ライフサイエンスコーパス: heat-labile

1 le components, one heat stable and the other heat labile.

2 ptimal at 7.2 to 7.5 and a pI of 5.1 and was heat labile.

3 of temperature, rendering the material more heat labile.

4 hole-cell preparations of H. ducreyi and was heat labile.

5 xins, Ehx activity was calcium dependent and heat labile.

6 t activity of DnaK and co-chaperones and are heat-labile.

7 ther collagenous proteins, is SDS-stable but heat-labile.

8 The asgE mutant yielded about 10-fold less heat-labile A-factor and about twofold less heat-stable

9 sgE cells is in the production or release of heat-labile A-factor.

10 Sequence comparisons of heat-stable and heat-labile AGPases identified an N-terminal motif uniqu

11 ngle knockouts with truncation in either the heat-labile (alt) or the heat-stable (ast) cytotonic ent

12 tor(s), which can survive freeze-thawing, is heat labile and also can be inhibited by superoxide dism

13 llowing: it was proteinaceous in that it was heat labile and destroyed by proteinase K; it was a glyc

14 ng activity in Alternaria extract was highly heat labile and had an M(r) of approximately 60 kDa.

15 Two types of enterotoxins, one heat labile and the other heat stable, are known to caus

16 two are cytotonic enterotoxins, one of them heat labile and the other heat stable.

17 mmunity and that this inhibitory activity is heat labile and trypsin sensitive and not attributable t

18 nd likely a protein, because its activity is heat labile and trypsin sensitive.

19 AAG activity was heat labile and was abolished by pronase or acid-glycine

20 10407 strain (colonization factor antigen I; heat-labile and heat-stable enterotoxin positive) and th

21 cells, which suggested the presence of both heat-labile and heat-stable factors.

22 like colonies were selected and screened for heat-labile and heat-stable toxins by GM1 enzyme-linked

23 This trophic activity was heat-labile and not present in parietal cortex cultures

24 s in the wild-type and countin(-) S>10Ks are heat-labile and protease-sensitive, suggesting that they

25 had a more complicated CD spectrum, and was heat-labile and susceptible to proteinase digestion.

26 e watery diarrhea through the elaboration of heat-labile and/or heat-stable toxins.

27 be blocked by monensin and brefeldin A, was heat labile, and appeared in a pattern different from th

28 glutathione, was proteinase K sensitive and heat labile, and could utilize ferric chloride, ferric c

29 nder both neutral and acidic conditions, was heat labile, and had a molecular weight of approximately

30 ited osteoclast differentiation was soluble, heat labile, and resided in the lower molecular weight (

31 at the cytosolic inhibitor is nondialyzable, heat-labile, and trypsin-sensitive, thereby identifying

32 e CF antibody response is directed against a heat-labile antigen that has chitinase activity, hereaft

33 The polymerase was found to be heat labile at 40 degrees C in the absence of added vRNA

34 the NFATc1-dependent production of a soluble heat-labile autocrine factor that is capable of promotin

35 factor in animal and plant cell extracts was heat labile but relatively insensitive to protease inhib

36 Second, a heat-labile but polymyxin B-insensitive factor present i

37 erotoxigenic B. fragilis, or ETBF) produce a heat-labile ca. 20-kDa protein toxin (BFT).

38 hed this binding, thus suggesting a role for heat-labile complement components.

39 nd cyclin A-CDK activities upon release from heat-labile complexes.

40 nucleatum and the Candida species involved a heat-labile component on F. nucleatum and a mannan-conta

41 Moreover, RRVs appear to carry a heat-labile component that actively suppresses activatio

42 Xenopus oocyte maturation, is the essential heat-labile component.

43 t expansins account for the pH-sensitive and heat-labile components of wall stress relaxation.

44 ng that the endotoxicity was associated with heat-labile components.

45 e enzymatic activity of CTA1, stabilized the heat-labile conformation of CTA1 and protected it from i

46 e that an HIV-inducing factor, distinct from heat-labile cytokines, present in the female genital muc

47 It is heat-labile, dependent upon divalent cations (Mg2+) for

48 A number of PPCPs were determined to be heat labile during laboratory simulation of the renderin

49 g both vaccines in a single formulation with heat-labile E. coli enterotoxin mutants reduced the resp

50 ining protein polysaccharide conjugates with heat-labile E. coli enterotoxin mutants, for infants and

51 ce factors, including heat-stable (estA) and heat-labile (eltA) enterotoxin genes.

