ライフサイエンスコーパス: hairy

1 In contrast, long-range repressors such as Hairy act over distances >1 kb.

2 Fibrillar, or "hairy," adhesives have evolved multiple times independen

3 Antirrhinum (snapdragons), which has evolved hairy alpine-adapted species or lowland species with a r

4 ved Hairy/Enhancer of Split family repressor Hairy, analyzing histone marks and gene expression in Dr

5 es that differentiate this species from its 'hairy' ancestors.

6 o a prepattern of repression, established by Hairy and Dl, which unfolds progressively during larval

7 h signaling and induced the transcription of hairy and E (spl)-1 (HES)-1, by Notch-independent mechan

8 evels of cyclin A2, cyclin B1, cyclin E2 and hairy and enhancer of split (Hes) 1.

9 Hairy and enhancer of split 1 (Hes-1) is a mammalian tra

10 ated HLF knockdown revealed the NOTCH target Hairy and Enhancer of Split 1 (HES1) and the cyclin-depe

11 t Notch1, but not Notch2, activation induced hairy and enhancer of split 1 (HES1) expression and gene

12 by NOTCH receptors, which signal through the Hairy and Enhancer of Split 1 (HES1) transcription regul

13 EBNA2 and EBNALP, EBNALP was associated with hairy and enhancer of split 1 (hes1), cd21, cd23, and ar

14 helix-loop-helix (bHLH) transcription factor hairy and enhancer of split 1 (HES1).

15 and autoimmune diseases, activate the Notch1-hairy and enhancer of split 1 (Notch1-HES1) axis in targ

16 ession of the Notch transcriptional effector Hairy and Enhancer of Split 1 that enables this dual rol

17 n of the biliary stem-cell signaling pathway hairy and enhancer of split 1/pancreatic duodenal homeob

18 A higher hairy and enhancer of split 1:mouse atonal homolog 1 rat

19 ed by expression of the transcription factor Hairy and Enhancer of Split 3 (HES3) and sensitivity to

20 the expression of the specific target genes hairy and enhancer of split 3 (Hes3) and Sonic hedgehog

21 MEM106B, PDIA6 and the Notch signaling genes hairy and enhancer of split 4 (HES4) and JAGGED2, suppor

22 TAp63 transactivates hairy and enhancer of split 5 (Hes5) and atonal homolog

23 The initial efforts identify that hairy and enhancer of split 6 (Hes6), a novel transcript

24 ty shift assays, we show that Pax3 regulates Hairy and enhancer of split homolog-1 (Hes1) and Neuroge

25 HES1 (hairy and enhancer of split) is a transcription factor t

26 We identify transcriptional repressor - hairy and enhancer of split-1 (HES1) - as a mediator of

27 ression, as well as downstream targets HES1 (hairy and enhancer of split-1) and CCND1 (cyclin D1).

28 F-kappaB but also results in decreased Hes1 (hairy and enhancer of split-1), a negative regulator of

29 re accompanied by a reduction in Notch-1 and Hairy and enhancer of split-5 (Hes-5) mRNA and protein l

30 k from NOTCH3 receptor signaling through the Hairy and enhancer of Split-5 (HES-5) protein to smooth

31 Notch induces Hairy and Enhancer of Split-related with YRPW motif (Hey

32 The afferent inputs from hairy and glabrous skin are distinct with respect to bot

33 reshold mechanoreceptors that innervate both hairy and glabrous skin.

34 using the prototypical long-range repressor Hairy and the short-range repressor Knirps.

