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1 lture-initiating cell assay cells (primitive hematopoietic cells).
2 of induced RASGRP1 overexpression in primary hematopoietic cells.
3 s during short-term or long-term tracking of hematopoietic cells.
4 uled out a requirement for PHD2 signaling in hematopoietic cells.
5 ing and bone marrow (BM) retention of normal hematopoietic cells.
6 nine nucleotide exchange factor expressed in hematopoietic cells.
7 elic inactivation of the respective genes in hematopoietic cells.
8 entiation state in both normal and malignant hematopoietic cells.
9 ysis of human-specific pathogens that infect hematopoietic cells.
10 IS mice, had minimal effects on normal human hematopoietic cells.
11 was not attributable to lymphocytes or other hematopoietic cells.
12 ecial type of proteasome mainly expressed in hematopoietic cells.
13  was not induced by transfer of Ttc7-mutated hematopoietic cells.
14 plants are repopulated with autologous human hematopoietic cells.
15 is of DB818-treated murine Hoxa9-transformed hematopoietic cells.
16 d Nom1, are essential for the maintenance of hematopoietic cells.
17 n of leukemic cells without impacting normal hematopoietic cells.
18 re mainly caused by enhanced accumulation of hematopoietic cells.
19 esis pathway expression compared with normal hematopoietic cells.
20 minant of HCMV tropism for select subsets of hematopoietic cells.
21 mage, is compromised in Foxo3(-/-) primitive hematopoietic cells.
22 ase Atad3a hyperactivated mitophagy in mouse hematopoietic cells.
23 ated mice lacking LIFR in either CD11c(+) or hematopoietic cells.
24 diation preconditioning to ablate endogenous hematopoietic cells.
25 liferation, survival, and differentiation of hematopoietic cells.
26 to the development and lineage commitment of hematopoietic cells.
27 provides a distinct competitive advantage to hematopoietic cells.
28   In these models, Nf1 haploinsufficiency in hematopoietic cells accelerated tumor onset and increase
29 nscription factor predominantly expressed in hematopoietic cells, acts as an essential regulator of h
30 to SP and NK-A to protect the most primitive hematopoietic cell and also to maintain immune/hematopoi
31  safely induces immune tolerance to combined hematopoietic cell and organ allografts in humans.
32 current knowledge on cortactin expression in hematopoietic cells and discusses the functional implica
33 cells cause an expanded population of mutant hematopoietic cells and drive an aberrant inflammatory s
34 ggering apoptosis in specific cells, such as hematopoietic cells and endothelium.
35 -/-) mice have decreased MHC-I expression on hematopoietic cells and fewer CD8(+) T cells prior to in
36  a ubiquitous pathogen that latently infects hematopoietic cells and has the ability to reactivate wh
37 s (PI3Ks) gamma and delta are key enzymes in hematopoietic cells and have been seen as high-value tar
38 ppressor, which regulates the homeostasis of hematopoietic cells and immune responses.
39 py on PI-PLCbeta1 inositide signaling, using hematopoietic cells and MDS samples.
40  T cells was dependent on TLR9 expression on hematopoietic cells and partially dependent on receptor
41 tibodies inhibited leukemic, but not normal, hematopoietic cells and synergized with other antileukem
42 netic screen, comparing normal primary mouse hematopoietic cells and their malignant counterparts in
43        Chimeric mice deficient for Notch3 in hematopoietic cells and/or resident tissue cells were ge
44              As Gdf11 is expressed by mature hematopoietic cells, and erythroid precursor cell expres
45 esis (enterocytes, hepatocytes, macrophages, hematopoietic cells, and in the case of pregnancy, place
46 e is expressed in many cell types, including hematopoietic cells, and is a member of the Tec kinase f
47  rate of protein synthesis relative to other hematopoietic cells, and it can be easily adapted to qua
48 ersistence of higher VCN in the repopulating hematopoietic cells are associated with better outcome.
