ライフサイエンスコーパス: herpes

1 s provide protection from symptomatic ocular herpes.

2 ated with protective immunity against ocular herpes.

3 sed immunotherapy to combat recurrent ocular herpes.

4 mmune checkpoints to combat recurrent ocular herpes.

5 ionship exists between tuberculosis risk and herpes coinfection.

6 established HLA Tg rabbit model of recurrent herpes highlight that blocking immune checkpoints combin

7 will help design future T-cell-based ocular herpes immunotherapeutic vaccines.IMPORTANCE A staggerin

8 t immune checkpoint targets to combat ocular herpes.IMPORTANCE HSV-specific tissue-resident memory CD

9 e immune response against symptomatic ocular herpes.IMPORTANCE We investigated the protective role of

10 a significant reduction in recurrent ocular herpes in HLA transgenic (Tg) rabbit model.

11 mportant strategy to combat recurrent ocular herpes in the clinic.

12 associated with protection against recurrent herpes infection and disease.

13 ated with protective immunity against ocular herpes infection and disease.IMPORTANCE We report that n

14 tiviral CD8(+) T cell exhaustion during SYMP herpes infection and pave the way to targeting immune ch

15 ucosa and provide protection against genital herpes infection in mice.

16 d, these cells protect hosts against genital herpes infection.

17 press replication or reactivation of chronic herpes infections.

18 viruses in the Herpesviridae family, causes herpes labialis (cold sores) and keratitis (inflammation

19 Typical herpes lesion site biopsy (TLSB) and cervical biopsy spe

20 severity and frequency of recurrent genital herpes lesions.

21 Herpes ocular infections was suspected.

22 in the guinea pig model of recurrent genital herpes, of subunit vaccine candidates that were based on

23 isms of protection against recurrent genital herpes remain to be fully elucidated.

24 ere highly prevalent in bile after LT, while herpes simpex virus 1 and 2 (HSV-1, HSV-2), varicella-zo

25 Fatal herpes simplex encephalitis (HSE) results from immune pa

26 infection (including cytomegalovirus [CMV], herpes simplex I/II or varicella zoster virus [HSV/VZV],

27 Ocular herpes simplex keratitis (HSK) is a consequence of viral

28 Herpes simplex keratitis (HSK), caused by herpes simplex

29 r clinical trial of 106 patients with active herpes simplex stromal keratitis who had not received an

30 flammation and in shortening the duration of herpes simplex stromal keratitis.

31 be structural organization of the virions of Herpes Simplex Type 1 viruses and bacteriophage MS2.

32 umanized monoclonal IgG antibody against the herpes simplex viral protein glycoprotein D (gD) was rad

33 We developed a herpes simplex viral vector to rapidly yet transiently o

34 erpesviridae-positive, which included 9 with herpes simplex virus (8.8%), 5 with varicella-zoster vir

35 al fluid polymerase chain reaction (PCR) for herpes simplex virus (HSV) and varicella zoster virus wa

36 Herpes simplex virus (HSV) can cause severe infection in

37 Neonatal herpes simplex virus (HSV) disease results in unacceptab

38 the TG into the brain stem.IMPORTANCE Latent herpes simplex virus (HSV) DNA has been detected in the

39 Herpes simplex virus (HSV) establishes latency in neuron

40 Herpes simplex virus (HSV) establishes lifelong latent i

41 Herpes simplex virus (HSV) glycoprotein C (gC) functions

42 We previously reported that herpes simplex virus (HSV) glycoprotein K (gK) binds to

43 of protein-protein interactions between four herpes simplex virus (HSV) glycoproteins (gD, gH/gL, and

44 The herpes simplex virus (HSV) heterodimer gE/gI and another

45 umcision was associated with reduced odds of herpes simplex virus (HSV) infection among MSM overall (

46 Numerous cases point to the link between herpes simplex virus (HSV) infection and multifocal CNS

47 ance frequently complicates the treatment of herpes simplex virus (HSV) infections in immunocompromis

48 Herpes simplex virus (HSV) is a common and often benign

49 Herpes simplex virus (HSV) is a neuroinvasive virus that

50 Herpes simplex virus (HSV) is among the most prevalent v

51 eity of cells expressing the LATs.IMPORTANCE Herpes simplex virus (HSV) is responsible for significan

52 Herpes simplex virus (HSV) is the main cause of viral en

53 d microparticles and a replication-defective herpes simplex virus (HSV) recombinant vector.

