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1 delta transposon in the hafE gene eliminated hemadsorption.
2  strains and serovars also differed in their hemadsorption abilities that positively correlated with
3 e, but no such pretreatment was required for hemadsorption activity (HAD) at 22 or 37 degrees C.
4                  Although conservation of NA hemadsorption activity among avian virus NAs suggests th
5             To determine the conservation of hemadsorption activity among different NAs, we have exam
6  Kong/7/75 N2 NA abolished the high level of hemadsorption activity exhibited by the wild-type protei
7                          NAs which possessed hemadsorption activity for chicken erythrocytes (RBCs) w
8 s of influenza A viruses exhibit significant hemadsorption activity that localizes to a site distinct
9 H stalk chimeras are surface-expressed, show hemadsorption activity, and trigger MV F.
10      A swine virus N1 NA exhibited only weak hemadsorption activity, while in human virus N1 and N2 N
11  equine virus N8 NA possessed high levels of hemadsorption activity.
12  glycoprotein that has been shown to mediate hemadsorption and be involved in host immunomodulation a
13 formation, we found that 4-GU-DANA prevented hemadsorption and fusion of persistently infected cells
14                                          The hemadsorption and neuraminidase activities of the chimer
15                                              Hemadsorption and neuraminidase activity analysis confir
16 ild-type levels resulted in reacquisition of hemadsorption and normal levels of HMW1, HMW3, and P65.
17                                      Fusion, hemadsorption, and neuraminidase activities were demonst
18 ctive reactivity for NR6(DeltaEGFR) in mixed hemadsorption assays, fluorescence-activated cell sortin
19 namely, measurements of intracellular pH and hemadsorption assays, which confirmed the much reduced a
20 in, we found that 4-GU-DANA had no effect on hemadsorption but did inhibit HA2b-red blood cell fusion
21 ls inoculated intramuscularly with 10(2) 50% hemadsorption doses (HAD(50)) of ASFV-G- A137R remained
22  even when administered to pigs at 10(6) 50% hemadsorption doses (HAD50).
23 ecific antibodies that blocked M. pneumoniae hemadsorption (HA) and binding to the sialylated glycopr
24                          Characterization of hemadsorption (HA)-negative mutants has resulted in iden
25 ed by a sialic acid binding site, termed the hemadsorption (Hb) site, which is discrete from the enzy
26 rface and were functional as demonstrated by hemadsorption, hemolysis, and NA assays.
27  than for reducing plaque number or blocking hemadsorption indicate the particular efficacy of these
28        However, these transformants remained hemadsorption negative, suggesting that HMW1 is required
29        The P30 adhesin genes of spontaneous, hemadsorption-negative (HA-) class II Mycoplasma pneumon
30 erein result in loss of these proteins and a hemadsorption-negative (HA-) phenotype, prompting the de
31 igen-based assays such as the observation of hemadsorption or cytopathic effect.
32 and has been shown to be responsible for the hemadsorption phenomenon observed for ASFV-infected cell
33            However, this deletion mutant was hemadsorption positive, indicating that P28 may not be r
34 on of this disruption plasmid into wild-type hemadsorption-positive (HA+) M. genitalium cells permitt
35                         However, none of the hemadsorption-positive NAs could bind equine, swine, or
36  pulmonis UAB CT V-1 variants that differ in hemadsorption properties.
37                  Mutagenesis of the putative hemadsorption site of A/duck/Hong Kong/7/75 N2 NA abolis
38 species lack molecules, recognized by the NA hemadsorption site, present on human and chicken RBCs.