphocytes in gut-associated lymphatic tissue (GALT).

61 ted: first, massive early replication in the gut-associated lymphatic tissue, including intestinal Pe

62 n, pDCs migrate from peripheral blood to the gut-associated lymphatic tissue, where they may contribu

63 is variable and incomplete, particularly in gut-associated lymphatic tissues (GALT).

64 phocytes represent a substantial fraction of gut-associated lymphocytes, but their function in mucosa

65 of immunological and microbiota profiles in Gut Associated Lymphoid Tissue (GALT) with the effects i

66 lt rabbits comparable with those produced in gut associated lymphoid tissues of young rabbits.

67 in tissue resident T cells of intestines and gut associated lymphoid tissues, which demonstrated pred

68 hocytes located in both primary lymphoid and gut-associated lymphoid compartments.

69 is prevalent in peripheral blood, liver, and gut-associated lymphoid organs and scarce in the spleen,

70 on of the formation of Peyer's patches (PP), gut-associated lymphoid organs that have a key role in t

71 eneration of virus-specific Ab production by gut-associated lymphoid tissue (GALT) after i.m. immuniz

72 ) T cells to T reg cells occurs primarily in gut-associated lymphoid tissue (GALT) after oral exposur

73 pinpoint the origins of Peyer's patches and gut-associated lymphoid tissue (GALT) and describe locat

74 ing CD4(+) T cells preferentially traffic to gut-associated lymphoid tissue (GALT) and have a key rol

75 duced by B-cell receptor engagement in human gut-associated lymphoid tissue (GALT) and involvement of

76 High levels of viral replication occur in gut-associated lymphoid tissue (GALT) and other lymphoid

77 EL) are a critical effector component of the gut-associated lymphoid tissue (GALT) and play an import

78 s, the transitional 2 (T2) B cells, homes to gut-associated lymphoid tissue (GALT) and that most T2 B

79 y of parenteral nutrition-induced changes in gut-associated lymphoid tissue (GALT) and upper respirat

80 sly or intragastrically had small intestinal gut-associated lymphoid tissue (GALT) atrophy along with

81 We, therefore, examined lymph node (LN) and gut-associated lymphoid tissue (GALT) biopsies from full

82 We hypothesized that T cells primed in the gut-associated lymphoid tissue (GALT) by a specific anti

83 d genes is important for colonization of the gut-associated lymphoid tissue (GALT) by S. typhimurium.

84 The predictive value of acute gut-associated lymphoid tissue (GALT) CD4+ T cell deplet

85 In contrast, in gut-associated lymphoid tissue (GALT) derived from indiv

86 The gut-associated lymphoid tissue (GALT) faces a considerab

87 this study, we show that p38alpha regulates gut-associated lymphoid tissue (GALT) formation in a non

88 and memory T cells from peripheral blood and gut-associated lymphoid tissue (GALT) from eight patient

89 Gut-associated lymphoid tissue (GALT) harbors the majori

90 Effects of therapy interruption on gut-associated lymphoid tissue (GALT) have not been well

91 n, and gastrointestinal tract (including the gut-associated lymphoid tissue (GALT) in rhesus monkeys

92 The role of lymphocytes in the gut-associated lymphoid tissue (GALT) in the production

93 Gut-associated lymphoid tissue (GALT) is a major site of

94 Gut-associated lymphoid tissue (GALT) is a sensor region

95 Gut-associated lymphoid tissue (GALT) is a significant b

96 Gut-associated lymphoid tissue (GALT) is an early target

97 Gut-associated lymphoid tissue (GALT) is an early target

98 Although the gut-associated lymphoid tissue (GALT) is an important ea

99 The gut-associated lymphoid tissue (GALT) is constantly expo

100 and profound depletion of CD4(+) T cells in gut-associated lymphoid tissue (GALT) of animals infecte

101 acytomas that develop "spontaneously" in the gut-associated lymphoid tissue (GALT) of interleukin-6 t

102 d B cells are a predominant component of the gut-associated lymphoid tissue (GALT) they may play a ro

103 Diet influences the ability of gut-associated lymphoid tissue (GALT) to maintain mucosa

104 otably a network of draining lymph nodes and gut-associated lymphoid tissue (GALT) to screen for infe

105 The cytokine profile in the gut-associated lymphoid tissue (GALT) was measured.

106 Somatic diversification occurs in gut-associated lymphoid tissue (GALT), and by about 1-2

107 IELs), previously stimulated to cycle in the gut-associated lymphoid tissue (GALT), proliferated in t

108 V-1-induced depletion of CD4+ T cells in the gut-associated lymphoid tissue (GALT), we first determin

109 ng infection of lymphocytes that home to the gut-associated lymphoid tissue (GALT).