52 The B subunit of Escherichia coli heat labile enterotoxin (LT-B) is a potent oral immunoge

53 genic E. coli use OMVs to deliver bundles of heat labile enterotoxin to host cells.

54 h the catalytic domains of diphtheria toxin, heat labile enterotoxin, and pertussis toxin, all three

55 h diarrhea caused by neither V. cholerae nor heat labile enterotoxin-producing Escherichia coli.

56 with a double-mutant of the Escherichia coli heat-labile enterotoxin (dmLT) adjuvant using microneedl

57 administered with or without a double-mutant heat-labile enterotoxin (dmLT) as an adjuvant, in Bangla

58 within the B5 structure of Escherichia coli heat-labile enterotoxin (EtxB) at around pH 5.0.

59 ceptor-binding B subunit of Escherichia coli heat-labile enterotoxin (EtxB) is a highly stable, nonto

60 The B-subunit pentamer of Escherichia coli heat-labile enterotoxin (EtxB) is an exceptionally stabl

61 or without 5 microgram of the mutant E. coli heat-labile enterotoxin (LT(R192G)) as a mucosal adjuvan

62 se (rUre) plus 25 microg of Escherichia coli heat-labile enterotoxin (LT) (n = 26) or placebo plus LT

63 Escherichia coli heat-labile enterotoxin (LT) and cholera toxin (CT) were

64 e and mutant forms of cholera toxin (CT) and heat-labile enterotoxin (LT) are effective adjuvants for

65 ion factors (CFs) such as CFA/I fimbriae and heat-labile enterotoxin (LT) are important virulence fac

66 Cholera toxin (CT) and heat-labile enterotoxin (LT) are powerful mucosal adjuva

67 Cholera toxin (CT) and Escherichia coli heat-labile enterotoxin (LT) are structurally similar AB

68 Cholera toxin (CT) and Escherichia coli heat-labile enterotoxin (LT) are two closely related mul

69 ization factor CS6 and cholera toxin (CT) or heat-labile enterotoxin (LT) as the adjuvant induced imm

70 Heat-labile enterotoxin (LT) displayed adjuvant properti

71 ry of inactivated influenza vaccine with the heat-labile enterotoxin (LT) from enterotoxigenic Escher

72 cholera toxin (CT) from Vibrio cholerae and heat-labile enterotoxin (LT) from enterotoxigenic Escher

73 cretion of two highly homologous substrates: heat-labile enterotoxin (LT) from enterotoxigenic Escher

74 Cholera toxin (CT) and the heat-labile enterotoxin (LT) from Escherichia coli are h

75 It appears that toxins such as the heat-labile enterotoxin (LT) from Escherichia coli can h

76 immunization allows safe delivery of native heat-labile enterotoxin (LT) from Escherichia coli via a

77 ety, and feasibility of a vaccine containing heat-labile enterotoxin (LT) from ETEC delivered to the

78 piglets were used to study the importance of heat-labile enterotoxin (LT) in infection with an ETEC s

79 Heat-labile enterotoxin (LT) is an important virulence f

80 y Vibrio cholerae across the outer membrane, heat-labile enterotoxin (LT) is retained on the surface

81 Heat-labile enterotoxin (LT) is secreted from ETEC via v

82 tion of these antigens with Escherichia coli heat-labile enterotoxin (LT) mutant R192G (LT R192G) or

83 Protein and mRNA levels of heat-labile enterotoxin (LT) of Escherichia coli are hig

84 s cholera toxin (CT) and the closely related heat-labile enterotoxin (LT) of Escherichia coli, the la

85 nt ETEC colonization factor CS6, either with heat-labile enterotoxin (LT) or patches containing CS6 a

86 various strains of Vibrio cholerae, and the heat-labile enterotoxin (LT) produced by some enterotoxi

87 Given recent evidence suggesting that the heat-labile enterotoxin (LT) provides a colonization adv

88 inding of the B subunits of Escherichia coli heat-labile enterotoxin (LT) to epithelial cells lining

