ライフサイエンスコーパス: gut-associated lymphoid tissue

1 early immune responses in the periphery and gut-associated lymphoid tissue.

2 a typhimurium to deliver DNA directly to the gut-associated lymphoid tissue.

3 regions of colonization and necrosis of the gut-associated lymphoid tissue.

4 c immune responses, in a location other than gut-associated lymphoid tissue.

5 fever in mice have been shown to target the gut-associated lymphoid tissue.

6 f cytokine production in discrete regions of gut-associated lymphoid tissue.

7 us distribution of components of the diffuse gut-associated lymphoid tissue.

8 and that this diversification occurs in the gut-associated lymphoid tissue.

9 us distribution of components of the diffuse gut-associated lymphoid tissue.

10 oid tissues from normal mice, chiefly in the gut-associated lymphoid tissue.

11 elium of high endothelial venules in gut and gut-associated lymphoid tissue.

12 lo branch and are selectively recruited into gut-associated lymphoid tissue.

13 he numbers of CD4(+) and IL-17(+) T cells in gut-associated lymphoid tissue.

14 ells (LSEC) supported migration into gut and gut-associated lymphoid tissue.

15 dendritic cells, B cells and T cells in the gut-associated lymphoid tissue.

16 vation or HIV-specific responses in PBMCs or gut-associated lymphoid tissue.

17 correlated with active viral replication in gut-associated lymphoid tissue.

18 -1) results in the dissemination of virus to gut-associated lymphoid tissue.

19 of Peyer's patches, a major component of the gut-associated lymphoid tissue.

20 e of IgA production by B cells, derived from gut-associated lymphoid tissues.

21 tinal epithelium and delivered to underlying gut-associated lymphoid tissues.

22 eted delivery of recombinant antigens to the gut-associated lymphoid tissues.

23 o the RA-dependent homing receptor switch in gut-associated lymphoid tissues.

24 otavirus (RV)-specific Ab-secreting cells in gut-associated lymphoid tissues.

25 ally populates epithelia and lung as well as gut-associated lymphoid tissues.

26 levels of target engagement and exposure in gut-associated lymphoid tissues.

27 e aberrant immune responses are initiated in gut-associated lymphoid tissues.

28 l tetramers to track SFB-specific B cells in gut-associated lymphoid tissues.

29 nd MHC class II molecule presentation in the gut-associated lymphoid tissues.

30 l immunity through antigen-processing by the gut-associated lymphoid tissues.

31 epithelial cells (IECs) and the interior of gut-associated lymphoid tissues.

32 etention to promote B cell egress from these gut-associated lymphoid tissues.

33 prominent role in iT(reg) cell generation in gut-associated lymphoid tissues.

34 increase in the frequency of T(H)17 cells in gut-associated lymphoid tissues.

35 be induced in the absence of the spleen and gut-associated lymphoid tissues.

36 lation is present at a high frequency in the gut-associated lymphoid tissue and appears to mediate a

37 somatic hypermutation occur in young rabbit gut-associated lymphoid tissue and are thought to divers

38 nic Salmonella infection is localized to the gut-associated lymphoid tissue and does not extend effic

39 ches aimed at immune reconstitution of human gut-associated lymphoid tissue and for the development,

40 ave compared the responses of T cells in the gut-associated lymphoid tissue and in the periphery to i

41 ets of CD4(+) T cells from peripheral blood, gut-associated lymphoid tissue and lymph node tissue spe

42 this nutritional intervention targeting the gut-associated lymphoid tissue and microbiota.

43 nfection with depletion of CD4(+) T cells in gut-associated lymphoid tissue and other pathologic sequ

44 ntly required on potential reservoirs in the gut-associated lymphoid tissue and the central nervous s

45 me paracellular pathway, in concert with the gut-associated lymphoid tissue and the neuroendocrine ne

46 direct impact on the host defense system of gut-associated lymphoid tissue and therefore has potenti

47 vector-modified CD4 T cells was measured in gut-associated lymphoid tissue and was correlated with e

48 nodes (MLN) are structural components of the gut-associated lymphoid tissues and contribute to the in

49 monstrate a B cell subset that is induced in gut-associated lymphoid tissues and is characterized by

50 ss in mouse colon epithelium led to enlarged gut-associated lymphoid tissues and recruitment of immun

51 dCAM-1 RNA is restricted to mucosal tissues, gut-associated lymphoid tissues and spleen.

