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1 artition and Portal Vein Ligation for Staged hepatectomy).
2 unit of autologous blood before living donor hepatectomy.
3 llowed liver function recovery after partial hepatectomy.
4 eration process following two-thirds partial hepatectomy.
5 rioperative outcomes for patients undergoing hepatectomy.
6 r2 knockout (Itpr2(-/-) ) mice following 67% hepatectomy.
7 ion as the primary energy source early after hepatectomy.
8 splantation, ulcerative colitis, and partial hepatectomy.
9 tion and reduced steatosis following partial hepatectomy.
10 e injected with Concanvalin A before partial hepatectomy.
11               Patient overall survival after hepatectomy.
12 lates growth and metabolic adaptations after hepatectomy.
13  drinking water of mice subjected to partial hepatectomy.
14 ia-reperfusion injury and regeneration after hepatectomy.
15 us and overall complications following major hepatectomy.
16 eneration in 157 patients undergoing partial hepatectomy.
17  model of 2-acetylaminofluorene with partial hepatectomy.
18 nd HPC-associated biliary regeneration after hepatectomy.
19        MSCs supported survival after partial hepatectomy.
20 PP treatment delayed liver weight gain after hepatectomy.
21 olization, if used, types of transection and hepatectomy.
22 ignaling, was strongly induced after partial hepatectomy.
23 neration and clinical outcomes after partial hepatectomy.
24 decades, morbidity is still high after major hepatectomy.
25 ers and the in vivo activation after partial hepatectomy.
26 nce to the 10-fold volume removal of partial hepatectomy.
27 elated deaths occurred within 365 days after hepatectomy.
28 nefit from adjuvant CIK cell treatment after hepatectomy.
29 nce to the 10-fold volume removal of partial hepatectomy.
30 ferred to surgical teams to be evaluated for hepatectomy.
31 h higher rate of proliferation after partial hepatectomy.
32 e recovery of the living donor after partial hepatectomy.
33 blockade on liver regeneration after partial hepatectomy.
34 luded all adult patients undergoing elective hepatectomy.
35 nalyze surgeons' anticipation of the risk of hepatectomy.
36 fferent time points until 72 h after partial hepatectomy.
37 generation when applied 2 h prior to partial hepatectomy.
38 ion of SB-258719 sixteen hours after partial hepatectomy.
39 (mitotic index in HE sections) after partial hepatectomy.
40 Wistar rats were subjected to 60-70% partial hepatectomy.
41 hepatectomy liver failure (PHLF) after major hepatectomy.
42 nd 59 of these donors underwent living donor hepatectomy.
43 the selection of patients undergoing partial hepatectomy.
44 lism during liver regeneration after partial hepatectomy.
45 9 proteolytically cleaved VEGF after partial hepatectomy.
46  PHLF remain clinical challenges after major hepatectomy.
47 ent predictive biomarker of PHLF after major hepatectomy.
48 ting similar outcome as other types of major hepatectomies.
49 n display similar safety and feasibility for hepatectomies.
50 tectomy (20%, 23%, 30%, P < 0.0001), 2-stage hepatectomy (0%, 3%, 4%, P < 0.001), need for portal vei
51 evidenced by increases in the rates of major hepatectomy (20%, 23%, 30%, P < 0.0001), 2-stage hepatec
52       Patients were treated with an extended hepatectomy (202 [39.3%]), a hemihepatectomy (180 [35.0%
53 n hemihepatectomy (48.8%), or hemi-/extended hepatectomy (36.3%).
54 eration was compared with that after partial hepatectomy (70%).
55 rsus 16%, open proctectomy 13% vs 17%, major hepatectomy 8% versus 12%, pancreatoduodenectomy 16% ver
56                       Most underwent a major hepatectomy (82%), often accompanied by lymphadenectomy
57 Although ALPPS is now an established 2-stage hepatectomy additional data are warranted to further ref
58 w conceptual technique of laparoscopic right hepatectomy allowing for low blood loss and morbidity.
59 ared to 28 and 12 months for patients in the hepatectomy alone group (control).