89 In addition, physiologically active heat-labile enterotoxin (LT) was associated with ETEC ve

90 the A and B subunits of the Escherichia coli heat-labile enterotoxin (LT) were evaluated for their ab

91 Escherichia coli heat-labile enterotoxin (LT), an oligomeric protein with

92 acterial exotoxins, such as Escherichia coli heat-labile enterotoxin (LT), exert strong immunostimula

93 y strains that express K88 (F4)(+) fimbriae, heat-labile enterotoxin (LT), heat-stable enterotoxin b

94 that a major effector molecule of ETEC, the heat-labile enterotoxin (LT), may enhance these interact

95 immunized with urease plus Escherichia coli heat-labile enterotoxin (LT), or parenterally immunized

96 Heat-labile enterotoxin (LT), produced by enterotoxigeni

97 natural receptor for cholera toxin (CT) and heat-labile enterotoxin (LT), which are the causative ag

98 a CF antigen (CFA)/I-, CFA/II-, CFA/IV-, or heat-labile enterotoxin (LT)-ETEC diarrheal episode duri

99 Among its virulence factors, ETEC produces heat-labile enterotoxin (LT).

100 es the ADP-ribosyltransferase toxin known as heat-labile enterotoxin (LT).

101 trate safe application of a patch containing heat-labile enterotoxin (LT, derived from Escherichia co

102 hether a nontoxic mutant of Escherichia coli heat-labile enterotoxin (LT-K63) and CpG1826 as model ad

103 in combination with the E. coli B subunit of heat-labile enterotoxin (LTB).

104 Escherichia coli type IIa heat-labile enterotoxin (LTIIa) binds in vitro with high

105 n the presence of wild-type Escherichia coli heat-labile enterotoxin (LTwt), LTR72, a partially inact

106 (CT) or a detoxified mutant Escherichia coli heat-labile enterotoxin (R192G LT) were intranasally adm

107 txB) and its close relative Escherichia coli heat-labile enterotoxin (rEtxB) to act as mucosal adjuva

108 (shortest distance 26 A), cholera toxin and heat-labile enterotoxin (shortest distance 31 A), anti-H

109 n the presence of an Escherichia coli mutant heat-labile enterotoxin [mLT(R192G)] or cholera toxin su

110 ce and evaluated a modified Escherichia coli heat-labile enterotoxin adjuvant, LT(R192G), for augment

111 on 56 produced CTB variants that had 7 or 12 heat-labile enterotoxin B residue substitutions in the a

112 ycoproteins and recombinant Escherichia coli heat-labile enterotoxin B subunit (rEtxB) as an adjuvant

113 bound weakly, if at all, to Escherichia coli heat-labile enterotoxin B subunit.

114 tant form of the CT-related Escherichia coli heat-labile enterotoxin designated LT(R192G), or CpG oli

115 fusing ctxB and the related Escherichia coli heat-labile enterotoxin eltB genes at codon 56 produced

116 (T13I), a novel i.d. adjuvant of the type II heat-labile enterotoxin family, elicited strong systemic

117 Topical delivery of strong adjuvants such as heat-labile enterotoxin from Escherichia coli (LT) induc

118 ction render both CT and the closely related heat-labile enterotoxin from Escherichia coli (LT) unsta

119 Using the recognition process between heat-labile enterotoxin from Escherichia coli and gangli

120 immunized in conjunction with mutant (R192G) heat-labile enterotoxin from Escherichia coli.

121 or 2xAbeta1-15 plus mutant Escherichia coli heat-labile enterotoxin LT(R192G) adjuvant.

122 h or without the attenuated Escherichia coli heat-labile enterotoxin LT(R192G) as an adjuvant, was ex

123 ence factor released by this pathogen is the heat-labile enterotoxin LT, which upsets the balance of

124 The carrier moiety of heat-labile enterotoxin of Escherichia coli (EtxB) is fo

125 of immunostimulating (IS) patches containing heat-labile enterotoxin of Escherichia coli (LT) enhance

126 ty, immunogenicity, and adjuvanticity of the heat-labile enterotoxin of Escherichia coli (LT) was exa

127 understanding the adjuvant properties of the heat-labile enterotoxin of Escherichia coli (LT).

