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1 manent, semipermanent, temporary, or gradual hair dyes.
2 ade framework for accurate and interpretable hair dye analysis.
3                                        Using hair dye and rhodamine labeling we established that the
4 igated associations between use of synthetic hair dyes and risk of brain tumors in a hospital-based c
5 is are chemicals and agricultural exposures, hair dyes, and blood transfusions.
6 s, medical conditions, pesticides, solvents, hair dyes, and diet.
7  history of hematopoietic cancer, and use of hair dyes are associated with t(14;18)-defined subsets o
8                         Certain chemicals in hair dyes are known animal carcinogens.
9 ch on Cancer reported that some chemicals in hair dyes are probably carcinogenic to those exposed to
10 ed as never-users or ever-users of permanent hair dye, based on self-report.
11                  For women who started using hair dye before 1980 as compared with never users, a sta
12 use was observed among women who began using hair dye before 1980.
13 ainly observed among women who started using hair dyes before 1980.
14 m for identifying oxidative and nonoxidative hair dye colorants and predicting their perceptual color
15 ological mechanisms through which the use of hair dye could be associated with human metabolism and c
16 lfide showing the strongest association with hair dye (effect size (beta) = - 0.263; FDR adjusted p-v
17              Although use of permanent brown hair dye for 20 or more years was associated with glioma
18  observed association reflects the change in hair dye formula contents during the past two decades or
19                              Personal use of hair dye has been inconsistently linked to risk of non-H
20                    For women who began using hair dye in 1980 or afterward, increased FL risk was lim
21 mbranous ventricular septal defect, 5.5%; 7) hair dye in multiple/multiplex membranous ventricular se
22 tions were found for women who started using hair dyes in 1980 or afterward.
23 ommon allergens include fragrance chemicals, hair dyes, metals, rubber accelerators and preservatives
24 es (pOR=2.2, 95% CI=1.0-4.7), have ever used hair dyes (pOR=4.5, 95% CI=1.3-15.4), and have had a gre
25 drogen peroxide concentrations in commercial hair dye products, which is a significant issue in the h
26 e (PPD) is a strong contact allergen used in hair dye that is known to cause allergic contact dermati
27       Compared with never-users of permanent hair dye, the age-adjusted relative risks (RR) for the d
28 ecause of the complexity of chemicals in the hair dye, the consecutive reaction monitoring of PhIP at
29 odds ratio was for 15 or more years of using hair dyes up to six times per year (odds ratio = 2.4, 95
30  pathway genes modifies the relation between hair dye use and risk of non-Hodgkin lymphoma.
31 tly increased risk was observed for starting hair dye use before 1980 (relative to never use) among w
32          Biological mechanisms through which hair dye use may be related to human metabolism and canc
33 obiotic metabolism in the carcinogenicity of hair dye use needs to be confirmed in larger studies.
34 he association between metabolite levels and hair dye use was estimated using linear regression, adju
35                                              Hair dye use was not associated with either subtype.
36                                  Duration of hair dye use was not related to risk of SLE.
37 es have suggested an association of personal hair dye use with bladder and hematopoietic cancers.
38 ent evidence for an association of synthetic hair dye use with glioma, meningioma, or acoustic neurom
39          We found no evidence that permanent hair dye use, age at first use, frequency of use, or dur
40 d nonpermanent (semipermanent and temporary) hair dye use.
41 r a lack of detailed information on personal hair-dye use in previous studies.
42         These results indicate that personal hair-dye use may play a role in risks of FL and CLL/SLL
43 nce interval (CI): 1.1, 1.4) associated with hair-dye use was observed among women who began using ha
44 -3alpha,17beta-diol disulfate was reduced in hair dye users (beta = - 0.492; FDR adjusted p-value = 0
45 irst serum metabolomic examination comparing hair dye users and nonusers in the Alpha-Tocopherol, Bet
46 ther pathways differed significantly between hair dye users and nonusers, including metabolites previ
47      In situ detection and identification of hair dyes using surface-enhanced Raman spectroscopy (SER