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1 -directed alertness in patients with chronic hemispatial neglect.
2 spheric asymmetries similar to those seen in hemispatial neglect.
3 king and contributes to our understanding of hemispatial neglect.
4 estigating spatial-processing impairments in hemispatial neglect.
5 been introduced as a promising treatment for hemispatial neglect.
6 he debilitating attention deficits common to hemispatial neglect.
7 to those observed in patients suffering from hemispatial neglect.
8 t temporo-parietal cortex commonly result in hemispatial neglect.
9 as well as those of the clinical syndrome of hemispatial neglect.
10 nts without cortical hypoperfusion showed no hemispatial neglect.
11  the visual abilities of human patients with hemispatial neglect.
12 g (PWI), and detailed testing for aphasia or hemispatial neglect.
13 ents with schizophrenia, cerebral palsy, and hemispatial neglect.
14 tern is consistent with data from those with hemispatial neglect after stroke and raises the possibil
15      The site of lesion responsible for left hemispatial neglect after stroke has been intensely deba
16 ine-scale functional architecture underlying hemispatial neglect, an attentional deficit caused by pa
17     Lesions of parietal cortex often lead to hemispatial neglect, an impairment of choices of targets
18 research challenges previous explanations of hemispatial neglect and enhances our understanding of ho
19 te right subcortical infarcts using tests of hemispatial neglect and magnetic resonance diffusion-wei
20 l attention deficits in stroke patients with hemispatial neglect are correlated at 2 weeks postonset
21 udy, we test the hypothesis that recovery of hemispatial neglect correlates with a return of network
22 e hypothesized that most cases of aphasia or hemispatial neglect due to acute, subcortical infarct ca
23 ected attention and recovery from multimodal hemispatial neglect following cortical lesions of medial
24                                              Hemispatial neglect following right-hemisphere stroke is
25 attention-deficit hyperactivity disorder and hemispatial neglect following right-hemisphere stroke.
26 rch and selective attention in patients with hemispatial neglect following stroke.
27 ssessments of right-hemisphere patients with hemispatial neglect have revealed significant overall de
28 ent prospects of neurological disorders like hemispatial neglect, hemianopia and other deficits after
29 ispheric effects of spatial attention (e.g., hemispatial neglect, hemifield capacity limits) are well
30                                              Hemispatial Neglect (HN) is a failure to allocate attent
31 schizophrenia suggest a subtle form of right hemispatial neglect in acutely ill patients but not in c
32 rotigotine would have a beneficial effect on hemispatial neglect in stroke patients.
33 d in directed attention and its dysfunction, hemispatial neglect, in the rat.
34 e that sometimes follows brain injury called hemispatial neglect, in which patients exhibit attention
35 strate that abnormal network connectivity in hemispatial neglect is behaviorally relevant.
36 calable, and self-administered treatment for hemispatial neglect might serve as a useful addition to
37  environment and, with that facility lost in hemispatial neglect, search becomes uselessly stuck with
38  can cause patients to ignore half of space (hemispatial neglect syndrome), but only for right (not l
39 wed no difference in performance on tests of hemispatial neglect, volume of infarct or sex distributi
40          In bedside testing of patients with hemispatial neglect, we have found that extinction for c
41 we present a case series of 14 patients with hemispatial neglect who, when asked to clap, repeatedly
42                 Forty-nine participants with hemispatial neglect, who demonstrated significant spatia