128 Cholera toxin (CT) and the heat-labile enterotoxin of Escherichia coli (LT-I) are m

129 The type IIb heat-labile enterotoxin of Escherichia coli (LT-IIb) and

130 ly detoxified mucosal adjuvant, derived from heat-labile enterotoxin of Escherichia coli (LTK63), was

131 The structure and function LT-IIa, a type II heat-labile enterotoxin of Escherichia coli, are closely

132 amperometric biosensor for Escherichia coli heat-labile enterotoxin on a sol-gel thin-film electrode

133 to Th17-inducing adjuvants, such as type II heat-labile enterotoxin or cholera toxin, resulted in in

134 combination with the mutant Escherichia coli heat-labile enterotoxin R72 (LT-R72) induced significant

135 V. cholerae enterotoxin and Escherichia coli heat-labile enterotoxin reversed passive latex agglutina

136 Heat-labile enterotoxin subunit B (LTB) is a noncatalyti

137 The nCT and heat-labile enterotoxin type 1 (LTh-1) redirected TT int

138 of cholera toxin as a surrogate for E. coli heat-labile enterotoxin was ineffective against ETEC inf

139 erum IgG and IgM antibody titers against the heat-labile enterotoxin were equivalent in the H10407- a

140 stable toxin (with or without genes encoding heat-labile enterotoxin), and Shigella spp.

141 n (ST-ETEC; with or without co-expression of heat-labile enterotoxin), and Shigella.

142 The B subunit of heat-labile enterotoxin, a periplasmic protein of Escher

143 short-circuit responses to Escherichia coli heat-labile enterotoxin, heat-stable enterotoxin, and Vi

144 Bromelain was 62% effective in preventing heat-labile enterotoxin-induced secretion, 51% effective

145 obacter spp. (OR: 1.46; 95% CI: 1.11, 1.91), heat-labile enterotoxin-producing E. coli (OR: 1.55; 95%

146 n factor antigens as well as the immunogenic heat-labile enterotoxin.

147 bitor for cholera toxin and Escherichia coli heat-labile enterotoxin.

148 mmetry, independent of the expression of the heat-labile enterotoxin.

149 a coli (LT-I) are members of the serogroup I heat-labile enterotoxins (HLT) and can serve as systemic

150 Cholera toxin (CT) and the type II heat-labile enterotoxins (HLT) LT-IIa and LT-IIb act as

151 Two families of bacterial heat-labile enterotoxins (HLTs) have been described: the

152 antigens (CFAs), and enterotoxins, including heat-labile enterotoxins (LT) and heat-stable enterotoxi

153 Cholera toxin (CT) and the type II heat-labile enterotoxins (LT-IIa and LT-IIb) are potent

154 The type II heat-labile enterotoxins (LT-IIa and LT-IIb) of Escheric

155 ectious diarrhea, produce heat-stable and/or heat-labile enterotoxins and at least 25 different colon

156 e Vibrio cholerae-Escherichia coli family of heat-labile enterotoxins having an AB5 structure.

157 LT-IIa and LT-IIb, the type II heat-labile enterotoxins of Escherichia coli, are closel

158 nction to cholera toxin and LT-I, the type I heat-labile enterotoxins of Vibrio cholerae and E. coli,

159 ctions of cholera toxin and LT-I, the type I heat-labile enterotoxins of Vibrio cholerae and enteroto

160 ly that includes the B subunits of the AB(5) heat-labile enterotoxins, cholera toxin, pertussis toxin

161 The type II heat-labile enterotoxins, LT-IIa and LT-IIb, exhibit pot

162 The heat-labile enterotoxins, such as cholera toxin (CT), an

163 inant human resistin was able to protect the heat-labile enzymes citrate synthase and Nde1 from therm

164 The ability of 2 mutants of heat-labile Escherichia coli enterotoxin (LTK63 and LTR7

165 field isolates examined produce one or more heat-labile exotoxins and may suggest that M. ovis plays

166 e amino acids and peptides, and at least two heat-labile extracellular proteases.

167 ng by enhancing recruitment of Integrator or Heat Labile Factor to snRNA or RDH genes, respectively.

168 monstrated that SCs, but not OECs, secrete a heat labile factor(s) that inhibits oligodendrocyte myel

169 nucleoprotein (U7 snRNP) and an unidentified heat-labile factor (HLF).