54 Herpes simplex virus (HSV) requires fusion between the v

55 The herpes simplex virus (HSV) single-strand DNA binding pro

56 s, systemic antimicrobial use, imaging data, herpes simplex virus (HSV) testing, and overall hospital

57 h as immunotherapy with oncolytic engineered herpes simplex virus (HSV) therapy, are urgently warrant

58 terial meningoencephalitis, 6% influenza, 6% herpes simplex virus (HSV), and 6% Mycoplasma pneumoniae

59 Anal swabs were collected to test HPV, herpes simplex virus (HSV), and 7 STIs.

60 to, HIV, respiratory syncytial virus (RSV), herpes simplex virus (HSV), and measles.

61 on of certain viruses, including poliovirus, herpes simplex virus (HSV), cytomegalovirus (CMV), and i

62 rather diverse glycan specificities such as Herpes Simplex Virus (HSV), Influenza A Virus (IAV), and

63 h diagnosed with-congenital cytomegalovirus, herpes simplex virus (HSV), varicella zoster virus (VZV)

64 The ubiquitous human pathogens, herpes simplex virus (HSV)-1 and HSV-2, are distinct vir

65 Herpes simplex virus (HSV)-1 proteins pUL7 and pUL51 for

66 ation in STING are unexpectedly resistant to Herpes Simplex Virus (HSV)-1, despite lacking STING-indu

67 TDRD7 is a viral restriction factor against herpes simplex virus (HSV-1).

68 One compound was also active against herpes simplex virus (HSV1 and HSV2), and another compou

69 ineered to constitutively express the type I Herpes Simplex Virus (HSV1) HSV-1 receptor, nectin-1, to

70 ockout (KO) mice were infected ocularly with herpes simplex virus 1 (HSV-1) (strain McKrae).

71 In the case of the herpes simplex virus 1 (HSV-1) 0DeltaNLS vaccine, the co

72 Herpes simplex virus 1 (HSV-1) and HSV-2 can efficiently

73 ng assembly of the neurotropic herpesviruses herpes simplex virus 1 (HSV-1) and pseudorabies virus (P

74 strate that these cells can be infected with herpes simplex virus 1 (HSV-1) at a multiplicity of infe

75 rtive infections in HeLa cells infected with herpes simplex virus 1 (HSV-1) at high multiplicity of i

76 ILCs isolated from mice can be infected with herpes simplex virus 1 (HSV-1) but that subsequent repli

77 Herpes simplex virus 1 (HSV-1) can induce damage in brai

78 Herpes simplex virus 1 (HSV-1) can infect virtually all

79 Herpes simplex virus 1 (HSV-1) causes a lifelong infecti

80 Herpes simplex virus 1 (HSV-1) causes significant morbid

81 Herpes simplex virus 1 (HSV-1) cycles between phases of

82 rs such as bovine herpesvirus 1 (BoHV-1) and herpes simplex virus 1 (HSV-1) establish and maintain li

83 bility to reactivate from latency.IMPORTANCE Herpes simplex virus 1 (HSV-1) establishes a lifelong in

84 Herpes simplex virus 1 (HSV-1) establishes a lifelong la

85 Following acute infection, herpes simplex virus 1 (HSV-1) establishes lifelong late

86 Herpes simplex virus 1 (HSV-1) establishes lifelong late

87 induced reactivation from latency.IMPORTANCE Herpes simplex virus 1 (HSV-1) establishes lifelong late

88 Chronic viruses such as herpes simplex virus 1 (HSV-1) evade the hosts' immune s

89 An earlier report showed that herpes simplex virus 1 (HSV-1) expresses two microRNAs (

90 During all stages of infection, herpes simplex virus 1 (HSV-1) expresses viral microRNAs

91 is one of several cellular receptors used by herpes simplex virus 1 (HSV-1) for cell entry.