60 artition and Portal vein ligation for Staged hepatectomy (ALPPS) has been tested in various indicatio
61 artition and Portal Vein Ligation for Staged Hepatectomy (ALPPS) occurred over time and is associated
62 artition and portal vein ligation for staged hepatectomy (ALPPS) show clinically distinct recovery pa
63 artition and portal vein ligation for staged hepatectomy (ALPPS), a 2-stage hepatectomy procedure, ha
64 artition and Portal Vein Ligation for Staged Hepatectomy (ALPPS), available after stage-1, either to
65   Moreover, liver regeneration after partial hepatectomy also depended upon the formation of InsP3 in
66 gment, crystallizable) to mice after partial hepatectomy and acetaminophen intoxication, and measured
67 egeneration, which is required after partial hepatectomy and acute or chronic liver injury.
68 urs prior to and sixteen hours after partial hepatectomy and by intraperitoneal administration of SB-
69   Regenerative growth was induced by partial hepatectomy and exposure to carbon tetrachloride.
70  better 5-year overall survival from initial hepatectomy and from liver recurrence in the PSH than in
71 r transplantation by pure laparoscopic total hepatectomy and liver graft implantation using a preexis
72                  The patient underwent total hepatectomy and living donor liver transplantation (LDLT
73 -258719 and SB-269970) at 16 h after partial hepatectomy and peaked at 32 h ([(3)H]-thymidine incorpo
74 of liver regeneration after extended partial hepatectomy and portal vein ligation for multiple biloba
75 y augmented liver regeneration after partial hepatectomy and portal vein ligation, and increased the
76 Epac2 are deleted, were subjected to partial hepatectomy and the regenerating liver was analyzed with
77 mportant to help improve the safety of donor hepatectomy and to provide a database for informed conse
78 suffered from residual biliary calculi after hepatectomy and underwnet POC from August 2016 to June 2
79 ted 20 years ago, has been described for all hepatectomies, and is considered as the reference techni
80 diofrequency (RF) ablation, partial surgical hepatectomy, and a sham operation and to inhibit HCC rec
81 ogram who underwent pancreaticoduodenectomy, hepatectomy, and colectomy at a single academic institut
82 phagectomy, total/partial gastrectomy, major hepatectomy, and pancreatectomy were identified using th
83                  She underwent an open right hepatectomy, and pathology surprisingly revealed a bilia
84 of portal hypertension, planned extension of hepatectomy, and the MELD score.
85 ression-free survival, conversion to partial hepatectomy, and viable HCC within the tumor specimen.
86 n otherwise recoverable surgical injury (30% hepatectomy), antibiotics, and a short period of starvat
87                 Nonanatomical tissue-sparing hepatectomies are associated with worse DFS in patients
88     We used liver regeneration after partial hepatectomy as a physiological stress response model.
89 h HCC hepatocellular carcinoma who underwent hepatectomy at one institution were evaluated with insti
90 ected from 55 patients who underwent partial hepatectomy at the Royal Infirmary Edinburgh between Dec
91 entially amenable to curative extended right hepatectomy but insufficient size of the future liver re
92 tiple and smaller CRLM, underwent more major hepatectomies, but had less extrahepatic disease.
93 efficient early cytokine response to partial hepatectomy, but is inhibitory to later growth factor ac
94 cal role in liver regeneration after partial hepatectomy, but their role in TCPOBOP-induced direct hy
95 accelerated liver regeneration after partial hepatectomy by 40%, whereas systemic MMP inhibition impa
96 nd of hepatocyte proliferation after partial hepatectomy by preventing increases in growth hormone re
97 p38gamma induces proliferation after partial hepatectomy by promoting the phosphorylation of retinobl
98 hepatectomy performed, and hospital type and hepatectomy caseload were retrieved.
99                                      Partial hepatectomy causes a transient increase in p21 in a subp
100 s preconditioned with retrorsine and partial hepatectomy, cell transplantation after ETN pretreatment
101 sion-free survival (PFS) for patients in the hepatectomy/CIK combination group were 41 and 16 months,
102 ectomy, pancreatectomy, pulmonary resection, hepatectomy, colectomy, and cystectomy) between 2010 and
103 uperior estimation of patient survival after hepatectomy compared with current staging systems.
104 bited hepatocyte proliferation after partial hepatectomy, confirming its pivotal role in cell cycle p
105                       In response to partial hepatectomy, deletion of Epac1 and/or Epac2 led to incre
106               Thus, after two-thirds partial hepatectomy, DeltaEGFR livers displayed lower and delaye
107              Consecutive patients undergoing hepatectomy during 1998 to 2015 at 1 institution were an
108                     ALPPS is a novel 2-stage hepatectomy enabling resection of extensive hepatic tumo
109 ortality was similar to conventional 2-stage hepatectomies for CRLM.
