ライフサイエンスコーパス: hallucination

1 t frequently presented directions (a form of hallucinations).

2 f human consciousness and clinical models of hallucination.

3 drowsiness, confusion, loss of balance, and hallucination.

4 lus when only noise is present: a perceptual hallucination.

5 e psychological treatment of auditory verbal hallucinations.

6 nsomnia leads to a reduction in paranoia and hallucinations.

7 sual recognition to social vision and visual hallucinations.

8 ring current models for mechanisms of visual hallucinations.

9 tem atrophy, including a patient with visual hallucinations.

10 sence of concurrent persecutory ideation and hallucinations.

11 ng stimulus onset predisposes an observer to hallucinations.

12 eep in patients with persistent delusions or hallucinations.

13 cataplexy, sleep paralysis and sleep-related hallucinations.

14 later emergence of persecutory ideation and hallucinations.

15 clinical symptoms of schizophrenia, such as hallucinations.

16 ucinations, relative to those without visual hallucinations.

17 reduced connectivity in Parkinson's disease hallucinations.

18 components of a model of oscillating visual hallucinations.

19 arent-rated negative symptoms and lowest for hallucinations.

20 during this phase that ketamine users report hallucinations.

21 fluctuations and clozapine is effective for hallucinations.

22 yperactivity in sensory cortex that leads to hallucinations.

23 m underlying 5-HT2A receptor-mediated visual hallucinations.

24 ual perceptual alterations, including visual hallucinations.

25 vercompensate in the case of CBS, leading to hallucinations.

26 psychotic hallucinations, and true psychotic hallucinations.

27 back mechanisms can enhance patterned visual hallucinations.

28 the observed regularity of geometric visual hallucinations.

29 en more pronounced and bizarre delusions and hallucinations.

30 These are experienced as geometric visual hallucinations.

31 function in patients with and without visual hallucinations.

32 de for reporting of persistent formed visual hallucinations.

33 cutive function, were associated with visual hallucinations.

34 linical study of whom 32% experienced visual hallucinations.

35 primary outcome is an improvement in visual hallucinations.

36 mechanisms and when and how to treat visual hallucinations.

37 uraging a more empathic approach to clinical hallucinations.

38 into the causal neuroanatomical substrate of hallucinations.

39 e of prior beliefs as a critical elicitor of hallucinations.

40 cal application was limited by dysphoria and hallucinations.

41 p dysfunction in schizophrenia patients with hallucinations.

42 ucinations, but not the subgroup with visual hallucinations.

43 brain circuit that mediated the conditioned hallucinations.

44 for severe (77.8%) compared with mild (36%) hallucinations.

45 nia was a mediator of change in paranoia and hallucinations.

46 n symptoms (fatigue, myoclonus, sedation and hallucinations, 0 = best and 40 = worst possible) betwee

47 mean difference 0.3, 95% CI -2.0 to 2.6) and hallucinations (-1.9, -6.5 to 2.7).

48 Among patients with baseline hallucinations, 13 of 17 (76.5%) who discontinued risper

49 ents for ADS-5102 vs placebo included visual hallucinations (15 [23.8%] vs 1 [1.7%]), peripheral edem

50 atients with Parkinson's disease (81 without hallucinations, 19 with visual hallucinations) and 34 he

51 confusion (50%), movement disorder (38%) and hallucinations (25%) were common.

52 omnia 89.7%, confusion 65.5%, amnesia 55.6%, hallucinations 51.9%), dysautonomia (hyperhidrosis 86.2%

53 an interesting perspective for understanding hallucinations accompanying sleep paralysis (SP; "night-

54 tigation of Captagon and its metabolites for hallucination addiction, and also analyzed the cell sign

55 ents with schizophrenia who have experienced hallucinations against patients who have not, matched on

56 Delusion, hallucination, agitation, anxiety, apathy, motor-disturb

57 ences in ocular pathology between the visual hallucination and non-visual hallucination groups.