92 Reactivation of herpes simplex virus 1 (HSV-1) from neurons in sensory g

93 The Us11 protein encoded by herpes simplex virus 1 (HSV-1) functions to impair autop

94 Herpes simplex virus 1 (HSV-1) genes are transcribed by

95 i derived from the long-arm component of the herpes simplex virus 1 (HSV-1) genome, (iv) pUL36 serves

96 Herpes simplex virus 1 (HSV-1) has infected more than 80

97 e predicted 80 open reading frames (ORFs) of herpes simplex virus 1 (HSV-1) have been intensively stu

98 emethylation of histone H3K9 associated with herpes simplex virus 1 (HSV-1) immediate early (IE) prom

99 The herpes simplex virus 1 (HSV-1) immediate early protein I

100 the role of CD8(+) T cells in the control of herpes simplex virus 1 (HSV-1) infection and disease is

101 es have established a potential link between herpes simplex virus 1 (HSV-1) infection and the develop

102 Analysis of Herpes Simplex virus 1 (HSV-1) infection by population-a

103 onses following vaccination in resistance to herpes simplex virus 1 (HSV-1) infection continues to be

104 We have previously shown that herpes simplex virus 1 (HSV-1) infection results in the

105 We recently reported that herpes simplex virus 1 (HSV-1) infection suppresses CD80

106 (-/-) mice were highly susceptible to ocular herpes simplex virus 1 (HSV-1) infection, independent of

107 Herpes simplex virus 1 (HSV-1) infects mucosal epithelia

108 Herpes simplex virus 1 (HSV-1) infects several types of

109 The envelope glycoprotein I (gI) of herpes simplex virus 1 (HSV-1) is a critical mediator of

110 Herpes simplex virus 1 (HSV-1) is a leading cause of inf

111 t of proteolipids in this process.IMPORTANCE Herpes simplex virus 1 (HSV-1) is a neurotropic pathogen

112 amatic displacement of the portal.IMPORTANCE Herpes simplex virus 1 (HSV-1) is the causative agent of

113 Utilizing a mouse model of herpes simplex virus 1 (HSV-1) keratitis, we found that

114 High rates of wild-type (WT) herpes simplex virus 1 (HSV-1) latency reactivation depe

115 Corneal infection with herpes simplex virus 1 (HSV-1) leads to infection of tri

116 pectrometry approach, we have shown that the herpes simplex virus 1 (HSV-1) neurovirulence- and autop

117 le effect on cell-intrinsic immunity against herpes simplex virus 1 (HSV-1) or gammaherpesvirus 68 (g

118 ering number of the world population harbors herpes simplex virus 1 (HSV-1) potentially leading to bl

119 stimulates bovine herpesvirus 1 (BoHV-1) and herpes simplex virus 1 (HSV-1) reactivation.

120 terograde transneuronal tracers derived from herpes simplex virus 1 (HSV-1) strain 129 (H129) are imp

121 The features of herpes simplex virus 1 (HSV-1) strain 129 (H129), includ

122 We used herpes simplex virus 1 (HSV-1) to infect the human DRG-d

123 stressful stimuli.IMPORTANCE The ability of herpes simplex virus 1 (HSV-1) to periodically reactivat

124 nd transmission to nerve endings, capsids of herpes simplex virus 1 (HSV-1) travel retrogradely withi

125 Herpes simplex virus 1 (HSV-1) triggers both the cyclic

126 Here, we show that herpes simplex virus 1 (HSV-1) virions travel in associa

127 sidering the three groups, the prevalence of herpes simplex virus 1 (HSV-1) were 9% in saliva and 5%

128 e proportion of the world population harbors herpes simplex virus 1 (HSV-1), a major cause of infecti

129 protein 0 (ICP0), an E3 ubiquitin ligase of herpes simplex virus 1 (HSV-1), can derepress viral gene

