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1 artition and Portal Vein Ligation for Staged hepatectomy).
2 unit of autologous blood before living donor hepatectomy.
3 llowed liver function recovery after partial hepatectomy.
4 eration process following two-thirds partial hepatectomy.
5 rioperative outcomes for patients undergoing hepatectomy.
6 r2 knockout (Itpr2(-/-) ) mice following 67% hepatectomy.
7 ion as the primary energy source early after hepatectomy.
8 splantation, ulcerative colitis, and partial hepatectomy.
9 tion and reduced steatosis following partial hepatectomy.
10 e injected with Concanvalin A before partial hepatectomy.
11 Patient overall survival after hepatectomy.
12 lates growth and metabolic adaptations after hepatectomy.
13 drinking water of mice subjected to partial hepatectomy.
14 ia-reperfusion injury and regeneration after hepatectomy.
15 us and overall complications following major hepatectomy.
16 eneration in 157 patients undergoing partial hepatectomy.
17 model of 2-acetylaminofluorene with partial hepatectomy.
18 nd HPC-associated biliary regeneration after hepatectomy.
19 MSCs supported survival after partial hepatectomy.
20 PP treatment delayed liver weight gain after hepatectomy.
21 olization, if used, types of transection and hepatectomy.
22 ignaling, was strongly induced after partial hepatectomy.
23 neration and clinical outcomes after partial hepatectomy.
24 decades, morbidity is still high after major hepatectomy.
25 ers and the in vivo activation after partial hepatectomy.
26 nce to the 10-fold volume removal of partial hepatectomy.
27 elated deaths occurred within 365 days after hepatectomy.
28 nefit from adjuvant CIK cell treatment after hepatectomy.
29 nce to the 10-fold volume removal of partial hepatectomy.
30 ferred to surgical teams to be evaluated for hepatectomy.
31 h higher rate of proliferation after partial hepatectomy.
32 e recovery of the living donor after partial hepatectomy.
33 blockade on liver regeneration after partial hepatectomy.
34 luded all adult patients undergoing elective hepatectomy.
35 nalyze surgeons' anticipation of the risk of hepatectomy.
36 fferent time points until 72 h after partial hepatectomy.
37 generation when applied 2 h prior to partial hepatectomy.
38 ion of SB-258719 sixteen hours after partial hepatectomy.
39 (mitotic index in HE sections) after partial hepatectomy.
40 Wistar rats were subjected to 60-70% partial hepatectomy.
41 hepatectomy liver failure (PHLF) after major hepatectomy.
42 nd 59 of these donors underwent living donor hepatectomy.
43 the selection of patients undergoing partial hepatectomy.
44 lism during liver regeneration after partial hepatectomy.
45 9 proteolytically cleaved VEGF after partial hepatectomy.
46 PHLF remain clinical challenges after major hepatectomy.
47 ent predictive biomarker of PHLF after major hepatectomy.
48 ting similar outcome as other types of major hepatectomies.
49 n display similar safety and feasibility for hepatectomies.
50 tectomy (20%, 23%, 30%, P < 0.0001), 2-stage hepatectomy (0%, 3%, 4%, P < 0.001), need for portal vei
51 evidenced by increases in the rates of major hepatectomy (20%, 23%, 30%, P < 0.0001), 2-stage hepatec
55 rsus 16%, open proctectomy 13% vs 17%, major hepatectomy 8% versus 12%, pancreatoduodenectomy 16% ver
57 Although ALPPS is now an established 2-stage hepatectomy additional data are warranted to further ref
58 w conceptual technique of laparoscopic right hepatectomy allowing for low blood loss and morbidity.
