ライフサイエンスコーパス: half-time

1 vitro, showed an approximately 3-fold longer half time.

2 ion of various severities did not affect the half-time.

3 mean liver half-time exceeded the mean tumor half-time.

4 K viruses reconstituted the capsid-uncoating half-time.

5 d rapidly from the blood pool, with a 12-min half-time.

6 es in [Ca(2+) ]m occurred more slowly (decay half time = 1.17 +/- 0.07 s).

7 blood and plasma was initially rapid (blood half-time, 1.31 +/- 0.81 min; plasma, 1.07 +/- 0.66 min;

8 sustained depolarization slowly inactivated (half-time = 10-15 min) ROMK channels that had been engin

9 ompared with that from wild-type xenografts (half-time, 12 +/- 1 h) and other organs (half-time, 2.6-

10 change was found in gastric emptying (median half-time 137 vs. 132 vs. 125 min, p = 0.52) or nutritio

11 heart 2.6 times more slowly than PHEN (mean half-time 155+/-52 versus 55+/-10 min, respectively; P <

12 ts (half-time, 12 +/- 1 h) and other organs (half-time, 2.6-21 h).

13 nvolvement exhibited a faster mean clearance half-time (28.6 minutes) than did children without evide

14 by either a prolonged terminal phase (blood half-time, 285 +/- 148.49 min; plasma, 240 +/- 84.85 min

15 f at least two species of free radicals with half-times: 3.3 and 125.4 h.

16 iving IVIG had a significantly shorter serum half-time (41.6 +/- 14.0 h) than those without (65.2 +/-

17 2 children had a mean radioaerosol clearance half-time (58.1 minutes; 162 studies in 108 children) si

18 4)I and to the retention and slow clearance (half-time, 63 +/- 6 h) of MIBG from transduced xenograft

19 its activation rate was surprisingly faster (half time about 20 s).

20 nstant of about 40 s, and it refills slowly (half-time about 8 min) as nascent vesicles bud from rand

21 meter volume, generator output rate, wash-in half-time, administration time).

22 Secondary end points included ADP recovery half-time (ADP t(1/2;) energetic marker), iron status, s

23 s suggested by a 2-fold increase in recovery half-time after fluorescence photobleaching.

24 D52 and RAD54 leaves unchanged the rejoining half times and the contribution of the slow component, a

25 se-deficient mutant displayed 4.5-s recovery half-time and a reduction in the mobile fraction to 30%.

26 linkage to ankyrin in resealed cells with a half-time and first order rate constant of 12 +/- 3.6 mi

27 isplaces endogenous ankyrin from IOVs with a half-time and first order rate constant of 42 +/- 14 min

28 Proximal colonic emptying half-time and geometric center at 48 hours were also acc

29 in a dual-phase solid-liquid study (emptying half-time and percentage emptying at 1, 2, 3, and 4 h),

30 d a quadratic relation between the diffusive half-time and the averaged mean curvature of the membran

31 abeled antibody formats showed shorter blood half-times and higher liver uptake than the radiolabeled

32 The half-time appears to vary inversely with the anatomic or

33 ) is reduced, flash excitation induces fast (half-time approximately 0.1 ms) oxidation of heme b(H),

34 E3BD folded fairly rapidly at 298K (folding half-time approximately 25 micros) and BBL and POB folde

35 and BBL and POB folded even faster (folding half-times approximately 3-5 micros).

36 hibits equally rapid dynamics of the Z ring (half time, approximately 8 s).

37 male smokers were predicted to be one and a half times as old as their chronological age compared to

38 The half-time associated with the change in sweat chloride,

39 at elevated temperatures, we infer that the half-time at 25 degrees C for spontaneous hydrolytic dec

40 ol transfer (% s(-1)) was unprecedented, the half-time at 37 degrees C being ~1 s.

41 The half-time at the nonstenotic mitral valve was less than

42 Whole-body retention half-time averaged 198 +/- 75 h.

43 ent of variation (15.2%, 5.8%, and 19.0% for half-time, cell volume [V(o)], and hydraulic conductivit

44 d exponentially, with the dose-rate decrease half-time corresponding to the effective clearance half-

45 ing detection limits, extending dissociation half-times, decreasing drug dosages and increasing drug

46 The half-time disappearance rate of I-gp120 from brain was 1

47 The pressure half-time Doppler echocardiography method is a practical

48 ose of normal tissues, except the mean liver half-time exceeded the mean tumor half-time.

