ライフサイエンスコーパス: hemorrhage

1 lial/endothelial cell dysfunction (edema and hemorrhage).

2 of significant subinternal limiting membrane hemorrhage).

3 ubarachnoid hemorrhage with intraventricular hemorrhage).

4 cluding patients diagnosed with subarachnoid hemorrhage).

5 in ischemic stroke and 42% in intracerebral hemorrhage.

6 ral microbleeds (CMBs) as a marker of occult hemorrhage.

7 and treatment of the underlying mechanism of hemorrhage.

8 d with long-term outcome after intracerebral hemorrhage.

9 less, CNS blood vessels were intact, without hemorrhage.

10 LFL was observed as an evolving retinal hemorrhage.

11 ents, 88 lobar, and 40 deep intraparenchymal hemorrhage.

12 ssociated hemolysis, or after a subarachnoid hemorrhage.

13 olar wall rupture may be sufficient to cause hemorrhage.

14 odalities for treatment of diabetic vitreous hemorrhage.

15 age in patients with aneurysmal subarachnoid hemorrhage.

16 are patients with hypofibrinogenemia-induced hemorrhage.

17 by 2 months of age primarily due to internal hemorrhage.

18 ar blood vessels that often lead to cerebral hemorrhage.

19 dence in the initial evaluation of suspected hemorrhage.

20 or pre-retinal hemorrhage, and mild vitreous hemorrhage.

21 schemic stroke, and 2,974 with intracerebral hemorrhage.

22 IVA was given with complete clearance of the hemorrhage.

23 ularizations elsewhere (NVEs) and preretinal hemorrhage.

24 icted poor outcome in lobar intraparenchymal hemorrhage.

25 gow Outcome Scale) in lobar intraparenchymal hemorrhage.

26 es, and 4% (95% CI, 0.01-0.1%) intracerebral hemorrhage.

27 t be assessed in patients with intracerebral hemorrhage.

28 are common vascular anomalies causing brain hemorrhage.

29 ing 53 with sepsis and 59 with intracerebral hemorrhage.

30 hospitalization for bleeding or intracranial hemorrhage.

31 hance associations with disease severity and hemorrhage.

32 sculature, which result in aneurysms and eye hemorrhages.

33 Twelve of the RCNs were active with exudates/hemorrhages.

34 nocytes was found in 39 (93%) white-centered hemorrhages.

35 patients with thick versus thin subarachnoid hemorrhage (1.92 vs 1.99 mg/dL; p = 0.022).

36 ied Fisher scale (grades: 0, no radiographic hemorrhage; 1, thin [< 1 mm in depth] subarachnoid hemor

37 of gastrointestinal bleeding or intracranial hemorrhage (12.9 per 1000 person-years) compared with th

38 7]; P = .29), or progression of intracranial hemorrhage (16% vs 20%; difference, -5.4% [95% CI, -12.8

39 hage; 1, thin [< 1 mm in depth] subarachnoid hemorrhage; 2, thin subarachnoid hemorrhage with intrave

40 Subarachnoid hemorrhage (20%) was the most common type of intracrania

41 Within 1 year of intracerebral hemorrhage, 224 (56%) of 402 patients died.

42 brain injury (44%), followed by intracranial hemorrhage (24%), and ischemic infarct (16%).

43 ; P = 0.003), higher proportion with macular hemorrhage (25.5% vs. 13.2%; P = 0.014), and fewer anti-

44 atitis (PEP) (16.1% vs. 6.4%, p = 0.17), and hemorrhage (3.2% vs. 8.5%, p = 0.35).

45 (1.1%), ischemic stroke (1.9%), intracranial hemorrhage (3.5%), and brain death (1.6%).

46 ubarachnoid hemorrhage with intraventricular hemorrhage; 3, thick [>= 1 mm] subarachnoid hemorrhage;

47 , subretinal hemorrhage (51%), and choroidal hemorrhage (30%).

48 intracerebral hemorrhage (8%), and subdural hemorrhage (4%).

49 hemorrhage; 3, thick [>= 1 mm] subarachnoid hemorrhage; 4, thick subarachnoid hemorrhage with intrav

50 auma (62%), lens expulsion (54%), subretinal hemorrhage (51%), and choroidal hemorrhage (30%).