60 artition and Portal vein ligation for Staged hepatectomy (ALPPS) has been tested in various indicatio
61 artition and Portal Vein Ligation for Staged Hepatectomy (ALPPS) occurred over time and is associated
62 artition and portal vein ligation for staged hepatectomy (ALPPS) show clinically distinct recovery pa
63 artition and portal vein ligation for staged hepatectomy (ALPPS), a 2-stage hepatectomy procedure, ha
64 artition and Portal Vein Ligation for Staged Hepatectomy (ALPPS), available after stage-1, either to
65 Moreover, liver regeneration after partial hepatectomy also depended upon the formation of InsP3 in
66 gment, crystallizable) to mice after partial hepatectomy and acetaminophen intoxication, and measured
68 urs prior to and sixteen hours after partial hepatectomy and by intraperitoneal administration of SB-
70 better 5-year overall survival from initial hepatectomy and from liver recurrence in the PSH than in
71 r transplantation by pure laparoscopic total hepatectomy and liver graft implantation using a preexis
73 -258719 and SB-269970) at 16 h after partial hepatectomy and peaked at 32 h ([(3)H]-thymidine incorpo
74 of liver regeneration after extended partial hepatectomy and portal vein ligation for multiple biloba
75 y augmented liver regeneration after partial hepatectomy and portal vein ligation, and increased the
76 Epac2 are deleted, were subjected to partial hepatectomy and the regenerating liver was analyzed with
77 mportant to help improve the safety of donor hepatectomy and to provide a database for informed conse
78 suffered from residual biliary calculi after hepatectomy and underwnet POC from August 2016 to June 2
79 ted 20 years ago, has been described for all hepatectomies, and is considered as the reference techni
80 diofrequency (RF) ablation, partial surgical hepatectomy, and a sham operation and to inhibit HCC rec
81 ogram who underwent pancreaticoduodenectomy, hepatectomy, and colectomy at a single academic institut
82 phagectomy, total/partial gastrectomy, major hepatectomy, and pancreatectomy were identified using th
85 ression-free survival, conversion to partial hepatectomy, and viable HCC within the tumor specimen.
86 n otherwise recoverable surgical injury (30% hepatectomy), antibiotics, and a short period of starvat
89 h HCC hepatocellular carcinoma who underwent hepatectomy at one institution were evaluated with insti
90 ected from 55 patients who underwent partial hepatectomy at the Royal Infirmary Edinburgh between Dec
91 entially amenable to curative extended right hepatectomy but insufficient size of the future liver re
93 efficient early cytokine response to partial hepatectomy, but is inhibitory to later growth factor ac
94 cal role in liver regeneration after partial hepatectomy, but their role in TCPOBOP-induced direct hy
95 accelerated liver regeneration after partial hepatectomy by 40%, whereas systemic MMP inhibition impa
96 nd of hepatocyte proliferation after partial hepatectomy by preventing increases in growth hormone re
97 p38gamma induces proliferation after partial hepatectomy by promoting the phosphorylation of retinobl
100 s preconditioned with retrorsine and partial hepatectomy, cell transplantation after ETN pretreatment
101 sion-free survival (PFS) for patients in the hepatectomy/CIK combination group were 41 and 16 months,
102 ectomy, pancreatectomy, pulmonary resection, hepatectomy, colectomy, and cystectomy) between 2010 and
104 bited hepatocyte proliferation after partial hepatectomy, confirming its pivotal role in cell cycle p
112 phagectomy, gastrectomy, pancreatectomy, and hepatectomy for cancer between 2012 and 2017 were identi
114 o stimulate liver regeneration after partial hepatectomy for colorectal liver metastases (CRLM).
115 anuary 1992 and December 2012, who underwent hepatectomy for CRLM and resection of synchronous EHD.
118 endent prognostic factor for mortality after hepatectomy for HCC in European patients and could be us
124 ging results have been reported with partial hepatectomy for solitary metastases, with percutaneous h
125 ver of wild type mice in response to partial hepatectomy, further supporting a role for these protein
127 erioperative care and surgical technique for hepatectomy have improved, the indications for and compl
128 on-PSH was a risk of noncandidacy for repeat hepatectomy (hazard ratio: 8.18, confidence interval: 1.