35 Strikingly, five TFs (nf-kb, egr, pax6, hairy, and clockwork orange) were predicted to co-regula

36 trate that the products of the genes stripe, hairy, and extramacrochaetae contribute to rescue by ant

37 le genes even-skipped, odd-skipped, runt and hairy are all expressed as early blastoderm pair-rule st

38 Hairy attachment structures (fossula spongiosa), present

39 Transcriptome analysis of hairy B. villosa leaves indicated higher expression of s

40 r, transcripts of the TRY inhibitory gene in hairy B. villosa were surprisingly high relative to B. n

41 The gain of eccrine sweat glands in hairy body skin has empowered humans to run marathons an

42 e expression differences in an exceptionally hairy Brassica species compared with a glabrous species

43 y, which disrupts the repressive activity of Hairy by inhibiting the recruitment of its cofactor Grou

44 eukaemia is reviewed, focussing first on the hairy cell itself and then on its interactions with the

45 arge fraction of melanomas, thyroid cancers, hairy cell leukaemias and, to a smaller extent, a wide s

46 ia (29), T-cell prolymphocytic leukemia (3), hairy cell leukemia (1), adult T-cell leukemia/lymphoma

47 V600E mutation was reported in all cases of hairy cell leukemia (HCL) but not in other peripheral B-

48 BRAFV600E mutation was recently detected in hairy cell leukemia (HCL) by whole exome sequencing.

49 tic leukemia (CLL) cases, 100% (32 of 32) of hairy cell leukemia (HCL) cases, 15% (5 of 34) of mantle

50 has been the standard first-line therapy of hairy cell leukemia (HCL) for 30 years.

51 Hairy cell leukemia (HCL) is a chronic B-cell leukemia n

52 Hairy cell leukemia (HCL) is a chronic mature B-cell neo

53 Hairy cell leukemia (HCL) is a distinct clinicopathologi

54 Hairy cell leukemia (HCL) is a distinct clinicopathologi

55 Hairy cell leukemia (HCL) is a rare, indolent B-cell dis

56 Hairy cell leukemia (HCL) is characterized by underexpre

57 Hairy cell leukemia (HCL) is marked by near 100% mutatio

58 Hairy cell leukemia (HCL) shows unique clinicopathologic

59 iral oncogene homolog B1 (BRAF) mutations in hairy cell leukemia (HCL) subsets, demonstrating that BR

60 To compare hairy cell leukemia (HCL) with chronic lymphocytic leuke

61 produce high response rates in patients with hairy cell leukemia (HCL), a significant number of patie

62 ), 7 of 7 follicular lymphoma (FL), 13 of 17 hairy cell leukemia (HCL), and 2 of 3 mantle cell lympho

63 target on B-cell malignancies, particularly hairy cell leukemia (HCL), but its soluble extracellular

64 e (BRAF V600E) is the key driver mutation in hairy cell leukemia (HCL), suggesting opportunities for

65 defining the BRAF-V600E driving mutation in hairy cell leukemia (HCL),provide extensive laboratory s

66 ious CDKN1B mutation in 16% of patients with hairy cell leukemia (HCL).

67 recently described as a molecular marker of hairy cell leukemia (HCL).

68 duces protracted remissions in patients with hairy cell leukemia (HCL).

69 nd also occurs in the hematopoietic neoplasm hairy cell leukemia (HCL).

70 ates when used as first-line monotherapy for hairy cell leukemia (HCL); however, patients continue to

71 low-grade follicular lymphoma (FL; n = 44), hairy cell leukemia (HCL; n = 15), and reactive lymphoid

72 cally, this paper describes the frequency of hairy cell leukemia among all adult leukemias and charac

73 ons is helpful in the diagnosis of classical hairy cell leukemia and a number of histiocytic neoplasm

74 ate of complete remissions in drug-resistant hairy cell leukemia and has a lower response rate in ped

75 RITs have shown efficacy in refractory hairy cell leukemia and in some children with acute lymp

76 ladribine is best known for the treatment of hairy cell leukemia and other lymphoid cancers, it also

77 The description of hairy cell leukemia as a specific clinical entity was pu

78 HA22 is very active in drug-resistant hairy cell leukemia but is less active in children with

79 V600E-expressing human primary melanoma and hairy cell leukemia cells.

80 The Hairy Cell Leukemia Foundation convened an international

81 ibine followed by rituximab in patients with hairy cell leukemia including the vari-ant form (HCLv).