51 In contrast, rates of intracerebral hemorrhage (6% vs 8%; p = 0.35) did not differ.

52 -5.6% to 11.0%]) or symptomatic intracranial hemorrhage (7 [4.7%] vs 2 [1.3%]; unadjusted risk differ

53 unadjusted risk for symptomatic intracranial hemorrhage (7.7% versus 4.8%) and in-hospital mortality

54 patients [100%] vs 12 of 17 patients without hemorrhage [71%], P = .01) and development of the acute

55 ranial hemorrhage, followed by intracerebral hemorrhage (8%), and subdural hemorrhage (4%).

56 .9% vs 30.6%, P = 0.400), postpancreatectomy hemorrhage (9.1% vs 5.1%, P = 0.352), delayed gastric em

57 le performing vitrectomy for severe vitreous hemorrhage, a point of strong adherence between a old he

58 intracranial aneurysm leads to subarachnoid hemorrhage, a severe type of stroke.

59 table/potentially preventable (P/PP) deaths, hemorrhage accounted for 55.1%.

60 sed probability of symptomatic intracerebral hemorrhage (adjusted OR per 30 minutes increase in time

61 y perfusion and reduced intestinal edema and hemorrhage after BD.

62 subhyaloid, outer plexiform, and subretinal hemorrhages after 2 minutes of chest compressions.

63 of a retinal tear, pre-retinal and vitreous hemorrhages after completing a session of whole-body vib

64 eatic fistula, bile leak, postpancreatectomy hemorrhage (all ISGPS grade B/C), severe complications (

65 ing 53 with sepsis and 59 with intracerebral hemorrhage, along with 53 control participants.

66 tricular tachycardia, upper gastrointestinal hemorrhage, anaphylactic reaction, acute kidney injury,

67 between asymptomatic neonates with subdural hemorrhage and control neonates.

68 Ripk3 deficiency reduces cerebral hemorrhage and delays the onset of neural damage mediate

69 with the occurrence of side effects such as hemorrhage and edema.

70 of hepatic decompensation (ascites, variceal hemorrhage and hepatic encephalopathy), which defines th

71 TIC exacerbates hemorrhage and is associated with higher morbidity and m

72 ment of all-cause mortality of intracerebral hemorrhage and ischemic stroke patients admitted to the

73 Patients with lobar intraparenchymal hemorrhage and lateralized rhythmic delta activity were

74 ly activates necroptosis to promote cerebral hemorrhage and neuroinflammation.

75 e, a point of strong adherence between a old hemorrhage and retinal surface was identified and manage

76 y nets, increasing the risk of intracerebral hemorrhage and stroke.

77 At month 6, the presence of hemorrhage and the change in central subfield thickness

78 ied risks of serious side effects, including hemorrhages and cerebrospinal fluid leakage.

79 ression to PDR including the surface area of hemorrhages and the distance of hemorrhages from the ONH

80 g 10 with sepsis and five with intracerebral hemorrhage, and 11 healthy controls.

81 n imaging findings such as ischemic infarct, hemorrhage, and acute hemorrhagic necrotizing encephalop

82 (95% CI = 59-457%) in risk of intracerebral hemorrhage, and an increase in white matter hyperintensi

83 AS), increases the risk of preterm delivery, hemorrhage, and death.

84 greater infarct expansion, increased cardiac hemorrhage, and delayed neutrophil accumulation, which r

85 d inflammation resolution, increased cardiac hemorrhage, and enhanced cardiac dysfunction.

86 tter, indirect hyperbilirubinaemia, cerebral hemorrhage, and mental status change (in two [12%] patie

87 temporal retinal tear, inferior pre-retinal hemorrhage, and mild vitreous hemorrhage.

88 daptive immunity, coagulation abnormalities, hemorrhage, and multiorgan failure with up to 33% case f

89 on and fundus photography were evaluated for hemorrhage, and spectral-domain (SD) OCT images from HAR

90 dilated and sclerotic segments, perivenular hemorrhages, and foci of phlebitis.

91 inding, reduced VE-cad levels, microvascular hemorrhaging, and decreased survival.

92 normal blood cells, predisposing patients to hemorrhage, anemia, and infections.