49        We show that metabolic alterations in hematopoietic cells are fundamental to the pathogenesis
50  marrow transplantation experiments identify hematopoietic cells as the predominant source of plasma
51 numbers and impaired B cell development in a hematopoietic cell-autonomous manner.
52 has been until recently considered absent in hematopoietic cells because these cells express the cort
53 that Grasp55 plays a role in Myc-transformed hematopoietic cells but not in normal hematopoietic cell
54 ion in the host and to a lesser extent donor hematopoietic cells by overexpressing the RA-catabolizin
55 , rather than a local role for Tmprss6-/- in hematopoietic cells, contributes to the MEP lineage comm
56                                  Nkx2.3(-/-) hematopoietic cells could not rescue wild-type mice from
57        These results establish that IUHCT of hematopoietic cells decorated with GSK3 inhibitor-loaded
58    Taken together, these results reveal that hematopoietic cell-derived Gdf11 is largely dispensable
59                        In addition, although hematopoietic cell-derived Gdf11 may contribute to the p
60 ess functions of the Id3 and/or Tet2 gene in hematopoietic cell development and clonal hematopoiesis.
61 e the causal role of heterochromatin loss in hematopoietic cell development.
62 tt lymphoma, including genes associated with hematopoietic cell differentiation (FLI1, BCL11A) or B-c
63 dies identify a pathogenic role for Hsp70 in hematopoietic cells during allergic airway inflammation;
64              We identified 28 subsets of non-hematopoietic cells during homeostasis, 14 of which expr
65 ost HSCs, enabling rapid and efficient donor hematopoietic cell engraftment.
66 y limits the activation and proliferation of hematopoietic cells, enhances the gammaherpesvirus-drive
67 ted biological functions and pathways during hematopoietic cell expansion and development.
68 which is unresponsive to transplant due to a hematopoietic cell-extrinsic mechanism.
69 ritical regulator of inflammation controlled hematopoietic cell fate decisions of HSCs.
70 neurotrophins have the potential to regulate hematopoietic cell fate decisions.
71 ing a role for O-GlcNAcylation in regulating hematopoietic cell fate.
72 ught to examine whether macrophages or other hematopoietic cells found in the embryo prior to HSC gen
73                           Transplantation of hematopoietic cells from a healthy individual (allogenei
74 e substantially greater in baboons receiving hematopoietic cells from a pig expressing high levels of
75                         We hypothesized that hematopoietic cells from a restricted lineage would be m
76 y chimeras that lacked GILT in both TECs and hematopoietic cells had a high conventional T/Treg cell
77 oietic and nonhematopoietic cells, Map3k8 in hematopoietic cells has a more dominant role.
78 ntinuous quantitative single-cell imaging of hematopoietic cells has been used to solve decades-long
79                   However, RAS activation in hematopoietic cells has immunologic effects that diverge
80 raftment of normal and diseased human immune/hematopoietic cells has made in vivo functional characte
81 ymphomas (EBV-PTLD) after transplantation of hematopoietic cell (HCT) or solid organ (SOT) but is una
82  General Hospital, we pioneered simultaneous hematopoietic cell (HCT)/kidney transplantation from HLA
83 the maintenance and expansion of multipotent hematopoietic cells, i.e. hematopoietic stem cells (HSCs
84 acted with nascent and emerging intra-aortic hematopoietic cells (IAHCs).
85 itional deletion of Bmal1 in endothelium and hematopoietic cells in murine models of microvascular an
86         Total body irradiation (TBI) damages hematopoietic cells in the bone marrow and thymus; howev
87 ive regulator of the CXCR4 retention axis of hematopoietic cells in the bone marrow.
88                     Progenitors of the first hematopoietic cells in the mouse arise in the early embr
89  By transplantation of lentiviral-transduced hematopoietic cells in the Mpl-deficient mouse model, we
90 fferentially interacts with murine and human hematopoietic cells in these mouse models and how these
91 d infer cellular dynamics of differentiating hematopoietic cells in vitro and in vivo.
