ライフサイエンスコーパス: hepatitis A virus

1 as identified recently as a receptor for the hepatitis A virus.

2 ths were Salmonella Typhi, Taenia solium and hepatitis A virus.

3 fectively prevented with vaccination against hepatitis A virus.

4 rent infection as the community reservoir of hepatitis A virus.

5 e genome is conserved among strains, such as hepatitis A virus.

6 solate, respectively, of the HM175 strain of hepatitis A virus.

7 the parameters for the ubiquitination of the hepatitis A virus 3C protease are K(m) = 20 +/- 5 microm

8 A high seroprevalence of hepatitis A virus (81%) among human immunodeficiency vir

9 we amplified virtually the entire genomes of hepatitis A virus (a member of the Picornaviridae family

10 Here, we show that hepatitis A virus, a positive-strand RNA virus responsib

11 and genetic depletion studies, we show that hepatitis A virus, an hepatotropic picornavirus, broadly

12 andomization, 1414 (31%) were susceptible to hepatitis A virus and 1090 were eligible for the per-pro

13 However, recent work on hepatitis A virus and hepatitis E virus challenges this

14 multiple human maladies, yet currently, only hepatitis A virus and poliovirus can be controlled with

15 ad through the oral-fecal route (salmonella, hepatitis A virus), and organisms spread through direct

16 , Chlamydia pneumoniae, Helicobacter pylori, hepatitis A virus, and herpes simplex virus-1 were measu

17 Prevalence of antibodies to hepatitis A virus (anti-HAV) and hepatitis E virus (anti

18 fter vaccination were tested for antibody to hepatitis A virus (anti-HAV) by ELISA.

19 f passively transferred maternal antibody to hepatitis A virus (anti-HAV) on the duration of seroposi

20 Antibodies to hepatitis A virus (anti-HAV) were measured in children f

21 es of the encephalomyocarditis virus and the hepatitis A virus are both type III substrates for the m

22 rhinovirus, encephalomyocarditis virus, and hepatitis A virus) are morphologically similar, comprisi

23 enteric viruses, such as Human Norovirus and Hepatitis A Virus, are readily transmitted via the fecal

24 ne response to vaccinations directed against Hepatitis A virus as well as SARS-CoV-2.

25 cally linked to asthma and is a receptor for hepatitis A virus, but the endogenous ligand of TIM-1 is

26 an KIM-1 exhibits homology to a monkey gene, hepatitis A virus cell receptor 1 (HAVcr-1), which was i

27 The study was performed on Hepatitis A Virus Cell Receptor-1 protein, also known as

28 lar injury [KIM-1 (kidney injury molecule-1)/Hepatitis A virus cellular receptor 1 ( Havcr1)] were ev

29 The hepatitis A virus cellular receptor 1 (HAVcr-1) cDNA cod

30 The hepatitis A virus cellular receptor 1 (HAVcr-1) cDNA was

31 e toxin binds to protein receptors including hepatitis A virus cellular receptor 1 (HAVCR1), but the

32 The hepatitis A virus cellular receptor 1 (HAVCR1/TIM1), a m

33 expression of the inhibitory receptors PD-1, hepatitis A virus cellular receptor 2 (TIM3), lymphocyte

34 in-containing protein 3 (TIM3, also known as hepatitis A virus cellular receptor 2), and their respec

35 e disease in countries with very low and low hepatitis A virus endemicity, and universal childhood va

36 munized sequentially with tetanus toxoid and hepatitis A virus failed to develop antibody to either a

37 Sequences of hepatitis A virus from all 170 patients who were tested

38 Hepatitis A virus genotype IB was identified in 144 of 1

39 Hepatitis A virus genotype IB, uncommon in the Americas,

40 dministered to persons after exposure to the hepatitis A virus has not been compared directly with im

41 Herein, we show that hepatitis A virus (HAV) 3C protease (3Cpro) cleaves NEMO

42 Vaccination provides long-term immunity to hepatitis A virus (HAV) among the general population, bu

43 lages in rural Alaska between 2 epidemics of hepatitis A virus (HAV) and after the second epidemic (1

44 ess the protective 190/4 epitope, they bound hepatitis A virus (HAV) and gained limited susceptibilit