82 Hairy cell leukemia is an uncommon hematologic malignanc

83 Enormous progress in the treatment of hairy cell leukemia over the last five decades has emerg

84 Immunophenotypic features in classic hairy cell leukemia show that the leukemic cells express

85 utcome of young patients with a diagnosis of hairy cell leukemia treated with cladribine.

86 Hairy cell leukemia variant (HCLv) presents with high di

87 notoxin treatment and report that samples of hairy cell leukemia with high levels of Bim protein resp

88 ity against chronic lymphocytic leukemia and hairy cell leukemia, in general, monoclonal antibodies h

89 al blood samples obtained from patients with hairy cell leukemia, marginal zone lymphoma, and chronic

90 lymphoma, CLL, acute lymphoblastic leukemia, hairy cell leukemia, non-Hodgkin lymphoma (including rit

91 d many complete remissions in drug-resistant Hairy cell leukemia, where many treatment cycles can be

92 ts profound impact on the natural history of hairy cell leukemia, with responses approaching 100% and

93 1 in both leukemia and lymphoma, and BRAF in hairy cell leukemia.

94 ll known and revolutionized the treatment of hairy cell leukemia.

95 signaling important for the pathogenesis of hairy cell leukemia.

96 ted kinase (ERK) in the hairy cells (HCs) of hairy cell leukemia.

97 time the clinical and pathologic features of hairy cell leukemia.

98 The biology of hairy-cell leukaemia is reviewed, focussing first on the

99 BRAF inhibitor vemurafenib in patients with hairy-cell leukemia that had relapsed after treatment wi

100 d many complete remissions in drug-resistant hairy-cell leukemia when several cycles of the agent can

101 B-Raf kinase (BRAF mutations) are present in hairy-cell leukemia, cutaneous melanoma, thyroid carcino

102 BRAF V600E is the genetic lesion underlying hairy-cell leukemia.

103 tive in patients with relapsed or refractory hairy-cell leukemia.

104 isms driving variant and IGHV4-34-expressing hairy-cell leukemias, we performed whole-exome sequencin

105 ellular signal-regulated kinase (ERK) in the hairy cells (HCs) of hairy cell leukemia.

106 Hairy cells are highly activated clonal B cells related

107 ete response (CR), defined as presence of no hairy cells in BM and blood with normalization of counts

108 r signal-regulated kinase phosphorylation of hairy cells in vivo.

109 The activation of hairy cells makes them unusually sensitive to interferon

110 s have yielded cellulose nanocrystals (CNC), hairy CNC (HCNC), and cellulose nanofibrils (CNF) with d

111 third-party observers, in the context of the Hairy-Downy game.

112 transcription of Notch target genes, such as Hairy Enhancer of Split (Hes) and Hes-related with YRPF

113 Expression of NOTCH and hairy enhancer of split (HES) proteins was assessed in p

114 sette subfamily G member 2 (ABCG2), p63, and hairy enhancer of split 1 (Hes1) were chosen as corneoli

115 cient mice showed reduced gene expression of hairy enhancer of split 1, an intestinal progenitor cell

116 he promoter that regulates the expression of Hairy Enhancer of Split 5, an inhibitor of neurite forma

117 Hairy enhancer of split-1 was a Notch1-downstream gene e

118 transcription of Notch target genes, such as hairy enhancer of split-related with YRPW motif (Hey)1 a

119 tors (Notch1-4), or through the Notch target Hairy/Enhancer of Split 1 (HES1), consistently leads to

120 Notch effector, and represses Notch-induced Hairy/Enhancer of Split 1 activity.

121 Therefore, we studied the conserved Hairy/Enhancer of Split family repressor Hairy, analyzin

122 Expression of the hairy/enhancer of split transcription factor hes6, is al

123 Hairy/Enhancer of Split-1, which is expressed in Clara c

124 a reduction in endodermal expression of the hairy/enhancer of split-related gene, her5, at mid to la

125 ing analysis and identified that Hey1 of the hairy/Enhancer of split-related repressor protein basic

126 The Notch target Hairy/Enhancer of Split1 (HES1) is sufficient to reprodu

127 ivity or deleting the downstream target gene Hairy/Enhancer-of-split 1, results in an increase in Ley

128 ch-regulated targets, including genes of the hairy/enhancer-of-split family in organisms ranging from

129 lences Notch, Neuralized and all three Ciona Hairy/Enhancer-of-Split genes.

