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1 ssing within 1 h, both in Alu repeats and in heterochromatic alpha-satellite DNA.
2   These epigenetic alterations affect mainly heterochromatic and bivalent regions and provide possibl
3 eloped an in vivo single DSB system for both heterochromatic and euchromatic loci in Drosophila melan
4                                      In both heterochromatic and euchromatic segments of the genome,
5  contrast to theories that Y chromosomes are heterochromatic and gene poor, the mouse MSY is 99.9% eu
6 ning Drosophila melanogaster Y chromosome is heterochromatic and has few genes.
7 euchromatic and intronic regions compared to heterochromatic and intergenic regions, respectively, an
8  D. melanogaster revealed previously unknown heterochromatic and telomeric transition sequences, and
9 somes are gene-poor, repeat-rich and largely heterochromatic and therefore represent a difficult targ
10 ciated with the nuclear lamina are generally heterochromatic and transcriptionally repressed.
11         The inactivated X chromosome becomes heterochromatic and visits a specific nuclear compartmen
12 Conversely, repair in regions classified as "heterochromatic" and "repressed" was relatively low at e
13 egions of the genome (genic, intergenic, and heterochromatic) and at different distances ( approximat
14  the Drosophila Y chromosome is degenerated, heterochromatic, and contains few genes, increasing evid
15 ated gene HvS40 and altering distribution of heterochromatic areas in the nuclei.
16 tion and reorganization of euchromatic A and heterochromatic B compartments.
17      Previous work in animals has shown that heterochromatic breaks are translocated out of the heter
18                                     However, heterochromatic centromeres remained recombination-suppr
19  are characterized by a lack or reduction of heterochromatic CHH methylation and enrichment of CHH me
20  (ORs) genes requires the formation of large heterochromatic chromatin domains that sequester the OR
21 ditionally, PELP1 positively correlates with heterochromatic chromatin marks and negatively correlate
22        We found a drastic loss of H3K9me2 at heterochromatic chromocenters in vim1/2/3 nuclei.
23 r show that silenced clusters associate with heterochromatic chromosomal domains toward the periphery
24 s from new juxtapositions of euchromatic and heterochromatic chromosomal regions, and manifests as st
25              In Drosophila, transcripts from heterochromatic clusters are processed into primary piRN
26 We show that Rhino binds specifically to the heterochromatic clusters that produce piRNA precursors,
27 v) increase in the number of pericentromeric heterochromatic clusters; and (v) increase in the freque
28 atin and heterochromatin, which implies that heterochromatic compaction does not reduce MNase accessi
29 ns that switched from the euchromatic to the heterochromatic compartment during neutrophil differenti
30 tion involved genomic regions located in the heterochromatic compartment in both progenitors and neut
31  for the concept that induced changes to the heterochromatic component of the genome are coincident w
32 tem, identifying certain endogenous genes in heterochromatic contexts as privileged while foreign DNA
33 ysfunction in laminopathies is relaxation of heterochromatic DNA [1].
34 e a variation on this strategy for repairing heterochromatic DNA damage.
35                                              Heterochromatic DNA domains have important roles in the
36 orylation of S824 is necessary for repair of heterochromatic DNA lesions and similar to cells express
37 show that lack of histone H1, which elevates heterochromatic DNA methylation in somatic tissues, does
38 X RELATED PROTEIN 5 and 6 (ATXR5/6) regulate heterochromatic DNA replication and genome stability.
39       Males often contain substantially more heterochromatic DNA than females, due to the presence of
40 affold for the anchoring of highly condensed heterochromatic DNA to the nuclear envelope, thereby est
41 ization of Tet1, but not of Tet2 and Tet3 at heterochromatic DNA.
42 for defining the boundary between active and heterochromatic DNA.
43 chromatic breaks are translocated out of the heterochromatic domain for repair.
44  this prediction, we found that shortening a heterochromatic domain in Saccharomyces had no impact on
45 360 can enhance silencing of a reporter in a heterochromatic domain of Drosophila melanogaster.
