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1 ovirus TKs of rhesus monkey rhadinovirus and herpesvirus saimiri.
2 the gene 50 transcripts encoded by KSHV and herpesvirus saimiri.
3 KSHV) (formal name, human herpesvirus 8) and herpesvirus saimiri.
4 7 encode transcriptional regulating genes in herpesvirus saimiri.
5 of Epstein-Barr virus and the ORF65 gene of herpesvirus saimiri.
6 Kaposi's sarcoma-associated herpesvirus and herpesvirus saimiri.
7 s derived from a rhesus monkey infected with herpesvirus saimiri.
8 me not conserved with Epstein-Barr virus and herpesvirus saimiri.
9 of Tip function in T cell transformation by herpesvirus saimiri.
10 cell transformation by subgroup A strains of herpesvirus saimiri.
11 letion mutant, point mutant, and recombinant herpesvirus saimiri.
12 elete the reporter gene from the recombinant herpesvirus saimiri.
14 e to the other known human herpesviruses and herpesvirus saimiri, a closely related gammaherpesvirus
16 ad been transformed to permanent growth with Herpesvirus saimiri, an oncogenic virus of nonhuman prim
18 KSHV is a gamma herpesvirus with homology to herpesvirus Saimiri and Epstein-Barr virus, both of whic
19 ally related to human gammaherpesviruses and herpesvirus saimiri and has been reported to be associat
20 s to a G-protein-coupled receptor encoded by herpesvirus Saimiri and to human interleukin-8 receptors
21 gamma-herpesviruses, Epstein-Barr virus and Herpesvirus saimiri, and are present in the lesions of m
22 erved that ORF45s of rhesus rhadinovirus and herpesvirus saimiri are found exclusively in the nucleus
26 wo noncoding RNAs expressed by the oncogenic Herpesvirus saimiri, bind host microRNAs miR-142-3p, miR
27 ologs including KSbcl-2, BHRF1, and ORF16 of herpesvirus saimiri contain poorly conserved Bcl-2 homol
33 Tyrosine kinase interacting protein (Tip) of Herpesvirus saimiri (HVS) activates the lymphoid-specifi
34 ty to the virus-encoded cyclin (v-cyclin) of herpesvirus saimiri (HVS) and 31% identity and 53% simil
35 T-cell transformation by the DNA tumor virus herpesvirus saimiri (HVS) and designated tyrosine kinase
36 of the saimiri transforming protein (STP) of herpesvirus saimiri (HVS) and of K1 of KSHV, other membe
39 product of open reading frame 14 (orf14) of herpesvirus saimiri (HVS) exhibits significant homology
41 gy and colinearity with the right end of the herpesvirus saimiri (HVS) genome and more limited homolo
48 eracting protein (Tip) of the T lymphotropic Herpesvirus saimiri (HVS) is constitutively present in l
50 tyrosine kinase-interacting protein (Tip) of herpesvirus saimiri (HVS) is required for binding to the
51 any other herpesvirus, including the gamma-2 herpesvirus saimiri (HVS) of New World squirrel monkeys.
52 Deletion of the terminal repeats (TR) from herpesvirus saimiri (HVS) renders it unable to produce i
53 T-cell transformation by the DNA tumor virus herpesvirus saimiri (HVS) strain 484, designated tyrosin
54 nsforming protein oncogene, called STP-A, of herpesvirus saimiri (HVS) subgroup A is not required for
58 ies of cyclins encoded by two herpesviruses, herpesvirus saimiri (HVS) which can transform blood lymp
59 nuclear RNAs of the Sm class are encoded by Herpesvirus saimiri (HVS), a gamma Herpesvirus that caus
61 s (KSHV; also known as human herpesvirus 8), herpesvirus saimiri (HVS), and Epstein-Barr virus (EBV).
63 r the left end of the genome in RRV26-95 and herpesvirus saimiri (HVS), but in KSHV the DHFR gene is
64 's sarcoma-associated herpesvirus (KSHV) and herpesvirus saimiri (HVS), has been shown to encode a la
65 ng the saimiri transforming protein (STP) of herpesvirus saimiri (HVS), Kaposi's sarcoma-associated h
66 m the PBL of normal donors by infection with Herpesvirus saimiri (HVS), to evaluate functional proper
67 re, using a close homolog of KSHV ORF57 from herpesvirus saimiri (HVS), we determined the crystal str
68 conserved snRNAs encoded by the lymphotropic Herpesvirus saimiri (HVS), we determined the specific se
69 evious studies have demonstrated that Tip of herpesvirus saimiri (HVS), which is a T-lymphotropic tum
71 binant alpha-defensin-1 and CAF derived from herpesvirus saimiri (HVS)-transformed CD8(+) cells inhib
78 t of SAP defects on TCR signal transduction, herpesvirus saimiri-immortalized CD4 Th cells from XLP p
79 to induce bystander apoptosis was tested in herpesvirus saimiri-immortalized primary CD4(+) T cells
82 onstrating that, unlike the ORF57 homolog in herpesvirus saimiri, nucleolar trafficking is not requir
83 entify Sp140L as a restriction target of the herpesvirus saimiri ORF3 protein, implying a role for Sp
84 least 81 ORFs, including 66 with homology to herpesvirus saimiri ORFs, and 5 internal repeat regions
85 en reading frame 16 (ORF16) of the oncogenic herpesvirus saimiri protects cells from heterologous vir
87 the engineered reporter genes cloned within herpesvirus saimiri sequences, recombinant viruses were
90 imiri transforming protein (STP) oncogene of Herpesvirus saimiri subgroup A strain 11 (STP-A11) is no
91 lines derived by transformation of PBMC with herpesvirus saimiri, suggesting that this phenomenon is
94 tein found in the cell culture medium of the herpesvirus saimiri-transformed CD8(+) T-cell line, K#1
95 ORF-2, a 32-kDa viral protein expressed by herpesvirus saimiri-transformed lymphocytes, is essentia
96 a 56-kDa cellular protein in untransformed, herpesvirus saimiri-transformed, and Jurkat lymphocytes.
99 -class small nuclear RNAs, called HSURs (for Herpesvirus saimiri U RNAs), that are abundantly express
100 hen permissive cells were cotransfected with herpesvirus saimiri virion DNA and one of the engineered
102 back mechanism modulating gene expression in herpesvirus saimiri, whereby ORF 50a transcription is do
103 er herpesviruses, the Epstein-Barr virus and herpesvirus saimiri, which are known to be associated wi