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1  stress and elevated CO(2) in the absence of herbicide.
2 s of algae induced by the presence of Diuron herbicide.
3 hosate, the most widely applied agricultural herbicide.
4  leading to the development of new non-toxic herbicides.
5 ncentrations, on the sensitivity of weeds to herbicides.
6 ronmentally relevant concentrations of these herbicides.
7 active ingredient of thousands of commercial herbicides.
8 ed to identify new modes of action and novel herbicides.
9 tential of such compounds as antibiotics and herbicides.
10 and would be more useful for the delivery of herbicides.
11 overies were achieved with RSDs<20% for most herbicides.
12 ncluding incineration and the manufacture of herbicides.
13 ed clover, and alfalfa were not treated with herbicides.
14  weed growth and were developed as bleaching herbicides.
15 ndervine environment have relied on chemical herbicides.
16 e important for the sustainable use of these herbicides.
17 xemplified by the evolution of resistance to herbicides.
18  evaluate the toxic effects of a wildly-used herbicide 2,4-D (2,4-dichlorophenoxyacetic acid) on the
19                        The synthetic auxinic herbicide 2,4-dichlorophenoxyacetic acid (2,4-D) has bee
20  followed by the fungicide propiconazole and herbicide 2,4-dichlorophenoxyacetic acid (2,4-D).
21 low-herbicide regime as for the conventional-herbicide 2-year rotation.
22 veral pesticides including the commonly used herbicides 2,4-D and glyphosate, the insecticides permet
23 legged partridge (Alectoris rufa) eggs to an herbicide (2,4-D) and a fungicide (tebuconazole) applied
24 d evidence of associations with RCC for four herbicides (2,4,5-T, atrazine, cyanazine, and paraquat)
25 It is the target for more than 50 commercial herbicides(2).
26                            Seven pesticides (herbicides: 2,4-D and simazine; insecticides: carbaryl,
27 tion x herbicide combinations except the low-herbicide 3-year rotation, which contained approximately
28 oisomers of chiral synthetic auxins and AOPP herbicides, 3) spectroscopic data that demonstrate the c
29 16 fungicides (52%), 8 insecticides (26%), 2 herbicides (6%), 3 pesticide derivatives (10%), 1 insect
30 ne common contaminants (five antibiotics, an herbicide, a beta-blocker, an antidepressant, and an ant
31  Despite the continued use of picloram as an herbicide, a transporter for picloram was not known unti
32 er exposed to each pesticide alone, a mix of herbicides, a mix of insecticides, or a mix of all five
33 s as well as other insecticides, fungicides, herbicides, acaricides, growth regulators and veterinary
34  and (ii) compound-specific standards of the herbicides acetochlor and S-metolachlor (Aceto2, Metola2
35           Glyphosate is the most widely-used herbicide active ingredient, with $11 billion spent annu
36 ons in genes encoding the protein targets of herbicides, affecting the binding of the herbicide eithe
37 rine insecticides (DDT and lindane) and four herbicides (alachlor, metolachlor, 2,4-D, and pendimetha
38 opionate and aryloxyphenoxypropionate (AOPP) herbicides, albeit with varying substrate enantioselecti
39 stance mechanism and suggested that residual herbicides, alongside chemical diversity, were important
40                            Adsorption of the herbicide amitrole in aqueous solution on granular activ
41  or Rox(V)) have been extensively used as an herbicide and growth enhancers in animal husbandry, resp
42           These evolutionary processes drive herbicide and herbicide-resistant crop development and r
43 ost common genetically engineered traits are herbicide and insect resistance.
44                             Excessive use of herbicide and insecticide causes bioaccumulation in the
45         Portable systems for rapid assays of herbicide and insecticide residues have attracted promin
46 uence the photochemistry of its coformulated herbicide and suggest that such mixture effects should b
47         The models developed to identify the herbicide and the plant phenological stage at which it w
48                  Resistance to 157 different herbicides and 88% of known sites of action has been obs
49 mechanisms of plant resistance to picolinate herbicides and also shed light on the possible endogenou
50                                 Agricultural herbicides and daidzein inputs were primarily via upstre
51 ith an overall detection of 34 PPPs and with herbicides and fungicides found in equal proportions.