49 Half-dose and half-time FA-guided PDT were both effective and safe in

50 n in muscles from wild-type mice (relaxation half-time fell from approximately 150 to approximately 9

51 A NPs as compared to the CPX NS with release half times following the order: CPX NS < CPX-Zn NS << CP

52 e ADF/cofilin protein actophorin reduced the half time for both dissociation of gamma-phosphate from

53 erimental conditions, we have found that the half time for fluorescence recovery of E. coli Z rings i

54 The half time for modification (t0.5) for the wild-type prot

55 The half time for the slow component of the loss with age wa

56 l: 2.45, 5.89) and approximately two and one-half times for stage IIA or stage IIB disease (odds rati

57 only a limited effect on the biodegradation half times for three compounds when tested at environmen

58 steine 822 may be responsible for the longer half-time for acid recovery observed with pantoprazole.

59 The half-time for activation by LSS was approximately 5 s.

60 At 0 degree C, the half-time for addition of the first G residue is about 3

61 however, mts1 had only a small affect on the half-time for assembly and predominately affected the ex

62 The half-time for dispersal of proregion-EGFP following WPB

63 The half-time for dispersal of VWF-EGFP from extracellular p

64 ilibrium binding occurred within 2 h and the half-time for dissociation was approximately 4 h.

65 The half-time for dissociation was approximately 9.6 h with

66 The apparent half-time for fluorescence decay (t(1/2)) in PSII(-Mn) i

67 This is comparable with the 9-s half-time for FtsZ turnover in vivo and the 8-s turnover

68 ence of this negative regulatory domain, the half-time for fusion of an entire population of lipid ve

69 The half-time for gastric emptying was determined by scintig

70 The half-time for kinetic exchange of the 3HT_6M complex was

71 In addition, the half-time for MT turnover was decreased in cells treated

72 rom monomer, our results agree with the 16-h half-time for N-site GTP in vitro and 33 h half-life for

73 Moreover, a small increase in the half-time for photoactivation of the oxygen-evolving com

74 g depolarizations, with 3 muM BBG increasing half-time for recovery (measured at -120 mV) from 24 to

75 caffeine application and also increased the half-time for recovery of [Ca2+]i to resting values.

76 The half-time for recovery of function from desensitization

77 es Mn(II) --> Mn(III) photooxidation, with a half-time for regeneration of the Mn(II) complex of ca.

78 elaxation, shorter time to peak pressure and half-time for relaxation than wild-type hearts.

79 ctin nucleating factor (ANF), decreasing the half-time for spontaneous actin polymerization.

80 Specifically, the half-time for subunit turnover in vitro at pH 7.7 was 42

81 The half-time for the accumulation of GLUT4-eGFP (enhanced g

82 The inferred half-time for the decay of this productive conformation

83 e is placed by a deoxyguanylate residue, the half-time for the first addition increases from about 3

84 The half-time for the liquid crystalline phase to switch is

85 nges in the glycosylation state of SUR1, the half-time for the maturation of K(ATP) channels, includi

86 Binding was rapid, with a half-time for the on-rate <1.8 min and for the off-rate

87 (t(12) = approximately 100 micros), but the half-time for the reduction of cytochrome f is increased

88 In 1.0 M PO4(3-), the calculated half-time for water oxidation is approximately 7 mus.

89 Because 20 h corresponds to eight half-times for dissociation, only monomer would be prese

90 . actinomycetemcomitans and achieved shorter half-times for killing than control PMNs when assayed at

91 Based on comparatively slow half-times for phosphorylation and dephosphorylation, ph

92 ars, several orders of magnitude longer than half-times for spontaneous decomposition of other enviro

93 Half-times for the fluorescence increase are consistent

94 als as dictated by their effective clearance half-times from the mouse femurs.

95 Overall, the gastric emptying half-time (GE t50) was 54.7 +/- 3.8 min.

96 nd liquids by scintigraphy (gastric emptying half-time [GE t(1/2)]); gastric volumes by single-photon

97 under-the-curve and delayed gastric-emptying half-time (geometric mean: 285; 95% CI: 184, 442, compar

98 umber of synapses was three to three and one-half times greater onto the somata of dorsal striatal sp

99 the half-dose group and 2 of 26 eyes in the half-time group had recurrence of CSC during follow-up;

100 d) = 48 fM and slower dissociation kinetics (half-time > 5 days) than those for the streptavidin-biot

101 dmission within 72 hrs were nearly two and a half times higher on days when > or =9 patients were adm

102 an extraordinarily fast recovery of KRas WT [half-time (HT), approximately 1.3 s] compared to HRas, R