92 formed hematopoietic cells but not in normal hematopoietic cells in vivo.
93 nant state in normally differentiating human hematopoietic cells in which MYC expression has been der
94                         Here we show that in hematopoietic cells, including multiple myeloma (MM), ly
95                     On Mx1-Cre activation in hematopoietic cells induced by poly (I:C) injection, all
96 rrent Tet2 loss and Nras(G12D) expression in hematopoietic cells induced myeloid transformation, with
97 focused on the molecular mechanisms by which hematopoietic cells initiate and maintain innate and ada
98                 Thus, activation of NLRP3 in hematopoietic cells initiates IL-1beta-driven paracrine
99                           Our data suggest a hematopoietic cell-intrinsic role of Lsh in B cell devel
100 analyses suggest that CD70 expressed by host hematopoietic cells is involved in the control of allore
101 he hypomethylation phenotype of Dnmt3a (-/-) hematopoietic cells is reversible, we developed an induc
102 RAGE expressed on stromal cells, rather than hematopoietic cells, is critical to induction of asthma/
103 nt activation of an Src family kinase (SFK), hematopoietic cell kinase (HCK), specifically in DCs in
104  gene programs underlying the development of hematopoietic cell lineages.
105 he effects of anti-cancer agents on multiple hematopoietic cell lineages.
106 the different DDR outcomes utilized by human hematopoietic cell lines and primary human HSPC.
107 and compared the replication of the virus in hematopoietic cell lines with that of FeLV-A 61E by meas
108 ression pattern of metabolic pathways of two hematopoietic cell lines, we find that the relative posi
109 cytotoxic agents in C57BL/6 mice in vivo and hematopoietic cell lines.
110 erotic lesion formation, which suggests that hematopoietic cells mediate the proatherosclerotic pheno
111 DOCK8, a guanine exchange factor involved in hematopoietic cell migration, lead to immunodeficiency a
112                 We used both an experimental hematopoietic cell model of latency and cells from natur
113        Gene set enrichment analysis predicts hematopoietic cell number and cell infiltration are modu
114                         Here we show that in hematopoietic cells, Nup98 binds predominantly to transc
115                                              Hematopoietic cells of the marrow are regionally decimat
116 se variants confers a selective advantage to hematopoietic cells on DNA damage.
117 ypes could be complemented by wild-type (WT) hematopoietic cells or administration of exosomes produc
118  immune cells, such as natural killer cells, hematopoietic cells or induced pluripotent stem cells, t
119                                              Hematopoietic-cell or solid-organ transplant recipients
120  maribavir dose-blinded trial, recipients of hematopoietic-cell or solid-organ transplants (>=18 year
121 for clearing CMV viremia among recipients of hematopoietic-cell or solid-organ transplants.
122 fluorescence microscopy were used to analyze hematopoietic cell population dynamics and the cellular
123              Immune pathway heterogeneity of hematopoietic cell populations in breast tumors correspo
124 onstrate that ectopic expression of Snai1 in hematopoietic cells predisposes mice to AML development.
125 n GPS are known to be synthesized outside of hematopoietic cells, predominantly in the liver.
126 at Fbxl8 antagonizes cell cycle progression, hematopoietic cell proliferation, and oncogene-induced t
127 ecular bone and collagen fibers that replace hematopoietic cells, resulting in abnormal bone marrow f
128 ted by RIPK1, RIPK3, and MLKL kinases but in hematopoietic cells RIPK1 has anti-inflammatory roles an
129  single-cell gene-activity profiles of human hematopoietic cells showed that LTA induces genes restri
130 ics by dedicator of cytokinesis 2 (DOCK2), a hematopoietic cell-specific actin effector protein, has
131                                              Hematopoietic cell-specific deletion of PTN suppressed C
132 etic system, we generated a mouse model with hematopoietic cell-specific knockout of Hopx (Hopx(-/-))
133 se these cells express the cortactin homolog hematopoietic cell-specific lyn substrate-1.