110 in 2 expert centers practicing 1- or 2-stage hepatectomy for bCRLM.
111                           Outcomes following hepatectomy for BRAF-mut CRLM have not been well studied
112 phagectomy, gastrectomy, pancreatectomy, and hepatectomy for cancer between 2012 and 2017 were identi
113  by next-generation sequencing who underwent hepatectomy for CLM (2005-2015).
114 o stimulate liver regeneration after partial hepatectomy for colorectal liver metastases (CRLM).
115 anuary 1992 and December 2012, who underwent hepatectomy for CRLM and resection of synchronous EHD.
116                   All patients who underwent hepatectomy for CRLM with complete resection and known B
117               Of 4124 patients who underwent hepatectomy for CRLM, 1497 had complete resection and kn
118 endent prognostic factor for mortality after hepatectomy for HCC in European patients and could be us
119       All consecutive patients who underwent hepatectomy for HCC in our institution, between February
120             Among 198 patients who underwent hepatectomy for HCC, 109 patients had an available compu
121 ia could be used to evaluate patients before hepatectomy for HCC.
122                   Adults who underwent right hepatectomy for LDLT between 2000 and 2018 were analyzed
123                         ALPPS is a two-stage hepatectomy for patients with extensive liver tumors wit
124 ging results have been reported with partial hepatectomy for solitary metastases, with percutaneous h
125 ver of wild type mice in response to partial hepatectomy, further supporting a role for these protein
126 ctomy: >=16/yr, pancreatectomy: >=26/yr, and hepatectomy: &gt;=76/yr).
127 erioperative care and surgical technique for hepatectomy have improved, the indications for and compl
128 on-PSH was a risk of noncandidacy for repeat hepatectomy (hazard ratio: 8.18, confidence interval: 1.
129 eplete hepatocyte alphavbeta8, after partial hepatectomy, hepatocyte proliferation and liver-to-body
130 om the hepatic vein during pure laparoscopic hepatectomy; however, there is a risk of pulmonary gas e
131 95% CI: 1.141-6.024; P = 0.024), right-sided hepatectomy (HR: 2.143, 95% CI: 1.544-2.975; P < 0.001),
132 lysis of seven time points following partial hepatectomy identified the epigenetic regulator UHRF1, w
133 del) that underwent RF ablation, 35% partial hepatectomy (ie, left lobectomy), or a sham operation (c
134 ection in 67 consecutive patients with major hepatectomy (ie, resection of >=3 liver segments).
135  to include all patients undergoing elective hepatectomies in an observational study.
136 nsecutive adult patients undergoing elective hepatectomy in 9 HPB centers.
137 in normal and steatotic livers after partial hepatectomy in a rodent model.
138                                              Hepatectomy in G6PD-deficient donors is associated with
139 hed literature regarding the safety of donor hepatectomy in G6PDd individuals.
140 e and AICAR improved survival after extended hepatectomy in mice challenged with a Western diet, indi
141  on liver regeneration following 70% partial hepatectomy in mice lacking the Cip/Kip inhibitors p21(C
142 nalization also occurred following a partial hepatectomy in mice.
143 accelerated liver regeneration after partial hepatectomy in mice.
144                   We have shown that partial hepatectomy in multidrug resistance 2 knockout (Mdr2(-/-
145 n intention-to-treat analysis 1- and 2-stage hepatectomy in patients with bCRLM achieve comparable OS
146                               Survival after hepatectomy in patients with double mutation of APC and
147 tation of 2-acetylaminofluorene with partial hepatectomy in rats or on feeding a 3,5-diethoxycarbonyl
148 models of 2-acetylaminofluorene with partial hepatectomy in rats, and 3,5-diethoxycarbonyl-1,4-dihydr
149 iferation that can be observed after partial hepatectomy in rats.
150 stimulating liver regeneration after partial hepatectomy in rodents and humans.
151 were compared between patients who underwent hepatectomy in the eras 1998 to 2003, 2004 to 2009, and
152                   On multivariable analysis, hepatectomy in the most recent era 2010 to 2015 was asso
153                         We performed partial hepatectomy in WT and liver-specific Sirt1-deficient mic
154      compare liver regeneration upon partial hepatectomy in young and adult mice.
155 ity rates compared with conventional 2-stage hepatectomy including portal vein embolization.