103 We determine the diffusive half-time, i.e., the time that is required to reduce the

104 The biologic half-time in tumors was greater than in lungs.

105 This half time is 20 times slower than that for control dye-s

106 This half-time is substantially longer than that of exocytic

107 romagnetic exchange interaction is one and a half times larger than the ferromagnetic one, a magnetic

108 0 s), liquid-only gastric emptying (emptying half-time), liquid and solid emptying in a dual-phase so

109 age, it takes a human expert about one and a half times longer to decide an image is normal than to r

110 n <10 s after stimulation and decayed with a half time <20 s, indicating that the time course of bulk

111 iated endocytosis from clathrin-coated pits (half-time <10 min).

112 tide dissociates rapidly from alphaIIbbeta3 (half-time <2 min), consistent with the priming window de

113 ension after quick recovery) was analysed by half-time measurement and also by exponential curve fitt

114 acquired for MVA assessment by the pressure half-time method and by two 3D quantification methods (m

115 ntervention, the median MVAs by the pressure half-time method, mitral valve quantification software,

116 percentage gastric emptying (%GE) at 90 min, half-time (min) based on an exponential fit, and clearan

117 The Pd-Pt nanodendrites were two and a half times more active on the basis of equivalent Pt mas

118 CI, 6.46-10.73); cancer decedents, one and a half times more likely (OR, 1.57 [95% CI, 1.25-1.96]); a

119 change or explantation were nearly two and a half times more likely to have a final best-corrected vi

120 , although equilibration is very slow with a half-time near 6 h.

121 The determination of the half-time obviates the need for the commonly used exerci

122 cycles, and an exceptionally low adsorption half time of 10.6 min were achieved.

123 rm is a primary event, which proceeds with a half time of 14 min, and is followed by authentic proces

124 iency, electron injection kinetics exhibit a half time of 150 ps.

125 ls, but the slow component, operating with a half time of 18.4 h, is still able to rejoin the majorit

126 ynamic, continually remodeling itself with a half time of 30 s, similar to microtubules in the mitoti

127 dissociating from the mother filament with a half time of 500 seconds.

128 which peaked at 8+/-3 pA and declined with a half time of 7+/-2 ms.

129 CEF appeared with a half time of about 20 min after exposure to H2O2, sugges

130 y fast turnover, decaying in the dark with a half time of about 6 h.

131 om temperature at high concentrations with a half time of approximately 1 h.

132 ctal epithelial cells are turned over with a half time of approximately 1month in adult virgin mice.

133 unced slow relaxation component with a decay half time of more than 50 s was measured for deltat > 10

134 h of protein-polyphenol interactions and (a) half time of protein decay in gastric conditions (R(2)=0

135 lathrin- and K(+)-independent process with a half time of several seconds.

136 t DT40 cells rejoin IR-induced DNA DSBs with half times of 13 min and 4.5 h and contributions by the

137 e to a change of energy intake is slow, with half times of about 1 year.

138 dynamic gas exchange methods to characterise half times of stomatal opening and closing in response t

139 The half times of the slow component of the cell loss after

140 The half times of the slow component were only 26 years afte

141 proceeded faster than its exit, i.e., with a half-time of <1 s at 37 degrees C.

142 s, hydrolytic dechlorination proceeds with a half-time of 0.18 s.

143 into two phases; the first occurring with a half-time of 0.8 s, the second with a half-time of 14 s,

144 -type HELLS is very dynamic, with a recovery half-time of 0.8s and a mobile protein fraction of 61%.

145 (1/2)) approximately 26 h) with an estimated half-time of 1.2 h.

146 f-time, Td, equal to the effective clearance half-time of 117mSn(4+)DTPA from the femur (222 h).

147 The concentration (%ID/g) and biologic half-time of 131-Lym-1 in tumor were about twice those i

148 The effective half-time of 131I measured in athyrotic patients was use

149 with a half-time of 0.8 s, the second with a half-time of 14 s, possibly reflecting the differential

150 oth a slower response to AMP-PNP (activation half-time of 140 +/- 20 s vs. 21 +/- 4 s for wild type)

151 d on repolarization and decays slowly with a half-time of 150-300 ms.

152 This expression was reversible with a half-time of 17.2 +/- 0.5 h.

153 tion constant of 87 pM, an inhibitor release half-time of 17.2 min, and a Km/Kd ratio of 70,100.

