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1 ds (DPPH, ORAC and erythrocyte resistance to haemolysis).
2 linked to microvascular red blood cell (RBC) haemolysis.
3 PNH and clinically significant extravascular haemolysis.
4 other for 1-14 days) would cause significant haemolysis.
5 PNH and clinically significant extravascular haemolysis.
6 o sustained reticulocytopenia, to near-fatal haemolysis.
7 ompromised, the RBCs are more susceptible to haemolysis.
8 rds hydrogen peroxide and on surface-induced haemolysis.
9 altering red blood cell rheology and causing haemolysis.
10 nd no evidence of hepatitis, cholestasis, or haemolysis.
11 c sucrose solutions at pH 6) supported their haemolysis.
12 Cytochalasin B prevented haemolysis.
13 of individuals at risk of primaquine-induced haemolysis.
14 ver, abnormal liver function tests, and mild haemolysis.
15 EC necroptosis and complement-dependent RBC haemolysis.
16 site reactions (in 20 [26%] of 77 patients), haemolysis (15 [19%]), nasopharyngitis (12 [16%]), and d
17 flavonoid that potentiated copper-triggered haemolysis (155 +/- 81 % at twice the amount of Cu(2+)),
19 lation of eight mutants that failed to cause haemolysis, all of which had transposon insertions in ge
21 significant improvements in haemoglobin and haemolysis among patients with pyruvate kinase deficienc
22 nillin or 5-hydroxymethyl, and urea) reduced haemolysis, an effect not due to increased oxygen affini
24 ubarachnoid haemorrhage is often followed by haemolysis and concomitant oxidative stress, and is freq
25 hiphos on trout health through intravascular haemolysis and consequently from pathophysiologic proces
27 nto the mechanism of streptolysin S-mediated haemolysis and have implications for the development of
33 12 patients reported serious adverse events; haemolysis and pyrexia were the most common (each occurr
34 oderate (2438 m) altitude increased rates of haemolysis and right ventricular systolic pressures in m
36 oposed direct link between contact-dependent haemolysis and Shigella entry, and demonstrate that IpaB
39 considerable antimicrobial activity, minimal haemolysis, and low cytotoxicity, we introduced the natu
40 of 41 through to week 16 due to breakthrough haemolysis, and ten [13%] of 77 due to severe treatment-
41 hildren showed evidence of treatment-related haemolysis, and the mean maximum decrease in haemoglobin
46 due to clinically significant extravascular haemolysis can affect patients with paroxysmal nocturnal
47 may result in a ten-fold increase in sample haemolysis, compared to the recommended guideline proced
51 progressive changes in the profile of their haemolysis curves, as the curves migrated towards lower
52 showed no change in profile of the migrating haemolysis curves, suggesting that their PCl distributio
53 son, haptoglobin, NO(x) , ovotransferrin and haemolysis differed significantly between breeding and n
54 on (2 [6%]), and one (3%) each with anaemia, haemolysis, fatigue, and a neurological, metabolic, resp
55 on of patients with vivax malaria at risk of haemolysis following 8-aminoquinoline radical cure is su
56 ched the threshold of clinically significant haemolysis (fractional haematocrit reduction >25%) in G6
57 PNH and clinically significant extravascular haemolysis (haemoglobin <=9.5 g/dL; absolute reticulocyt
63 2438 m) altitude would have a higher rate of haemolysis, impaired cardiac function and reduced exerci
68 he potential to cause clinically significant haemolysis in G6PD heterozygous females who are reported
70 The 8-aminoquinolines cause dose-dependent haemolysis in glucose-6-phosphate dehydrogenase deficien
72 primaquine or tafenoquine) is complicated by haemolysis in individuals with glucose-6-phosphate dehyd
77 ial was designed to measure efficacy and not haemolysis in relation to G6PD genotype and that the het
79 to week 72, changes in the concentration of haemolysis markers (absolute and percentage reticulocyte
80 etic deletion of Mlkl from ECs decreased RBC haemolysis, microvascular obstruction and reduced ischae
81 scending dose regimen was stopped because of haemolysis (n=1) and asymptomatic increases in transamin
83 ss the 48-week trials, clinical breakthrough haemolysis occurred in seven (7%) of 96 iptacopan-treate
91 ignificantly reduced listeriolysin O-induced haemolysis (p < 0.05), and ameliorated H(2)O(2)-induced
92 that at temperatures < or = 15 degrees C the haemolysis rate was significantly inhibited with little
95 induced systemic inflammation and apoptosis, haemolysis, rhabdomyolysis, smoke inhalation injury, dru
96 Many acute and chronic anaemias, including haemolysis, sepsis and genetic bone marrow failure disea
99 the serious adverse events were significant haemolysis (three in the 7-day group and one in the 14-d
100 ale-biased macrophage concentration, BKA and haemolysis titers, but only during the breeding season.
101 sferrin concentration, haemagglutination and haemolysis titres increased 12 weeks into the dry season
102 in concentrations, and haemagglutination and haemolysis titres) over two annual cycles of wet and dry
103 in concentrations, and haemagglutination and haemolysis titres), body mass and primary moult, fortnig
104 r [11%] of 37), chest pain (two [5%] of 37), haemolysis (two [5%] of 37), and neutropenia (two [5%] o
105 designed to characterise primaquine-induced haemolysis using a holistic Bayesian analysis of all pub
111 e rate of sample laboratory rejection due to haemolysis when commonly practiced deviations from the g
113 adherence to the vascular endothelium and by haemolysis, which results in endothelial cell activation
114 rent work addresses the hypothesis that this haemolysis will provide a novel diagnostic and prognosti