52                We show consistent effects of herbicides and insecticides on ecosystem function, and s
53                    However, in comparison to herbicides and insecticides, the exposure to and effects
54 enoxypropionate synthetic auxin or with AOPP herbicides and of AAD-2, which has the opposite (S)-enan
55 is at the base of the exceptional potency of herbicides and potentially a target for the discovery of
56        Terbuthylazine (TBA) is a widely used herbicide, and its heterogeneous reaction with OH radica
57                                    Nutrient, herbicide, and sediment loading from agricultural fields
58 a target for the development of antibiotics, herbicides, and antimalarial drugs.
59 oducts, such as pharmaceuticals, pesticides, herbicides, and crop protection agents.
60 ll molecules are used commercially as drugs, herbicides, and fungicides in different systems, but in
61 of most water, minimal use of pesticides and herbicides, and no nutrient losses.
62  organophosphorus insecticides, phenoxy-acid herbicides, and triazine herbicide) to inhibit butyrylch
63 is biosynthetic route a promising target for herbicides, antifungal agents, and antibiotics.
64                                              Herbicide application did not significantly increase res
65 vering of soil with sand or irrigation after herbicide application.
66 2) levels (750 ppm) prior to, and following, herbicide application.
67 differences between seeds due to pre-harvest herbicides application.
68 rant (GMHT) crops, for example, might reduce herbicide applications and increase crop yields, but rem
69 asing crop rotation diversity while reducing herbicide applications may maintain effective weed contr
70    All were managed with conventional or low herbicide applications.
71 % of maize, 43% of soybean and 45% of cotton herbicide applications.
72                                         Five herbicides applied at three moments of the plant reprodu
73 mmon in no-till agriculture situations where herbicides are involved in crop management.
74 tlas demonstrates why some widely used auxin herbicides are not, or are very poor substrates.
75   During wheat cultivation, glyphosate-based herbicides are recommended to be applied a week prior to
76                                  When parent herbicides are still applied, metabolites are constantly
77                                              Herbicides are the most commonly used means of controlli
78 b-DHAD is inhibited by a recently discovered herbicide, aspterric acid, that, given the essentiality
79                                    Moreover, herbicide assays and sequencing revealed that French teo
80 pea seeds produced using different desiccant herbicides at harvest anticipation.
81 rt approach enabled chip-HPLC-MS analysis of herbicides at the highest pressure and speed.
82  weeks) exposed to two concentrations of the herbicide atrazine (100 and 250 mug L(-1)).
83                              The widely-used herbicide atrazine (ATZ) is detected in ground and surfa
84 generational effects promoted by widely used herbicide atrazine (ATZ).
85                             For example, the herbicide atrazine affects host defences (e.g. resistanc
86 was used for probing toxicological effect of herbicide atrazine on the algae's lipidome, demonstratin
87                                          The herbicide atrazine, a suspected endocrine disrupting che
88 se-dependent responses to the PSII-inhibitor herbicides atrazine and diuron, frequently detected in w
89 iotics that frequently contaminate hives-two herbicides (atrazine and glyphosate) and three fungicide
90 amine the single and combined effects of two herbicides (atrazine, glyphosate), and three insecticide
91 tope analysis (Cl-CSIA) of three chlorinated herbicides, atrazine, acetochlor, and metolachlor, which
92 o six pesticides (insecticide: clothianidin; herbicides: atrazine, S-metolachlor; fungicides: azoxyst
93            No AUX1 activity was observed for herbicides based on benzoic acid (dicamba), pyridinyloxy
94                                         Some herbicides bind multiple proteins, making the evolution
95  mass spectrometry also show that when these herbicides bind, thiamine diphosphate (ThDP) is modified
96  pyrimidinyl group inserted deepest into the herbicide binding site.
97 iazolinone ring is inserted deepest into the herbicide binding site.
98 ns reduced toxicity and system dependence on herbicides by 25-51%.
99 tes that pre-harvest use of glyphosate-based herbicides can cause significant differences in wheat pr
100 and classified as glyphosate, non-glyphosate herbicides, chemical lepidopteran insecticides, biologic
101 hloridazon (DPC), the main metabolite of the herbicide chloridazon (CLZ), is more water soluble and p
102  (DPC), the major degradation product of the herbicide chloridazon.