129 eplete hepatocyte alphavbeta8, after partial hepatectomy, hepatocyte proliferation and liver-to-body
130 om the hepatic vein during pure laparoscopic hepatectomy; however, there is a risk of pulmonary gas e
131 95% CI: 1.141-6.024; P = 0.024), right-sided hepatectomy (HR: 2.143, 95% CI: 1.544-2.975; P < 0.001),
132 lysis of seven time points following partial hepatectomy identified the epigenetic regulator UHRF1, w
133 del) that underwent RF ablation, 35% partial hepatectomy (ie, left lobectomy), or a sham operation (c
140 e and AICAR improved survival after extended hepatectomy in mice challenged with a Western diet, indi
141 on liver regeneration following 70% partial hepatectomy in mice lacking the Cip/Kip inhibitors p21(C
145 n intention-to-treat analysis 1- and 2-stage hepatectomy in patients with bCRLM achieve comparable OS
147 tation of 2-acetylaminofluorene with partial hepatectomy in rats or on feeding a 3,5-diethoxycarbonyl
148 models of 2-acetylaminofluorene with partial hepatectomy in rats, and 3,5-diethoxycarbonyl-1,4-dihydr
151 were compared between patients who underwent hepatectomy in the eras 1998 to 2003, 2004 to 2009, and
158 sts that AhR functionality following partial hepatectomy is dependent on a p21(Cip1)-regulated signal
163 ious consequences for normal livers, partial hepatectomy leads to severe liver necrosis and reduced h
164 derwent PSH and 144 patients underwent right hepatectomy, left hepatectomy, or left lateral sectionec
165 has enabled extensive liver resection, post-hepatectomy liver failure remains one of the most lethal
166 mathematical framework which described post-hepatectomy liver regeneration in each patient by incorp
167 The pure laparoscopic approach in right hepatectomy (LRH) for living donor liver transplantation
170 ent studies of complications following donor hepatectomy may not be generalizable to all hospitals pe
172 l vein revascularization who underwent lobar hepatectomy, median OS was not reached yet exceeded 24.5
179 ost commonly performed procedure was ex vivo hepatectomy (n = 18), followed by ex vivo resection and
180 65 postoperative days in patients undergoing hepatectomy (n = 2811) and/or pancreatectomy (n = 1092)
181 proctectomy (n = 24,925; median = 6), major hepatectomy (n = 9,805; median = 6), pancreatoduodenecto
182 dent association with LD was found for major hepatectomy (odds ratio [OR], 2.41; 95% CI, 1.17-4.30; P
187 More specifically, we have performed partial hepatectomy on mice with genetic deficiency in C3, the m
188 of the time from aortic perfusion to end of hepatectomy on outcomes after DCD LT in the United Kingd
189 reased innate immunity in mice after partial hepatectomy or acetaminophen-induced injury, with reside
190 , such as rodent LR after two-thirds partial hepatectomy or administration of damaging chemicals (CCl
193 tocyte proliferation following liver partial hepatectomy or damage resulting from carbon tetrachlorid
194 Thirty-nine patients undergoing partial hepatectomy or liver transplantation for HCC were consen
196 ical more limited resections such as central hepatectomy or sectionectomies may provide an alternativ
197 4 patients underwent right hepatectomy, left hepatectomy, or left lateral sectionectomy (non-PSH grou
199 ificantly influenced the risk of death after hepatectomy (P < 0.001) and pancreatectomy (P < 0.001).
200 male sex (P = .03), and no history of prior hepatectomy (P = .04) or hepatic arterial infusion chemo
201 caused higher postresection PVP after right hepatectomy (P = 0.04), the Pringle maneuver was associa
202 er the second procedure, with extended right hepatectomy performed at day 23 after transplantation.
203 sociated conditions, indication for surgery, hepatectomy performed, and hospital type and hepatectomy
205 nd Cdk2 for liver regeneration after partial hepatectomy (PH) by generating corresponding double- and
206 e of cell proliferation that follows partial hepatectomy (PH) identified approximately 1,400 mammalia
207 ), standard (60%), or extended (80%) partial hepatectomy (PH) in mice with and without liver steatosi
209 intrinsic hepatic innervation after partial hepatectomy (PH) in rats and the presence and pattern of
210 UND & AIMS: Liver regeneration after partial hepatectomy (PH) increases the protein folding burden at
213 groups) weeks of treatment with G49, partial hepatectomy (PH) was performed, and all mice were mainta
214 iating liver regeneration (LR) after partial hepatectomy (PH), by regulating expression of Cyclin-D1.