134 to blood cell phenotypes, delineate relevant hematopoietic cell states influenced by regulatory genet
135 ctional expression of cortactin in different hematopoietic cells, such as macrophages, dendritic cell
136 the importance of cytokine-directed cues for hematopoietic cell survival and differentiation, the imp
137 stem cells, but not in normal CD34(+)CD38(-) hematopoietic cells, T cells, or vital tissues.
138                 Clonal expansions of mutated hematopoietic cells, termed clonal hematopoiesis, are co
139 ts a specific, nonredundant role for RAC2 in hematopoietic cells that discriminates RAC2 from the rel
140 ed a conditioning regimen, infusion of donor hematopoietic cells, then immunosuppressive drugs and an
141 particles conjugated to the surface of donor hematopoietic cells to enhance their proliferation kinet
142  a pooled loss-of-function screen in primary hematopoietic cells to interrogate 389 candidate genes c
143 on factor in the terminal differentiation of hematopoietic cells to the monocytes has been well estab
144 tal stage in which HCMV infects, HCMV drives hematopoietic cells towards a weaker immune-responsive m
145           HE cells undergo an endothelial to hematopoietic cell transition, giving rise to HSPCs that
146 a significant complication facing allogeneic hematopoietic cell transplant (allo-HCT) recipients, as
147 use of morbidity and mortality in allogeneic hematopoietic cell transplant (allo-HCT) recipients.
148 tic cell transplant (autoHCT) and allogeneic hematopoietic cell transplant (alloHCT).
149 yeloid leukemia (AML), as well as autologous hematopoietic cell transplant (autoHCT) and allogeneic h
150                                              Hematopoietic cell transplant (HCT) and solid organ tran
151 eiving intensive chemotherapy and the use of hematopoietic cell transplant (HCT) for specific high-ri
152                                         Only hematopoietic cell transplant (HCT) has been shown to ha
153 ary graft-versus-host disease (cpGVHD) after hematopoietic cell transplant (HCT) manifests as progres
154                                              Hematopoietic cell transplant (HCT) recipients are frequ
155 rgies among solid organ transplant (SOT) and hematopoietic cell transplant (HCT) recipients are limit
156  guidelines recommend vaccinating allogeneic hematopoietic cell transplant (HCT) recipients at 3 mont
157  guidelines recommend vaccinating allogeneic hematopoietic cell transplant (HCT) recipients at 3 mont
158 and preventing disseminated viral disease in hematopoietic cell transplant (HCT) recipients but does
159          Cytomegalovirus (CMV) infections in hematopoietic cell transplant (HCT) recipients cause sub
160 solized RBV, we aimed to compare outcomes of hematopoietic cell transplant (HCT) recipients treated w
161 sample types, including (i) whole blood from hematopoietic cell transplant (HCT) recipients with and
162  associated with substantial morbidity among hematopoietic cell transplant (HCT) recipients, but the
163 sk patients with hematologic malignancies or hematopoietic cell transplant (HCT) recipients.
164 rding the impact of human bocavirus (BoV) in hematopoietic cell transplant (HCT) recipients.
165  is associated with significant mortality in hematopoietic cell transplant (HCT) recipients.
166 V) infection causes significant morbidity in hematopoietic cell transplant (HCT) recipients.
167 Seq) test for diagnosing pulmonary IMI after hematopoietic cell transplant (HCT).
168 h mortality in ex vivo T-cell depleted (TCD) hematopoietic cell transplant (HCT).
169 elapse mortality (NRM) after unrelated donor hematopoietic cell transplant (HCT).
