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1 pioneering spinal cord commissural axons is haptotactic.
4 a2 integrins were sufficient to inhibit both haptotactic and chemotactic migration induced by individ
5 hA2 receptor internalization, and suppressed haptotactic and chemotactic migration of prostate cancer
10 pled receptor kinase 6 (GRK6) is crucial for haptotactic but dispensable for chemotactic CCL21 gradie
11 tes and enhanced fibronectin (FN)-stimulated haptotactic cell migration equal to FAK-reconstituted ce
12 es, failure to potentiate integrin-dependent haptotactic cell migration in vitro, and accumulation of
13 These results support a model whereby (a) haptotactic cell migration on Ln-5 is regulated by conce
14 h factor-limiting conditions, Shc stimulates haptotactic cell migration without affecting anchorage-d
15 es its inhibitory function in ROCK-dependent haptotactic cell migration, as well as invasion of human
18 ibitory effect of GM3, together with CD9, on haptotactic cell motility was demonstrated by a few line
19 fibronectin, and N-cadherin; (iii) enhanced haptotactic cell motility; and (iv) converted epithelial
21 ails, which may provide both chemotactic and haptotactic cues for efficient CD8(+) T cell migration a
22 tween chemotactic (indirect stimulation) and haptotactic (direct stimulation) migration of two distin
24 d induced integrin alpha(v)beta(3)-dependent haptotactic endothelial cell adhesion and migration and
27 stability, suggesting that cells recognize a haptotactic gradient formed by a combination of laminins
28 raction suggests selective disruption of the haptotactic gradient may be an achievable therapeutic ap
29 oposed mechanisms include (1) formation of a haptotactic gradient that "guides" neutrophils to the ce
31 In vivo this could limit the formation of haptotactic gradients on endothelial heparan sulfate pro
32 inoglycans (GAGs) are essential for creating haptotactic gradients to guide the migration of leukocyt
33 igration on Ln-5, but only when migration is haptotactic (i.e., spontaneous or stimulated by alpha 3
36 ectionally spread, to polarize and to govern haptotactic migration along gradients of the adhesive li
37 xpressing cells displayed increased rates of haptotactic migration compared to those of LMP1-negative
38 th PF-573,228 inhibited both chemotactic and haptotactic migration concomitant with the inhibition of
40 Inhibition of MMPs significantly decreased haptotactic migration induced by individual growth facto
42 than tamoxifen at inhibiting chemotactic and haptotactic migration of both cell lines at all concentr
43 mobilized to tissues as demonstrated for the haptotactic migration of dendritic cells (DCs) toward hi
44 fashion, whereas immobilized PTN18 promotes haptotactic migration of glioblastoma cells in a PTPRZ1-
46 olecules and ECM proteins, thereby promoting haptotactic migration of T cells to sites of inflammatio
47 eviously demonstrated that in keratinocytes, haptotactic migration on laminin-5 was stimulated by ant
49 ion of CHL1 in HEK293 cells stimulated their haptotactic migration toward collagen I, fibronectin, la
50 ment to collagen I and laminin and increased haptotactic migration toward collagen I, relative to par
52 Axl as a novel regulator of endothelial cell haptotactic migration towards the matrix factor vitronec
53 ced lymphatic endothelial cord formation and haptotactic migration were suppressed by anti-alpha1 and
54 yed by absence of focal adhesions, decreased haptotactic migration while increasing random migration.
55 brain white matter were capable of promoting haptotactic migration, and this response was inhibitable
56 xperiments comparing the cell attachment and haptotactic migration-enhancing properties of DMP1 to BS
62 perse when Sp-Eph enhances adhesion, causing haptotactic movement to regions of higher ligand abundan
63 (GAGs) and chemokines drive the formation of haptotactic or immobilized gradients of chemokines at th
64 However, it is unclear how chemotactic and haptotactic pathways integrate with each other to drive
65 pecific abrogation of Arp2/3 interferes with haptotactic repositioning of platelets to microlesions,
66 ed that directional migration results from a haptotactic response of each cell to the gradient, a mod
68 thelial migration in vitro and inhibited the haptotactic response to wild-type CCL7, CXCL12, and CXCL
69 Based on these findings, we conceptualize haptotactic sensing as an exploration, with F-actin bund