130 Oncolytic herpes simplex virus 1 (HSV-1), devoid of the gamma(1)34

131 In cells infected with herpes simplex virus 1 (HSV-1), hnRNPA2B1 was quantitati

132 al studies of the prototypical herpesviruses herpes simplex virus 1 (HSV-1), HSV-2, human cytomegalov

133 ocused on understanding the biology of human herpes simplex virus 1 (HSV-1), no tool has been develop

134 sosome-terminal endocytic pathway.IMPORTANCE Herpes simplex virus 1 (HSV-1), the prototype alphaherpe

135 In herpes simplex virus 1 (HSV-1)-infected cells, hnRNPA2B1

136 the more distantly related alphaherpesvirus herpes simplex virus 1 (HSV-1).

137 5beta, are activated early in infection with herpes simplex virus 1 (HSV-1).

138 GLF5 in Epstein-Barr virus (EBV), and vhs in herpes simplex virus 1 (HSV-1).

139 irus that can enter via the plasma membrane, herpes simplex virus 1 (HSV-1).

140 titer than other alphaherpesviruses, such as herpes simplex virus 1 (HSV1) or pseudorabies virus (PRV

141 that are derived from the DNA polymerase of herpes simplex virus 1 (Pol peptides).

142 aherpesviruses (pseudorabies virus [PRV] and herpes simplex virus 1 [HSV-1]).

143 EBV], human herpesvirus 6A [HHV-6A], HHV-6B, herpes simplex virus 1 [HSV-1], HSV-2, JC virus [JCV], a

144 This VZV study also complemented prior herpes simplex virus 1 and pseudorabies virus studies in

145 presence of brefeldin A, while studies with herpes simplex virus 1 documented an impaired secondary

146 -oligosaccharyltransferase with NGI-1 causes herpes simplex virus 1 dysfunction.

147 This disease can occur after reactivation of herpes simplex virus 1 in the trigeminal ganglia, leadin

148 Furthermore, time-lapse imaging of herpes simplex virus 1 infected epithelial cells enabled

149 owth within tumor cells.IMPORTANCE Oncolytic herpes simplex virus 1 is a promising agent for cancer i

150 polyfunctional antibody responses.IMPORTANCE Herpes simplex virus 1 is the leading cause of infectiou

151 selectively kill senescent cells expressing herpes simplex virus 1 thymidine kinase (HSV-TK).

152 virus, influenza virus, dengue virus type 2, herpes simplex virus 1, and nonenveloped human adenoviru

153 stomatitis virus, Semliki Forest virus, and herpes simplex virus 1, elicit the neuronal expression o

154 and higher incidence of cytomegalovirus and herpes simplex virus 1, possibly influenced by demograph

155 adults worldwide are latently infected with herpes simplex virus 1.

156 Human immunodeficiency virus (HIV-1) and herpes simplex virus 2 (HSV-2) affect hundreds of millio

157 Reactivation of herpes simplex virus 2 (HSV-2) from latency causes viral

158 after the last virus inoculation.IMPORTANCE Herpes simplex virus 2 (HSV-2) infects nearly 500 millio

159 Herpes simplex virus 2 (HSV-2) is a common sexually tran

160 y(I.C) double-stranded RNA or infection with herpes simplex virus 2 (HSV-2).

161 in addition to one of three TAP inhibitors: herpes simplex virus 2 ICP47, bovine herpes virus 1 UL49

162 nced by certain microbial stimuli, including herpes simplex virus 2, and blocked by antibodies agains

163 Instead, upon secondary challenge with herpes simplex virus 2, circulating memory B cells that

164 Thirteen patients (62%) had herpes simplex virus and 8 patients (38%) had varicella

165 revious reports linking TLR3 deficiency with herpes simplex virus encephalitis.

166 Like all the herpesviruses, herpes simplex virus encodes machinery that enables it t

167 ly in stromal fibroblasts restores oncolytic herpes simplex virus function.