170 s of allogeneic (Allo) and autologous (Auto) hematopoietic cell transplant and CD19-directed chimeric
171 3 consecutive young males with cALD prior to hematopoietic cell transplant and its association with m
172 tiocytosis, congenital immunodeficiency, and hematopoietic cell transplant are independently associat
173                                              Hematopoietic cell transplant candidates and recipients
174 l infection within 100 days after allogeneic hematopoietic cell transplant increases risk of pulmonar
175 st disease (GVHD) occurring after allogeneic hematopoietic cell transplant is an allo-reactive T cell
176 med a retrospective review of 738 allogeneic hematopoietic cell transplant patients enrolled from 200
177 se of infectious complications in allogeneic hematopoietic cell transplant recipients (alloHCT).
178                         It is widely used in hematopoietic cell transplant recipients but is infreque
179 ains a major complication in solid organ and hematopoietic cell transplant recipients.
180 totoxic chemotherapy regimens and allogeneic hematopoietic cell transplant to achieve the best long-t
181 y immunocompromised diagnosis and history of hematopoietic cell transplant, and among survivors immun
182                       Among patients without hematopoietic cell transplant, congenital immunodeficien
183                    Among patients with prior hematopoietic cell transplant, liquid malignancy (adjust
184  of morbidity and mortality after allogeneic hematopoietic cell transplant.
185 lood specimens from recipients of allogeneic hematopoietic cell transplant.
186                                   Autologous hematopoietic cell transplantation (AHCT) consolidation
187 e-cycle melphalan 200 mg/m(2) and autologous hematopoietic cell transplantation (AHCT) followed by le
188                                   Allogeneic hematopoietic cell transplantation (allo-HCT) can cure t
189                          Although allogeneic hematopoietic cell transplantation (allo-HCT) is current
190                                   Allogeneic hematopoietic cell transplantation (allo-HCT) is indicat
191  into the intestinal mucosa after allogeneic hematopoietic cell transplantation (allo-HCT) triggers n
192  cells from a healthy individual (allogeneic hematopoietic cell transplantation (allo-HCT)) demonstra
193          In patients who received allogeneic hematopoietic cell transplantation (allo-HCT), we descri
194 which is the main complication of allogeneic hematopoietic cell transplantation (allo-HCT).
195 sus-host disease (GVHD) following allogeneic hematopoietic cell transplantation (allo-HCT).
196 fe-threatening complication after allogeneic hematopoietic cell transplantation (allo-HCT).
197  of morbidity and mortality after allogeneic hematopoietic cell transplantation (allo-HCT).
198 ntensive chemotherapy followed by allogeneic hematopoietic cell transplantation (alloHCT) and single-
199                                   Allogeneic hematopoietic cell transplantation (alloHCT) benefits in
200  graft-vs-tumor effects following allogeneic hematopoietic cell transplantation (alloHCT), but retros
201 in at risk for relapse even after allogeneic hematopoietic cell transplantation (alloHCT).
202 ory DLBCL who were ineligible for autologous hematopoietic cell transplantation (auto-HCT) or who had
203 depletion regimen (HR, 0.25), and allogeneic hematopoietic cell transplantation (HCT) after CAR T-cel
204 tranded DNA (dsDNA) viruses after allogeneic hematopoietic cell transplantation (HCT) are limited by
205                                         Post hematopoietic cell transplantation (HCT) autoimmune cyto
206                                   Allogeneic hematopoietic cell transplantation (HCT) benefits childr
207 er insight into humoral viral immunity after hematopoietic cell transplantation (HCT) could have pote
208 ematologic malignancies and those undergoing hematopoietic cell transplantation (HCT) face a complex
209 ary immunodeficiencies undergoing allogeneic hematopoietic cell transplantation (HCT) for difficult-t
210 ite improvements, mortality after allogeneic hematopoietic cell transplantation (HCT) for nonmalignan
211 st disease (GVHD) is higher after allogeneic hematopoietic cell transplantation (HCT) from unrelated
212                                              Hematopoietic cell transplantation (HCT) has been consid
213                                              Hematopoietic cell transplantation (HCT) has now been sh
214 ltivariate Cox model for OS using allogeneic hematopoietic cell transplantation (HCT) in first comple
215  nonrelapse mortality (NRM) after allogeneic hematopoietic cell transplantation (HCT) in the pediatri
216                                              Hematopoietic cell transplantation (HCT) is curative for
217 rimary cytomegalovirus (CMV) infection after hematopoietic cell transplantation (HCT) is poorly under
218                     Relapse after allogeneic hematopoietic cell transplantation (HCT) is the leading
219 gnificant morbidity and early mortality, and hematopoietic cell transplantation (HCT) is the only wid
220                                              Hematopoietic cell transplantation (HCT) is the primary
221 C) transfusion practices and their impact on hematopoietic cell transplantation (HCT) outcomes are po
222  psychological distress during their child's hematopoietic cell transplantation (HCT) process.