93 There was no evidence of hemorrhage, anemia, or AVMs in major organs to explain t

94 We conclude that the rates of thrombosis and hemorrhage appear to be similar following hospital disch

95 al extension, and a large proportion of such hemorrhages are likely to be a result of mechanisms othe

96 na, consistent with the finding that retinal hemorrhages are often found in multiple layers of the re

97 disorders (thrombocytopenia, thrombosis and hemorrhage) are risk factors for SARS-CoV-2-associated m

98 tinopathy in the multivariate model included hemorrhage area (odds ratio [OR], 2.63; 95% confidence i

99 aphy session, we classified intraparenchymal hemorrhage as isolated deep (no insular, subarachnoid, s

100 mon phenomenon after aneurysmal subarachnoid hemorrhage (aSAH) and contributes to neurocognitive decl

101 ents with high-grade aneurysmal subarachnoid hemorrhage (aSAH) is only insufficiently displayed by cu

102 sis differed between aneurysmal subarachnoid hemorrhage (aSAH) patients with surgical clipping and en

103 In aneurysmal subarachnoid hemorrhage (aSAH), accurate diagnosis of aneurysm is ess

104 dreaded sequelae of aneurysmal subarachnoid hemorrhage (aSAH), requiring timely intervention with th

105 ter Acute Stroke Due to Spontaneous Cerebral Hemorrhage Associated With Antiplatelet Therapy trial, o

106 perfusion and reducing intestinal edema and hemorrhage associated with BD.

107 m revealed tortuous veins and a flame shaped hemorrhage at 7 o'clock.

108 ble analyses identified presence of vitreous hemorrhage at baseline, increasing age, absence of epire

109 1.9, 14.2); bevacizumab-treated eyes without hemorrhage at month 6 had a mean VALS improvement of 3.2

110 follow-up, aflibercept-treated eyes without hemorrhage at month 6 had a mean VALS improvement of 8.0

111 I: -4.6, 11.0); and observation eyes without hemorrhage at month 8 had a mean VALS improvement of 13.

112 mHg; 95% CI, 1.02-1.12; P = 0.008), and disc hemorrhage at visit 1 (HR, 2.08; 95% CI, 1.07-4.04; P =

113 t study eyes (89% [973/1095]) showed macular hemorrhages at baseline, declining to 31% (319/1042) at

114 pillary alterations; however, intra-alveolar hemorrhages, bacterial deposition, and markers of coagul

115 ge >15 years of age with evidence of truncal hemorrhage below the diaphragm and decision for emergent

116 lts diagnosed with spontaneous intracerebral hemorrhage between June 1, 2010 and May 31, 2013.

117 ative pancreatic fistula, postpancreatectomy hemorrhage, bile leakage, delayed gastric emptying, woun

118 Delirium is common after intracerebral hemorrhage, but severe neurologic deficits may confound

119 Reduced area of hemorrhage by month 6 was observed in 70.7% (116 of 164)

120 nticoagulants were a factor in 3 retrobulbar hemorrhage cases.

121 lusion criteria included other intracerebral hemorrhage causes, anticoagulation, coagulopathy, or imm

122 content, fibrous cap structure, intraplaque hemorrhage), complementing the clinical lesion classific

123 ration before transfer and 34.9% experienced hemorrhage complications.

124 he CT scan, to identify retinal and vitreous hemorrhages consistent with TS.

125 elow the diaphragm and decision for emergent hemorrhage control intervention within 60 minutes of arr

126 a novel strategy to obtain earlier temporary hemorrhage control, supporting cardiac, and cerebral per

127 In 75, REBOA was utilized for temporary hemorrhage control.

128 proved diagnostic performance for predicting hemorrhage-control interventions and mortality compared

129 owing BOP by assessing the extent of surface hemorrhage/contusion, Hematoxylin and Eosin staining, an

130 demonstrated ~ 8.5 psi is the threshold for hemorrhage/contusion, up to 30 exposures.

131 ely collected at different time-points after hemorrhage: days 0-1 (acute); days 2-4 (pre-VSP); days 5

132 ome, in part by reducing BSCB disruption and hemorrhage decreasing cytotoxic neuroinflammation and at

133 indings that would bias toward PPV (vitreous hemorrhage, dense cataract, proliferative vitreoretinopa

134 The origin of blood in glaucoma-related disc hemorrhages (DH) remains unknown.

135 l 47 to 53 consists of 3 main features: deep hemorrhages (DH), venous beading (VB), and intraretinal

136 old girl with bilateral multilayered retinal hemorrhages due to SBS.

