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1 rotonin transporter, and is unlikely to be a hallucinogen.
2 t little is known of how the CLA responds to hallucinogens.
3 erotonin receptor (5-HTR) agonists including hallucinogens.
4 on of both serotonergic and non-serotonergic hallucinogens.
5 of these scarcely characterized serotonergic hallucinogens.
6 phenethylamines derived from the 2C class of hallucinogens.
7 , which can be mimicked by administration of hallucinogens.
8 e one of the most potent naturally occurring hallucinogens.
9 anisms responsible for the unique effects of hallucinogens.
10 signaling pattern and behavioral response to hallucinogens.
11 ion of the more flexible phenethylamine type hallucinogens.
12 amate is a common mechanism in the action of hallucinogens.
13 become more widely used in the U.S. as legal hallucinogens.
14 get responsible for the actions of classical hallucinogens.
15 n, synthesize, extract, identify, and ingest hallucinogens.
16 c illness and the psychotomimetic effects of hallucinogens.
17 ast new light on the mechanisms of action of hallucinogens.
18 sedatives, stimulants, cocaine, opiates, and hallucinogens.
19 sed stereotypical behavioral response to the hallucinogen 2,5-dimethoxy-4-iodoamphetamine (DOI), an a
20 ed high prevalence of prior cocaine (35.5%), hallucinogen (49.4%), heroin (8.7%), and inhalant (30.4%
21 art in the behavioural effects of tryptamine hallucinogens(5), particularly 5-methoxy-N,N-dimethyltry
22 2A)R subtype has been shown to be central to hallucinogen action, yet the precise mechanisms mediatin
23 gs used in this context include serotonergic hallucinogens, amphetamine, and NMDA receptor antagonist
25 ing-methylated derivatives of the well-known hallucinogen and entactogen MDA (1a) were synthesized an
26 ic acid diethylamide (LSD) is a prototypical hallucinogen and its psychedelic actions are exerted thr
27 ecstasy is currently classified as a type of hallucinogen and its withdrawal is not recognized in the
28 ions for the inactivity of 6-fluoro-DET as a hallucinogen and to determine the effects of fluorinatio
31 d essential role in mediating the actions of hallucinogens and atypical antipsychotic drugs at 5-HT(2
32 serotonin receptors respond to the action of hallucinogens and atypical antipsychotic drugs, we have
34 t a low addictive potential similar to other hallucinogens and consistent with kappa opiate receptor
35 irus, with increased head-twitch response to hallucinogens and diminished antipsychotic-like effect o
36 ing in the mPFC to the behavioral actions of hallucinogens and further support the targeting of mGlu2
38 receptor (GPCR) that mediates the effects of hallucinogens and is the target of a number of commonly
39 cts were identified for structurally similar hallucinogens and nonhallucinogens and found to correspo
40 Associations between ED visits involving hallucinogens and SSD were estimated using cause-specifi
42 lie in the characterization of serotonergic hallucinogens and the screening of these compounds in bi
43 LT, mescaline) and of a selected group of 2C hallucinogens and their corresponding NBOMes, with EC50
45 ng middle-stage drugs (including stimulants, hallucinogens, and inhaled drugs) at t2 were increased f
46 ith such pharmacologically distinct ligands, hallucinogens, and nonhallucinogens obtained from all-at
47 The 5-HT(2A) receptor is a target of several hallucinogens, antipsychotics, anxiolytics, and antidepr
49 ences between psychedelic and nonpsychedelic hallucinogens as well as entactogens, and their behavior
51 receptor (5-HT2AR) is the target of classic hallucinogens, atypical antipsychotics, and psychoplasto
54 Due to the possible metabolism of certain hallucinogens by MAO-A, which would cause a bias in the
55 utic potential, and in drugs of abuse (e.g., hallucinogens, central stimulants, empathogens), the lat
57 ed PFC population activity, the serotonergic hallucinogen DOI dose-dependently decreased population a
59 d mental health conditions, individuals with hallucinogen ED visits had a greater risk of SSD compare
60 esults have led to the widely held idea that hallucinogens elicit their effect by modulating synaptic
67 , unambiguous evidence of the consumption of hallucinogens has not been reported from any rock art si
69 norin A, the most potent naturally occurring hallucinogen, has attracted an increasing amount of atte
71 n amphetamine, cannabis, club drug, cocaine, hallucinogen, heroin, nonheroin opioid, sedative/tranqui
73 otentially promising clinical application of hallucinogens in the treatment of anxiety reactive to ad
76 of HTR2A bound to 25-CN-NBOH-a prototypical hallucinogen-in complex with an engineered Galphaq heter
78 0.6 mg/kg, i.p.), which is a phenethylamine hallucinogen, increased glutamate to 206% above saline-t
81 hallucinations are hallmarks of serotonergic hallucinogen-induced altered states of consciousness.