110 on between commensal intestinal bacteria and gut-associated lymphoid tissue (GALT).

111 ad to rapid depletion of CD4(+) T cells from gut-associated lymphoid tissue (GALT).

112 ification occurs in the appendix, which is a gut-associated lymphoid tissue (GALT).

113 ciated viral sanctuaries, including BCFs and gut-associated lymphoid tissue (GALT).

114 ed accumulation and homing of Tconv cells in gut-associated lymphoid tissue (GALT).

115 cyte trafficking and adhesion in the gut and gut-associated lymphoid tissue (GALT).

116 ntegrin alpha4beta7 and can be identified in gut-associated lymphoid tissue (GALT).

117 lation is present at a high frequency in the gut-associated lymphoid tissue and appears to mediate a

118 somatic hypermutation occur in young rabbit gut-associated lymphoid tissue and are thought to divers

119 nic Salmonella infection is localized to the gut-associated lymphoid tissue and does not extend effic

120 ches aimed at immune reconstitution of human gut-associated lymphoid tissue and for the development,

121 ave compared the responses of T cells in the gut-associated lymphoid tissue and in the periphery to i

122 ets of CD4(+) T cells from peripheral blood, gut-associated lymphoid tissue and lymph node tissue spe

123 this nutritional intervention targeting the gut-associated lymphoid tissue and microbiota.

124 nfection with depletion of CD4(+) T cells in gut-associated lymphoid tissue and other pathologic sequ

125 ntly required on potential reservoirs in the gut-associated lymphoid tissue and the central nervous s

126 me paracellular pathway, in concert with the gut-associated lymphoid tissue and the neuroendocrine ne

127 direct impact on the host defense system of gut-associated lymphoid tissue and therefore has potenti

128 vector-modified CD4 T cells was measured in gut-associated lymphoid tissue and was correlated with e

129 in organized lymphoid nodules of the colonic gut-associated lymphoid tissue at 14 days; double-labeli

130 rphine inhibits Ag-specific IgA responses in gut-associated lymphoid tissue at least partially throug

131 lthough both decreased IgA production due to gut-associated lymphoid tissue atrophy and impaired muco

132 soluble oral Ags do not require an organized gut-associated lymphoid tissue but are most likely induc

133 mechanism via which functionally specialized gut-associated lymphoid tissue DCs can extend the repert

134 All the mutants colonized the gut-associated lymphoid tissue efficiently, but capaciti

135 hypothesized that, because teleost SALT and gut-associated lymphoid tissue have probably been subjec

136 is likely to affect local gut integrity and gut-associated lymphoid tissue homeostasis, which in tur

137 olute numbers of Tregs declined in blood and gut-associated lymphoid tissue in patients with chronic

138 We examined the cytokine microenvironment in gut-associated lymphoid tissue in response to orally adm

139 l exposure to SEB, suggesting a role for the gut-associated lymphoid tissue in the gastrointestinal m

140 prevalent, although virus genomes persist in gut-associated lymphoid tissue in the majority of indivi

141 mic autoimmune responses to the formation of gut-associated lymphoid tissue in the neonatal period of

142 that elevated interferon-gamma, produced by gut-associated lymphoid tissue in the small intestine, i

143 Gut-associated lymphoid tissue is central to the product

144 Since gut-associated lymphoid tissue is likely to play an impo

145 The migration of lymphocytes to gut-associated lymphoid tissue is mediated by integrin a

146 Gut-associated lymphoid tissue is the dominant site for

147 Gut-associated lymphoid tissue is the major reservoir of

148 ronic HIV infection results in impairment of gut-associated lymphoid tissue leading to systemic immun

149 iated antiviral immunity in association with gut-associated lymphoid tissue mass atrophy.

150 which can influence immune responses in the gut-associated lymphoid tissue of a neonate.