137 Improvement in hemorrhage during follow-up was associated with visual a

138 Relative to eyes with hemorrhage during follow-up, aflibercept-treated eyes wi

139 s (modified Fisher Scale (mFS), Subarachnoid Hemorrhage Early Brain Edema Score) (P < 0.05).

140 al hemorrhage (sICH), and major extracranial hemorrhage (ECH) within 90 days.

141 with both ischemic stroke and intracerebral hemorrhage, especially compared with other populations o

142 ive Surgery Plus Alteplase for Intracerebral Hemorrhage Evacuation Phase III trial.

143 strain caused high mortality, intra-alveolar hemorrhages, extensive alveolar septal sequestration of

144 Greater tumor thickness, vitreous hemorrhage, exudative retinal detachment, and poor basel

145 diagnostic brain CT for acute intracerebral hemorrhage features and SVD biomarkers.

146 0%) was the most common type of intracranial hemorrhage, followed by intracerebral hemorrhage (8%), a

147 thalmitis, retinal detachment, and choroidal hemorrhage following EK procedures is low.

148 postoperative endophthalmitis and choroidal hemorrhage following EK was 0.03% and 0.05%, respectivel

149 ent is the third documented case of vitreous hemorrhage following whole-body vibration training, he i

150 ascular dysregulation may contribute to disc hemorrhage formation in ocular hypertension.

151 ifferentiation of small foci of intracranial hemorrhage from calcium and improved diagnostic accuracy

152 ifferentiation of small foci of intracranial hemorrhage from calcium.

153 Vitreous hemorrhage from proliferative diabetic retinopathy can c

154 g 205 adults with vison loss due to vitreous hemorrhage from proliferative diabetic retinopathy who w

155 Among participants whose eyes had vitreous hemorrhage from proliferative diabetic retinopathy, ther

156 Acer2 deficiency causes hemorrhages from the maternal vasculature in the junctio

157 al [CI], 1.25-5.53), and greater distance of hemorrhages from the ONH (OR, 1.24; 95% CI, 0.97-1.59).

158 face area of hemorrhages and the distance of hemorrhages from the ONH.

159 placenta previa/abuptio placenta/ante-partum hemorrhage, further significant factors, more importantl

160 ithout TS, those with TS had a higher Fisher Hemorrhage Grade and a lower mean (+/-standard deviation

161 All individual DR lesions (hemorrhage [H], microaneurysm [ma], cotton wool spot [CW

162 ve pancreatic fistula and postpancreatectomy hemorrhage had the greatest independent impact on mortal

163 Six weeks later, retinal hemorrhages had substantially resolved, and there was op

164 eosinophilic pneumonia and diffuse alveolar hemorrhage, have also been reported.

165 Compared with the reference group, vitreous hemorrhage (hazard ratio, 2.53 [P < 0.001] and 2.80 [P =

166 : 1.09 to 1.46; p = 0.0017) and intracranial hemorrhage (HR: 1.30; 95% CI: 1.07 to 1.59; p = 0.0094).

167 Participants at increased risk for variceal hemorrhage (HVPG >=12 mm Hg) had a higher mean SHAPE gra

168 Rates of secondary intraventricular hemorrhage, hydrocephalus, and thromboembolic events wer

169 Intracerebral hemorrhage (ICH) accounts for a disproportionate amount

170 for supratentorial spontaneous intracerebral hemorrhage (ICH) and whether it is modified by key basel

171 MCE), ischemic stroke (IS) and intracerebral hemorrhage (ICH) in a cohort of Chinese adults.

172 Intracerebral hemorrhage (ICH) is a devastating form of stroke affecti

173 Spontaneous deep intracerebral hemorrhage (ICH) is a devastating subtype of stroke with

174 Intracerebral hemorrhage (ICH) is an especially feared complication in

175 BIs), multiple sclerosis (MS), intracerebral hemorrhage (ICH), and neuromyelitis optica (NMO).

176 cholesterol levels and risk of intracerebral hemorrhage (ICH), but it remains unclear whether this as

177 SCs) transplantation following intracerebral hemorrhage (ICH).

178 nt in patients presenting with intracerebral hemorrhage (ICH).