46 dary elements at the edges of a 20-kb silent heterochromatic domain where nucleosomes are methylated
47  of asymmetric tetramer inheritance within a heterochromatic domain would have the potential to desta
48 l model to test the properties of an induced heterochromatic domain.
49 es impaired the de novo establishment of the heterochromatic domain.
50                                              Heterochromatic domains are complex structures composed
51                                              Heterochromatic domains are enriched with repressive his
52     Two recent papers in Nature propose that heterochromatic domains are organized into phase-separat
53                                              Heterochromatic domains containing histone H3 lysine 9 m
54                             We conclude that heterochromatic domains form via phase separation, and m
55 n efficiency of origins in late-replicating, heterochromatic domains is only 25% lower than in early-
56 itioning of chromosomes into euchromatic and heterochromatic domains requires mechanisms that specify
57      After removal of the HP1alpha stimulus, heterochromatic domains were heritably transmitted, undi
58 corporated macroH2A retargets the same large heterochromatic domains where macroH2A was already enric
59   This model makes a prediction that shorter heterochromatic domains would experience unbalanced tetr
60 ro- and euchromatin: H1 is necessary to form heterochromatic domains yet dispensable for silencing of
61 in firing time, including that of origins in heterochromatic domains, was established in late G1 phas
62 ns between both constitutive and facultative heterochromatic domains, with the strongest interactions
63 H3 lysine 9 (H3K9) is methylated (H3K9me) at heterochromatic domains.
64 decreasing interactions between constitutive heterochromatic domains.
65 nappreciated roles of NDR positioning within heterochromatic domains.
66 A enzymatic activity is required to relocate heterochromatic double-strand breaks outside the domain,
67 (dKDM4A) histone demethylase is required for heterochromatic DSB mobility.
68 oral kinetics and pathway utilization during heterochromatic DSB repair depend on dKDM4A-dependent de
69                                              Heterochromatic DSBs display specialized temporal and sp
70 hful homologous recombination (HR) repair of heterochromatic DSBs relies on the relocalization of DSB
71 hought to be the main pathway used to repair heterochromatic DSBs, direct tests of this hypothesis ar
72            ACF1-SNF2H requires RNF20 to bind heterochromatic DSBs, underlies RNF20-mediated chromatin
73 g (NHEJ) repair pathways but exclusively for heterochromatic DSBs.
74 orylation (pKAP-1) and CHD3.1 dispersal from heterochromatic DSBs; however, how heterochromatin compa
75 tion and via a mechanism distinct from known heterochromatic effects on origin function.
76 may contribute to adaptation of genes to the heterochromatic environment and, hence, facilitate the e
77                    Our data suggest that the heterochromatic environment at the gDMR reinforces silen
78 past, including increased repeat-content and heterochromatic environment.
79 are strongly correlated with euchromatic and heterochromatic epigenetic histone modifications, respec
80                      Global establishment of heterochromatic features occurs following MZT and requir
81                           Recruitment of the heterochromatic features SMCHD1, macroH2A, H3K27me3, and
82 lates with the ability to recruit additional heterochromatic features.
83 Mu1c(Col) was intergenic and associated with heterochromatic features.
84 e performed whole-genome analyses of several heterochromatic features: dimethylation of lysine 9 and
85     MP density was measured using customized heterochromatic flicker photometery.
86 -wavelength fundus autofluorescence [AF] and heterochromatic flicker photometry [HFP]), and serum con
87 POD) measured noninvasively using customized heterochromatic flicker photometry and blood samples gen
88                      MPOD was measured using heterochromatic flicker photometry in 10 eyes (5 patient
89 eccentricity was measured using a customized heterochromatic flicker photometry technique.
90 juvenile sharks, made using single flash and heterochromatic flicker photometry under conditions of d
91                                              Heterochromatic flicker photometry was used to measure t
92               We measured MPOD by customized heterochromatic flicker photometry.
93 l layers characterizes senescence-associated heterochromatic foci (SAHF) formation in human fibroblas
94 f heterochromatin into senescence-associated heterochromatic foci (SAHFs).