58 past large-sample investigations of auditory hallucination and suggest potentially important new find

59 onse (>25% reduction in scores on delusions, hallucinations and agitation domains of the Neuropsychia

60 allucinations, and the link between auditory hallucinations and characterological entities.

61 symptoms than patients with PDD and had more hallucinations and cognitive fluctuations.

62 ), than Parkinson's disease patients without hallucinations and controls.

63 tic of positive symptoms, including auditory hallucinations and delusions of control.

64 rceptual-inference hierarchy may explain why hallucinations and delusions tend to cluster together ye

65 refers to the spectrum of illusions, formed hallucinations and delusions that occur in PD.

66 risperidone better controlled mean levels of hallucinations and delusions throughout follow-up (beta

67 Early emergence of comorbid hallucinations and delusions were observed in 57% of ind

68 arkinson's disease psychosis, which includes hallucinations and delusions, is frequent and debilitati

69 Despite their effectiveness in treating hallucinations and delusions, prolonged exposure to anti

70 ther with neuropsychiatric features, such as hallucinations and delusions, were scored and compared a

71 f positive symptoms in the disorder, such as hallucinations and delusions.

72 This framework may explain hallucinations and delusions.

73 nificant differences favoring citalopram for hallucinations and favoring placebo for sleep/nighttime

74 Visual hallucinations and illusions are thought to be caused by

75 contribute to the pathophysiology of visual hallucinations and may explain their predominantly visua

76 ut also in fact driven by, top-down effects: hallucinations and mental imagery.

77 15%-59%), with modest shared environment for hallucinations and negative symptoms (17%-24%) and signi

78 dorsolateral prefrontal cortex could affect hallucinations and negative symptoms, respectively.

79 her delineate an association between musical hallucinations and neurodegenerative disease.

80 ent adverse neuropsychiatric events, such as hallucinations and night terrors, reported by HIV patien

81 xole group had serious events, two of which (hallucinations and orthostatic hypotension) were deemed

82 The neural mechanisms that produce hallucinations and other psychotic symptoms remain uncle

83 mise for treating refractory auditory verbal hallucinations and other selected manifestations of schi

84 on selectivity provides a mechanism for both hallucinations and perception.

85 ctivity in Parkinson's disease patients with hallucinations and performed an analysis of controllabil

86 tive decline with new-onset recurrent visual hallucinations and progressive lethargy.

87 ce occurs in most patients with delusions or hallucinations and should be treated as a clinical probl

88 ew the demographics of subjects with musical hallucinations and to determine the prevalence of neurol

89 elopment of therapies specifically targeting hallucinations and visuoperceptive functions.

90 e (81 without hallucinations, 19 with visual hallucinations) and 34 healthy age-matched controls.

91 o positive (i.e., subthreshold delusions and hallucinations) and negative (i.e., subthreshold social

92 ith a psychotic disorder and auditory verbal hallucinations, and 50 healthy control subjects underwen

93 higher order visual areas can induce complex hallucinations, and also reaffirm direct electrical brai

94 es persecutory ideation, auditory and visual hallucinations, and diagnosis of probable psychosis.

95 ore features of DLB (dementia, parkinsonism, hallucinations, and fluctuations) with striatal dopamine

96 ible neuronal events at the origin of visual hallucinations, and further suggests that brain oscillat

97 linked to cognitive dysfunction, audiovisual hallucinations, and negative affective states akin to th

98 clude myoclonic seizures, auditory or visual hallucinations, and renal failure.

99 evaluable items were most often orientation, hallucinations, and speech or mood content.

100 ation, Hoehn and Yahr score, fluctuations or hallucinations, and striatal DAT availability as measure

101 atic and multisensorial features of auditory hallucinations, and the link between auditory hallucinat

102 , quasi-psychotic delusions, quasi-psychotic hallucinations, and true psychotic hallucinations.