170 Here, we show that a heat-labile factor greater than 100 kDa in the cytoplasm

171 BMSC CM contained a heat-labile factor that increased BMMs osteoclastogenesi

172 ts of rim-pattern MAbs were synergistic with heat-labile factors in normal human serum for opsonizati

173 ing joint formation requires RAG1, RAG2, and heat-labile factors present in the nuclear extract of no

174 Astrocytes release soluble, heat-labile factors that increase barrier properties and

175 label from [32P-adenylate]NAD to one or more heat-labile factors within extracts prepared from severa

176 Originally described as a heat-labile fraction of the serum responsible for the op

177 Ala222Val (677C-->T) polymorphism encodes a heat-labile gene product that is associated with elevate

178 El Tor hemolysin of Vibrio cholerae and the heat-labile hemolysin of Vibrio mimicus.

179 in activity, indicating the involvement of a heat-labile high-molecular-weight component in Ped-2E9 c

180 ative examination of the heat-stable (O) and heat-labile (HL) serogrouping results for 9,024 sporadic

181 enterotoxin positive) and that production of heat-labile holotoxin provides a significant advantage i

182 d 50% activity remaining at 0 degrees C) and heat labile (inactivated within 10 min at 37 degrees C).

183 y was shown to be highly protease sensitive, heat labile, independent of divalent cations, and extrem

184 The heat-labile inhibitor has been identified as 2,4-dihydro

185 Purification of the most heat-labile inhibitor to homogeneity yielded two prepara

186 The enzyme is heat labile, is soluble, and requires oxygen for activit

187 igenic Escherichia coli (ETEC) produces both heat-labile (LT) and heat-stable (ST) enterotoxins and i

188 Adhesins and enterotoxins, including heat-labile (LT) and heat-stable (STa) toxins, are the k

189 . coli (E. coli O39), which was negative for heat-labile (LT) and heat-stable (STa, STb) ETEC toxins,

190 virulence, in particular the plasmid-encoded heat-labile (LT) and heat-stable enterotoxins and the co

191 Although heat-stable (ST) and heat-labile (LT) enterotoxins produced by enterotoxigeni

192 enes that encode the heat-stable (ST) and/or heat-labile (LT) enterotoxins, as well as surface struct

193 , diphtheria, pertussis and Escherichia coli heat-labile (LT) toxins, and all produce disease by alte

194 oides fragilis (ETBF) cells produce a 20-kDa heat-labile metalloprotease toxin which is potentially i

195 , suggesting an endotoxin-like property of a heat-labile molecule(s) of the spirochete.

196 vated temperatures that may adversely affect heat-labile molecules and macromolecules.

197 Heat-labile MOM proteins were required for this enhanced

198 d this limitation is often attributed to the heat-labile nature of Rca.

199 tion, and activity are all influenced by the heat-labile nature of the isolated toxin A chain.

200 Fibroblasts immortalized with the same heat-labile oncogene do not display reduced levels of cy

201 us erythematosus (SLE), a study of the serum heat-labile opsonic capacity (HLOC) in such patients was

202 intestinal epithelium and the elaboration of heat-labile or heat-stable toxins which induce a secreto

203 y enterotoxins (such as cholera toxin or the heat-labile or stable enterotoxins produced by Escherich

204 Monoclonal antibody (mAb) F78 recognizes a heat-labile particle composed of Sm core proteins design

205 representatives were discovered as the most heat labile phenolic compounds of cloudy apple juice.

206 nd applied with the aim to find out the most heat labile phenolic constituents in cloudy apple juice.

207 ore, MKP-3 and MKP-1 appeared to be critical heat-labile phosphatases involved in the activation of E

208 The heat-labile plasma protein, fibronectin, inhibited PF4 b

209 of the spliceosomal snRNPs, and an essential heat labile processing factor has been identified as sym

210 ound that necrotic cardiomyocytes released a heat-labile proinflammatory signal activating MAPKs and

211 cteroides fragilis that produce a ca. 20-kDa heat-labile protein toxin (termed B. fragilis toxin [BFT

212 rm a complex in porcine aqueous humor with a heat-labile protein(s).

213 ting as a molecular chaperone and protecting heat-labile proteins from thermal aggregation and inacti

214 ting as a molecular chaperone and protecting heat-labile proteins from thermal aggregation in a non-s

215 thermal aggregation and inactivation of the heat-labile proteins, citrate synthase and malate dehydr

216 istent with their additional regulation by a heat-labile repressor.