223 prophylaxis after matched-related allogeneic hematopoietic cell transplantation (HCT) recently showed
224 sing polymerase chain reaction in allogeneic hematopoietic cell transplantation (HCT) recipients who
225 eria are associated with adverse outcomes in hematopoietic cell transplantation (HCT) recipients.
226  (CMV) is the most common viral infection in hematopoietic cell transplantation (HCT) recipients.
227 ersus placebo in CMV-seropositive allogeneic hematopoietic cell transplantation (HCT) recipients.
228                                              Hematopoietic cell transplantation (HCT) remains the onl
229 am infection (BSI) on outcomes of allogeneic hematopoietic cell transplantation (HCT) utilizing the C
230 been effective in preventing acute GvHD post hematopoietic cell transplantation (HCT), its efficacy a
231 (GvHD) is a major complication of allogeneic hematopoietic cell transplantation (HCT), mediated prima
232 rent treatment options, including allogeneic hematopoietic cell transplantation (HCT), often fail to
233           In patients who undergo allogeneic hematopoietic cell transplantation (HCT), post-HCT clone
234 -6B) frequently reactivates after allogeneic hematopoietic cell transplantation (HCT).
235 d nonmyeloablative conditioning regimens for hematopoietic cell transplantation (HCT).
236 e (aGVHD) hinders the efficacy of allogeneic hematopoietic cell transplantation (HCT).
237 nked to poor outcomes in patients undergoing hematopoietic cell transplantation (HCT).
238  has had a marked impact on human allogeneic hematopoietic cell transplantation (HCT).
239 ll-recognized after myeloablative allogeneic hematopoietic cell transplantation (HCT).
240 Ss) are life-threatening complications after hematopoietic cell transplantation (HCT).
241 onditioning for HLA class I or II mismatched hematopoietic cell transplantation (HCT).
242  with symptomatic hemorrhagic cystitis after hematopoietic cell transplantation (HCT).
243 , 113 patients with ALL underwent allogeneic hematopoietic cell transplantation (HCT); frequency of S
244  major barrier to engraftment after in utero hematopoietic cell transplantation (IUHCT).
245          Strategies to enhance the safety of hematopoietic cell transplantation and to improve patien
246                                   Allogeneic hematopoietic cell transplantation at the time of second
247                                              Hematopoietic cell transplantation can correct hematolog
248                    The success of allogeneic hematopoietic cell transplantation depends heavily on th
249  depletion of donor CD4+ T cells early after hematopoietic cell transplantation effectively prevents
250 e is a major cause of death after allogeneic hematopoietic cell transplantation for acute leukemias.
251 t event-free survival (EFS) after allogeneic hematopoietic cell transplantation for sickle cell disea
252 s in a cohort of 2531 patients who underwent hematopoietic cell transplantation from 2010 to 2015 aft
253 versus-host disease (aGVHD) after allogeneic hematopoietic cell transplantation have poor prognosis,
254                                   Allogeneic hematopoietic cell transplantation involves consideratio
255                                   Allogeneic hematopoietic cell transplantation is indicated for refr
256 re previously treated with either autologous hematopoietic cell transplantation or two or more prior
257 sociated with higher mortality in allogeneic hematopoietic cell transplantation recipients with LRTD.