168 monly found bacterial pigment in controlling herpes simplex virus infection, for which diverse and mu

169 During Herpes Simplex Virus infection, viral replication compar

170 prevention of neonatal infections.IMPORTANCE Herpes simplex virus is among the most serious infection

171 activity during HSV-1 infections.IMPORTANCE Herpes simplex virus persists lifelong in neurons and ca

172 w that respiratory syncytial virus (RSV) and herpes simplex virus type 1 (HSV-1) accumulate a rich an

173 Here we report that GA inhibits Herpes simplex virus type 1 (HSV-1) by inhibition of bot

174 This study aimed at characterizing herpes simplex virus type 1 (HSV-1) epidemiology in the

175 Orolabial herpes simplex virus type 1 (HSV-1) infection has a wide

176 Herpes simplex keratitis (HSK), caused by herpes simplex virus type 1 (HSV-1) infection, is the co

177 In herpes simplex virus type 1 (HSV-1) infection, the coupl

178 ls associated with protective and pathogenic herpes simplex virus type 1 (HSV-1) infections remains u

179 Here we use direct RNA-seq to profile the herpes simplex virus type 1 (HSV-1) transcriptome during

180 asis of genome packaging and organization in herpes simplex virus type 1 (HSV-1), we developed sequen

181 Herpes simplex virus type 1 (HSV-1)-infected corneas can

182 t protective functions during infection with herpes simplex virus type 1 (HSV-1).

183 (mDCs) are not permissive for infection with herpes simplex virus type 1 (HSV-1).

184 Herpes simplex virus type 1 (HSV1) infection has been as

185 unravel the complexity of the interactome of herpes simplex virus type 1 (HSV1), the prototypical her

186 The herpes simplex virus type 1 thymidine kinase (HSV1-tk) g

187 une responses to chronic infections, such as herpes simplex virus type 2 (HSV-2) in HIV/HSV-coinfecte

188 f infection were seen in smokers, those with herpes simplex virus type 2 (HSV-2) infection, men who h

189 n to be at least tripled in individuals with herpes simplex virus type 2 (HSV-2) infection.

190 Genital infection with herpes simplex virus type 2 (HSV-2) is common and increa

191 This study investigated herpes simplex virus type 2 (HSV-2) seroprevalence utili

192 Interestingly, cells infected with herpes simplex virus type-1 (HSV-1) incorporated EdC and

193 Herpes simplex virus type-1 (HSV-1), one of the most wid

194 ed by the development of acyclovir-resistant herpes simplex virus viremia, primary graft failure, and

195 lium, Trichomonas vaginalis, adenovirus, and herpes simplex virus were absent.

196 ed virus, often an adeno-associated virus or herpes simplex virus, among many other types.

197 es were investigated for Epstein-Barr virus, herpes simplex virus, and HCMV-specific immunoglobulin G

198 For herpes simplex virus, this process requires the products

199 al response to HCMV, but not Epstein-Barr or herpes simplex virus, was associated with increased risk

200 Herpes simplex virus-1 (HSV-1) establishes a latent infe

201 gy is a powerful host defense that restricts herpes simplex virus-1 (HSV-1) pathogenesis in neurons.

202 Herpes simplex virus-1 (HSV-1) replicates within the nuc

203 Neurotropic viruses, such as herpes simplex virus-1 (HSV-1), also rely on cellular AK

204 Infection by viruses, including herpes simplex virus-1 (HSV-1), and cellular stresses ca

205 Concordantly, GPX4 deficiency inhibited herpes simplex virus-1 (HSV-1)-induced innate antiviral

206 f several other pathogenic viruses including Herpes Simplex Virus-1 and-2, Vaccinia virus, and Zika v

207 ared to class B infections (cytomegalovirus, herpes simplex virus-1/2, human herpesvirus 8, hepatitis

208 e mechanisms underlying rapid elimination of herpes simplex virus-2 (HSV-2) in the human genital trac

209 ells.IMPORTANCE We study the pathogenesis of herpes simplex virus-mediated corneal disease.

210 -Barr virus (EBV), cytomegalovirus (CMV) and Herpes simplex virus.