103 neurocognitive processes in auditory verbal hallucinations, and, consequently, it is proposed that p

104 s with hallucinations, particularly auditory hallucinations, antipsychotic discontinuation should be

105 offer a rare and unique opportunity to study hallucinations apart from confounding clinical factors,

106 Visual hallucinations are common in older people and are especi

107 Visual hallucinations are common in Parkinson's disease and are

108 Hallucinations are common in psychiatric disorders, and

109 However, irrespective of mechanism, visual hallucinations are difficult to treat.

110 These results suggest that perceptual hallucinations are due to an imprecise and biased state

111 Visual hallucinations are frequent, disabling complications of

112 Our findings provide insights into how hallucinations are generated, with breakdown of a key st

113 Intrusive memories, images, and hallucinations are hallmark symptoms of psychiatric diso

114 Visual illusions and hallucinations are hallmarks of serotonergic hallucinoge

115 Once hallucinations are identified, physical, cognitive and o

116 ngs suggest early onset illusions and formed hallucinations are linked to amyloid pathology in PD and

117 bances in psychosis, such as auditory verbal hallucinations, are associated with increased baseline a

118 s, defined as the occurrence of delusions or hallucinations, are frequent in Alzheimer disease (AD),

119 s or no hallucinations, patients with severe hallucinations as a presenting symptom at baseline had a

120 in reducing the severity of auditory verbal hallucinations as well as negative symptoms.

121 omnia Severity Index (ISI) and delusions and hallucinations assessed by the Psychotic Symptoms Rating

122 ary outcome was reduction in auditory verbal hallucinations at 12 weeks, measured by total score on t

123 sed KOR agonist, does not cause dysphoria or hallucinations at therapeutic doses in humans.

124 inant effects induced by LSD included visual hallucinations, audiovisual synesthesia, and positively

125 (rTMS) for the treatment of auditory verbal hallucinations (AVH), an update is provided on the effic

126 ients with pharmacoresistant auditory verbal hallucinations (AVH).

127 hizophrenia who report daily auditory verbal hallucinations (AVHs) and sociodemographically matched h

128 Auditory/verbal hallucinations (AVHs) are accompanied by activation in W

129 nic patients with or without auditory verbal hallucinations (AVHs) to that of normal controls remain

130 they included a reduction in the severity of hallucinations but an increase in the severity of sleep/

131 t was present for the subgroup with auditory hallucinations, but not the subgroup with visual halluci

132 connectivity changes with Parkinson's visual hallucinations, but the biological factors underlying se

133 n sulcal length increasing the likelihood of hallucinations by 19.9%, regardless of the sensory modal

134 tural markers that predict the occurrence of hallucinations by comparing patients with schizophrenia

135 eceptors may lead to the formation of visual hallucinations by increasing cortical excitability and a

136 Visual hallucinations can be a major hallmark of late stage dis

137 Here we show that visual hallucinations can be induced in the normal population b

138 els have revealed that the geometry of these hallucinations can be related to functional neuro-anatom

139 Our results demonstrate that hallucinations can be studied objectively, and they shar

140 This case series shows that musical hallucinations can occur in association with a wide vari

141 The primary outcome measure was the Hallucination Change Score (HCS).

142 as quantitative traits (self-rated paranoia, hallucinations, cognitive disorganization, grandiosity,

143 and had an extremely low frequency of visual hallucinations compared with Parkinson's disease.

144 effect of AVATAR therapy on auditory verbal hallucinations, compared with a supportive counselling c

145 tter atrophy patterns associated with visual hallucinations, comparing Parkinson's disease hallucinat

146 between abnormal visual function and visual hallucinations, considering current models for mechanism

147 ween-group d = -0.66) and positive symptoms (hallucinations, delusions, disorganization) (adjusted me

148 itions experience persistent auditory verbal hallucinations, despite treatment.

149 Delusion and hallucination did not.

150 a second experiment, we find that simulated hallucinations do not evoke the temporal distortion comm

151 and maintenance of persecutory ideation and hallucinations during 18 months of follow-up in the 2000

152 symptoms), and had enduring auditory verbal hallucinations during the previous 12 months, despite co

153 allucinators, all of whom experienced visual hallucinations, exhibited grey matter atrophy with signi

154 ess, delusions, unusual thought content, and hallucinations (F(1,26.69) = 12.42, p = .002; Cohen's f

155 eduncular hallucinosis, a syndrome of visual hallucinations following subcortical lesions long hypoth

156 from three centres in the UK who had command hallucinations for at least 6 months leading to major ep

157 mprovements relative to sham stimulation for hallucination frequency and clinical global improvement

158 Hallucination frequency, total auditory hallucination ra

159 Area Spt acts as a gateway to the hallucination-generating cerebral network.

160 Patients in the visual hallucinations group had similar disease duration but ha

161 ween the visual hallucination and non-visual hallucination groups.