151 e, spontaneous germinal centers developed in gut-associated lymphoid tissue of LMP2A mice, indicating

152 between 4 and 9 d, but were not detected in gut-associated lymphoid tissue or lymph nodes.

153 ral replication, similar to lymphoid tissue, gut-associated lymphoid tissue or semen.

154 ontributing to the HIV-induced impairment of gut-associated lymphoid tissue structure and function, e

155 esenteric lymph nodes, splenic follicles and gut-associated lymphoid tissue that demonstrate high lev

156 tent progenitor (MPP) cell population in the gut-associated lymphoid tissue that promotes T(H)2 cytok

157 homeostasis is maintained by the response of gut-associated lymphoid tissue to bacteria transported a

158 Therefore, modulating gut-associated lymphoid tissue to boost Tr1 cells may be

159 eost SALT structurally resembles that of the gut-associated lymphoid tissue, and it possesses a diver

160 The best studied is gut-associated lymphoid tissue, but distinct epithelium-

161 opies in bulk and resting CD4 T cells and in gut-associated lymphoid tissue, CD4 T-cell-associated HI

162 IgA is shaped by local cues provided by the gut-associated lymphoid tissue, driven by the constantly

163 T560, a mouse B lymphoma that originated in gut-associated lymphoid tissue, expresses receptors that

164 its role on CD4 T cells, specifically in the gut-associated lymphoid tissue, is less well understood.

165 nhanced restoration of CD4(+) T cells within gut-associated lymphoid tissue, respectively.

166 ages in systemic lymphoid tissues, including gut-associated lymphoid tissue, the major site of HIV-1

167 eption can be associated with enlargement of gut-associated lymphoid tissue, we studied the capacity

168 nd on the gastrointestinal immune system and gut-associated lymphoid tissue, which may be active site

169 vation or HIV-specific responses in PBMCs or gut-associated lymphoid tissue.

170 correlated with active viral replication in gut-associated lymphoid tissue.

171 -1) results in the dissemination of virus to gut-associated lymphoid tissue.

172 of Peyer's patches, a major component of the gut-associated lymphoid tissue.

173 a typhimurium to deliver DNA directly to the gut-associated lymphoid tissue.

174 regions of colonization and necrosis of the gut-associated lymphoid tissue.

175 c immune responses, in a location other than gut-associated lymphoid tissue.

176 fever in mice have been shown to target the gut-associated lymphoid tissue.

177 f cytokine production in discrete regions of gut-associated lymphoid tissue.

178 us distribution of components of the diffuse gut-associated lymphoid tissue.

179 and that this diversification occurs in the gut-associated lymphoid tissue.

180 us distribution of components of the diffuse gut-associated lymphoid tissue.

181 oid tissues from normal mice, chiefly in the gut-associated lymphoid tissue.

182 elium of high endothelial venules in gut and gut-associated lymphoid tissue.

183 lo branch and are selectively recruited into gut-associated lymphoid tissue.

184 dendritic cells, B cells and T cells in the gut-associated lymphoid tissue.

185 he numbers of CD4(+) and IL-17(+) T cells in gut-associated lymphoid tissue.

186 ells (LSEC) supported migration into gut and gut-associated lymphoid tissue.

187 early immune responses in the periphery and gut-associated lymphoid tissue.

188 grins are involved in the homing of cells to gut-associated lymphoid tissues (GALT) and inflamed tiss

189 osal health and general wellbeing, maintains gut-associated lymphoid tissues (GALT) in a chronically

190 e accumulation of some prion diseases in the gut-associated lymphoid tissues (GALT) is important for

191 upon follicular dendritic cells (FDC) within gut-associated lymphoid tissues (GALT) is important for

192 Gut-associated lymphoid tissues (GALT) represent major s

193 help from gluten-specific CD4(+) T cells in gut-associated lymphoid tissues (GALT) via the formation

194 ction on the number of lymphoid cells in the gut-associated lymphoid tissues (GALT) were determined.

195 ecies generate their preimmune repertoire in gut-associated lymphoid tissues (GALT), compensating a r

196 Here we show that the absence of gut-associated lymphoid tissues (GALT), such as Peyer's

197 chain (gammac) (Rag-gammac(-/-)), which lack gut-associated lymphoid tissues (GALT), such as Peyer's

198 nal bacteria are required for development of gut-associated lymphoid tissues (GALT), which mediate a

199 s in part because of residual replication in gut-associated lymphoid tissues (GALT).