103 e antibacterial agent chloroxylenol, and the herbicide chloroxynil.
104 tance (NTSR) mechanisms have evolved to most herbicide classes.
105 cals and the triazine and phenoxyacetic acid herbicide classes.
106  soybean was <25 kg ha(-1) in all rotation x herbicide combinations except the low-herbicide 3-year r
107  agricultural communities may be affected by herbicides commonly used for weed management via several
108 g TWA concentrations that was independent of herbicide concentrations ranging over 2 orders of magnit
109 lleles are robust substitutes for antibiotic/herbicide-dependent marker genes as well as surprisingly
110 y environmental analysis and more especially herbicide detection.
111 lic detoxification, but genes encoding these herbicide-detoxifying enzymes have yet to be identified.
112 e by inducing the expression and activity of herbicide-detoxifying enzymes.
113                         Other pesticides and herbicides did not contribute to photoluminescence recov
114 e fed leaf material that had been exposed to herbicides did not.
115                          The strength of the herbicide disruption effect is characterized further by
116  for two reference toxicants, copper and the herbicide diuron, for tropical marine species.
117 that exposure to glyphosate (a commonly used herbicide) does not result in glyphosate bioaccumulation
118                  Progenies were subjected to herbicide dose-response studies following drift selectio
119                                              Herbicide drift exposure rapidly selected for Amaranthus
120                                     Repeated herbicide drift exposure to weeds could be detrimental t
121 lants were used for the subsequent rounds of herbicide drift exposure.
122 hemp populations were recurrently exposed to herbicide drift in a wind tunnel study over two generati
123 ormation is available on the consequences of herbicide drift in non-target plants.
124                                The impact of herbicide drift in sensitive crops is extensively invest
125                            In both biotypes, herbicide effectiveness was reduced when plants were sub
126  of herbicides, affecting the binding of the herbicide either at or near catalytic domains or in regi
127 n of 2,4-Dichlorophenoxy Acetic Acid (2,4-D) herbicide either in standard solution and spiked real sa
128 6%), stability and a LOD (0.3ppb) lower than herbicide emission limits.
129 o 2,4-D, glyphosate and ammonium glufosinate herbicides (event DAS-444O6-6) and insect-resistant due
130                  Insects directly exposed to herbicides experienced high mortality; while those fed l
131                              Five commercial herbicide families inhibit acetohydroxyacid synthase (AH
132 he sulfonylamino-carbonyl-triazolinone (SCT) herbicide families, revealing the structural basis for t
133            Glyphosate is the most widespread herbicide for weed management, being extensively used in
134 commonly added to commercial chloroacetamide herbicide formulations and widely used worldwide, but th
135 large number of biocidal active ingredients (herbicides, fungicides, and insecticides); we then selec
136 PA commissioned screening of 320 pesticides, herbicides, fungicides, and other chemicals in a series
137 nmentally relevant doses of glyphosate-based herbicides (GBH) is still a debatable and unresolved mat
138               Controversial glyphosate-based herbicides (GBHs) are the most frequently used herbicide
139 rbicides (GBHs) are the most frequently used herbicides globally.
140 sed agricultural pesticides worldwide is the herbicide glyphosate (N-(phosphonomethyl)glycine), commo
141 itive response to RT, whereas the use of the herbicide glyphosate did not significantly affect earthw
142 ubsequently determined to be tolerant to the herbicide glyphosate in greenhouse spray tests.
143 phosphate and in plants is the target of the herbicide glyphosate.
144  commonly used fungicide carbendazim and the herbicide glyphosate.
145 rganic acids of general interest such as the herbicides glyphosate, ethephon, and fosamine and arseni
146    The first two trials tested the effect of herbicide (glyphosate) application on plant survival and
147 c variation) to the world's most widely used herbicide (glyphosate) in a major, economically damaging
148                                 A mixture of herbicides had no effect on microbial diversity or struc
149            The selection pressure exerted by herbicides has led to the repeated evolution of herbicid
150 ocedure for analysis of difficult high polar herbicides (HPH) in diverse foods of plant origin.