130 Here, we show that the NOTCH target Hairy/Enhancer-of-Split Related with YRPW Motif 1 (HEY1)

131 g to enhanced expression of the Notch target hairy/enhancer-of-split related with YRPW motif protein

132 Here, we reveal that Hey2, a hairy/enhancer-of-split-related basic helix-loop-helix t

133 Most strikingly, Hairy exhibits biochemical activity on many loci that ar

134 , using numerical simulations, we show that "hairy" (f-star) or DNA grafted on nanocubes provides a g

135 was not effective in induction of Kr-h1 and Hairy gene expression in vitro in fat bodies of female m

136 directs spatially dynamic expression of the Hairy gene her5.

137 imer mediates JH III activation of Kr-h1 and Hairy genes in the context of light-dependent circadian

138 nt Physalis pubescens L., commonly named as "hairy groundcherry" in English and "Deng-Long-Cao" in Ch

139 Three genes, extramacrochaetae (emc), hairy (h) and stripe (sr), involved in repression have b

140 We find that a single gene, Hairy (H), which is needed to repress trichome fate, und

141 ese roles are distinct from the bHLH protein Hairy (H), which we show restricts atonal (ato) expressi

142 tous digital dermatitis (PDD), also known as hairy heel wart, is a growing cause of lameness of cows

143 ant resulted in inflammation and swelling of hairy hind foot skin in rats, a transient thermal hypera

144 as found to be significantly enhanced in the hairy hindpaw skin and its receptor GDNF family receptor

145 Western blot and real-time PCR analysis of hairy hindpaw skin and L2/L3 DRGs after saphenous nerve

146 Real-time PCR analysis of L2/L3 DRGs and hairy hindpaw skin at various times after saphenous nerv

147 In contrast, Hairy induces widespread histone deacetylation and inhib

148 sized pollen grains that can easily stick to hairy insects for pollination to nanoscale virus particl

149 g bone involvement (80%-95%), as well as the hairy kidney appearance on computed tomography scan (63%

150 odds ratio (OR) = 0.68, p = 0.0035) and oral hairy leukoplakia (adjusted OR = 0.67, p = 0.033) when c

151 Epstein-Barr virus (EBV) causes hairy leukoplakia (HL), a benign lesion of oral epitheli

152 as nasopharyngeal carcinoma (NPC), and oral hairy leukoplakia (OHL) lesions that have lytic infectio

153 ription from Zp, with all Z(+) cells in oral hairy leukoplakia being BLIMP1(+).

154 ses are due to lytic infection (such as oral hairy leukoplakia) or latent infection (such as nasophar

155 Here we demonstrate that the Arabidopsis HAIRY MERISTEM (HAM) family of transcription regulators

156 eviously found that in Arabidopsis SAMs, the HAIRY MERISTEM (HAM) family transcription factors form a

157 The Petunia hybrida HAIRY MERISTEM (HAM) gene, a member of the GRAS family o

158 including its specific expression signature, hairy morphology, and antiapoptotic behavior.

159 Monodispersed hairy nanocomposites with typical 2 nm (isophthalic acid

160 Polymer-grafted nanoparticles, often called hairy nanoparticles (HNPs), are an intriguing class of n

161 Hairy nanoparticles are employed as multifunctional node

162 ng the BclA protein that contributes to the 'hairy nap' layer.

163 BclA and related proteins form the hairy nap, and require ExsFA (BxpB) for their localizati

164 a 2D-crystalline basal layer, overlaid by a hairy nap.