52 eost SALT structurally resembles that of the gut-associated lymphoid tissue, and it possesses a diver

53 e, a carrier molecule to deliver antigens to gut-associated lymphoid tissues, and possibly an adjuvan

54 ) infections are acquired mucosally, and the gut-associated lymphoid tissues are important sites for

55 in organized lymphoid nodules of the colonic gut-associated lymphoid tissue at 14 days; double-labeli

56 rphine inhibits Ag-specific IgA responses in gut-associated lymphoid tissue at least partially throug

57 lthough both decreased IgA production due to gut-associated lymphoid tissue atrophy and impaired muco

58 soluble oral Ags do not require an organized gut-associated lymphoid tissue but are most likely induc

59 The best studied is gut-associated lymphoid tissue, but distinct epithelium-

60 indicate that T helper cells were induced in gut-associated lymphoid tissues by i.g. immunization wit

61 opies in bulk and resting CD4 T cells and in gut-associated lymphoid tissue, CD4 T-cell-associated HI

62 (CD4(+) CD69(+)) T lymphocytes was found in gut-associated lymphoid tissues collected from animals w

63 mechanism via which functionally specialized gut-associated lymphoid tissue DCs can extend the repert

64 bone marrow, blood, spleen, lymph nodes, and gut-associated lymphoid tissues depleted by day 7, inclu

65 IgA is shaped by local cues provided by the gut-associated lymphoid tissue, driven by the constantly

66 All the mutants colonized the gut-associated lymphoid tissue efficiently, but capaciti

67 T560, a mouse B lymphoma that originated in gut-associated lymphoid tissue, expresses receptors that

68 In mammals that use gut-associated lymphoid tissues for expansion and somati

69 he physiologic conduit for antigens to reach gut associated-lymphoid tissues, for penetration of the

70 of immunological and microbiota profiles in Gut Associated Lymphoid Tissue (GALT) with the effects i

71 eneration of virus-specific Ab production by gut-associated lymphoid tissue (GALT) after i.m. immuniz

72 ) T cells to T reg cells occurs primarily in gut-associated lymphoid tissue (GALT) after oral exposur

73 pinpoint the origins of Peyer's patches and gut-associated lymphoid tissue (GALT) and describe locat

74 ing CD4(+) T cells preferentially traffic to gut-associated lymphoid tissue (GALT) and have a key rol

75 duced by B-cell receptor engagement in human gut-associated lymphoid tissue (GALT) and involvement of

76 High levels of viral replication occur in gut-associated lymphoid tissue (GALT) and other lymphoid

77 EL) are a critical effector component of the gut-associated lymphoid tissue (GALT) and play an import

78 s, the transitional 2 (T2) B cells, homes to gut-associated lymphoid tissue (GALT) and that most T2 B

79 y of parenteral nutrition-induced changes in gut-associated lymphoid tissue (GALT) and upper respirat

80 sly or intragastrically had small intestinal gut-associated lymphoid tissue (GALT) atrophy along with

81 We, therefore, examined lymph node (LN) and gut-associated lymphoid tissue (GALT) biopsies from full

82 We hypothesized that T cells primed in the gut-associated lymphoid tissue (GALT) by a specific anti

83 d genes is important for colonization of the gut-associated lymphoid tissue (GALT) by S. typhimurium.

84 The predictive value of acute gut-associated lymphoid tissue (GALT) CD4+ T cell deplet

85 In contrast, in gut-associated lymphoid tissue (GALT) derived from indiv

86 The gut-associated lymphoid tissue (GALT) faces a considerab

87 this study, we show that p38alpha regulates gut-associated lymphoid tissue (GALT) formation in a non

88 and memory T cells from peripheral blood and gut-associated lymphoid tissue (GALT) from eight patient

89 Gut-associated lymphoid tissue (GALT) harbors the majori

90 Effects of therapy interruption on gut-associated lymphoid tissue (GALT) have not been well

91 n, and gastrointestinal tract (including the gut-associated lymphoid tissue (GALT) in rhesus monkeys