154 ODCase turns its substrate over with a half-time of 18 ms, in a reaction that proceeds in its a

155 rted from the cell surface to the TGN with a half-time of 20-25 min, six to seven times faster than L

156 Ca2+] amplitude of 289 +/- 3 nM with a decay half-time of 20.8 +/- 0.2 ms and a full width at half-ma

157 o proceed from the N719 triplet state with a half-time of 200 +/- 60 ps and quantum yield of 84 +/- 5

158 aged 23% +/- 11% and cleared with a biologic half-time of 215 +/- 107 h (43 patients) or longer (5 pa

159 ed 2.4% +/- 2.0% and cleared with a biologic half-time of 243 +/- 144 h (27 patients) or longer (9 pa

160 aged 22% +/- 12% and cleared with a biologic half-time of 271 +/- 185 h (36 patients) or longer (6 pa

161 ch cleared monoexponentially with a biologic half-time of 293 +/- 157 h (33 patients) or did not clea

162 ynamic, continually remodeling itself with a half-time of 30 s.

163 caused disassembly of protofilaments with a half-time of 5 s.

164 that disulfide reduction: (i) occurs with a half-time of 6 h after folate-FRET endocytosis, (ii) beg

165 tion constant of 30 pM, an inhibitor release half-time of 64 min, and a Km/Kd ratio of 203,300.

166 The uptake and biological half-time of 67Cu-21T-BAT-Lym-1 in tumors were greater t

167 secreted from transfected HeLa cells with a half-time of 7 h and the rate-limiting step was exported

168 s were mixed, a FRET signal developed with a half-time of 7 s, demonstrating a rapid and continuous d

169 These dynamics occurred with a half-time of 7 to 8 min and were accompanied by state tr

170 tion is rapid and profound, occurring with a half-time of 70 s.

171 a conversion occurred at 37 degrees C with a half-time of 73 h.

172 reaction that proceeds in its absence with a half-time of 78 million years in neutral solution.

173 increased back to pre-stimulus values with a half-time of 8-10 min.

174 w orientation, which is close to the in vivo half-time of 9.6 days.

175 eady state) was similar between groups; mean half-time of 92 plus minus 4 min (NS between groups).

176 on recovered to 83% of control values with a half-time of about 10.5 min.

177 rature and racemizes at 199 degrees C with a half-time of about 11 h.

178 lmodulin was removed was even slower, with a half-time of about 20 min.

179 ence of insulin, IRAP was endocytosed with a half-time of about 3-5 min.

180 ls of similar morphology as Abeta42, but the half-time of aggregation (t1/2) increases exponentially

181 flet of liposomes and proteoliposomes with a half-time of approximately 0.1 min.

182 (125)I-MCP-1 has a blood clearance half-time of approximately 10 minutes and circulates in

183 SP rapidly - with a half-time of approximately 10 s with intracellular GTPga

184 ster than those determined by the LS method (half-time of approximately 120 micros vs half-time of ap

185 HPV31 entry was remarkably slow, with a half-time of approximately 14 h, whereas the entry half-

186 as does the truncation mutant CaSRDelta868 (half-time of approximately 16 h).

187 od (half-time of approximately 120 micros vs half-time of approximately 235 micros, in the presence o

188 BC) approximated a monoexponential response (half-time of approximately 26 s).

189 III3-FRET fit a first-order reaction with a half-time of approximately 30 s; the kinetics with desta

190 of less than 10 mum(2), with a concentration half-time of approximately 40 s.

191 PIP(2) was resynthesized with a half-time of approximately 50 s.

192 ach the PVC via early endosomes (EEs) with a half-time of approximately 60 min.

193 r GTP association, both with PLD2), it has a half-time of approximately 7 min, is enhanced with incre

194 1alpha carboxyl terminus matures completely (half-time of approximately 8 h) and without a lag period

195 elationship between Ca2+ wave period and the half-time of basal [Ca2+]SR recovery following Ca2+ rele

196 First, the half-time of conversion for both de novo and seeded kine

197 e concentration of mCAH3 increased, with the half-time of CP at the barbed end decreasing from approx

198 l (V(H)) of -30 mV, the enzyme decreased the half-time of decay and charge transfer of EPSCs by 32 an

199 tized in the continuous presence of agonist (half-time of desensitization, t(1/2), 2.13 s).

200 The decay half-time of each mutant correlated with the yield of th

201 igher maximal concentration and has a longer half-time of elimination than 3F-2.