200 g cells, which are generated from B cells in gut-associated lymphoid tissues (GALT).

201 mune responses were assessed in the neonatal gut-associated lymphoid tissues (GALT).

202 an enhanced homing capacity to draining and gut-associated lymphoid tissues (GALTs) after systemic a

203 re associated with immune cell activation in gut-associated lymphoid tissues (GALTs) and significant

204 Gut-associated lymphoid tissues (GALTs) interact with in

205 ere we describe a method for isolating human gut-associated lymphoid tissues (GALTs) that allows unpr

206 nmental localization in intestine and in the gut-associated lymphoid tissues (GALTs).

207 tor from activated cells derived from bovine gut-associated lymphoid tissues (Peyer's patch and mesen

208 nodes (MLN) are structural components of the gut-associated lymphoid tissues and contribute to the in

209 monstrate a B cell subset that is induced in gut-associated lymphoid tissues and is characterized by

210 ss in mouse colon epithelium led to enlarged gut-associated lymphoid tissues and recruitment of immun

211 dCAM-1 RNA is restricted to mucosal tissues, gut-associated lymphoid tissues and spleen.

212 ) infections are acquired mucosally, and the gut-associated lymphoid tissues are important sites for

213 indicate that T helper cells were induced in gut-associated lymphoid tissues by i.g. immunization wit

214 (CD4(+) CD69(+)) T lymphocytes was found in gut-associated lymphoid tissues collected from animals w

215 bone marrow, blood, spleen, lymph nodes, and gut-associated lymphoid tissues depleted by day 7, inclu

216 In mammals that use gut-associated lymphoid tissues for expansion and somati

217 T cell priming by dendritic cells (DC) from gut-associated lymphoid tissues gives rise to effector c

218 cent studies have shown that human and mouse gut-associated lymphoid tissues harbor a unique NK cell

219 Lymphocyte trafficking into gut-associated lymphoid tissues is mediated by interacti

220 Under physiological conditions the gut-associated lymphoid tissues not only prevent the ind

221 lymph nodes, how immune cells in the gut and gut-associated lymphoid tissues respond to IL-2C is not

222 rating phagocytes, including macrophages, in gut-associated lymphoid tissues that are not observed in

223 es and is responsible for T-cell homing into gut-associated lymphoid tissues through its binding to m

224 int) mice, dendritic cell recruitment in the gut-associated lymphoid tissues was normalized, and tumo

225 3 showed that most of the donor cells in the gut-associated lymphoid tissues were rapidly replaced, b

226 e, a carrier molecule to deliver antigens to gut-associated lymphoid tissues, and possibly an adjuvan

227 In the local microenvironment of gut-associated lymphoid tissues, inflammatory cytokines

228 delivery of plant-synthesized insulin to the gut-associated lymphoid tissues, insulin was linked to t

229 on was monitored by enumerating listeriae in gut-associated lymphoid tissues, livers, and spleens.

230 velopment of antigen-specific T cells in the gut-associated lymphoid tissues, mice were immunized i.g

231 ut) inflamed sites such as the lung into the gut-associated lymphoid tissues, Peyer's patches, and th

232 Orally ingested proteins can stimulate gut-associated lymphoid tissues, potentially inducing to

233 CTL were also detected in rectal washes and gut-associated lymphoid tissues, respectively.

234 ymphocytes to peripheral lymph nodes and the gut-associated lymphoid tissues, respectively.

235 prominent role in iT(reg) cell generation in gut-associated lymphoid tissues.

236 increase in the frequency of T(H)17 cells in gut-associated lymphoid tissues.

237 be induced in the absence of the spleen and gut-associated lymphoid tissues.

238 tinal epithelium and delivered to underlying gut-associated lymphoid tissues.

239 eted delivery of recombinant antigens to the gut-associated lymphoid tissues.

240 otavirus (RV)-specific Ab-secreting cells in gut-associated lymphoid tissues.

241 levels of target engagement and exposure in gut-associated lymphoid tissues.

242 e aberrant immune responses are initiated in gut-associated lymphoid tissues.

243 l tetramers to track SFB-specific B cells in gut-associated lymphoid tissues.

244 e of IgA production by B cells, derived from gut-associated lymphoid tissues.