92 The role of lymphocytes in the gut-associated lymphoid tissue (GALT) in the production

93 Gut-associated lymphoid tissue (GALT) is a major site of

94 Gut-associated lymphoid tissue (GALT) is a sensor region

95 Gut-associated lymphoid tissue (GALT) is a significant b

96 Gut-associated lymphoid tissue (GALT) is an early target

97 Gut-associated lymphoid tissue (GALT) is an early target

98 Although the gut-associated lymphoid tissue (GALT) is an important ea

99 The gut-associated lymphoid tissue (GALT) is constantly expo

100 and profound depletion of CD4(+) T cells in gut-associated lymphoid tissue (GALT) of animals infecte

101 acytomas that develop "spontaneously" in the gut-associated lymphoid tissue (GALT) of interleukin-6 t

102 d B cells are a predominant component of the gut-associated lymphoid tissue (GALT) they may play a ro

103 Diet influences the ability of gut-associated lymphoid tissue (GALT) to maintain mucosa

104 otably a network of draining lymph nodes and gut-associated lymphoid tissue (GALT) to screen for infe

105 The cytokine profile in the gut-associated lymphoid tissue (GALT) was measured.

106 Somatic diversification occurs in gut-associated lymphoid tissue (GALT), and by about 1-2

107 IELs), previously stimulated to cycle in the gut-associated lymphoid tissue (GALT), proliferated in t

108 V-1-induced depletion of CD4+ T cells in the gut-associated lymphoid tissue (GALT), we first determin

109 ification occurs in the appendix, which is a gut-associated lymphoid tissue (GALT).

110 ciated viral sanctuaries, including BCFs and gut-associated lymphoid tissue (GALT).

111 ed accumulation and homing of Tconv cells in gut-associated lymphoid tissue (GALT).

112 cyte trafficking and adhesion in the gut and gut-associated lymphoid tissue (GALT).

113 ntegrin alpha4beta7 and can be identified in gut-associated lymphoid tissue (GALT).

114 ng infection of lymphocytes that home to the gut-associated lymphoid tissue (GALT).

115 on between commensal intestinal bacteria and gut-associated lymphoid tissue (GALT).

116 ad to rapid depletion of CD4(+) T cells from gut-associated lymphoid tissue (GALT).

117 grins are involved in the homing of cells to gut-associated lymphoid tissues (GALT) and inflamed tiss

118 osal health and general wellbeing, maintains gut-associated lymphoid tissues (GALT) in a chronically

119 e accumulation of some prion diseases in the gut-associated lymphoid tissues (GALT) is important for

120 upon follicular dendritic cells (FDC) within gut-associated lymphoid tissues (GALT) is important for

121 Gut-associated lymphoid tissues (GALT) represent major s

122 help from gluten-specific CD4(+) T cells in gut-associated lymphoid tissues (GALT) via the formation

123 ction on the number of lymphoid cells in the gut-associated lymphoid tissues (GALT) were determined.

124 ecies generate their preimmune repertoire in gut-associated lymphoid tissues (GALT), compensating a r

125 Here we show that the absence of gut-associated lymphoid tissues (GALT), such as Peyer's

126 chain (gammac) (Rag-gammac(-/-)), which lack gut-associated lymphoid tissues (GALT), such as Peyer's

127 nal bacteria are required for development of gut-associated lymphoid tissues (GALT), which mediate a

128 mune responses were assessed in the neonatal gut-associated lymphoid tissues (GALT).

129 s in part because of residual replication in gut-associated lymphoid tissues (GALT).

130 g cells, which are generated from B cells in gut-associated lymphoid tissues (GALT).

131 an enhanced homing capacity to draining and gut-associated lymphoid tissues (GALTs) after systemic a

132 re associated with immune cell activation in gut-associated lymphoid tissues (GALTs) and significant

133 Gut-associated lymphoid tissues (GALTs) interact with in

134 ere we describe a method for isolating human gut-associated lymphoid tissues (GALTs) that allows unpr

135 nmental localization in intestine and in the gut-associated lymphoid tissues (GALTs).