45 ZCCHC14 is an essential host factor for both hepatitis A virus (HAV) and hepatitis B virus (HBV).

46 Hepatitis A virus (HAV) and hepatitis C virus (HCV) are

47 a child who died of FVH upon infection with hepatitis A virus (HAV) at age 11 yr and who was homozyg

48 irus B3, human rhinovirus 14, mengovirus, or hepatitis A virus (HAV) capsid proteins were supplied in

49 To determine whether hepatitis A virus (HAV) could tolerate the insertion of

50 Hepatitis A virus (HAV) differs from other members of th

51 Hepatitis A virus (HAV) encodes a single polyprotein whi

52 s (eHAV) are infectious and the only form of hepatitis A virus (HAV) found circulating in blood durin

53 little is known of the mechanisms underlying hepatitis A virus (HAV) genome replication.

54 Hepatitis A virus (HAV) has been adapted to grow efficie

55 Hepatitis A virus (HAV) immunoassays use cell culture-de

56 Fecal excretion of hepatitis A virus (HAV) in 18 patients with HAV infectio

57 deoxynojirimycin] inhibit the replication of hepatitis A virus (HAV) in cell culture (50% inhibitory

58 Replication of hepatitis A virus (HAV) in cultured cells is inefficient

59 r highly specific and sensitive detection of hepatitis A virus (HAV) in food and water are of particu

60 The possible association between hepatitis A virus (HAV) infection and coronary artery di

61 Hepatitis A virus (HAV) infection can stimulate the prod

62 Acute liver failure (ALF) due to hepatitis A virus (HAV) infection is an uncommon but pot

63 We describe a murine model of hepatitis A virus (HAV) infection that recapitulates cri

64 Hepatitis A virus (HAV) infection typically resolves wit

65 on has dramatically reduced the incidence of hepatitis A virus (HAV) infection, but new infections co

66 n of HEV infection has broad similarities to hepatitis A virus (HAV) infection, with most cases being

67 ed prospectively in natural and experimental hepatitis A virus (HAV) infection.

68 Hepatitis A virus (HAV) infects African green monkey kid

69 Hepatitis A virus (HAV) infects African green monkey kid

70 Remarkably, the hepatitis A virus (HAV) IRES requires eIF4E for its tran

71 IMPORTANCE Hepatitis A virus (HAV) is a common cause of viral hepat

72 Hepatitis A virus (HAV) is a common infection that is tr

73 Hepatitis A virus (HAV) is a hepatotropic picornavirus t

74 Hepatitis A virus (HAV) is an ancient and ubiquitous hum

75 Hepatitis A virus (HAV) is an hepatotropic human picorna

76 Although hepatitis A virus (HAV) is associated only with acute he

77 Unlike other picornaviruses, hepatitis A virus (HAV) is cloaked in host membranes whe

78 Hepatitis A virus (HAV) is naturally transmitted by the

79 the high-risk groups for vaccination against hepatitis A virus (HAV) is unlikely to have a significan

80 The genetic relatedness of hepatitis A virus (HAV) isolates was determined to ident

81 The current Hepatitis A virus (HAV) molecular epidemiology in Israel

82 Coinfection with hepatitis A virus (HAV) or hepatitis B virus (HBV) in pa

83 The human homolog of TIM-1 is the hepatitis A virus (HAV) receptor, which may explain the

84 Hepatitis A virus (HAV) remains enigmatic, despite 1.4 m

85 A common cause of acute hepatitis in humans, hepatitis A virus (HAV) replicates within hepatocytes wi

86 unclear whether NK cells are elevated due to hepatitis A virus (HAV) replication and ongoing associat

87 apy for hepatitis A, and many aspects of the hepatitis A virus (HAV) replication cycle remain to be e

88 and accurate molecular diagnostic tools for hepatitis A virus (HAV) RNA detection, subgenotype ident

89 Initiation of translation of hepatitis A virus (HAV) RNA occurs by internal entry and

90 otein poly(rC) binding protein 2 (PCBP2) for hepatitis A virus (HAV) RNA translation.

91 Hepatitis A virus (HAV) superinfection in persons with h

92 Hepatitis A virus (HAV) superinfection is associated wit

93 However, hepatitis A virus (HAV) temporarily inhibits Treg-cell f

94 ody (MAb) 190/4, which blocks the binding of hepatitis A virus (HAV) to AGMK cells.