162 r multiple comparisons, patients with visual hallucinations had significantly greater disease duratio

163 Auditory verbal hallucinations have attracted a great deal of scientific

164 Musical hallucinations have been linked to multiple associated c

165 agonists, including sedation, dysphoria, and hallucinations, have limited their clinical use.

166 Auditory verbal hallucinations (hearing voices) are typically associated

167 he absence of a sensory signal, also termed "hallucinations." Here, we investigated whether spontaneo

168 oral gyrus of either hemisphere and auditory hallucination; (ii) left superior-/middle-temporal gyri

169 Visual hallucinations, illusions and extrapyramidal tract signs

170 e correlated strongly with ratings of visual hallucinations, implying that intrinsic brain activity e

171 motor impairment correlated positively with hallucination improvement following rW rTMS.

172 continence in 50% and dementia in 39%.Visual hallucinations in 0%.

173 induced radial (<10 Hz) or spiral (10-20 Hz) hallucinations in a behavioral experiment involving full

174 is has implications for the understanding of hallucinations in clinical and non-clinical populations,

175 lovian learning task, we induced conditioned hallucinations in four groups of people who differed ort

176 s strong, overly precise priors can engender hallucinations in healthy subjects and that individuals

177 l connectivity changes that accompany visual hallucinations in Parkinson's disease and the organizati

178 dementia, which are common co-morbidities of hallucinations in Parkinson's disease and whose neuroana

179 The exact pathogenesis of visual hallucinations in Parkinson's disease is not known but a

180 t structural MRI differences associated with hallucinations in Parkinson's disease may permit earlier

181 ic and ophthalmological correlates of visual hallucinations in Parkinson's disease, the combined data

182 pathways have all been posited as causes of hallucinations in Parkinson's disease.

183 y contributing to the pathogenesis of visual hallucinations in Parkinson's disease.

184 ehension during movements and drive auditory hallucinations in pathological states, the synaptic orga

185 tome, and lead to the hypothesis that visual hallucinations in patients with COS may be because of de

186 Acting on command hallucinations in psychosis can have serious consequence

187 Auditory hallucinations in schizophrenia are alleviated by antips

188 rs) with persistent distressing delusions or hallucinations in the context of insomnia and a schizoph

189 Within the FTLD group, hallucinations in the initial years of the disease were

190 d a research programme to investigate visual hallucinations in the key and high burden areas of eye d

191 The resulting paranoia and hallucinations in turn cause further stress, and eventua

192 The phenomenon of musical hallucinations, in which individuals perceive music in t

193 Structural lesions associated with musical hallucinations involved both hemispheres with a preferen

194 f the variation in subjective experiences of hallucination is central to psychiatry, yet systematic e

195 The presence of hallucinations is a key diagnostic symptom of schizophre

196 r regimes where activity that corresponds to hallucinations is induced by both spatially homogeneous

197 toms, i.e., the presence of delusions and/or hallucinations, is a frequent complication of AD.

198 which plays the role in the pathogenesis of hallucinations, is activated by DOI in MIA offspring onl

199 The beneficial effect on hallucinations lasted for up to 3 months.

200 ocation dictated the sensory modality of the hallucination: lesions causing visual hallucinations wer

201 determines both the structure and content of hallucination-like events.

202 Our results suggest that lesions causing hallucinations localize to a single common brain network

203 Overall, the Hallucination Machine offers a valuable new technique fo

204 e we describe such a tool, which we call the Hallucination Machine.

205 nts illustrate potential applications of the Hallucination Machine.