83 ns Software categories (alcohol-, cannabis-, hallucinogen-, inhalant-, opioid-, sedative-, stimulant-
84 ies (disorders related to alcohol, cannabis, hallucinogens, inhalants, opioids, sedatives, stimulants
86 semisynthetic ergoline alkaloid analogue and hallucinogen, is a potent psychoplastogen with promising
91 valuated using in vivo behavioral assays for hallucinogen-like and 5-HT(1A) agonist activity and in v
92 DOI from saline were employed to assess the hallucinogen-like behavioral properties of these rigid t
93 receptors, making it one of the most potent hallucinogen-like phenylalkylamine derivatives reported
95 y of representatives of different classes of hallucinogens (LSD, 5-MeO-DALT, mescaline) and of a sele
97 report biochemical studies showing that the hallucinogen lysergic acid diethylamide, its precursor e
99 While a frequently raised concern is that hallucinogens may be associated with an increased risk o
100 enewed interest in the clinical potential of hallucinogens may lead people with depression to a gener
101 nderstanding of the response to serotonergic hallucinogens may provide mechanistic insights into perc
104 ction is strikingly similar to that of other hallucinogens, necessitating studies of direct compariso
105 uced by various related compounds, including hallucinogens (perceptual), entheogens (spiritual), and
107 suicide, persistent psychotic disorders, or hallucinogen persisting perception disorders following a
109 sidered an integral component of ritualistic hallucinogen practices, attenuates 25I-NBOH induced HTR
110 f 11 days, and the group was subdivided into hallucinogen-preferring users (n = 10) and MDMA-preferri
113 d the effects of acute administration of the hallucinogen psilocybin (.16 mg/kg) versus placebo on am
115 After the deletion of Nurr1 in the CLA, both hallucinogen receptor expression and signaling are lost.
116 et search techniques, the authors located 81 hallucinogen-related sites and categorized the informati
117 e and abuse/dependence of cannabis, cocaine, hallucinogens, sedatives, stimulants, and opiates was as
118 We will discuss marijuana, cocaine, opioids, hallucinogens, solvents and the newer so-called rave or
119 ic drugs, and activation of mGluR2 abolishes hallucinogen-specific signalling and behavioural respons
120 ur classes of psychoactive natural products: hallucinogens, stimulants, cannabinoids, and opioids.
126 posed mechanism of action for phenethylamine hallucinogens, that such compounds must be full agonists
127 Given the potential therapeutic actions of hallucinogens, these findings could accelerate the disco
129 gs and reduce the ability of psychotomimetic hallucinogens to increase glutamatergic transmission.
130 ortion with SSD at 3 years, 208 of 5217 with hallucinogen use [3.99%] vs 13 639 of 9 239 075 in the g
131 tudy, individuals with an ED visit involving hallucinogen use had a greater risk of developing an SSD
134 ut a partner had higher rates of alcohol and hallucinogen use, while those with same-sex sexual exper
140 ividuals with an incident ED visit involving hallucinogens were compared with members of the general
141 Annual rates of incident ED visits involving hallucinogens were stable between 2008 and 2012 and then