156                                The number of hepatectomies increased in each era (794 in 1998 to 2003
157                                        Major hepatectomy increased PVP by 26.9% (P = 0.001), markedly
158 sts that AhR functionality following partial hepatectomy is dependent on a p21(Cip1)-regulated signal
159             Liver regrowth following partial hepatectomy is enabled by proliferation of hepatocytes t
160 ial for retrievability, and graft removal by hepatectomy is impractical.
161                               A second-stage hepatectomy is performed as soon as the graft has regene
162                                    Two-stage hepatectomy is the surgical strategy mostly chosen for t
163 ious consequences for normal livers, partial hepatectomy leads to severe liver necrosis and reduced h
164 derwent PSH and 144 patients underwent right hepatectomy, left hepatectomy, or left lateral sectionec
165  has enabled extensive liver resection, post-hepatectomy liver failure remains one of the most lethal
166  mathematical framework which described post-hepatectomy liver regeneration in each patient by incorp
167      The pure laparoscopic approach in right hepatectomy (LRH) for living donor liver transplantation
168                             Diabetes, repeat-hepatectomy, major-hepatectomy, synchronous-major-proced
169 KRAS-mutated CRLM, more extensive anatomical hepatectomies may be warranted.
170 ent studies of complications following donor hepatectomy may not be generalizable to all hospitals pe
171                                 During total hepatectomy, median blood loss was 500 mL, and no patien
172 l vein revascularization who underwent lobar hepatectomy, median OS was not reached yet exceeded 24.5
173             To prevent drops in oxygen after hepatectomy, mice were pretreated with inositol trispyro
174 short together with efficient cooling during hepatectomy might improve outcome.
175                       The retrorsine-partial hepatectomy model was used for liver repopulation studie
176                              In the extended hepatectomy model, liver-selective MMP-9 inhibition rest
177 sed liver repopulation in retrorsine/partial hepatectomy model.
178                     After two-thirds partial hepatectomy, mutant mice (n = 5) displayed increased liv
179 ost commonly performed procedure was ex vivo hepatectomy (n = 18), followed by ex vivo resection and
180 65 postoperative days in patients undergoing hepatectomy (n = 2811) and/or pancreatectomy (n = 1092)
181  proctectomy (n = 24,925; median = 6), major hepatectomy (n = 9,805; median = 6), pancreatoduodenecto
182 dent association with LD was found for major hepatectomy (odds ratio [OR], 2.41; 95% CI, 1.17-4.30; P
183 donors undergoing PLDRH and open donor right hepatectomy (ODRH).
184                                      Partial hepatectomy of the adult mammalian liver activates compe
185                                      Partial hepatectomy of Uhrf1(HepKO) livers resulted in early and
186                         We performed partial hepatectomies on wild-type C57BL/6, CD45.1, Tcrd(-/-), o
187 More specifically, we have performed partial hepatectomy on mice with genetic deficiency in C3, the m
188  of the time from aortic perfusion to end of hepatectomy on outcomes after DCD LT in the United Kingd
189 reased innate immunity in mice after partial hepatectomy or acetaminophen-induced injury, with reside
190 , such as rodent LR after two-thirds partial hepatectomy or administration of damaging chemicals (CCl
191 ional replicative capacity following partial hepatectomy or chemical injuries.
192                                              Hepatectomy or completion pancreatectomy was accomplishe
193 tocyte proliferation following liver partial hepatectomy or damage resulting from carbon tetrachlorid
194      Thirty-nine patients undergoing partial hepatectomy or liver transplantation for HCC were consen
195 or patients' >/=18 years who underwent wedge hepatectomy or lobectomy from 2000 to 2014.
196 ical more limited resections such as central hepatectomy or sectionectomies may provide an alternativ
197 4 patients underwent right hepatectomy, left hepatectomy, or left lateral sectionectomy (non-PSH grou
198 re it with a successive cohort of open right hepatectomies (ORH) for LDLT.
199 ificantly influenced the risk of death after hepatectomy (P < 0.001) and pancreatectomy (P < 0.001).
200  male sex (P = .03), and no history of prior hepatectomy (P = .04) or hepatic arterial infusion chemo
201  caused higher postresection PVP after right hepatectomy (P = 0.04), the Pringle maneuver was associa
202 er the second procedure, with extended right hepatectomy performed at day 23 after transplantation.
