ライフサイエンスコーパス: hard exudate

1 ated with increased risk of CSME and retinal hard exudate.

2 east in some cases it resolves with residual hard exudate.

3 al pigment epithelium alterations with dense hard exudates.

4 e associated with the development of retinal hard exudates.

5 d with serous retinal detachment and retinal hard exudates.

6 loss is likely to be associated with retinal hard exudate and serum lipid abnormalities.

7 Hard exudates and CME regressed completely in 15 of 15 a

8 id not show significant risk in worsening of hard exudates and severity of DME in the lipid-lowering

9 clinically significant macular edema (CSME), hard exudates, and other diabetic retinopathy (DR) end p

10 larization, a complete resolution of retinal hard exudates, and the development of a self-limited vit

11 ange in CDVA, CST, intraocular pressure, and hard exudate area over time.

12 Hard exudate area was assessed from color fundus stereop

13 es from autopsy specimens have characterized hard exudates as a composition of lipid-laden macrophage

14 d exudate scores, and ETDRS scores minus the hard exudate component.

15 and triglycerides) with development of CSME, hard exudate, DR progression, and development of prolife

16 retinal detachment had resolved, but retinal hard exudates had increased.

17 s (FVs) in the choriocapillaris, presence of hard exudate (HE), and cataract were determined.

18 ological outcomes between eyes with baseline hard exudates (HE) and all other eyes among patients wit

19 inopathy Study protocol) for the presence of hard exudates (HE), retinal thickening (RT), clinically

20 Hard exudates (HEs) are the classical sign of diabetic r

21 ith higher risk of incident CSME and macular hard exudate in the DCCT cohort.

22 they were mild increase in CFT, presence of hard exudates in center subfield, and absence of renal d

23 However, the characteristics of hard exudates in living patients have not been examined

24 This differentiation of retinal hard exudates in patients by AO-SLO may help in understa

25 y (AO-SLO) to examine the characteristics of hard exudates in patients with retinal vascular diseases

26 acy of radially arranged sectors affected by hard exudates in the detection of CSME.

27 mained attached, but progressive increase in hard exudates in the inferotemporal quadrant was noted i

28 on-wool spots, intraretinal hemorrhages, and hard exudates in the macula were observed by ophthalmosc

29 bles morphological classification of retinal hard exudates into two types, which could not be disting

30 retinal hemorrhages and/ or microaneurysms, hard exudates, new vessels, fibrous proliferations, and

31 Clinically, this appeared as hard exudate on funduscopic images.

32 than the thickness in regions with irregular hard exudates (P = 0.01 -->0.02).

33 tinal mass, and vitreous seeds resolved, the hard exudates persisted for more than 2 years after the

34 Hard exudates regressed completely in 14 of 15 eyes and

35 he macular edema had completely resolved and hard exudates regressed slowly in 100% of patients.

36 lipid-lowering group compared with placebo (hard exudates: relative risk, 1.00; 95% CI, 0.47-2.11; P

37 ed visual acuity (BCVA), presence of retinal hard exudates, retinal detachment (RD), cystoid macular

38 and RR 1.95, P for trend = 0.03 for LDL) and hard exudate (RR 2.44, P for trend = 0.0004 for total-to

39 t Diabetic Retinopathy Study (ETDRS) scores, hard exudate scores, and ETDRS scores minus the hard exu

40 Similarly, for the development of retinal hard exudates, the RR for the top vs bottom quintile of

41 The retinal thickness in regions with round hard exudates was significantly greater than the thickne

42 Retinal hard exudate, which is accompanied by macular edema, is

43 The cases of hard exudates within 1 disc diameter (DD) of the fovea (