202 he K(d) for ethylene binding to ETR1 and the half-time of ethylene dissociation from ETR1 do not unde

203 -daily relamorelin significantly reduced the half-time of gastric emptying and vomiting, as well as n

204 The primary end point was the half-time of gastric emptying.

205 (210)Pb from foliage operate with a maximum half-time of greater than 370 days (P > 99%), which is a

206 (v) The half-time of HbNO in vivo is approximately 40 min.

207 ime of approximately 14 h, whereas the entry half-time of HPV16 was 4 h.

208 rast to these relatively modest effects, the half-time of inactivation of the mutant was reduced by m

209 HR) are approximately 89% identical, but the half-time of internalization of agonist mediated by the

210 However, the half-time of intracellular [Ca2+] decay was significantl

211 hat Pex18p is constitutively degraded with a half-time of less than 10 min in wild-type Saccharomyces

212 duced receptor destabilization and increased half-time of ligand dissociation.

213 antidepressant treatment both increases the half-time of maximum recovery of GFP-Galphas and decreas

214 -170-decorated MT plus ends could reduce the half-time of melanosome aggregation by ~50%.

215 f P(700)(+)/P(700) is 447 mV and the in vivo half-time of P(700)(+) reduction by its natural donor, p

216 The half-time of recovery of acid secretion in rats followin

217 Over the course of 20 contractions the half-time of relaxation increased from 36.5 +/- 0.09 ms

218 are wortmannin-insensitive and occur with a half-time of roughly 5 min.

219 unexpectedly stable state with an unfolding half-time of several days.

220 y after peptide addition with a deactivation half-time of several minutes.

221 l transfer by 2.9-fold and by decreasing the half-time of sterol transfer by 10-fold (from 11.6 to 1.

222 ance gene) through the BDP, the evolutionary half-time of the gene of interest increases 4-10 times,

223 e experimental results, showing an increased half-time of the recorded events, are complemented by an

224 , 6-minute walk), muscle aerobic metabolism (half-time of VO2 and near infrared recovery), and forear

225 relatively rapid reaction, with an apparent half-time of ~1 s.

226 MCA4b or C28 is much slower, with an overall half-time of ~10 min.

227 definitely at 77 K, (*)CH(3) quenches with a half-time of ~2 min at this temperature.

228 issociation of the enzyme-substrate complex (half-time of ~30 s) is rate-limiting for the investigate

229 rin and GGA1 cycle on and off membranes with half-times of 10-20 s, independently of vesicle budding.

230 293 to 15.8 and 18.1 nM, respectively, with half-times of 2-3 minutes.

231 as significantly faster than that of WT with half-times of 25.1 +/- 1.8 and 32.2 +/- 2.5 min (mean +/

232 ation rate and leaf water potential (typical half-times of 30 min versus 1-2 h).

233 eclined from the peak in 1965 with effective half-times of 6-8 years, only interrupted by a temporary

234 0.88, and 0.79 cGy/h for dose rate decrease half-times of 62, 255, and 425 h and infinity, respectiv

235 gh iron-sulfur cluster F(X) is biphasic with half-times of approximately 5-30 ns ("fast" phase) and a

236 domain (450 nm) recovers spontaneously with half-times of approximately 50 s.

237 7.4, 20 degrees C, and in 0.3% SDS (w/v) the half-times of bR denaturation were 19.2 min for the wild

238 (WT) mice, nor were the delays in onset and half-times of Ca2+ sensitization, initiated by flash pho

239 ocessing in PC1/3 nulls with prolongation of half-times of conversion by 7- and 10-fold for proinsuli

240 The half-times of cyclization have been determined for both

241 We derived approximate expressions for the half-times of degradation and aggregation and show that

242 80Y and alpha7-P180F were studied and showed half-times of desensitization of 650 and 160 ms, respect

243 iciency in DNA-PK activity did not alter the half-times of either of these components of rejoining bu

244 e of dithiothreitol (DTT), and shows typical half-times of equilibration in the 10 h range.

245 smic tails are significantly different, with half-times of ligand internalization ranging from less t

246 is suppressed in HeLa cervical cancer cells, half-times of procaspase-9 processing, as well as the mo

247 The half-times of response are 8.8+/-0.2 seconds at 15 mM HC

248 ent exhibited two similar kinetic pools, the half-times of which were consistent with protein (t(b)(1

249 onset of desensitization dramatically, with half-times of ~930 and 700 ms, respectively, compared to

250 A plot of the phase 2b rate (or 1/half-time) of tension decay after stretch versus [P(i)]

251 fect gastric emptying ( approximately 30-min half time) or pH (mean +/- SD: 4.4 +/- 0.8) at 90 min.

