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1 is related to the number and distribution of hamartomatous brain growths (cortical tubers) that chara
2 ocytes to form hair follicles and stimulated hamartomatous changes.
3 ulin (FLCN) cause Birt-Hogg-Dube syndrome, a hamartomatous disease marked by mitochondria-rich kidney
4 tuberous sclerosis complex (TSC), a dominant hamartomatous disorder that often presents with mental r
5 , Lhermitte-Duclos disease (LDD) caused by a hamartomatous enlargement of the cerebellum, ataxia, sei
6 Cowden's disease, which are characterized by hamartomatous growth or cancer.
7 nt trait characterized by the development of hamartomatous growths in many organs.
8 ifferentiation results in the development of hamartomatous growths in many organs.
9 disorder characterised by the development of hamartomatous growths in many organs.
10 d characterized by the development of benign hamartomatous growths in multiple organ systems.
11 ease demonstrates a widespread potential for hamartomatous growths in multiple organ systems.
12 ps the region for Cowden disease, a distinct hamartomatous intestinal polyposis syndrome with increas
13 sphatase and tensin homolog (PTEN) generates hamartomatous intestinal polyps with epithelial and stro
14  disorder that results in the development of hamartomatous lesions in a variety of organ systems.
15 on of TSC2 can be critical in development of hamartomatous lesions in TSC and cancer pathogenesis.
16 lly been regarded as nonneoplastic, possibly hamartomatous lesions, but the pathogenesis of FGPs in b
17 ltisystem growth of benign tumours and other hamartomatous lesions, which leads to diverse and debili
18 terized by seizures, mental retardation, and hamartomatous lesions.
19 lyps, 1 or more of which was hyperplastic or hamartomatous (N = 397), were prospectively recruited.
20 tionally have been regarded as nondysplastic hamartomatous or hyperplastic lesions, but their pathoge
21 astrointestinal polyps, including at least 1 hamartomatous or hyperplastic/serrated polyp.
22 ngliocytoma of the cerebellum, is an unusual hamartomatous overgrowth disorder.
23 ry disease characterized by gastrointestinal hamartomatous polyposis and increased cancer susceptibil
24 an-Riley-Ruvalcaba syndrome (BRRS) is a rare hamartomatous polyposis condition with features of macro
25 sis, MUTYH-associated polyposis, and certain hamartomatous polyposis conditions.
26                                Patients with hamartomatous polyposis in the colon associated with gan
27 0) is an autosomal dominant gastrointestinal hamartomatous polyposis syndrome in which patients are a
28                          The best understood hamartomatous polyposis syndrome is Peutz-Jeghers syndro
29 nile polyposis (JP) is an autosomal dominant hamartomatous polyposis syndrome where affected individu
30 e pharmacological treatment identified for a hamartomatous polyposis syndrome.
31                                              Hamartomatous polyposis syndromes (HPS) account for a sm
32                                              Hamartomatous polyposis syndromes are a group of clinica
33                         The gastrointestinal hamartomatous polyposis syndromes are rare, autosomal do
34 rectal cancer (HNPCC) or Lynch syndrome, the hamartomatous polyposis syndromes, and certain other rar
35                                    The three hamartomatous polyposis syndromes, Bannayan-Riley-Ruvalc
36            Due to the relative rarity of the hamartomatous polyposis syndromes, recommendations for m
37 testinal features that are classic for other hamartomatous polyposis syndromes, such as Bannayan-Rile
38 otein phosphatase and a protein kinase cause hamartomatous polyposis syndromes, which are characteris
39 essment, and management of patients with the hamartomatous polyposis syndromes, with a focus on endos
40     We sought to determine the prevalence of hamartomatous polyposis-associated mutations in the susc
41 ssociated with risk of germline mutations in hamartomatous-polyposis associated genes.
42 ile polyposis syndrome (JPS) is an inherited hamartomatous-polyposis syndrome with a risk for colon c
43 shared clinical features of gastrointestinal hamartomatous polyps among the three syndromes and the c
44  aim of this review is to categorize gastric hamartomatous polyps and aid in the identification of hi
45  disorder characterized by predisposition to hamartomatous polyps and gastrointestinal cancer.
46 disease characterized by a predisposition to hamartomatous polyps and gastrointestinal cancer.
47 d disorder characterized by gastrointestinal hamartomatous polyps and mucocutaneous melanin pigmentat
48              Juvenile polyps are regarded as hamartomatous polyps and occur in sporadic and familial
49                             Gastrointestinal hamartomatous polyps are also present in Cowden syndrome
50                   Though most of the gastric hamartomatous polyps are benign, certain types are assoc
51 e characterized by multiple gastrointestinal hamartomatous polyps in the absence of the extraintestin
52    Mutant mice developed rectal bleeding and hamartomatous polyps in the colorectum.
53 autosomal-dominant disorder characterized by hamartomatous polyps in the gastrointestinal tract and b
54 in which the predominant finding is multiple hamartomatous polyps in the gastrointestinal tract.
55 B1 methylation was found in four of 22 (18%) hamartomatous polyps lesions.
56 utosomal dominant condition characterized by hamartomatous polyps of the gastrointestinal tract and a
57  with incomplete penetrance characterized by hamartomatous polyps of the gastrointestinal tract and a
58 er which is characterized by the presence of hamartomatous polyps throughout the gastrointestinal tra
59 n disorder in which individuals have typical hamartomatous polyps within the gastrointestinal tract.
60 minant condition characterized by intestinal hamartomatous polyps, mucocutaneous melanin deposition,
61       The focus of this review is on gastric hamartomatous polyps, which are relatively rare and diag
62 ial polyposis syndromes and gastric inverted hamartomatous polyps.
63 ologic characteristics of their polyps, each hamartomatous syndrome carries an elevated risk for canc
64                     Pathways involved in the hamartomatous syndromes include those of vascular endoth
65                   These results suggest that hamartomatous TSC skin tumors are induced by paracrine f
66 autosomal-dominant disorder characterized by hamartomatous tumors of the skin, kidneys, brain, and lu
67 ith tuberous sclerosis complex (TSC) develop hamartomatous tumors showing loss of function of the tum
68 disease tuberous sclerosis, characterized by hamartomatous tumors, results from mutations in either T