95 nucleoporins are required for aggregation of heterochromatic foci and epigenetic inheritance.
96 ells and show that H1 overexpression creates heterochromatic foci in the VC progenitor cell.
97 ne H2A, is a marker of senescence-associated heterochromatic foci that synergizes with DNA methylatio
98  decreased H3K9 dimethylation, dispersion of heterochromatic foci, and derepression of MET-2 targets.
99 s, the repaired MeCP2 protein is enriched in heterochromatic foci, reflecting restoration of normal M
100 nt chromosomes converge in a small number of heterochromatic foci.
101              All three factors colocalize in heterochromatic foci.
102 cells destabilizes the senescence-associated heterochromatic foci.
103 nd its binding partner E2FA are recruited to heterochromatic gammaH2AX-labelled DNA damage foci in an
104  quantitative evaluation of the stability of heterochromatic gene repression.
105 e delete this trigger, dramatically altering heterochromatic gene silencing and chromatin architectur
106 ons outside of DNA replication, including in heterochromatic gene silencing and telomere maintenance.
107 he repetitive rDNA arrays are thought to use heterochromatic gene silencing as a mechanism for metabo
108      Here we show that RNA editing regulates heterochromatic gene silencing in Drosophila.
109                                              Heterochromatic gene silencing is an important form of g
110 y partially dependent on known components of heterochromatic gene silencing, implying that a distinct
111 and chromatin structure indicative of a more heterochromatic genome.
112  T cells revealed DNA hypomethylation in the heterochromatic genomic compartment, as well as reactiva
113  neutrophil genomes were highly enriched for heterochromatic genomic interactions across vast genomic
114 Ms by analyzing histone H3 purified from the heterochromatic germline micronucleus of the model organ
115 ed as either flickering full-field colors or heterochromatic gratings.
116 incident with the presence of high levels of heterochromatic H3 lysine nine trimethylation (H3K9me3).
117                        In parallel, level of heterochromatic H3K9me2 decreases at this gene.
118 ssociated changes in spatial distribution of heterochromatic H3K9me2 patterns in the nuclei.
119 in type I latency, as well as an increase in heterochromatic H3K9me3 at these sites.
120 ectopic alphoid DNA integration site removed heterochromatic H3K9me3 modification and was sufficient
121  of transposons is mediated by 24-nucleotide heterochromatic (het)siRNAs, RDR2, DCL3 and AGO4.
122 of histone H3 lysine 9 (H3K9) methylation, a heterochromatic histone mark, from immediate early and l
123 ation rates, displayed similar decreases for heterochromatic histone marks.
124  fewer mature oligodendrocytes and decreased heterochromatic histone marks.
125  depend on dKDM4A-dependent demethylation of heterochromatic histone marks.
126  the literature around the role of the major heterochromatic histone methyltransferase Suv39h1 in the
127                 Cyclin D1 and Dicer maintain heterochromatic histone modification (Tri-m-H3K9).
128 ation H3K9ac and a corresponding increase in heterochromatic histone modification H3K9me3 at the LMP2
129 gle cells and correlates positively with the heterochromatic histone modification H3K9me3.
130 NA bases to several megabases in the case of heterochromatic histone modifications.
131 -regulated interplay between euchromatic and heterochromatic histone modifications.
132  and mitosis, the contribution and extent of heterochromatic histone posttranslational modifications
133               Unexpectedly, silencing of the heterochromatic HML and HMR loci was not lost during agi
134                                        Maize heterochromatic knobs cheat female meiosis by forming ne
135 drive system that transforms typically inert heterochromatic knobs into centromere-like domains (neoc
136 petitive genomic features such as telomeres, heterochromatic knobs, and centromeres.
137 rnal structure of seven centromeres and five heterochromatic knobs, showing that the major tandem rep
138 lncRNA transcription in both euchromatic and heterochromatic lncRNA-based gene silencing processes.