245 o the RA-dependent homing receptor switch in gut-associated lymphoid tissues.

246 ally populates epithelia and lung as well as gut-associated lymphoid tissues.

247 nd MHC class II molecule presentation in the gut-associated lymphoid tissues.

248 l immunity through antigen-processing by the gut-associated lymphoid tissues.

249 epithelial cells (IECs) and the interior of gut-associated lymphoid tissues.

250 etention to promote B cell egress from these gut-associated lymphoid tissues.

251 he physiologic conduit for antigens to reach gut associated-lymphoid tissues, for penetration of the

252 a(4)beta(7) interactions, as is the case for gut-associated lymphoreticular tissue.

253 system consists of immune cells in organized gut-associated lymphoreticular tissues (GALT) and diffus

254 associated immune alterations occur first in gut-associated lymphoreticular tissues, and thus nasal d

255 A reduction in gut-associated lymphoreticular tissues, intestinal antig

256 160-bp genome and compared it to other human gut-associated M. smithii strains and other Archaea.

257 ne tumor necrosis factor alpha (TNFalpha) in gut-associated macrophages and the liver.

258 Recent findings point to gut-associated mechanisms in the control of T1D pathogen

259 Using endogenous TGF-beta, DCs from gut-associated mesenteric lymph nodes were capable of di

260 Insect gut-associated microbes modulating plant defenses have b

261 robic food chain that characterizes resident gut-associated microbial communities along with the winn

262 A disruption of gut-associated microbial communities by antibiotic treat

263 The composition of gut-associated microbial communities changes during inte

264 long-standing evolutionary association with gut-associated microbial communities has given rise to a

265 lso performed rapid real-time runs to assess gut-associated microbial communities in critically ill a

266 estinal epithelium separates host tissue and gut-associated microbial communities.

267 ve as a model for understanding more complex gut-associated microbial communities.

268 Perturbation of the gut-associated microbial community may underlie many hum

269 Age-dependent changes of the gut-associated microbiome have been linked to increased

270 nmental radiation exposures and small mammal gut-associated microbiomes (fungal and bacterial) in the

271 axon abundance and sub-species diversisty in gut-associated microbiomes are new feature space to util

272 transmission patterns of oral-associated and gut-associated microbiota need not be the same.

273 Gut-associated microbiota of ants include Rhizobiales ba

274 in gut-draining lymph nodes (LNs) to induce gut-associated mucosal immunity.

275 found that taxon-abundance distributions of gut-associated multi-person microbiomes exhibited genera

276 cterium mandapamensis symbiosis is a binary, gut-associated mutualism that serves as a powerful model

277 The collapse of the gut-associated MZB maturational axis in severe SLE affir

278 gut-resident pathogenic bacteria, control of gut-associated opportunistic infections, and survival of

279 l environment in the colonization success of gut-associated opportunistic pathogens with implications

280 ic organism, and Escherichia coli, the model gut-associated organism and an opportunistic pathogen, i

281 ntegrin facilitate entry of T cells into the gut-associated organized lymphoid tissues such as the me

282 SIgA from the intestinal lumen to underlying gut-associated organized lymphoid tissues.

283 or lamina propria but not for generating the gut-associated organized lymphoid tissues.

284 only the Enterobacteriaceae family of human gut-associated Proteobacteria maintain a GUS operon unde

285 ke family of proteins toward BSH activity in gut-associated species.

286 madeltaT17 cell expansion and downregulating gut-associated T-bet.

287 alling, leading to reversible remodelling of gut-associated terminal tracheal cells and intestinal st

288 We observe an Ahr-independent increase in gut-associated Th17s in stressed mice, indicating that t

289 To identify the gut-associated tick aspartic hemoglobinase, this work fo

290 e of the major IL-27 cellular sources in the gut-associated tissue.

291 SIV-specific immune responses in either the gut-associated tissues or PBMC, were induced in six of t

292 vaccine (OPV or Sabin vaccine) replicates in gut-associated tissues, eliciting mucosa and systemic im

293 cific immunity that remains localized to the gut-associated tissues.

294 irus localized predominantly to lymphoid and gut-associated tissues.

295 ich is also reflected as a local decrease of gut-associated Treg.

296 evils, has greatly expanded our knowledge of gut-associated viruses in devils and provides important