ライフサイエンスコーパス: hepatic artery thrombosis

1 he primary graft and 1 from complications of hepatic artery thrombosis).

2 lure due to chronic rejection and late onset hepatic artery thrombosis.

3 nsplantation, was frequently associated with hepatic artery thrombosis.

4 ation is associated with a high incidence of hepatic artery thrombosis.

5 thrombosis) enable prediction of subsequent hepatic artery thrombosis.

6 No patient had evidence of hepatic artery thrombosis.

7 No differences were observed in rates of hepatic artery thrombosis.

8 he most common etiologies of graft loss were hepatic artery thrombosis (33.4%), acute or chronic reje

9 days following primary transplant) included hepatic artery thrombosis (5), chronic rejection (4), se

10 and the main cause for retransplantation was hepatic artery thrombosis (6 [2%] vs 17 [6%]).

11 ow Protein S may be causally associated with hepatic artery thrombosis after OLT.

12 ecurrent cytomegalovirus activation, one has hepatic artery thrombosis and one is likely to have pers

13 Hepatic artery thrombosis and portal vein thrombosis (PV

14 In an attempt to decrease the incidence of hepatic artery thrombosis and to increase collaboration

15 function, 50% for chronic rejection, 60% for hepatic artery thrombosis, and 60% for recurrent HCV.

16 Complications like acute rejection, hepatic artery thrombosis, and biliary issues still impa

17 -grafts were constructed for recipients with hepatic artery thrombosis, and double donor arteries wer

18 ence of ischemic type biliary strictures and hepatic artery thrombosis, and evaluated the causes of g

19 uded primary nonfunction, chronic rejection, hepatic artery thrombosis, and recurrent disease.

20 transplants; mainly for primary nonfunction, hepatic artery thrombosis, and recurrent primary disease

21 he main indication for retransplantation was hepatic artery thrombosis, and the major cause of death

22 Although duplex US remains a good screen for hepatic artery thrombosis, angiography is strongly recom

23 ed (more than 4 weeks after transplantation) hepatic artery thrombosis are less clearly defined.

24 er recipients who smoke have higher rates of hepatic artery thrombosis, biliary complications, and ma

25 allograft dysfunction, primary nonfunction, hepatic artery thrombosis, biliary ischemia, graft failu

26 itis C virus, longer length of stay at LDLT, hepatic artery thrombosis, biliary stricture, infection,

27 ve a significantly higher incidence of early hepatic artery thrombosis compared with non-FAP transpla

28 Early hepatic artery thrombosis (eHAT) after liver transplanta

29 reported lower rates of primary nonfunction, hepatic artery thrombosis, graft failure at 5 y, or reci

30 The incidence of hepatic artery thrombosis has decreased from 22% to 0% w

31 ansplant recipients were found to have early hepatic artery thrombosis (HAT) after a median of 7 post

32 tation, and outcome of management of delayed hepatic artery thrombosis (HAT) after liver transplant (

33 Hepatic artery thrombosis (HAT) after liver transplantat

34 Whilst causes of hepatic artery thrombosis (HAT) after liver transplantat

35 Hepatic artery thrombosis (HAT) after liver transplantat

36 igate whether center volume impacts the rate hepatic artery thrombosis (HAT) and patient survival aft

37 Although the clinical features of early hepatic artery thrombosis (HAT) are well defined, the fe

38 Hepatic artery thrombosis (HAT) can be a devastating com

39 Hepatic artery thrombosis (HAT) did not occur in any pat

40 The incidence of hepatic artery thrombosis (HAT) following orthotopic liv

41 Hepatic artery thrombosis (HAT) increases morbidity and

42 Hepatic artery thrombosis (HAT) is a cause of morbidity

43 trahepatic biliary strictures (IHBS) without hepatic artery thrombosis (HAT) is a serious complicatio

44 Hepatic artery thrombosis (HAT) is a significant cause o

45 Hepatic artery thrombosis (HAT) remains a devastating co

46 The most common predisposing factor was hepatic artery thrombosis (HAT), which occurred in eight

47 g retransplantation for either NAS (n = 18), hepatic artery thrombosis (HAT; n = 13), or nonbiliary g

48 reasons (primary graft nonfunction (PGNF) or hepatic artery thrombosis [HAT]), survival was significa

49 Other causes for re-OLT included hepatic artery thrombosis in 10 (2.6%), chronic rejectio

50 e believe that emergent revascularization of hepatic artery thrombosis in asymptomatic patients and r

51 ical implications in the prevention of early hepatic artery thrombosis in FAP patients after liver tr

52 A single vascular complication occurred (one hepatic artery thrombosis in group B).

53 nastomosis and should lower the incidence of hepatic artery thrombosis in the small-caliber left hepa

54 An increased rate of graft failure due to hepatic artery thrombosis <=14 days from initial LT was

55 I >25 kg/m 2 ; for the recipient: absence of hepatic artery thrombosis, mechanical ventilation, glome

56 ibed as an independent risk factor for early hepatic artery thrombosis, more studies to understand th

57 ventional angiography or surgery: transplant hepatic artery thrombosis (n = 3) or stenosis (n = 3), p

58 acute rejection at day 7, the development of hepatic artery thrombosis, nonanastomotic biliary strict

59 Reoperations identified two cases of hepatic artery thrombosis not previously identified by d

60 In addition, we reported two cases of hepatic artery thrombosis, one case of wound dehiscence

61 ly because of technical difficulties such as hepatic artery thrombosis or as a result of early allogr

62 hepatic artery flow with higher incidence of hepatic artery thrombosis (P = 0.043) and biliary compli

63 titis C (P<0.0001), as well as occurrence of hepatic artery thrombosis (P=0.0018) and prolonged cold

64 The incidence of hepatic artery thrombosis, portal vein stenosis/thrombos

65 There were no significant differences in hepatic artery thrombosis, portal vein thrombosis, prima

66 ant centers in North America with the lowest hepatic artery thrombosis rate and biliary complication

67 en advocated and has resulted in a decreased hepatic artery thrombosis rate in both the adult and ped

68 d similar ICU stay length (p = .22), 3-month hepatic artery thrombosis rates (4.4% vs 4.0%; p = .9),

69 Complications included one case each of hepatic artery thrombosis requiring retransplantation, b

70 reas 97 patients required retransplantation; hepatic artery thrombosis was the most common indication

71 uding those for primary nonfunction (PNF) or hepatic artery thrombosis were excluded.

72 biliary strictures, primary nonfunction and hepatic artery thrombosis were observed in the total coh

73 The occurrence is, partly, attributed to hepatic artery thrombosis, which is considered to be the

74 Early detection of hepatic artery thrombosis, with subsequent correction an