165 degrees C showed that addition of 1 wt % of hairy NPs into PAO led to significant reductions in coef

166 These hairy NPs represent a new type of lubricating oil additi

167 These hairy NPs showed exceptional stability in poly(alphaolef

168 The excellent lubricating properties of hairy NPs were further elucidated by the characterizatio

169 lymer brush-grafted inorganic nanoparticles (hairy NPs) as highly effective lubricant additives for f

170 polymers imparts in situ formation of robust hairy perovskite QDs permanently ligated by outer hydrop

171 actor strategy for in situ crafting a set of hairy perovskite QDs with precisely tunable size and exc

172 le control over the stability enhancement of hairy perovskite QDs.

173 on of smooth region and higher proportion of hairy region that is heavily branched with arabinan and

174 analyses revealed that endogenous levels of Hairy Related Transcription (HRT) factor 2 (HRT2) peaked

175 Here, we establish Gridlock (Grl), a Hairy-related basic helix-loop-helix (bHLH) transcriptio

176 We previously reported that mice lacking the hairy-related basic helix-loop-helix (bHLH) transcriptio

177 sion of the transcriptional repressors HRT1 (Hairy-related transcription factor 1) and HRT2, in a CBF

178 c helix-loop-helix transcriptional repressor Hairy-related transcription factor 2 (Hrt2) is expressed

179 Hairy-related transcription factor proteins, which antag

180 the expression of Notch downstream effectors hairy-related transcription factors.

181 atin immunoprecipitation, we have analyzed a Hairy-repressible gene in the embryo during activation a

182 In these assays, the Hairy repression domain did not exhibit previously descr

183 terference of isoflavone synthase in soybean hairy root composite plants.

184 Overexpression of MYB182 in hairy root culture and whole poplar plants led to reduce

185 yptamine biosynthesis in Catharanthus roseus hairy root culture eliminates all production of monoterp

186 , and increased nicotine accumulation in the hairy root culture media.

187 Peanut (Arachis hypogaea) hairy root cultures produce a diverse array of prenylate

188 lium under N-limited growth conditions using hairy root cultures.

189 transferase genes in elicitor-treated peanut hairy root cultures.

190 have been targeted at either whole plants or hairy root cultures.

191 onfirmed the importance of RolB and RolC for hairy root development by A. rhizogenes K599.

192 at are the causative agents of crown gall or hairy root disease.

193 related pathogens that cause crown gall and hairy root diseases, which result from integration and e

194 tants, yucca6 plants do not display short or hairy root phenotypes and lack morphological changes und

195 re identified and validated using transgenic hairy root system.

196 TL1a using complementation in plant mutants, hairy root transformation and microscopy.

197 However, hairy root transformation had notable influence on Sphin

198 The effect of hairy root transformation on rhizosphere bacterial commu

199 ducing PRP expression in Medicago truncatula hairy root tumors disrupted cortical and vascular patter

200 PRP2 in that it greatly reduced viability of hairy root tumors.

201 A hairy-root transformation system was employed to investi

202 ling of mttt8 mutant seeds and M. truncatula hairy roots (mttt8 mutant, mttt8 mutant complemented wit

203 Compared with control roots, CrWRKY1 hairy roots accumulated up to 3-fold higher levels of se

204 Overexpression of MtMATE69 in hairy roots altered Fe homeostasis and hormone levels un

205 and their conjugates in Medicago truncatula hairy roots and anthocyanin-overproducing tobacco (Nicot

206 ocyanin and PA accumulation in M. truncatula hairy roots and Arabidopsis thaliana seeds, respectively

207 ores anthocyanins and PAs in mttt8 plant and hairy roots and further enhances both productions in wil

208 Transcriptome analyses of overexpressing hairy roots and knockout mutants of MtMYB5 and MtMYB14 i

209 owever, when complex expression systems like hairy roots are used for production, multiple population

210 unt for the reduced nodulation in GmLEC2a-OE hairy roots but increased nodulation in GmWRI1b-OE hairy

211 Histochemical staining of transgenic hairy roots carrying the promoter-reporter constructs in

212 GmLEC2a-OE hairy roots displayed different or even opposite express

213 entified as a key player in the formation of hairy roots during the plant-A. rhizogenes interaction.