258 t disease (GvHD) is a common complication of hematopoietic cell transplantation that negatively impac
259 (GVHD) is a major complication of allogeneic hematopoietic cell transplantation that resembles autoim
260 induced hematopoietic syndrome is allogeneic hematopoietic cell transplantation, a therapy unavailabl
261 analysis of a nonhematologic neoplasm, after hematopoietic cell transplantation, or as a result of ge
262 d improved survival in a model of allogeneic hematopoietic cell transplantation, providing the ration
263 national Histocompatibility Working Group in Hematopoietic Cell Transplantation.
264 s, with and without censoring for allogeneic hematopoietic cell transplantation.
265 herapy regimens if ineligible for autologous hematopoietic cell transplantation.
266  the most serious complication of allogeneic hematopoietic cell transplantation.
267  three additional courses of chemotherapy or hematopoietic cell transplantation.
268  of morbidity and mortality after allogeneic hematopoietic cell transplantation.
269  life-threatening complication of allogeneic hematopoietic cell transplantation.
270  reduce the risk of relapse after allogeneic hematopoietic cell transplantation.
271 lly validating this model in unrelated donor hematopoietic cell transplantation.
272 iders who care for patients after allogeneic hematopoietic cell transplantation.
273 inib use, but its cytomegalovirus risk after hematopoietic-cell transplantation (HCT) is not known.
274 and its complications in adult patients with hematopoietic cell transplants (HCT), cancer (hematologi
275 ociated complications in adult patients with hematopoietic cell transplants (HCT), cancer, human immu
276                                           In hematopoietic cell transplants, alloreactive T cells med
277 atment, these responder patients, as well as hematopoietic cells treated with FeCl(3) and Deferasirox
278 n-regulation of MHC-I on a small fraction of hematopoietic cells triggered missing-self reactivity.
279 crophages (TAMs) represent the most abundant hematopoietic cell type in the solid tumor microenvironm
280 -Class to analyze 32 datasets from different hematopoietic cell types from the Blueprint Epigenome pr
281  the regulatory landscape of differentiating hematopoietic cell types in mouse.
282 u.1 regulation from those described in other hematopoietic cell types such as T cells which down-regu
283 45(+) AGM cells revealed predominance of two hematopoietic cell types, mannose-receptor positive macr
284 c stem cell niche and are thought to protect hematopoietic cells under stress.
285 ound that constitutive MDR1 expression among hematopoietic cells was observed in cytolytic lymphocyte
286 RA in nonhematopoietic cells, but not in the hematopoietic cells, was required for the development of
287 n in thymic epithelial cells (TECs), but not hematopoietic cells, was sufficient for complete deletio
288  molecular characteristics of the developing hematopoietic cells, we lack a complete understanding of
289  transcriptomic and metabolomic profiling of hematopoietic cells, we reveal that EVI1 overexpression
290 era experiments showed that CD137L-deficient hematopoietic cells were able to confer T1D resistance.
291                             Stromal, but not hematopoietic, cells were the essential source of Notch
292 ion mostly depends upon Notch3 expression in hematopoietic cells, which coincides with an enhanced pr
293       The Y chromosome is frequently lost in hematopoietic cells, which represents the most common so
294                                 Non-leukemic hematopoietic cells with DNMT3A(R882H) displayed focal m
295 tential (CHIP), the age-related expansion of hematopoietic cells with leukemogenic mutations without
296 t ZIKV infection was particularly evident in hematopoietic cells with microglia, the brain-resident m
297 NA in humans showed that clonal expansion of hematopoietic cells with somatic mutations in leukemogen
298 infected fibroblasts, endothelial cells, and hematopoietic cells, with some infected cells able to tr
299 ned by the continuous interactions of mobile hematopoietic cells within specialized microenvironments
300   Constitutive activation of beta-catenin in hematopoietic cells yielded lethal myeloid disease in a

 
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