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1  apoptosis in both S2 cells and H. virescens haemocytes.
2 t signalling is not autonomously required in haemocytes.
3 s proliferate and mature into differentiated haemocytes.
4  that is present in its inactive form inside haemocytes.
5 i-based immunity mediated by macrophage-like haemocytes.
6 , midgut, integument, testis, silk gland and haemocytes.
7 1 and Hr3, to repress ninjurins and disperse haemocytes.
8  Vsg is expressed in immune cells, including haemocytes and fat body cells.
9 eatment can increase cell number in cultured haemocytes and the Drosophila wing, respectively.
10 hat a reciprocal relationship exists between haemocytes and the VNC and that defects in nerve cord de
11                 Macrophage-like cells called haemocytes are key effectors of Drosophila cellular inna
12 is along the ventral nerve cord (VNC), where haemocytes are required for the correct development of t
13 , also provides the scaffold on which intact haemocytes assemble during encapsulation; a response tha
14 spersal, while stabilization of ninjurins in haemocytes blocks developmentally regulated SHC remodell
15  identify direct Notch targets in Drosophila haemocytes (blood cells), where Notch promotes crystal c
16  that antagonize ninjurin function in larval haemocytes cause premature SHC dispersal, while stabiliz
17  or localize to segmentally repeated sessile haemocyte compartments (SHCs).
18 wly cycling progenitor cells, whereas mature haemocytes comprising plasmatocytes, crystal cells and l
19 us immune responses (phenoloxidase activity, haemocyte concentration and melanization strength), alon
20                     Although PO activity and haemocyte count were weakly correlated across the whole
21 istent positive genetic correlations between haemocyte count, antibacterial and phenoloxidase activit
22 c effects on phenoloxidase (PO) activity and haemocyte count, both indicators of immune system activi
23 ween a suite of commonly used immune assays (haemocyte count, implant encapsulation, total phenoloxid
24 ity were substantially higher than those for haemocyte count, indicating that the different component
25 ivity in every case and substantially higher haemocyte counts in all treatments except unheated/low d
26           A higher temperature led to higher haemocyte counts when density was high and food quality
27 and heritability for components of immunity (haemocyte densities, proPhenoloxidase activity, resistan
28 two immune parameters related to resistance (haemocyte density and pre-immune challenge activity of p
29  genetic correlation between growth rate and haemocyte density was estimated).
30 g antibacterial histones together with other haemocyte-derived defence factors, but crucially, also p
31  cell size and cell-cycle progression in two haemocyte-derived Drosophila cell lines.
32   These include proteins that participate in haemocyte development, vesicle transport, actin cytoskel
33  mosquitoes constitutively release a soluble haemocyte differentiation factor into their haemolymph t
34  immune stimuli can be integrated to control haemocyte dispersal and immune function.
35    Our data reveal that the final pattern of haemocyte distribution, and the details and timing of it
36                                       Larval haemocytes exist either in circulation or localize to se
37                          We show that larval haemocytes express ninjurins, which are required for tar
38                                              Haemocytes from Vsg knockout Drosophila are resistant to
39                  We show that the failure of haemocyte migration along the VNC in slit mutants is not
40 at defects in nerve cord development prevent haemocyte migration along this structure.
41  VNC, further highlighting the importance of haemocyte migration for correct neural development.
42                                This block of haemocyte migration in turn disrupts the formation of th
43  and its receptor Robo are both required for haemocyte migration, but signalling is not autonomously
44 g epithelium, creating a physical barrier to haemocyte migration.
45 ontact inhibition plays in the patterning of haemocyte movements, we have mathematically analysed and
46 stimulation of innate immunity via increased haemocyte numbers compared to their constituent monother
47 Furthermore, we observed the same effects in haemocytes of H. virescens larvae, after TnBVank1 in viv
48 Vank1 in vivo transient transfection, and in haemocytes of parasitised larvae.
49 f functional in vitro assays on immunocytes (haemocytes) of the Mediterranean mussel Mytilus gallopro
50 rin cell-adhesion molecules as regulators of haemocyte recruitment to SHCs.
51 cently revealed that Drosophila macrophages (haemocytes) require contact inhibition for their uniform
52                                        Snail haemocyte RNA, extracted from parasite-challenged resist
53                                              Haemocytes take up dsRNA from infected cells and, throug
54  ninjurins, which are required for targeting haemocytes to SHCs.
55  proposed for SHCs, the mechanisms directing haemocytes to them are unclear.
56 in the environment, this interaction induces haemocyte trafficking into these tissues.
57 cells associated with host cilia and induced haemocyte trafficking.
58 bryonic development, Drosophila macrophages (haemocytes) undergo a series of stereotypical migrations