コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 ive mobility rehabilitation intervention for hemianopia.
2 ing the efficacy of prismatic treatments for hemianopia.
3 e could be visualized at longer durations of hemianopia.
4 All participants had homonymous hemianopia.
5 neuritis, and residual vision in homonymous hemianopia.
6 w pointed to the side contralateral to their hemianopia.
7 effective in restoring visual sensitivity in hemianopia.
8 ifically are critical for residual vision in hemianopia.
9 ere shifted contralaterally in patients with hemianopia alone and ipsilaterally in patients with hemi
14 training, shown to improve visual fields in hemianopia and optic nerve damage, might comprise such a
16 maging, with particular reference to work on hemianopia and the controversial suggestion of cortical
18 ients' test times were 40 seconds longer for hemianopias and 90 seconds longer for optic neuropathies
19 n the first SITA standard test by 0.82 dB in hemianopias and by 0.71 dB in optic neuropathy patients.
20 Examination demonstrated a right homonymous hemianopia, and magnetic resonance imaging revealed an e
21 unction in hemianopia.SIGNIFICANCE STATEMENT Hemianopia (blindness on one side of space) is widely co
22 patients with an incomplete right homonymous hemianopia, but no reading deficit, viewed single words
23 ted in patients with severe right homonymous hemianopia, commensurate with their inability to perform
25 eaders and have shown how a right homonymous hemianopia disrupts the motor preparation of reading sac
27 e 18 years or older with complete homonymous hemianopia for at least 3 months and without visual negl
33 in a group of seven patients with homonymous hemianopia; here a negative straight line relationship w
35 designed to examine the effect of homonymous hemianopia (HH) on detection of pedestrian figures in mu
36 ight patients with stroke-induced homonymous hemianopia (HH) were randomized into 2 training arms: in
37 The hypothesis that drivers with homonymous hemianopia (HH) would take a lane position that increase
40 al nerve fibre loss in congenital homonymous hemianopia in humans is well recognized from clinical ob
41 with concentric peripheral defect, temporal hemianopia, inferotemporal defect, near total loss, supe
43 of the OT occurs in acquired and congenital hemianopia, is correlated with visual field loss, and is
45 age to the primary visual cortex (V1) causes hemianopia, many patients retain some residual vision; k
47 sic symptoms (eg, motor weakness, dysphasia, hemianopia, monocular visual loss) but no consensus on s
48 ith left hemianopia, two patients with right hemianopia, nine patients with left hemi-neglect and nin
49 age >18 years at time of testing; homonymous hemianopia; no other ophthalmic or neurological disorder
50 ogic examination showed mild left homonymous hemianopia, normal gait with no cerebellar signs, and pr
54 Nine study participants (15%) demonstrated hemianopia or quadrantanopia, and an additional 36% had
55 Sensitivities were higher in patients with hemianopias or optic neuropathies using SITA standard co
57 t for the optic neuropathy patients; for the hemianopia patients the difference in values were betwee
60 seen with FDT testing in some patients with hemianopia, probably due to light scatter across the ver
61 ed, resulting in a complete right homonymous hemianopia, rightward saccades during text reading are d
63 tive method for restoring visual function in hemianopia.SIGNIFICANCE STATEMENT Hemianopia (blindness
64 row pointed to the side ipsilateral to their hemianopia than they did when the arrow pointed to the s
65 tween the Matrix and SAP in the detection of hemianopias, the sensitivity of SAP was higher, probably
66 ng line bisection in five patients with left hemianopia, two patients with right hemianopia, nine pat
68 nts with a relative homonymous or bitemporal hemianopia were tested with both conventional perimetry
70 re alexia ("alexia without agraphia") and no hemianopia, who read words slowly using a letter-by-lett