206 These results suggest that perceptual hallucinations may be due to an imprecise and biased sta

207 The cultural shaping of hallucinations may provide a rich case-study for linking

208 ndividuals with non-clinical auditory verbal hallucinations (most of them also experienced other non-

209 A subset of patients with early onset formed hallucinations (n=21) had reduced higher visual function

210 Within this single hallucination network, additional connections with the l

211 tions that have been associated with musical hallucinations: neurological, psychiatric, structural, d

212 Hallucinations occur in both normal and clinical populat

213 Psychotic symptoms, delusions and hallucinations, occur in approximately 50% of individual

214 We found that hallucinations occurred following lesions to a variety o

215 with the occurrence of sensory illusions or hallucinations (odds ratio: 8.68, P < 0.001) and eyeball

216 Hallucinations of musical notation may occur in a variet

217 oss of vision leads to complex, vivid visual hallucinations of objects, people, and whole scenes.

218 e highly distinctive 'presence' or 'passage' hallucinations of Parkinson's disease and can help to ex

219 experienced significantly fewer (P = 0.016) hallucinations of the most frequently presented directio

220 Unique aspects of SP hallucinations offer additional prospects for investigat

221 Compared with patients with mild hallucinations or no hallucinations, patients with sever

222 basimglurant, including three patients with hallucinations or psychosis.

223 is a single underlying mechanism for visual hallucinations or they have different disease-dependent

224 Auditory hallucinations--or voices--are a common feature of many

225 us activity that predisposed participants to hallucinations: overall lower prestimulus activity and a

226 us activity that predisposed participants to hallucinations: overall lower prestimulus activity and a

227 There were more postoperative hallucinations (p=0.01) and nightmares (p=0.03) with inc

228 sions), P2 (conceptual disorganisation), P3 (hallucinations), P5 (grandiosity), P6 (suspiciousness/pe

229 For patients with hallucinations, particularly auditory hallucinations, an

230 with patients with mild hallucinations or no hallucinations, patients with severe hallucinations as a

231 lnesses are associated with psychosis, i.e., hallucinations (perceptions in the absence of causative

232 Hallucinations, perceptions in the absence of objectivel

233 gest mixed-methods investigation of auditory hallucination phenomenology so far.

234 iated derealisation, olfactory and gustatory hallucinations, physical symptoms such as headaches, abd

235 hought mentioned above except true psychotic hallucinations plus marked superstitiousness, sixth sens

236 rthostatic hypotension, syncope, dyskinesia, hallucinations, prolongation of the QT interval, and imp

237 ch as psychotic or pharmacologically-induced hallucinations, provide a unique opportunity to examine

238 using severe neurological symptoms including hallucinations, psychosis, and seizures, and may result

239 he Psychotic Symptoms Rating Scales Auditory Hallucinations (PSYRATS-AH).

240 Hallucination frequency, total auditory hallucination rating scale score, and clinical global im

241 ubjects with Parkinson's disease with visual hallucinations, relative to those without visual halluci

242 ty and complexity, the mechanisms underlying hallucinations remain largely untested.

243 The brain regions responsible for hallucinations remain unclear.

244 ed with presence of persecutory ideation and hallucinations, remaining so after adjustment for sociod

245 ion between brain oscillations and geometric hallucinations remains a mystery.

246 al research on the phenomenology of auditory hallucinations remains scarce.

247 Not all hallucinations require intervention but for those that a

248 Recent theories propose that hallucinations result from an imbalance of prior expecta

249 eriencing subsequent subclinical paranoia or hallucinations rose by 133% and 92%, respectively.

250 Level of hallucination salience may usefully guide selection of W

251 s network, Parkinson's disease patients with hallucinations showed reduced controllability (less infl

252 accompanied by sleep-wake symptoms, such as hallucinations, sleep paralysis and disturbed sleep.

253 ve daytime sleepiness, cataplexy, hypnagonic hallucinations, sleep paralysis, and disturbed nocturnal

254 Hallucination strength peaked at ~11 Hz flicker and was

255 sual stimulation produces regular, geometric hallucinations such as radial or spiral patterns.