252 tic pools of NBD-cholesterol efflux but with half-times over 200% shorter than those of the cytoplasm

253 treatment effect on ascending colon emptying half-time (P = .015) and overall colonic transit at 48 h

254 rgetics as reflected by shorter PCr recovery half-times (PCr t(1/2)) on phosphorus magnetic resonance

255 All eyes that received half-time PDT showed complete resolution of SRF within 6

256 received half-dose PDT and 26 eyes received half-time PDT.

257 to evaluate the reliability of the pressure half-time (PHT) method for estimating mitral valve areas

258 Glycoside cleavage proceeds with half-times ranging from 4 years for 2'-deoxyinosine to 4

259 alysis of the size-dependent energy transfer half times revealed that the distinct relaxation rate co

260 LNCaP cells was unstable with a degradation half-time (t(1/2)) of 3 h at 37 degrees C.

261 bunits returned to the outer segments with a half-time (t(1/2)) of approximately 24 and 29 minutes, r

262 The penetration index (P.I.) and clearance half-time (T(1/2)) of DTPA were measured in each lung.

263 The half-time (t(1/2)) of release of retinal from VCOP was 7

264 occurs from two distinct kinetic pools: the half-times (t(1/2)) for the two pools in CHO-K1 cells ar

265 /- 0.5 mm/min, and K(+) release and reuptake half-times (t(1/2)) of 12 +/- 2 and 32 +/- 4 s, respecti

266 apparatuses was rapid and exponential, with half-times (t(1/2)) of 5 min (WP), less than 4 min (MB),

267 The half time, t(1/2), for the conversion to normal islet am

268 hly dynamic, which are evaluated by recovery half time (t1/2) in seconds, determined by fluorescence

269 The half-time (t1/2) to peak for the increase in [Ca2+]m was

270 Dose-rate decrease half-times (Td) (time required for 137Cs gamma ray dose

271 f 137Cs gamma-rays with a dose-rate decrease half-time, Td, equal to the effective clearance half-tim

272 ime corresponding to the effective clearance half-time (Te) of the radioactivity from the femoral bon

273 n rate annual woody cover increase two and a half times that of Australian savannas.

274 The HGNs exhibited energy transfer half times that ranged from 105 +/- 10 ps to 1010 +/- 80

275 Five nodes regressed with a half-time that was shorter than their observed effective

276 ferase concentrations of more than two and a half times the upper limit of normal, history of diabete

277 ore, the authors calculated gastric-emptying half-time, the time for half of the ingested solids or l

278 nation thresholds were on average five and a half times those of controls and were proportional to th

279 le mutant Ku70(-/-)/RAD54(-/-) shows similar half times to Ku70(-/-) cells.

280 ng the first 300 s of induction, 117% in the half-time to completion of induction, and 65% in intrins

281 In contrast, the half-time to maximal constriction was significantly incr

282 to initial graviresponse and the subsequent half-time to maximum steady state bending rate increased

283 n varied by orders of magnitude, from a 14-h half-time to no regression at all.

284 e of appearance in interstitial fluid (i.e., half-time to steady state) was similar between groups; m

285 i, but both the maximum bending rate and the half-time to the maximum rate were significantly reduced

286 ents were obtained by matching the simulated half-times to those observed experimentally and reported

287 ifference to fall to half its initial value (half-time) varied with the anatomic severity of the valv

288 r vesicles containing a single flippase, the half-time was 3.3 min.

289 held at -20 mV compared to -80 mV (refilling half-time was 38 +/- 10 and 20 +/- 6 s, respectively).

290 and 24% HbF in the HDE RBCs, the circulation half-time was 40, 80, and 180 hours, respectively.

291 The mean whole-body clearance half-time was 44.98 +/- 16.29 h.

292 Dopamine half-time was 5.4 +/- 0.7 min at normothermia, increased

293 rate, and cardiac output significantly, the half-time was affected only slightly.

294 In mild mitral stenosis, the half-time was approximately 100 msec; in moderate stenos

295 Pulmonary clearance half-time was measured by using conventional gamma camer

296 wice those in normal tissues, although tumor half-time was similar to that of the thyroid.

297 Half times were calculated from the exponential rate con

298 Proportionate reductions in equilibration half-time were observed for preparations with a higher a

299 ocreatine and adenosine diphosphate recovery half-times were almost twice as long as in controls (P <

300 g studies in vivo showed a range of turnover half-times with an average of 25 s for MtbFtsZ as compar