203 sociated conditions, indication for surgery, hepatectomy performed, and hospital type and hepatectomy
204                 Recent studies using partial hepatectomy (PH) and other experimental models of liver
205 nd Cdk2 for liver regeneration after partial hepatectomy (PH) by generating corresponding double- and
206 e of cell proliferation that follows partial hepatectomy (PH) identified approximately 1,400 mammalia
207 ), standard (60%), or extended (80%) partial hepatectomy (PH) in mice with and without liver steatosi
208                             Extended partial hepatectomy (PH) in patients is leading to portal hyperp
209  intrinsic hepatic innervation after partial hepatectomy (PH) in rats and the presence and pattern of
210 UND & AIMS: Liver regeneration after partial hepatectomy (PH) increases the protein folding burden at
211 during liver regeneration (LR) after partial hepatectomy (PH) is observed in several species.
212                         We performed partial hepatectomy (PH) to transgenic mice that overexpress SIR
213 groups) weeks of treatment with G49, partial hepatectomy (PH) was performed, and all mice were mainta
214 iating liver regeneration (LR) after partial hepatectomy (PH), by regulating expression of Cyclin-D1.
215 erent surgical procedures, including partial hepatectomy (PH), intraoperative portal vein ligation (P
216 involved in liver regeneration after partial hepatectomy (PH), to initiate growth, protect liver cell
217                  In conjunction with partial hepatectomy (PH), transplanted stem/progenitor cells eng
218 ells during liver regeneration after partial hepatectomy (PH).
219 position was recently reported after partial hepatectomy (PHx) in mice, but the role of fibrin(ogen)
220 a(-/-) (NSG) mice that had undergone partial hepatectomy (PHx) represented the best combination of en
221 aKlf6), cell proliferation following partial hepatectomy (PHx) was increased compared to controls.
222 ytes in culture, rat liver following partial hepatectomy (PHx), and hepatoma cell lines.
223 , we challenged liver function after partial hepatectomy (PHx), inducing acute proliferative and meta
224  liver regeneration after two-thirds partial hepatectomy (PHx).
225 a pneumoniae or Escherichia coli) or partial hepatectomy (PHx).
226 involved in liver regeneration after partial hepatectomy (PHX).
227                Pure laparoscopic donor right hepatectomy (PLDRH) is not a standard procedure for livi
228 urrently used in preclinical models: partial hepatectomy, portal ligature or embolization, and radiot
229 sion, within 1 hour after two-thirds partial hepatectomy (post-PH) in C57BL/6J mice.
230 oling is slow and livers are lukewarm during hepatectomy, potentially affecting outcome.
231 on for staged hepatectomy (ALPPS), a 2-stage hepatectomy procedure, has revolutionized the surgical m
232                   PTEN down-regulation after hepatectomy promotes the burning of TRAS-derived lipids
233 gment 2-3 transplantation with delayed total hepatectomy (RAPID) from living donor in a patient affec
234 artition and portal vein ligation for staged hepatectomy." RASPE induced safe and profound growth of
235 cal parenchymal resections including central hepatectomy (resection of segments 4, 5, and 8), right a
236 ificantly between patients in 1- and 2-stage hepatectomy, respectively: 37.2 and 34.5 months (P=0.6),
237 e and inhibition of cell proliferation after hepatectomy, resulting in liver failure.
238  inhibited in DeltaEGFR livers after partial hepatectomy, revealing a new function for EGFR kinase ac
239            A group of 30 patients with minor hepatectomy served as controls.
240                                              Hepatectomy specimen ex vivo MR imaging assisted in matc
241 ression and liver regeneration after partial hepatectomy, suggesting that DBC1/DN-DBC1 transitions pl
242          Diabetes, repeat-hepatectomy, major-hepatectomy, synchronous-major-procedure, inflow-occlusi
243  frequent use of laparoscopy in living donor hepatectomy, the laparoscopic approach has never been re
244                            Following partial hepatectomy, the liver initiates a regenerative program
245                            Following partial hepatectomy, the liver initiates a regenerative programm
246 t to surgeons with large experience in donor hepatectomy through a proctored learning curve.
247  (4.9 [3.5-5.9] vs 6.4 [4.3-12]; P < 0.001), hepatectomy time (70 [42-120] vs 81 [58-207]; P = 0.02),
248                No data exist to evaluate how hepatectomy time (HT), in the context of donation after
249 o investigate the relationship between donor hepatectomy time and post-transplant outcome in 12,974 r
250                                        Donor hepatectomy time had a similar effect on death-censored
251                                        Donor hepatectomy time impairs liver transplant outcome.
252 were more susceptible to the effect of donor hepatectomy time on death-censored graft survival.