256 Recent models for Parkinson's disease hallucinations suggest they arise due to a shift in the

257 encoding correlated with the magnitude of SZ hallucinations, task performance and an independent meas

258 ents with schizophrenia have auditory verbal hallucinations that are refractory to antipsychotic drug

259 1] years; P = .03), had more frequent visual hallucinations that did not achieve significance (9 of 1

260 ent in schizophrenia; (2) evidence regarding hallucinations that suggest they are not due to excessiv

261 EM-sleep-related phenomena (sleep paralysis, hallucinations) that intrude into wakefulness.

262 fMRI and iEEG, induced a topographic visual hallucination: the patient described seeing indoor and o

263 ducing positive symptoms (i.e. delusions and hallucinations), they do little to improve the disabling

264 Symptoms of psychosis, including hallucinations, thought insertion, strange experiences,

265 re assigned to cognitive therapy for command hallucinations + treatment as usual and 99 (50%) to trea

266 ) of 79 in the cognitive therapy for command hallucinations + treatment as usual group (odds ratio 0.

267 1: 1 ratio to cognitive therapy for command hallucinations + treatment as usual versus just treatmen

268 We studied 89 brain lesions causing hallucinations using a recently validated technique term

269 nd distinct hierarchical INT alterations for hallucinations versus delusions in the auditory and soma

270 s, and anterior cingulate metabolism; visual hallucinations (VH) with bilateral dorsolateral-frontal

271 the pathophysiological mechanisms of visual hallucinations (VHs) in patients with Parkinson disease

272 Visual hallucinations (VHs) occur in macular degeneration patie

273 onsequences, including anxiety, tachycardia, hallucinations, violent behavior, and psychosis.

274 The mean age at onset of the hallucinations was 56 years, ranging from 18 to 98 years

275 randomization, and the presence of baseline hallucinations was associated with a higher risk of rela

276 al vision, diminished tactile sensation, and hallucinations was persistently Bartonella koehlerae ser

277 which is hypothesized to account for visual hallucinations, we found connectivity with this region t

278 chniques and automated, data-driven methods, hallucinations were associated with specific brain morph

279 the thalamus while lesions causing auditory hallucinations were connected to the dentate nucleus in

280 of the hallucination: lesions causing visual hallucinations were connected to the lateral geniculate

281 th 75.5 +/- 8.0 years) and persistent visual hallucinations were documented in 63%.

282 yo medical record, 393 subjects with musical hallucinations were identified and divided into five cat

283 Visual hallucinations were more common in the group with neurol

284 ifiable groups (P < 0.001), whereas auditory hallucinations were more common in the psychiatric group

285 ase; resting tremor, pill-rolling tremor and hallucinations were more frequent in Lewy body disease.

286 Myoclonus, rigidity and hallucinations were more frequent in patients with sCJD

287 Patients with severe baseline hallucinations were more likely to relapse after randomi

288 ia and medication-refractory auditory verbal hallucinations were randomly allocated to receive 20 min

289 the four independent determinants of visual hallucinations were rapid eye movement sleep behavioural

290 Auditory verbal hallucinations were robustly reduced by tDCS relative to

291 s/signs, 3 (thought disorder, delusions, and hallucinations) were included in all diagnostic systems

292 nd the presence of visual and other auditory hallucinations, were evaluated independently in all five

293 n drive striking experiential changes (e.g., hallucinations) - which are experienced as involuntary -

294 completion is also critical for formation of hallucinations, which constitute a severe symptom of the

295 that some 5-HT(2A)R agonists induce LSD-like hallucinations, while others lack this psychoactive prop

296 mechanisms underlying its ability to induce hallucinations with greater duration and potency than cl

297 g the severity of persistent auditory verbal hallucinations, with a large effect size.

298 siological mechanism of illusions and formed hallucinations, with implications for their respective l

299 rse event (four due to psychotic disorder or hallucination within 10 days of start of the study drug)

300 n patients who go on to develop illusions or hallucinations within 3-4 years of follow-up in the Park