136 T cell priming by dendritic cells (DC) from gut-associated lymphoid tissues gives rise to effector c

137 cent studies have shown that human and mouse gut-associated lymphoid tissues harbor a unique NK cell

138 hypothesized that, because teleost SALT and gut-associated lymphoid tissue have probably been subjec

139 is likely to affect local gut integrity and gut-associated lymphoid tissue homeostasis, which in tur

140 olute numbers of Tregs declined in blood and gut-associated lymphoid tissue in patients with chronic

141 We examined the cytokine microenvironment in gut-associated lymphoid tissue in response to orally adm

142 l exposure to SEB, suggesting a role for the gut-associated lymphoid tissue in the gastrointestinal m

143 prevalent, although virus genomes persist in gut-associated lymphoid tissue in the majority of indivi

144 mic autoimmune responses to the formation of gut-associated lymphoid tissue in the neonatal period of

145 that elevated interferon-gamma, produced by gut-associated lymphoid tissue in the small intestine, i

146 In the local microenvironment of gut-associated lymphoid tissues, inflammatory cytokines

147 delivery of plant-synthesized insulin to the gut-associated lymphoid tissues, insulin was linked to t

148 Gut-associated lymphoid tissue is central to the product

149 Since gut-associated lymphoid tissue is likely to play an impo

150 The migration of lymphocytes to gut-associated lymphoid tissue is mediated by integrin a

151 Gut-associated lymphoid tissue is the dominant site for

152 Gut-associated lymphoid tissue is the major reservoir of

153 Lymphocyte trafficking into gut-associated lymphoid tissues is mediated by interacti

154 its role on CD4 T cells, specifically in the gut-associated lymphoid tissue, is less well understood.

155 ronic HIV infection results in impairment of gut-associated lymphoid tissue leading to systemic immun

156 on was monitored by enumerating listeriae in gut-associated lymphoid tissues, livers, and spleens.

157 iated antiviral immunity in association with gut-associated lymphoid tissue mass atrophy.

158 velopment of antigen-specific T cells in the gut-associated lymphoid tissues, mice were immunized i.g

159 Under physiological conditions the gut-associated lymphoid tissues not only prevent the ind

160 lt rabbits comparable with those produced in gut associated lymphoid tissues of young rabbits.

161 which can influence immune responses in the gut-associated lymphoid tissue of a neonate.

162 e, spontaneous germinal centers developed in gut-associated lymphoid tissue of LMP2A mice, indicating

163 between 4 and 9 d, but were not detected in gut-associated lymphoid tissue or lymph nodes.

164 ral replication, similar to lymphoid tissue, gut-associated lymphoid tissue or semen.

165 tor from activated cells derived from bovine gut-associated lymphoid tissues (Peyer's patch and mesen

166 ut) inflamed sites such as the lung into the gut-associated lymphoid tissues, Peyer's patches, and th

167 Orally ingested proteins can stimulate gut-associated lymphoid tissues, potentially inducing to

168 nhanced restoration of CD4(+) T cells within gut-associated lymphoid tissue, respectively.

169 CTL were also detected in rectal washes and gut-associated lymphoid tissues, respectively.

170 ymphocytes to peripheral lymph nodes and the gut-associated lymphoid tissues, respectively.

171 lymph nodes, how immune cells in the gut and gut-associated lymphoid tissues respond to IL-2C is not

172 ontributing to the HIV-induced impairment of gut-associated lymphoid tissue structure and function, e

173 esenteric lymph nodes, splenic follicles and gut-associated lymphoid tissue that demonstrate high lev

174 tent progenitor (MPP) cell population in the gut-associated lymphoid tissue that promotes T(H)2 cytok

175 rating phagocytes, including macrophages, in gut-associated lymphoid tissues that are not observed in

176 ages in systemic lymphoid tissues, including gut-associated lymphoid tissue, the major site of HIV-1

177 es and is responsible for T-cell homing into gut-associated lymphoid tissues through its binding to m

178 homeostasis is maintained by the response of gut-associated lymphoid tissue to bacteria transported a

179 Therefore, modulating gut-associated lymphoid tissue to boost Tr1 cells may be

180 int) mice, dendritic cell recruitment in the gut-associated lymphoid tissues was normalized, and tumo

181 eption can be associated with enlargement of gut-associated lymphoid tissue, we studied the capacity

182 3 showed that most of the donor cells in the gut-associated lymphoid tissues were rapidly replaced, b

183 in tissue resident T cells of intestines and gut associated lymphoid tissues, which demonstrated pred

184 nd on the gastrointestinal immune system and gut-associated lymphoid tissue, which may be active site