214 Medicago truncatula hairy roots expressing LaPT1 accumulated isowighteone, a

215 ase showed strong expression in the stele of hairy roots for all 4 PRP genes tested, with additional

216 Importantly, we leverage the microbial hairy roots for rapid, reproducible efficacy screening o

217 NUP1-RNAi hairy roots had reduced NUP1 mRNA accumulation levels, r

218 SPARENT TESTA 2 (TT2) in Medicago trunculata hairy roots induces both proanthocyanidin accumulation a

219 decreased the number of galls in transformed hairy roots inoculated with RKN.

220 Overexpression of MtMATE66 rendered hairy roots more tolerant to Al(3+) toxicity.

221 e accumulation of proanthocyanidins (PAs) in hairy roots of Medicago truncatula.

222 PAR was expressed ectopically in transformed hairy roots of Medicago.

223 ucted comparative transcriptomics on soybean hairy roots of the variety Williams 82 and imbibing seed

224 further supported by analysis of transgenic hairy roots overexpressing soybean GmWRI1b-OE and GmLEC2

225 GmLEC2a-OE hairy roots produced fewer nodules, in contrast to GmWRI

226 xpression of TSAR1 or TSAR2 in M. truncatula hairy roots resulted in elevated transcript levels of kn

227 the ectopic expression of MYB15 in grapevine hairy roots resulted in increased STS expression and in

228 Here, we report that plant hairy roots support the growth of fastidious pathogens l

229 The overexpression of CrWRKY1 in C. roseus hairy roots up-regulated several key TIA pathway genes,

230 Overexpression of CrMAPKK1 in C. roseus hairy roots upregulated TIA pathways genes and increased

231 gly induces PA accumulation in M. truncatula hairy roots, and both myb5 and myb14 mutants of M. trunc

232 analysis of par mutants and MtPAR-expressing hairy roots, coupled with yeast one-hybrid analysis, rev

233 fer leads to the formation of crown galls or hairy roots, due to expression of transferred T-DNA gene

234 C2-L3 was ectopically expressed in grapevine hairy roots, showing a reduction in proanthocyanidin con

235 Using composite plants with transgenic hairy roots, we show that RDN1 and RDN2 orthologs from d

236 ed in GmLEC2a-OE but increased in GmWRI1b-OE hairy roots, which may account for the reduced nodulatio

237 factor was observed in MYB182-overexpressing hairy roots.

238 , and membrane lipids compared to GmWRI1b-OE hairy roots.

239 s of phytoplasma-infected plants, by forming hairy roots.

240 to 4-methyl tryptophan inhibition of CrWRKY1 hairy roots.

241 ced fewer nodules, in contrast to GmWRI1b-OE hairy roots.

242 antly while chrysin glycosides accumulate in hairy roots.

243 ce when over-expressed in transgenic soybean hairy roots.

244 ic increase of TIA accumulation in C. roseus hairy roots.

245 roots but increased nodulation in GmWRI1b-OE hairy roots.

246 ivity from the microsomal fraction of peanut hairy roots.

247 rther enhances both productions in wild-type hairy roots.

248 hpRNA mediated gene silencing in transgenic, hairy roots.

249 or tanshinone production in elicited Danshen hairy roots.

250 Drosophila pair-rule genes even-skipped and hairy, show periodic expression in the posterior disc, c

251 the three major hair follicle types of trunk hairy skin (guard, awl/auchene, and zigzag hairs) is inn

252 tiling of LTMR subtype axonal projections in hairy skin and a remarkable degree of spatial precision

253 s, but also by those innervating whisker pad hairy skin and supraorbital vibrissae.

254 their relative patterns of axonal endings in hairy skin and the spinal cord.