253                Assessing the effect of donor hepatectomy time on outcome after transplantation.
254                                 Median donor hepatectomy time was 41 minutes [interquartile range (IQ
255                                        Donor hepatectomy time was defined as time between start of ao
256                                        Donor hepatectomy time was independently associated with graft
257 s donated after circulatory death had longer hepatectomy times than those from brain-dead donors [50
258 allowed for an increased percentage of major hepatectomies to be performed in a purely minimally inva
259                         We performed partial hepatectomies to test liver regeneration and then RNA-se
260 milieu of chronic inflammation links partial hepatectomy to accelerated hepatocarcinogenesis; this su
261 as to evaluate a modified technique of total hepatectomy to limit hazardous liver manipulation and im
262 anvalin A was injected 4 days before partial hepatectomy to natural killer T cells- deficient mice or
263 ral types and rounds of injury, ranging from hepatectomy to toxin-mediated damage.
264                             In mice, partial hepatectomy up-regulated expression of CCL20 and ligands
265                         ALPPS is a two-stage hepatectomy variant that increases resection rates and R
266                                 Laparoscopic hepatectomy was associated with lower intraoperative blo
267          A new conceptual technique of right hepatectomy was designed using evidence-based facts and
268                                       A left hepatectomy was done and dilated bile ducts filled with
269             Most important, LR after partial hepatectomy was impaired in caNrf2-transgenic mice as a
270                                       Repeat hepatectomy was more frequently performed in the PSH gro
271 tformin via oral gavage, after which a donor hepatectomy was performed followed by a standardized col
272                                   Concurrent hepatectomy was performed in 178 patients (38%), and whe
273                                    The right hepatectomy was performed.
274                Two weeks later completion of hepatectomy was performed.
275         The number of patient conversions to hepatectomy was seven for ACE and three for cTACE.
276  a model of liver regeneration after partial hepatectomy, we found that DN-DBC1 is down-regulated in
277        Between 2007 and 2012, a total of 680 hepatectomies were analyzed from a prospective database.
278 Between January 2013 and September 2013, 651 hepatectomies were included.
279 e 3- and 5-year overall survival rates after hepatectomy were 48.7% and 33.8%, respectively.
280 ancreaticoduodenectomy, subtotal and partial hepatectomy were analyzed.
281                    Complications after donor hepatectomy were categorized using International Classif
282 who underwent elective, adult-to-adult right hepatectomy were initially screened.
283 the collaborative study period, outcomes for hepatectomy were similar for LV and HV (85 vs 507 cases)
284 rvival (RFS) and overall survival (OS) after hepatectomy were worse in patients with double mutation
285 ber of lesions, clinical-risk score, 2-stage hepatectomy) were significantly worse in the HAI group;
286 icantly different in the first 2 years after hepatectomy when the interval was extended from 3 months
287                           ALPPS is a 2-stage hepatectomy, which incorporates parenchymal transection
288  Moreover, 2 reduced blood loss during liver hepatectomy, while 1 and aprotinin had no effect.
289 tients with HCC underwent various anatomical hepatectomies with the Glissonean pedicle approach betwe
290 93-4.62; P < .001), followed by extension of hepatectomy with (OR, 2.76; 95% CI, 1.85-4.77; P = .03)
291                Mice died at 15-18 days after hepatectomy with ascites, increased plasma ammonia, and
292              RASPE also allowed for extended hepatectomy with less risk of post-operative liver failu
293 nocarcinoma of right colon, underwent a left hepatectomy with ligation of right portal vein maintaini
294          Long-term outcomes after anatomical hepatectomy with the Glissonean pedicle approach in pati
295                                   Anatomical hepatectomy with the Glissonean pedicle approach was ach
296 the morbidity and mortality after anatomical hepatectomy with the Glissonean pedicle approach, and lo
297     ELLS during LT for PLD facilitates total hepatectomy with vena cava and caval flow preservation.
298 way were rapidly upregulated after two-third hepatectomy, with the ubiquitin ligase Nedd4-1 being a t
299 ally impedes tumorigenesis following partial hepatectomy without compromising survival or liver mass
300 ateral sectionectomy, and 3 underwent a left hepatectomy without middle hepatic vein procurement.

 
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