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1 tion as well as defective arteriogenesis and heart malformation.
2 itor pool can cause a spectrum of congenital heart malformation.
3 is-regulation in the aetiology of congenital heart malformation.
4 s one of the most common forms of congenital heart malformation.
5 phogenesis and the pathogenesis of the small heart malformation.
6 a mosaic mutation was not diabetic but had a heart malformation.
7 -blindness, balance disorders and congenital heart malformations.
8 lways are associated with complex congenital heart malformations.
9 ental disorder characterized by skeletal and heart malformations.
10 and the most common syndrome of conotruncal heart malformations.
11 n heterotaxy and related isolated congenital heart malformations.
12 d as the underlying cause of many congenital heart malformations.
13 H but not secondary PH because of congenital heart malformations.
14 y history of this defect or other congenital heart malformations.
15 epresent a significant portion of congenital heart malformations.
16 ing of cranial neural tube defects (NTD) and heart malformations.
17 ozygous mutations in Nkx2.5 that cause human heart malformations.
18 sting of biomaterials for complex congenital heart malformations.
19 child with heterotaxy and complex congenital heart malformations.
20 ch normalized antiviral responses, prevented heart malformations, ameliorated developmental delays, i
21 ardiac valve defects are a common congenital heart malformation and a significant clinical problem.
22 showed classic signs of PAH toxicity such as heart malformation and deformities in the jaw, fin, and
25 birth defects involving body wall, gut, and heart malformations and in mouse misplacement of head an
32 rred GRNs, we discovered that the Mef2c-null heart malformations are partly driven by increased activ
34 g the most frequently encountered congenital heart malformations are those with so-called single vent
36 Previous studies showed that conotruncal heart malformations can arise with the increase or decre
38 malies, abnormal branchial arch derivatives, heart malformations, diaphragmatic hernia, renal hypopla
40 mation of the epicardium and leads to severe heart malformations in developing zebrafish (Danio rerio
41 rgans not in the central nervous system, and heart malformations, in particular, might present diagno
43 cushions frequently contribute to congenital heart malformations including septal and valvular defect
45 te inviable, dying neonatally and exhibiting heart malformations involving the right ventricular outf
46 2-5(Cre) (Ezh2(NK)) caused lethal congenital heart malformations, namely, compact myocardial hypoplas
47 lot (TOF), the most common severe congenital heart malformation, occurs sporadically, without other a
48 enome-wide association study of a congenital heart malformation phenotype, provides evidence that com
50 l septal defect (ASD) is a common congenital heart malformation that occurs as an isolated anomaly in
51 mite counts, neural tube closure defects and heart malformation that presented between E9.5 and E13.5
52 has allowed patients with previously lethal heart malformations to survive and, in most cases, to th
53 er frequent findings included hip dysplasia, heart malformations, variable developmental delay, and h
54 rely affected individuals display congenital heart malformations, variable skeletal abnormalities inc
56 As in the knockout mice, the conotruncal heart malformations were accompanied by outflow tract ob
57 s conferring significant risk for individual heart malformations which are linked to genes governing