179 s emerged as a risk factor for intracerebral hemorrhage (ICH).

180 eir anticoagulant effects after intracranial hemorrhage (ICH).

181 el of hematoma clearance after intracerebral hemorrhage [ICH]), and (3) reduced proinflammatory respo

182 g Accelerated Resolution of Intraventricular Hemorrhage III trial and the Minimally Invasive Surgery

183 ion (aDelta: -1.00, P = 0.024), and vitreous hemorrhage in at least 1 eye (aDelta: -1.92, P = 0.021)

184 symptoms and prevention of life-threatening hemorrhage in immune thrombocytopenia (ITP) must be bala

185 ith higher rates of symptomatic intracranial hemorrhage in M2 occlusions only (OR = 4.40; 95% CI = 2.

186 gnesium levels are associated with extent of hemorrhage in patients with aneurysmal subarachnoid hemo

187 the treatment of gastro-esophageal variceal hemorrhage in patients with decompensated cirrhosis (fir

188 ive failure of clots decreases survival from hemorrhage in vivo.

189 s protective without increasing interstitial hemorrhages in the inflamed bowel or other organs.

190 to 30 [death]), progression of intracranial hemorrhage, incidence of seizures, and incidence of thro

191 primary and secondary prevention of cerebral hemorrhage include the treatment of hypertension, reduct

192 athology causing pulmonary consolidation and hemorrhage, increased mortality and specific modificatio

193 ear death and dependence after intracerebral hemorrhage, independent of known predictors of outcome.

194 microvascular obstruction (MVO), myocardial hemorrhage, infarct size, and clinical outcomes, after S

195 ot CFR, were associated with MVO, myocardial hemorrhage, infarct size, and clinical outcomes.

196 plications (i.e., endophthalmitis, choroidal hemorrhage, infectious keratitis, cystoid macular edema

197 e surface structure, presence of intraplaque hemorrhage (IPH), circle of Willis collaterals, and the

198 The efficient resolution of tissue hemorrhage is an important homeostatic function.

199 e findings suggest that intraretinal macular hemorrhage is an important indicator of disease severity

200 Noncompressible truncal hemorrhage is the leading cause of potentially preventab

201 rtant neurologic complications: intracranial hemorrhage, ischemic stroke, and/or brain death, as a co

202 cardiopulmonary disease) and uDCD (including hemorrhage, major polytrauma, burns, and poisoning).

203 ve pancreatic fistula and postpancreatectomy hemorrhage may have the greatest impact on in-hospital m

204 r, hypoxia/reoxygenation (H/R), our in vitro hemorrhage model, increased miR-19b expression in human

205 bleeding is the main cause of postoperative hemorrhage, most often no actively bleeding vessel can b

206 tion, contusion, diarrhea, peripheral edema, hemorrhage, muscle spasms, and pneumonia, as well as adv

207 ere irritation (n = 312) and subconjunctival hemorrhage (n = 284).

208 ing conjunctival hemorrhage (n = 5), retinal hemorrhage (n = 4), and vitreous floaters (n = 4).

209 participants (16/21), including conjunctival hemorrhage (n = 5), retinal hemorrhage (n = 4), and vitr

210 ration (n = 17), death (n = 13), retrobulbar hemorrhage (n = 7), optic nerve damage (n = 4), vascular

211 to only 5% in isolated deep intraparenchymal hemorrhage not extending to cortex/insula, subarachnoid,

212 Submacular hemorrhage occurred in 15 patients (36%) who were treatm

213 the control group; symptomatic intracranial hemorrhage occurred in 4.5% of the patients in each grou

214 Asymptomatic intracranial hemorrhage occurred in 51.4% of the patients in the thro

215 zures are frequent in lobar intraparenchymal hemorrhage, occurring in one in six monitored patients,

216 0.77; p = 0.011) and with thick subarachnoid hemorrhage (odds ratio 0.29 per 1 mg/dL increase; 95% CI

217 ter initial PPV were preoperative subretinal hemorrhage (odds ratio [OR], 5.73; P = 0.03), PVR found

218 r cohort presenting with delayed anastomotic hemorrhage of the donor duodenum (2.5%).