255 Light mechanical stimulation of hairy skin can induce a form of itch known as mechanical

256 The piloneural collar in mammalian hairy skin comprises an intricate pattern of circumferen

257 echanoafferents (C tactile, CT) in the human hairy skin have recently been linked to pleasant touch s

258 As hairy skin heals faster than nonhairy skin, we took bio-

259 ory neurons that detect pleasant stroking of hairy skin in vivo have not been reported.

260 ral axonal projections of the five principal hairy skin LTMR subtypes.

261 t of tactile stimulation differs between the hairy skin of the arm, and the glabrous skin of the palm

262 examined neuron had a receptive field on the hairy skin of the hindlimb and responded to noxious mech

263 y of cutaneous sensory arbor morphologies in hairy skin of the mouse using genetically-directed spars

264 from these cells provide innervation to the hairy skin of the perineal region and can be activated b

265 On the hairy skin of the upper limb, spatial acuity for pain an

266 mechanosensitive C fibers that innervate the hairy skin represent the neurobiological substrate for t

267 These neurons exclusively innervate hairy skin with large terminal arborizations that resemb

268 the upper lip (glabrous skin) and the chin (hairy skin).

269 ns are activated by massage-like stroking of hairy skin, but not by noxious punctate mechanical stimu

270 pheral nociceptor sensitization in hind foot hairy skin, but not glabrous skin, rapidly activates a d

271 In mouse hairy skin, lanceolate complexes associated with three t

272 In mouse hairy skin, Mrgprd, as marked by expression of green flu

273 In the glabrous and hairy skin, rare Merkel endings and transverse lanceolat

274 rogenitors that reside in the touch domes of hairy skin, termed touch dome progenitor cells (TDPCs).

275 In hairy skin, transient hyperalgesia was associated with s

276 afferent classes that innervate glabrous and hairy skin.

277 expressing neurons innervate exclusively the hairy skin.

278 ic sensory fibers that exclusively innervate hairy skin.

279 ic types of gentle mechanical stimulation on hairy skin.

280 c diblock DNA copolymers self-assemble into "hairy", star-like micelles, shown in the AFM image and t

281 Here, we show that expression of two other hairy stripes along the orthogonal A/P axis is establish

282 tio elastic micropillars spaced to mimic the hairy surface of bees.

283 nd cyclin D1, smoothening of leukemic cells' hairy surface, and, eventually, apoptosis.

284 Such bioinspired hairy surfaces could find applications in a variety of f

285 Nature relies on 'hairy' surfaces to protect blood capillaries from wear a

286 enhancers and long-range repressors such as Hairy that can dominantly inhibit distal elements.

287 ctors, many regions are targeted errantly by Hairy to modify the chromatin landscape.

288 rofile signature, change of morphology from "hairy" to "smooth," and eventually apoptosis.

289 ave been attributed to specialized feet with hairy toes that uncurl and peel in milliseconds.

290 found that three direct targets of ECR/USP--hairy, vrille, and Hr4--are required for cellular differ

291 ession of two JH-responsive genes, Kr-h1 and Hairy, was dependent on both the ratio of light to dark

292 itates the ubiquitylation of the HES protein Hairy, which disrupts the repressive activity of Hairy b

293 ila insulator proteins such as Suppressor of Hairy wing [SU(HW)] and Boundary Element Associated Fact

294 The Drosophila Suppressor of Hairy wing [Su(Hw)] insulator protein has an essential r

295 Suppressor of Hairy-wing [Su(Hw)] is a DNA-binding factor required for

296 The Drosophila Suppressor of Hairy-wing [Su(Hw)] protein is a globally expressed, mul

297 trovirus binds the zinc-finger Suppressor of Hairy-wing [Su(Hw)] protein that associates with hundred

298 gulation, we studiedDrosophila Suppressor of Hairy-wing [Su(Hw)], an exemplar multifunctional polydac

299 specific transcription factor (Suppressor of Hairy-wing) and a histone modification (H3K36me3).

300 luding compact body conformations, extremely hairy winter coats, and acute seasonal differences in me