95 lonal antibody (MAb) 190/4 blocks binding of hepatitis A virus (HAV) to the HAV cellular receptor 1 (

96 tion between homelessness and infection with hepatitis A virus (HAV) using a test-negative study desi

97 l responses (Seroresponse) and durability of hepatitis A virus (HAV) vaccination are reduced among hu

98 or Disease Control and Prevention recommends hepatitis A virus (HAV) vaccination for all children at

99 Hepatitis A virus (HAV) vaccination is recommended in ch

100 Universal 2-dose hepatitis A virus (HAV) vaccination of toddlers effectiv

101 ve no serological responses to their primary hepatitis A virus (HAV) vaccination or have seroreversio

102 h men (MSM) receiving two and three doses of hepatitis A virus (HAV) vaccine and HIV-uninfected MSM r

103 the safety and immunogenicity of 2 doses of hepatitis A virus (HAV) vaccine followed by a booster do

104 lity of virulent hepatitis E virus (HEV) and hepatitis A virus (HAV) was compared.

105 The AGM-27 strain OF hepatitis A virus (HAV) was originally isolated from an

106 ' nontranslated region in the replication of hepatitis A virus (HAV) was studied by analyzing the tra

107 uman (HM-175) and simian (AGM-27) strains of hepatitis A virus (HAV) were constructed to evaluate the

108 tream of the internal ribosome entry site of Hepatitis A virus (HAV) were studied, a 35mer (HAV-35),

109 Among these, hepatitis A virus (HAV), a common cause of acute hepatit

110 Here, we show that hepatitis A virus (HAV), a hepatotropic picornavirus, ab

111 f the internal ribosome entry site (IRES) of Hepatitis A virus (HAV), a picornavirus.

112 of positive-strand RNA viruses that includes hepatitis A virus (HAV), an ancient human pathogen that

113 Hepatitis A virus (HAV), an atypical member of the Picor

114 ed immunization for hepatitis B virus (HBV), hepatitis A virus (HAV), and COVID-19 during natalizumab

115 s known about the mechanism of cell entry of hepatitis A virus (HAV), and the identification of cellu

116 on, all participants were vaccinated against hepatitis A virus (HAV), and the increase of antibody ti

117 Many 'non-enveloped' viruses, including hepatitis A virus (HAV), are released non-lytically from

118 Hepatitis A virus (HAV), classified within the genus Hep

119 ition caused by hepatotropic viruses such as hepatitis A virus (HAV), hepatitis B virus (HBV), and HS

120 IgG antibodies to cytomegalovirus (CMV), hepatitis A virus (HAV), herpes simplex virus type 1 (HS

121 'NTR) of an attenuated, cell culture-adapted hepatitis A virus (HAV), HM175/P16, enhance growth in cu

122 d States was surveyed for antibody titers to hepatitis A virus (HAV), measles virus (MeV), and cytome

123 During infection with the hepatitis A virus (HAV), most patients develop mild or a

124 ad outbreaks of person-to-person transmitted hepatitis A virus (HAV), particularly among people who i

125 Human wild-type (wt) hepatitis A virus (HAV), the causative agent of acute he

126 y a stem-loop structure with cre function in hepatitis A virus (HAV), the type species of this genus,

127 Hepatitis A virus (HAV), unlike other picornaviruses, ha

128 ial serological studies were consistent with hepatitis A virus (HAV), with prozone phenomenon.

129 (HepA) vaccination recommendations in 2006, hepatitis A virus (HAV)-associated outbreaks have increa

130 rticipate in cargo delivery from exosomes of hepatitis A virus (HAV)-infected cells (exo-HAV) by clat

131 e period were negative for IgM antibodies to hepatitis A virus (HAV).

132 a cytopathogenic mutant (HM 175/24a) of the hepatitis A virus (HAV).

133 vaccine-preventable infection caused by the hepatitis A virus (HAV).

134 have had exposure and resultant immunity to hepatitis A virus (HAV).

135 se and control subjects were also tested for hepatitis A virus (HAV).

136 These results pertain to hepatitis A virus (HAV).

137 udies show that some picornaviruses, notably hepatitis A virus (HAV; genus Hepatovirus) and some memb

138 respond to a recall antigen and neoantigen (hepatitis A virus [HAV] vaccine) after 3 vaccinations.