219 ed SPK transplant patients, with anastomotic hemorrhage of the donor duodenum as a very late complica

220 treat population of HARBOR were analyzed for hemorrhage on DFE or fundus photography and exudative ac

221 e evaluated for 82 patients with evidence of hemorrhage on DFE or fundus photography at 3 months and

222 with resolution of vitreous and pre-retinal hemorrhages on 4-month follow up.

223 Small focal intracerebral hemorrhages only visible on exquisitely sensitive MRI se

224 ere normal, with no evidence of intracranial hemorrhage or edema.

225 ere normal, with no evidence of intracranial hemorrhage or edema.Her subsequent hospital course was c

226 fety outcome was a composite of intracranial hemorrhage or gastrointestinal bleeding.

227 outcomes included secondary intraventricular hemorrhage or hydrocephalus upon follow-up CT, thromboem

228 and lung disease that presents as pulmonary hemorrhage or interstitial lung disease (ILD).

229 was associated with symptomatic intracranial hemorrhage (OR = 3.01; 95% CI = 1.77-5.11; p < 0.0001).

230 d as seizures, ischemic stroke, intracranial hemorrhage, or brain death.Measurements and Main Results

231 as hepatic decompensation (ascites, variceal hemorrhage, or encephalopathy).

232 Subretinal hyperreflective material, macular hemorrhage, or RPE tear occurred in 14 of 47, 13 of 47,

233 hickness (P = 0.01) and presence of vitreous hemorrhage (P = 0.05).

234 17% of lobar and 5% of deep intraparenchymal hemorrhage (p = 0.09).

235 .57; 95% CI, 1.39-1.77) and in intracerebral hemorrhage patients compared with these groups (adjusted

236 ient among ischemic stroke and intracerebral hemorrhage patients stabilized.

237 ity in patients with aneurysmal subarachnoid hemorrhage, potentially through a hemostatic mechanism.

238 d gastric emptying (DGE), postpancreatectomy hemorrhage (PPH), bile leak, blood loss, reoperation, re

239 vement (p < 0.0001) had concomitant vitreous hemorrhage pre-op.

240 53 [95% CI, 0.05-1.02]; P=0.031), myocardial hemorrhage presence (odds ratio [OR], 3.20 [95% CI, 1.25

241 5% CI, -0.97 to -0.23]; P=0.002), myocardial hemorrhage presence (OR, 0.34 [95% CI, 0.15-0.75]; P=0.0

242 gains over 24 months, regardless of month 3 hemorrhage presence versus absence: 9.4 and 8.7 Early Tr

243 neumonia, serious infections, any infection, hemorrhage, renal failure, deep vein thrombosis, and unc

244 infection, liver diseases, gastrointestinal hemorrhage, renal failure, urinary tract calculus, chron

245 DR-related complications (including vitreous hemorrhage, retinal detachment, and neovascular glaucoma

246 d (kappa = 0.61, 0.48, and 0.52 for vitreous hemorrhage, retinal detachment, and neovascular glaucoma

247 rating room, endophthalmitis, suprachoroidal hemorrhage, retinal detachment, retinal tear, macular ed

248 e retina can explain the patterns of retinal hemorrhage (RH) seen in AHT.

249 patterns consistent with the diffuse retinal hemorrhages (RH) typically found in the posterior pole a

250 anial aneurysms (IA) and suffer subarachnoid hemorrhage (SAH) at younger ages than the general popula

251 ntribute to the pathogenesis of subarachnoid hemorrhage (SAH).

252 .0 [SD 18.4], 62% male, median intracerebral hemorrhage score 1.5 [interquartile range 1-2], delirium

253 Background Subdural hemorrhage (SDH) is thought to have a benign course in a

254 ing deaths, 292 (44.4%) were P/PP; of these, hemorrhage, sepsis, and traumatic brain injury accounted

255 of retinopathy of prematurity, intracranial hemorrhage, sepsis, necrotizing enterocolitis, bronchopu

256 ort the hypothesis that magnesium influences hemorrhage severity in patients with aneurysmal subarach

257 erum magnesium and radiographic subarachnoid hemorrhage severity.

258 t shock pulmonary endothelial dysfunction in hemorrhage shock.