211 that include the significant human pathogens herpes simplex viruses (HSV) and varicella zoster virus

212 ntain the same genetic variations.IMPORTANCE Herpes simplex viruses (HSV) infect a majority of adults

213 ults in Finland are seropositive carriers of herpes simplex viruses (HSV).

214 alphaherpesvirus related to human pathogens herpes simplex viruses 1 and 2 and varicella-zoster viru

215 transport of enveloped particles.IMPORTANCE Herpes simplex viruses 1 and 2 and varicella-zoster viru

216 Herpes simplex viruses bud into the nuclear membrane of

217 pesviruses such as varicella-zoster virus or herpes simplex viruses.

218 t human cells, DNA viruses such as vaccinia, herpes simplex, and adenovirus induced increased IFN pro

219 om patients with bacterial and viral (due to herpes simplex, varicella zoster, and enteroviruses) men

220 severity and frequency of recurrent genital herpes sores.

221 Herpes stromal keratitis (HSK) is a corneal chronic infl

222 blinding immunoinflammatory condition called herpes stromal keratitis (HSK), which involves the loss

223 n pathways play a fundamental role in ocular herpes T cell immunopathology and provide important immu

224 erging concept of developing an asymptomatic herpes vaccine that would boost effector memory CD4(+) a

225 the potential design of T-cell-based ocular herpes vaccines.

226 en AD or PD patients to those afflicted with herpes viral infections as to discover novel potential n

227 using anterograde transsynaptic conditional herpes viral tracing.

228 ch periodically replicate and produce viable herpes virions, particularly in anogenital and cervical

229 he latent phase, Kaposi's sarcoma-associated herpes virus (KSHV) maintains itself inside the host by

230 with infection by Kaposi sarcoma-associated herpes virus (KSHV).

231 RPP30) and a viral spike-in control (Phocine Herpes Virus 1 [PhHV-1]), which monitor sample quality a

232 bitors: herpes simplex virus 2 ICP47, bovine herpes virus 1 UL49.5, or rhesus cytomegalovirus Rh185.

233 irus (OR, 2.67; 95% CI, 1.75-4.36) and human herpes virus 6 (OR, 3.50; 95% CI, 1.15-10.63) were detec

234 The immune modulatory protein herpes virus entry mediator (HVEM) is one of several cel

235 CD160 and BTLA both bind to herpes virus entry mediator.

236 of members of the enveloped filo, alpha, and herpes virus families but not the flavivirus group and n

237 cting these mice with EGFP-expressing murine herpes virus-68 (MHV68-EGFP) caused occasional transient

238 However, the patient had evidence of human herpes virus-6B infection.

239 is divided into idiopathic MCD (iMCD), human herpes virus-8 (HHV8)-associated MCD (HHV8-MCD), and pol

240 obes that cause persistent infections (e.g., herpes viruses) do so by switching from fast-growing lyt

241 eloped viruses, including influenza viruses, herpes viruses, and coronaviruses.

242 t defense against pathogenic viruses such as herpes viruses, flaviviruses, retroviruses, and coronavi

243 HSATII RNA in human cells infected with two herpes viruses.

244 of S. purpurea extracts against both pox and herpes viruses.

245 ly, using a rabbit model of recurrent ocular herpes, we found that the combined blockade of PD-1 and

246 The primary reported risk factors for herpes zoster (HZ) are increasing age and immunodeficien

247 The incidence of herpes zoster (HZ) has been increasing in recent decades

248 dom (UK) due to concerns this could increase herpes zoster (HZ) incidence.

249 The primary reported risk factors for herpes zoster (HZ) include increasing age and immunodefi

250 compromised adults are at .increased risk of herpes zoster (HZ) infection and related complications.

251 ocompromised adults are at increased risk of herpes zoster (HZ) infection and related complications.

252 Efficacy of the live-attenuated herpes zoster (HZ) vaccine (ZVL) wanes substantially ove

253 The adjuvanted recombinant glycoprotein E herpes zoster (HZ) vaccine is superior to the live atten

254 Data on the epidemiology of herpes zoster (HZ), particularly in the unvaccinated imm

255 Data on the epidemiology of herpes zoster (HZ), particularly in the unvaccinated, im

256 and it can reactivate later in life, causing herpes zoster (HZ).