151 ntally relevant concentrations of widespread herbicides (i.e., 0.5-5 mug/L in water before extraction
152                In this study with the chiral herbicide imazamox, biphasic degradation was observed in
153 n factors (92, 96 and 98) determined for the herbicides, IMP, IMZ and IMT, respectively, were greatly
154                The widespread application of herbicides impacts surface water and groundwater.
155 w unrelenting use of a single mode of action herbicide in agricultural weed control drives genetic ad
156 hylethyl)-triazine-2,4-diamine), a prevalent herbicide in the United States, is often used along trib
157 eous determination of 120 various classes of herbicides in oilseed samples.
158 eveloped for the detection of photosynthetic herbicides in river water.
159 c activity to reduce alachlor, a chlorinated herbicide, in the aqueous medium at different pH values.
160 sistance to glyphosate, the most widely used herbicides, in several weed species, including common wa
161                                              Herbicides included fenoxaprop-p-ethyl (140 g ha(-1)), t
162 volatilization of carboxylic acid-containing herbicides including dicamba.
163                                              Herbicides increase crop yields by allowing weed control
164 nage weeds, the widespread adoption of these herbicides increased the risk for herbicide spray drift
165 usly implicated in increasing herbicide use, herbicide increases were more rapid in non-GE crops.
166                                Additionally, herbicides indirectly increased host susceptibility and
167            It is applied to study the Diuron herbicide influences on algal metabolism, evidencing flu
168 s organophosphates) and two pesticide types (herbicides, insecticides) applied at standardised enviro
169 nalyses have used flawed metrics to evaluate herbicide intensity and toxicity trends.
170  an association between chlorimuron ethyl, a herbicide introduced in 1986, and lung cancer that has n
171               The evolution of resistance to herbicides is a striking example of rapid, human-directe
172                      The popularity of these herbicides is due to their low application rates, high c
173 at (Triticum aestivum L.) to synthetic auxin herbicides is primarily due to rapid metabolic detoxific
174                              Reckless use of herbicides like butachlor (Buta) in the fields represent
175                            We found that the herbicide linuron amplifies astrocyte pro-inflammatory a
176 le for in situ degradation of the phenylurea herbicide linuron in a BPS matrix.
177                                       Third, herbicides may alter the quantity and composition of wee
178                                       First, herbicides may cause direct mortality to insects present
179 s suggests that a superior ability to resist herbicides may come at a cost to overall plant fitness.
180                                      Second, herbicides may induce plant defenses that increase resis
181             For example, enzymes involved in herbicide metabolism-based resistances include cytochrom
182  dark-light transitions, high light, and the herbicide methyl viologen, rapidly activated GCN2 kinase
183 ticide treatments (control, ethanol vehicle, herbicide mixture, and insecticide mixture) for 14 d to
184       Our results reveal that greater use of herbicide mixtures is associated with lower levels of sp
185 ported across many species and for all major herbicide modes of action.
186 ession in a pathway that is abolished by the herbicide norflurazon.
187 entration-dependent inhibition effect of the herbicide on photosynthesis was demonstrated.
188 ed to investigate the effects of an array of herbicides on the densities of several major pests found
189 imicrobial and anticancer drugs, pesticides, herbicides or fungicides.
190 : 1.3, 3.0)] insecticides and phenoxyacetate herbicides [OR=1.9 (95% CI: 1.2, 2.8)].
191 eneration of agricultural growth regulators, herbicides, or defense compounds.
192       The widely successful use of synthetic herbicides over the past 70 years has imposed strong and
193 method for determination of two bipyridylium herbicides, paraquat and diquat, in cowpeas by UPLC-MS/M
194 hich convey resistance to the broad-spectrum herbicide phosphinothricin (also known as glufosinate) v
195 n the biosynthetic pathway to the commercial herbicide phosphinothricin, uncovered an example of such
196 tance following recurrent-selection with low herbicide rates.
197                   Through bottom-up effects, herbicides reduce respiration and primary productivity b
198              Individual insecticides but not herbicides reduced the microbial diversity and richness
199 3- and 4-year rotations managed with the low-herbicide regime as for the conventional-herbicide 2-yea
200 ta from 2008-2015 showed that use of the low-herbicide regime reduced freshwater toxicity loads by 81
201 tation were managed with conventional or low-herbicide regimes.