215 erent surgical procedures, including partial hepatectomy (PH), intraoperative portal vein ligation (P
216 involved in liver regeneration after partial hepatectomy (PH), to initiate growth, protect liver cell
219 position was recently reported after partial hepatectomy (PHx) in mice, but the role of fibrin(ogen)
220 a(-/-) (NSG) mice that had undergone partial hepatectomy (PHx) represented the best combination of en
221 aKlf6), cell proliferation following partial hepatectomy (PHx) was increased compared to controls.
223 , we challenged liver function after partial hepatectomy (PHx), inducing acute proliferative and meta
228 urrently used in preclinical models: partial hepatectomy, portal ligature or embolization, and radiot
231 on for staged hepatectomy (ALPPS), a 2-stage hepatectomy procedure, has revolutionized the surgical m
233 gment 2-3 transplantation with delayed total hepatectomy (RAPID) from living donor in a patient affec
234 artition and portal vein ligation for staged hepatectomy." RASPE induced safe and profound growth of
235 cal parenchymal resections including central hepatectomy (resection of segments 4, 5, and 8), right a
236 ificantly between patients in 1- and 2-stage hepatectomy, respectively: 37.2 and 34.5 months (P=0.6),
238 inhibited in DeltaEGFR livers after partial hepatectomy, revealing a new function for EGFR kinase ac
241 ression and liver regeneration after partial hepatectomy, suggesting that DBC1/DN-DBC1 transitions pl
243 frequent use of laparoscopy in living donor hepatectomy, the laparoscopic approach has never been re
247 (4.9 [3.5-5.9] vs 6.4 [4.3-12]; P < 0.001), hepatectomy time (70 [42-120] vs 81 [58-207]; P = 0.02),
249 o investigate the relationship between donor hepatectomy time and post-transplant outcome in 12,974 r
257 s donated after circulatory death had longer hepatectomy times than those from brain-dead donors [50
258 allowed for an increased percentage of major hepatectomies to be performed in a purely minimally inva
260 milieu of chronic inflammation links partial hepatectomy to accelerated hepatocarcinogenesis; this su
261 as to evaluate a modified technique of total hepatectomy to limit hazardous liver manipulation and im
262 anvalin A was injected 4 days before partial hepatectomy to natural killer T cells- deficient mice or
271 tformin via oral gavage, after which a donor hepatectomy was performed followed by a standardized col
276 a model of liver regeneration after partial hepatectomy, we found that DN-DBC1 is down-regulated in
283 the collaborative study period, outcomes for hepatectomy were similar for LV and HV (85 vs 507 cases)
284 rvival (RFS) and overall survival (OS) after hepatectomy were worse in patients with double mutation
285 ber of lesions, clinical-risk score, 2-stage hepatectomy) were significantly worse in the HAI group;
286 icantly different in the first 2 years after hepatectomy when the interval was extended from 3 months
289 tients with HCC underwent various anatomical hepatectomies with the Glissonean pedicle approach betwe
290 93-4.62; P < .001), followed by extension of hepatectomy with (OR, 2.76; 95% CI, 1.85-4.77; P = .03)
293 nocarcinoma of right colon, underwent a left hepatectomy with ligation of right portal vein maintaini
296 the morbidity and mortality after anatomical hepatectomy with the Glissonean pedicle approach, and lo
297 ELLS during LT for PLD facilitates total hepatectomy with vena cava and caval flow preservation.
298 way were rapidly upregulated after two-third hepatectomy, with the ubiquitin ligase Nedd4-1 being a t
299 ally impedes tumorigenesis following partial hepatectomy without compromising survival or liver mass
300 ateral sectionectomy, and 3 underwent a left hepatectomy without middle hepatic vein procurement.