217 as several advantages compared with standard heat-labile reverse transcription methods.

218 Consequently, vaccines derived from heat-labile SAT viruses have been linked to the inductio

219 w-derived dendritic cells was facilitated by heat-labile serum component(s) independently of Dectin-1

220 ymorphonuclear leukocytes (CGD PMN) required heat-labile serum components (e.g., C3), and binding of

221 ytes was enhanced approximately 1000-fold by heat-labile serum components.

222 d uptake by CHO-CD14 cells involving another heat-labile serum factor(s) and cell-associated recognit

223 ects a cell surface abnormality and requires heat-labile serum factors.

224 uman lung microvascular EC lysates contained heat-labile sialidase activity for a fluorogenic substra

225 th heat-inactivated whole GBS bacteria and a heat-labile soluble factor released by GBS during growth

226 The IL-8-stimulating activity was heat labile, suggesting that the activity is a protein.

227 er formaldehyde treatment, suggesting that a heat-labile surface protein component is responsible for

228 a clear correlation between the reduction of heat-labile, surface-exposed Asn and Gln residues with t

229 erase I of E. coli and is substantially more heat labile than its most closely related homologs from

230 tested in the immunoassay and type 19A PS is heat labile, the pleural fluid samples were also tested

231 Based on previous reports that RCA is heat-labile, the Rubisco activation state was measured.

232 The inflammatory effect of cHSP60 was heat labile, thus excluding a role of contaminating LPS,

233 , the mitogenic activity of C pneumoniae was heat-labile, thus excluding a role of lipopolysaccharide

234 he catalytic subunit of the Escherichia coli heat labile toxin (LTA(1)) was studied after expression

235 B subunit homopentamers of cholera toxin and heat labile toxin as model GSL-binding proteins, the CaR

236 Some vaccine formulations containing the heat labile toxin B subunit (LTB) have been used in clin

237 in and the mucosal adjuvant Escherichia coli heat labile toxin LT(R192G) induces nearly complete prot

238 cholera toxin, ricin, botulinum toxin A, and heat labile toxin of E. coli).

239 ered with the Escherichia coli double mutant heat-labile toxin (dmLT) adjuvant afforded modest (10-30

240 ombined, coadministered with a double mutant heat-labile toxin (dmLT) from Escherichia coli, used as

241 multiple-mutated CT (mmCT) and double-mutant heat-labile toxin (dmLT) on human T cell responses.

242 ered with or without mutant Escherichia coli heat-labile toxin (LT(R192G)) as a mucosal adjuvant.

243 It has long been known that ETEC heat-labile toxin (LT) activates production of cAMP in t

244 erized virulence proteins, in particular the heat-labile toxin (LT) and colonization factors (CFs), t

245 ETEC-mediated diarrhea is orchestrated by heat-labile toxin (LT) and heat-stable toxins (STp and S

246 luated the adjuvanticity of Escherichia coli heat-labile toxin (LT) and LT-R192G.

247 -based adjuvants including cholera toxin and heat-labile toxin (LT) are powerful manipulators of muco

248 STa and heat-labile toxin (LT) are virulence determinants in ETE

249 patch delivery system (PDS) with or without heat-labile toxin (LT) from Escherichia coli or subcutan

250 skin-patch vaccine containing the pathogen's heat-labile toxin (LT) in a population of travellers to

251 nit (LTB) chimera admixed with double mutant heat-labile toxin (LT) LT-R192G/L211A (dmLT).

252 The heat-labile toxin (LT) of Escherichia coli is a potent m

253 In this study, we examined whether the ETEC heat-labile toxin (LT) or the heat-stable toxin (STa, al

254 colonization factor antigen I (CFA/I) and to heat-labile toxin (LT) were measured.

255 cytial virus (RSV), we used Escherichia coli heat-labile toxin (LT), and LTK63 (an LT mutant devoid o

256 istration to mice: alum, a derivative of the heat-labile toxin (LT), and the phase 1 flagellin of S.

