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1 ce interval: 0.80 to 1.3]; p = 0.93) over no hemoconcentration.
2 emia was associated with a small increase in hemoconcentration.
3  tertile were considered to have evidence of hemoconcentration.
4             It also reversed hypotension and hemoconcentration.
5 the increase in plasma lactate and prevented hemoconcentration.
6 e elevation of plasma lactate, and prevented hemoconcentration.
7 jury, with associated cardiac depression and hemoconcentration.
8 ction, and increases blood viscosity through hemoconcentration.
9  in catecholamine levels, platelet count and hemoconcentration.
10                                  PAF-induced hemoconcentration and arachidonic acid- and TPA-induced
11      Combining 2 indicators of decongestion, hemoconcentration and diuretic response improves risk pr
12 rized by transient and severe but reversible hemoconcentration and hypoalbuminemia caused by leakage
13 asation and vascular collapse accompanied by hemoconcentration and hypoalbuminemia.
14 minal pro-B-type natriuretic peptide levels, hemoconcentration, and clinical congestion score after 1
15 , such as diuretic efficiency, fluid output, hemoconcentration, and diuretic dose.
16 h in turn leads to hypotensive shock, severe hemoconcentration, and hypoalbuminemia.
17 pressure, oxygen extraction and consumption, hemoconcentration, and levels of TNF and eicosanoids.
18 gh bicarbonate, poor diuretic response, less hemoconcentration, and worsening heart failure (all P<0.
19                   Both diuretic response and hemoconcentration are indicators of decongestion and hav
20 then further dichotomized into early or late hemoconcentration by using the midway point of the hospi
21 ized by a rapidly evolving massive anasarca, hemoconcentration, cardiac dysfunction, multiple system
22  of furosemide on day 4 after admission) and hemoconcentration (change in hemoglobin at discharge or
23 atients with favorable diuretic response and hemoconcentration compared with all other patients.
24                                         Late hemoconcentration conferred a significant survival advan
25          Cardiovascular changes and profound hemoconcentration developed within 6 hrs from the start
26 zed by recurrent episodes of hypotension and hemoconcentration due to sudden transient extravasation
27 icating a reduction in intravascular volume, hemoconcentration during the treatment of decompensated
28 splayed both favorable diuretic response and hemoconcentration had a markedly lower risk of rehospita
29 nts with late hemoconcentration versus early hemoconcentration had higher average daily loop diuretic
30                                Patients with hemoconcentration had significantly lower 180-day mortal
31              Patients with late versus early hemoconcentration had similar baseline characteristics,
32 , hyperlipidemia, hypertension, hypotension, hemoconcentration, hemodilution, and hypercoagulable sta
33                      ALT was associated with hemoconcentration, hypotension, and circulatory collapse
34 venous pressures (p </= .007); indicators of hemoconcentration (i.e., sodium [p = .0004], albumin [p
35 s study sought to determine if the timing of hemoconcentration influences associated survival.
36                   Despite this relationship, hemoconcentration is associated with substantially impro
37                                              Hemoconcentration is significantly associated with measu
38                                              Hemoconcentration occurred in 422 (49.9%) patients (41.5
39                                         Only hemoconcentration occurring late in the hospitalization
40 rs was associated with a higher incidence of hemoconcentration (odds ratio, 3.1; P=0.015), and parado
41 al: 0.59 to 0.93]; p = 0.009), whereas early hemoconcentration offered no significant mortality benef
42 al dose diuretics was lower in patients with hemoconcentration (p < 0.01).
43 gs included tachycardia, hypotension, edema, hemoconcentration, profound leukocytosis, and absence of
44                       The group experiencing hemoconcentration received higher doses of loop diuretic
45 ilarly identify diuretic responsiveness, but hemoconcentration this early would not indicate euvolemi
46 the value of combining diuretic response and hemoconcentration to better predict patients at low risk
47                  However, patients with late hemoconcentration versus early hemoconcentration had hig
48                                              Hemoconcentration was defined as an increase in both hem
49                                              Hemoconcentration was mainly associated with better rena
50                                              Hemoconcentration was strongly associated with worsening
51 of participants experienced findings such as hemoconcentration, weight loss, reduction in amino termi
52                             Leukocytosis and hemoconcentration were noted in both cases prior to deat
53                         We hypothesized that hemoconcentration would be associated with worsening ren