202 ics of contrasting crop rotation systems and herbicide regimes.
203                              Most studies of herbicide resistance are performed reactively and focus
204  case of the eccDNA, demonstrate how evolved herbicide resistance can generate new insights into plan
205 edesign of plant cell walls, and deciphering herbicide resistance evolution can lead to the next gene
206 ng-term management as numerous weeds evolved herbicide resistance following recurrent-selection with
207 oncerns related to the use of antibiotic and herbicide resistance genes in the production of transgen
208 sformation are based on either antibiotic or herbicide resistance genes.
209                                              Herbicide resistance has been observed in Chloris trunca
210                                              Herbicide resistance has been studied for more than 60 y
211            Despite its multi-faceted nature, herbicide resistance has rarely been researched in a hol
212  Documenting the diversity of mechanisms for herbicide resistance in agricultural weeds is helpful fo
213                             The evolution of herbicide resistance in crop weeds presents one of the g
214 ection pressure, leading to the evolution of herbicide resistance in hundreds of weed species.
215 bicides has led to the repeated evolution of herbicide resistance in weeds.
216                                              Herbicide resistance in wild grasses is widespread in th
217 ackgrass (Alopecurus myosuroides), for which herbicide resistance is a major economic issue.
218                                              Herbicide resistance is a recurrent evolutionary event t
219 strates and discuss our findings in terms of herbicide resistance management.
220                             The evolution of herbicide resistance on contemporary timescales in turn
221 ates, with both agricultural populations and herbicide resistance only recently emerging in Canada.
222 revealed that French teosintes have acquired herbicide resistance via the introgression of a mutant h
223 -active assessment of adaptive potential for herbicide resistance, and provides compelling evidence o
224 n to a new climatic niche and acquisition of herbicide resistance, thereby fostering the establishmen
225 k was established linking viral infection to herbicide resistance, transcriptome sequencing showed a
226 notype specificity and measurable changes in herbicide resistance.
227             I then develop how one would use herbicide-resistance evolution as a model for studying e
228  circumvent the potential risk of antibiotic/herbicide-resistance gene transfer into neighboring plan
229                            The vast array of herbicide-resistance mechanisms for generalist (NTSR) an
230 ce, led us to sequence the transcriptomes of herbicide resistant and susceptible populations of black
231 wever, the role of gene flow in dispersal of herbicide-resistant alleles among weed populations is po
232 e evolutionary processes drive herbicide and herbicide-resistant crop development and resistance mana
233                                              Herbicide-resistant crops, therefore, are clearly not th
234 tant herbicide-target gene (ACC1) present in herbicide-resistant maize cultivars.
235 plant adaptation and to stimulate the use of herbicide-resistant plants as models for addressing key
236 ultural weed, Ipomoea purpurea, we show that herbicide-resistant populations self-fertilise more than
237       Here, I collate recent research on the herbicide-resistant problem in light of key questions an
238              I highlight discoveries made on herbicide-resistant weeds in three broad areas - the gen
239 ygen evolution, occurring in the presence of herbicides, results in a decrease of current signals by
240 e investigated the dissipation of the chiral herbicide S-metolachlor (SM) in soil in relation to its
241 oxacor is a safener paired with the high-use herbicide S-metolachlor.
242                                              Herbicide safeners are commonly applied in wheat to achi
243 ed for Amaranthus spp. biotypes with reduced herbicide sensitivity over two generations.
244 select Amaranthus spp. biotypes with reduced herbicide sensitivity.
245  of evolved resistance mechanisms, including herbicide sequestration in the vacuole, a rapid cell dea
246 ed to a more complex sample, the analysis of herbicides spiked in the wine matrix.
247 t the optimal control regime (combination of herbicide spray and fire) is difficult to implement, mea
248 n of these herbicides increased the risk for herbicide spray drift to surrounding vegetation.