257 TS), cholera toxin (CT) and Escherichia coli heat-labile toxin (LT), are powerful mucosal adjuvants.

258 colonizing the small intestine and producing heat-labile toxin (LT), heat-stable toxin (ST), or both

259 Escherichia coli heat-labile toxin (LT), or derivatives of LT carrying mu

260 l adhesins known as colonization factors and heat-labile toxin (LT).

261 enterocytes and for the optimal delivery of heat-labile toxin (LT).

262 cholera toxin (CT) and the Escherichia coli heat-labile toxin (LT-IIa), have been shown to enhance m

263 comprised of a pentameric B subunit of ETEC heat-labile toxin (LTB) in lieu of the CTB pentamer and

264 with either cholera toxin B-subunit (CTB) or heat-labile toxin (LTB), or phosphate-buffered saline (P

265 Native cholera toxin (nCT) and the heat-labile toxin 1 (nLT) of enterotoxigenic Escherichia

266 immunization with chimera plus single-mutant heat-labile toxin [LT(R192G)] elicited strong serum anti

267 ried between the symptomatic children (44.2% heat-labile toxin [LT], 38.5% heat-stable toxin [ST], an

268 ssessed a combination of F4 fimbriae and the heat-labile toxin and heat-stable toxin B enterotoxins.

269 all 103 amino acids of the Escherichia coli heat-labile toxin B subunit (LT-B) were assessed in mice

270 imbrial antigens, including CfaEB and a CfaE-heat-labile toxin B subunit (LTB) chimera admixed with d

271 onization factors and genetically detoxified heat-labile toxin from a human ETEC isolate (LTh).

272 that prior lung administration of a modified heat-labile toxin from Escherichia coli (LTK63) enhances

273 rog p.o. or rectally or 10 microg i.n.) plus heat-labile toxin from Escherichia coli as the mucosal a

274 The expression of soluble heat-labile toxin is subject to catabolite (glucose) act

275 istered intranasally with attenuated E. coli heat-labile toxin LT(R192G) also induced serum rotavirus

276 s particle, together with attenuated E. coli heat-labile toxin LT(R192G) as an adjuvant, reduces feca

277 a mucosal adjuvant, mutant Escherichia coli heat-labile toxin LT(R192G).

278 R192G), a genetically detoxified form of the heat-labile toxin of enterotoxigenic Escherichia coli.

279 ective antigen of Bacillus anthracis and the heat-labile toxin of Escherichia coli.

280 and that the T3SS effector EspB of EPEC, and heat-labile toxin of ETEC were secreted.

281 in expression because cells intoxicated with heat-labile toxin overproduce and release cAMP.

282 relieved the CRP-dependent repression of the heat-labile toxin promoter.

283 s to E. coli colonization factors and to the heat-labile toxin that induces the diarrhea.

284 ial adhesion and accelerated delivery of the heat-labile toxin, a principal ETEC virulence determinan

285 erial toxins: the cholera toxin, the E. coli heat-labile toxin, and three S. aureus toxins (the enter

286 heat-stable toxin (ST), with or without the heat-labile toxin, are among the four most important dia

287 In contrast to heat-labile toxin, CRP positively regulates the expressi

288 oadministered with a mutant Escherichia coli heat-labile toxin, LT-R192G (mLT) adjuvant.

289 asally (i.n.) with a mutant Escherichia coli heat-labile toxin, LT-R192G (mLT), as mucosal adjuvant.

290 ane vesicles have been shown to contain ETEC heat-labile toxin, the repression of nlpA may be an indi

291 pe 1 (CNF1) and CNF2, heat-stable toxin, and heat-labile toxin.

292 lude cholera toxin (CT) and Escherichia coli heat-labile toxin.

293 Cholera and Escherichia coli heat-labile toxins (CT and LT) require proteolysis of a

294 ETEC diarrhoea, and to assess the safety of heat-labile toxins from E coli (LT) delivered via patch.

295 as fluorescent chimeras of Escherichia coli heat-labile toxins LTI and LTIIb.

296 H. ducreyi produces two known heat-labile toxins, a hemolysin and a cytolethal distend

297 we used CT and the related Escherichia coli heat-labile type II enterotoxin LTIIb.

298 The factor was heat labile, was sensitive to trypsin treatment, and was

299 AGPases of cereal endosperms are heat labile, while those in other tissues, such as the p

300 nknown, although we have evidence that it is heat labile with molecular mass < 10 kD.