257 VZV causes varicella (chicken pox) and herpes zoster (shingles), while HCMV causes serious dise

258 Information on the risks of herpes zoster (zoster) preceding a cancer diagnosis and

259 d indirect influences on the epidemiology of herpes zoster among children.

260 rld-wide, reactivating in one-third to cause herpes zoster and occasionally chronic pain.

261 category, and the annual rates of change in herpes zoster by age category, in an interrupted time se

262 oups revealed a net increase of hospitalized herpes zoster cases in individuals aged 10-49 years afte

263 No herpes zoster cases or major adverse cardiac events incl

264 Although the annual incidence of herpes zoster in adults has continued to increase, the r

265 has a substantial population-level impact on herpes zoster in nonvaccinated age groups.

266 Estimated vaccine efficacy against herpes zoster in patients with solid tumour malignancies

267 lysis on impact of chickenpox vaccination on herpes zoster incidence and time trend, focusing on popu

268 The increase in age-adjusted herpes zoster incidence before implementation of chicken

269 In the antiretroviral therapy era, herpes zoster incidence continued to decline in people l

270 Herpes zoster incidence rates have continued to increase

271 Historic herpes zoster incidence trends in US adults have been ha

272 ccine effectiveness, duration of protection, herpes zoster incidence, and probability of postherpetic

273 tegories during 1996-2006 and 2007-2016, the herpes zoster incidences increased at annual rates of 1-

274 In 1991-1995, the herpes zoster incidences increased at annual rates of 4-

275 The annual incidences of herpes zoster increased throughout the period of 1991-20

276 sis showed a significant increase in risk of herpes zoster infection among patients who received JAK

277 meta-analysis, we found an increased risk of herpes zoster infection among patients with immune-media

278 Incidence rates of serious infections, herpes zoster infection, malignancy, and major cardiovas

279 mated incidence rates of serious infections, herpes zoster infection, non-melanoma skin cancer, other

280 Herpes zoster is linked to amyloid-associated diseases,

281 ion of chickenpox vaccination, incidences of herpes zoster may rise.

282 ied intention-to-treat population, confirmed herpes zoster occurred in 22 of 1328 (6.7 per 1000 perso

283 eatment of ophthalmoplegia in the setting of herpes zoster ophthalmicus (HZO) is controversial.

284 Herpes zoster ophthalmicus occurs primarily in elderly o

285 We report a case of herpes zoster ophthalmicus-related ophthalmoplegia (HZOR

286 Outcome measures were the incidences of herpes zoster per 100 000 person-years, by calendar year

287 The hypothesized increase in herpes zoster predicted from modelling of the exogenous

288 ng chemotherapy, but was not efficacious for herpes zoster prevention in patients with haematological

289 ccine was well tolerated and efficacious for herpes zoster prevention in patients with solid tumour m

290 ted varicella zoster virus (VZV) vaccine for herpes zoster prevention in patients with solid tumour o

291 We evaluated pediatric herpes zoster trends using administrative databases.

292 thod to assess the safety of live attenuated herpes zoster vaccination during 2011-2017 in US adults

293 tinued to increase since introduction of the herpes zoster vaccines.

294 rated and estimated vaccine efficacy against herpes zoster was 16.8% (95% CI -17.8 to 41.3).

295 Herpes zoster was reported by one (<1%) patient on conti

296 PORTANCE Varicella-zoster virus (VZV) causes herpes zoster, a major health issue in the aging and imm

297 human alphaherpesvirus causing varicella and herpes zoster, expresses 24 virally encoded sncRNA (VZVs

298 cts a transient increase in the incidence of herpes zoster, peaking in adults 15-35 years after the s

299 ting wild-type varicella delays the onset of herpes zoster, predicts a transient increase in the inci

300 development of novel treatments for painful herpes zoster.

301 matological malignancies is at high risk for herpes zoster.