249                                  Alternative herbicides, such as fomesafen, effectively controlled th
250 th response, nucleotide polymorphisms in the herbicide target (5-enolpyruvylshikimate-3-phosphate syn
251 S), has been identified as a novel, putative herbicide target.
252 resistance via the introgression of a mutant herbicide-target gene (ACC1) present in herbicide-resist
253 3-(3,4-dichlorophenyl)-1,1-dimethylurea), an herbicide that disrupts photosynthetic electron transfer
254            Glyphosate is an organophosphorus herbicide that is applied to the leaves of plants and cr
255                       Picloram is an auxinic herbicide that is widely used for controlling broad leaf
256                         Imazamox is a chiral herbicide that, in laboratory experiments in the dark, e
257 nhibitory activity of many of the commercial herbicides that target AHAS.
258 lved in the biosynthesis of the EPA-approved herbicide thaxtomin A.
259 nomethylglycine, PMG), the world's most used herbicide, the possibility of destruction of stockpiles
260        Applied to mode-of-action analysis of herbicides, the approach showed severe disturbance in th
261 he environmental fate and metabolism of this herbicide; the genes and enzymes responsible for biodegr
262 rematode exposure was observed with triazine herbicides, this effect was driven by increases in snail
263  to determine the effect of glyphosate-based herbicide timing on the chemistry of wheat gluten protei
264 cides, phenoxy-acid herbicides, and triazine herbicide) to inhibit butyrylcholinesterase, alkaline ph
265 ers are commonly applied in wheat to achieve herbicide tolerance by inducing the expression and activ
266 ation of ssODN and CRISPR/Cas9 to develop an herbicide tolerance trait in flax (Linum usitatissimum)
267                    While the introduction of herbicide tolerant crops provided growers new options to
268 l weed management with genetically modified, herbicide-tolerant (GMHT) crops, for example, might redu
269 ng 2 AADs have been deployed commercially in herbicide-tolerant crops.
270 s sp.) host-plants after the introduction of herbicide-tolerant crops.
271                                              Herbicide treatment had no significant impact on environ
272   Each was managed with conventional and low-herbicide treatments.
273 er of these, being instead linked to reduced herbicide uptake and/or translocation.
274 mats in the viticulture setting to eliminate herbicide usage.
275                                              Herbicides usage has resulted in resistance by weeds, al
276  increased, chronic toxicity associated with herbicide use decreased in two out of six crops, while a
277                                      Even as herbicide use increased, chronic toxicity associated wit
278                                Reductions in herbicide use intensity generally did not affect nutrien
279                            Here, I show that herbicide use intensity increased over the last 25 years
280                                              Herbicide use is among the most criticized aspects of mo
281 ave been previously implicated in increasing herbicide use, herbicide increases were more rapid in no
282 netically modified (GM) crops and associated herbicide use.
283 es glyphosate use but reduces non-glyphosate herbicide use; and adoption of GM insect-resistant crops
284            Isoproturon (IPU) is a phenylurea herbicide used to control broad-leaf grasses on grain fi
285 erstand environmental effects on efficacy of herbicides used to control goosegrass (Eleusine indica L
286 gence (POST) wheat-selective synthetic auxin herbicide, using alien substitution (the S genome of Aeg
287 nce standards (45) included pharmaceuticals, herbicides, vehicle-related compounds, plasticizers, and
288 ties that determine their ability to control herbicide volatilization are poorly understood.
289  may best predict their potential to prevent herbicide volatilization.
290                       Atrazine as case study herbicide was detected in a concentration range of 0.1 a
291 ectrophotometric determination of glyphosate herbicide was developed.
292 rage (TWA) concentrations and total loads of herbicides was assessed.
293 arations of chlorpheniramine and phenoxyacid herbicides were achieved without optimizing the MEKC con
294                                              Herbicides were applied to goosegrass maintained at soil
295  Trifluralin is a widely used dinitroaniline herbicide, which can persist in the environment and has
296 rsistent and even more polar than the parent herbicide, which increases the risk of groundwater conta
297 ts are selectively insensitive to picolinate herbicides, while pic30-3 is also defective in chlorate
298 d with a sensor for analysis of paraquat, an herbicide widely used over the world.
299 chanism implicated in enhanced metabolism of herbicides with diverse modes of action.
300  toward EDI compared to other pesticides and herbicides with similar structures such as diazinon, hep

 
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