139 Tuberculosis organisms, hepatitis A virus, hepatitis B virus, and measles virus

140 chimpanzee sera; sera from chimpanzees with hepatitis A virus, hepatitis B virus, or hepatitis C vir

141 actions can help rapidly detect and control hepatitis A virus illness caused by imported food.

142 ecovered from specimens from 117 people with hepatitis A virus illness.

143 Mutations which positively affect growth of hepatitis A virus in cell culture may negatively affect

144 uring serial passage of the HM-175 strain of hepatitis A virus in MRC-5 cell cultures in order to det

145 To better understand transmission of hepatitis A virus in such countries, the authors prospec

146 itis B virus now joins hepatitis C virus and hepatitis A virus in targeting the same innate immune re

147 ibility to immunoinflammatory conditions and hepatitis A virus-induced liver failure, which is though

148 der had a significantly higher prevalence of hepatitis A virus infection (37%) than children living i

149 Hepatitis A virus infection (HAV), hepatitis B virus inf

150 ica, DRB1*1301 is associated with protracted hepatitis A virus infection which may enhance exposure t

151 NK cells respond during the early stages of hepatitis A virus infection, we generated mice that are

152 In May, 2013, an outbreak of symptomatic hepatitis A virus infections occurred in the USA.

153 84- angstrom resolution crystal structure of hepatitis A virus IRES domain V (dV) in complex with a s

154 passively transferred maternal antibodies to hepatitis A virus (maternal anti-HAV) may lower the infa

155 l bacteria in the gastrointestinal tract and hepatitis A virus, may normally induce the development o

156 erved as controls, with infections by either hepatitis A virus or hepatitis B virus (HBV), or a nonin

157 tion from transcripts containing the IRES of hepatitis A virus or hepatitis C virus in BS-C-1 cells a

158 s community was largely spared from a recent hepatitis A virus outbreak in San Diego, California.

159 that the heterogeneous mixture of infectious hepatitis A virus particles (virions and provirions) typ

160 ore protein peptide, whereas no binding to a hepatitis A virus peptide control was observed.

161 When the growth kinetics of immature hepatitis A virus provirions and mature virions were mon

162 ing information, and did genetic analysis of hepatitis A virus recovered from patients' serum and sto

163 This virus family includes poliovirus, hepatitis A virus, rhinoviruses, and Coxsackieviruses.

164 Hepatitis A virus RNA detected in clinical specimens was

165 ad elevated titers of antibody to H. pylori, hepatitis A virus, rotavirus, and malaria at the outset,

166 unoglobulins A (IgA), G (IgG), and M (IgM)], hepatitis A virus, rotavirus, tetanus toxoid (IgG), and

167 ce (55% vs 55%) for patients with cirrhosis, hepatitis A virus screening (69% vs 56%), hepatitis C vi

168 patitis A (HepA) vaccine was assessed in 133 hepatitis A virus-seronegative, human immunodeficiency v

169 In contrast, hepatitis A virus seroprevalence increased with age.

170 idence interval: 0.8, 2.9) of H. pylori with hepatitis A virus seroprevalence that weakened after adj

171 tion since proteins of hepatitis C virus and hepatitis A virus similarly bind IPS-1 and target it for

172 ible for the normally asynchronous nature of hepatitis A virus uncoating kinetics.

173 lace, and postexposure prophylaxis with both hepatitis A virus vaccine and immunoglobulin was provide

174 to 18 years of age) to receive MenB-FHbp or hepatitis A virus vaccine and saline and assigned 3304 y

175 ore reactions at the injection site than the hepatitis A virus vaccine and saline.

176 were enrolled (HPV vaccine group n=3727 and hepatitis A virus vaccine control group n=3739).

177 trial phase, after which participants in the hepatitis A virus vaccine control group were provided th

178 through amplification and sequencing of the hepatitis A virus VP1-P2B junction region.

179 Symptomatic infection with hepatitis A virus was confirmed in 25 contacts receiving

180 No hepatitis A virus was detected in product B.

181 Hepatitis A virus was sequenced from serologic specimens

182 We conclude that for species such as hepatitis A virus with high levels of sequence conservat