259 ssociated with high symptomatic intracranial hemorrhage (sICH) (24%) and mortality (53%) rates.

260 nical thrombectomy, symptomatic intracranial hemorrhage (sICH), and favorable outcome (modified Ranki

261 systemic embolism, symptomatic intracerebral hemorrhage (sICH), and major extracranial hemorrhage (EC

262 ects of the initial injury like swelling and hemorrhaging, strategies for the induction of neuronal r

263 of preclinical and clinical nonintracerebral hemorrhage studies, adjunct 1-deamino-8-D-arginine vasop

264 taset is composed of annotations of the five hemorrhage subtypes (subarachnoid, intraventricular, sub

265 outcomes in an animal model of intracerebral hemorrhage, suggesting that this process could have biol

266 yos had fewer visible blood vessels and more hemorrhages than their wild-type littermates, which was

267 lateral lung ischemia from t = 0-70 min plus hemorrhage to a mean arterial blood pressure (MAP) of 30

268 ar, subdural, epidural, and intraparenchymal hemorrhage) typically encountered at brain CT.

269 Adult patients with intracerebral hemorrhage under antiplatelet treatment and follow-up CT

270 ion (2 U) within 60 minutes of intracerebral hemorrhage under antiplatelet treatment diagnosis on bra

271 early platelet transfusion in intracerebral hemorrhage under antiplatelet treatment.

272 and dichotomized (thick vs thin subarachnoid hemorrhage) univariate and adjusted logistic regression

273 underwent splenectomy followed by controlled hemorrhage until lactate reached 7.5-8.5 mmol/L.

274 = 4-6) 1 year after first-ever intracerebral hemorrhage using logistic regression, adjusting for know

275 ersus early vitrectomy for diabetic vitreous hemorrhage (VH).

276 temporal lobe involvement, intraparenchymal hemorrhage volume, and electrographic seizures predicted

277 The 30-day cumulative incidence of major hemorrhage was 0.7% (95% CI, 0.1-5.1) and of clinically

278 Vitreous hemorrhage was a risk factor for earlier retinal breaks

279 Extent of initial hemorrhage was graded semi-quantitatively on admission C

280 Mild chronic parenchymal hemorrhage was noted in 3 left superior PV lobes after I

281 cted on SD-OCT in more than 89% of eyes when hemorrhage was present on DFE or fundus photography.

282 T detected nAMD activity in 89% of eyes when hemorrhage was present on fundus photography.

283 vision gains with or without injection when hemorrhage was present without OCT-detectable fluid.

284 e elevated risk for symptomatic intracranial hemorrhage was seen only within the first 14 days (16.3%

285 Bilateral disease, higher IOP, and disc hemorrhage were confirmed as risk factors for deteriorat

286 stenosis, plaque ulceration, and intraplaque hemorrhage were not associated with symptomatic status.(

287 oration of tympanic membranes and middle ear hemorrhage were observed at 1 and 7 days, and were resto

288 genation patients, the rates of intracranial hemorrhage were similar between venoarterial extracorpor

289 Retinal hemorrhages were categorized according to size-those sma

290 ural effusion, pneumothoraces or perihepatic hemorrhages were relatively easy to treat.

291 at lung syndecan-1 mRNA is reduced following hemorrhage, whereas the molecular mechanism underlying t

292 with later (2010-2013) measures of infarct, hemorrhage, white matter hyperintensity (WMH) grade, bra

293 ith intracerebral, subarachnoid, or subdural hemorrhages who had at least 1 follow-up image within 24

294 ion of clearance of VH and rate of recurrent hemorrhage with any additional treatment in both groups.

295 barachnoid hemorrhage; 4, thick subarachnoid hemorrhage with intraventricular hemorrhage).

296 ubarachnoid hemorrhage; 2, thin subarachnoid hemorrhage with intraventricular hemorrhage; 3, thick [>

297 e association of a retinal tear and vitreous hemorrhage with whole-body vibration training.

298 gest that the rapid accumulation of cerebral hemorrhages, with accompanying fluid egress, may cause f

299 rovement at 3 days; symptomatic intracranial hemorrhage within 36 hours; and all-cause death.

300 This suggests that macular hemorrhages without OCT-detectable macular fluid may not