139 is recruited by the ATF/CREB homolog Atf1 to heterochromatic loci and promoters of stress-response ge
140                                     Multiple heterochromatic loci are often clustered into a higher o
141     In Arabidopsis, clustering of repetitive heterochromatic loci into so-called chromocenters is an
142 osons for silencing, but not the specialized heterochromatic loci that produce piRNA precursors.
143 NAs were concentrated at a smaller number of heterochromatic loci throughout the genome, especially a
144 that Fft3 suppresses turnover of histones at heterochromatic loci to facilitate epigenetic transmissi
145 sed copies of mating-type information at the heterochromatic loci, HMLalpha and HMRa, which are locat
146 ether to elicit transcriptional silencing at heterochromatic loci.
147 he loss of which causes dramatic increase in heterochromatic loci.
148 wi6 recruitment to centromeric and telomeric heterochromatic loci.
149  (H3K9), which recruits Swi6/HP1 proteins to heterochromatic loci.
150                   Here, we focus on a 180-kb heterochromatic locus producing Piwi-interacting RNAs (p
151 9me3 chromatin complex lies at the center of heterochromatic macromolecular assemblies and mediates m
152 ple model for the mitotic inheritance of the heterochromatic mark H3K27me1 and the protection of H3.3
153 ructure and leading to the dispersion of the heterochromatic mark H3K9me3.
154 e3, suggesting that dKDM4A demethylates this heterochromatic mark to facilitate repair.
155 ions and gene bodies marked by a facultative heterochromatic mark, which could explain the observed b
156                Progerin triggers loss of the heterochromatic marker H3K27me3, and premature senescenc
157 omatin specifically and acts in synergy with heterochromatic marks H3K9me2 and DNA methylation to mai
158 ) genomes are associated with the repressive heterochromatic marks H3K9me2/me3 and H3K27me3 during la
159 onstitutive H3K9me3 and facultative H3K27me3 heterochromatic marks in FRDA.
160 d to protect active chromatin from acquiring heterochromatic marks, such as dimethylated histone H3K9
161 ized by the presence of both euchromatic and heterochromatic marks.
162 athways in processing mRNAs of genes bearing heterochromatic marks.
163 irwise association between L1 insertions and heterochromatic marks.
164 chromatic domains, although all are close to heterochromatic masses.
165                These processes are linked to heterochromatic mo-2 minisatellite arrays, and require M
166 ent inactive X chromosome (Xi) enriched with heterochromatic modifications and XIST/Xist RNA, which e
167 Here we focus on the initial steps restoring heterochromatic modifications in the embryo.
168  of XIST/Xist RNA, and they lack the typical heterochromatic modifications of the Xi.
169  deletions that altered the distance between heterochromatic NDRs revealed a striking oscillatory rel
170 vation led to the unrestrained expression of heterochromatic noncoding RNAs (ncRNAs) that in turn tri
171 systematically altering the distance between heterochromatic nucleosome-depleted regions (NDRs), whic
172 tion complex on the neo-X and those becoming heterochromatic on the neo-Y show little overlap, possib
173 mark may be important for maintaining proper heterochromatic organization and, possibly, chromosome f
174 ith a central CENP-A(Cnp1) domain flanked by heterochromatic outer-repeat regions.
175  the number of crossovers between markers in heterochromatic pericentric regions that underwent demet
176 matic portion of the genome and 21 Mb in the heterochromatic portion, using a whole-genome shotgun as
177 re, we gained novel functional insight about heterochromatic PTMs by analyzing histone H3 purified fr
178 dant during schizogony and late gametocytes; heterochromatic PTMs mark early gametocytes.
179 ce-specifically in vitro, and localizes to a heterochromatic region in the COOLAIR promoter in vivo.
180          We observed the relocalization of a heterochromatic region, the mating-type region, from its
181 ne 9 acetylation (H3K9ac) is abundant in non-heterochromatic regions across all developmental stages.
182                       This was true even for heterochromatic regions analyzed by quantitative PCR.
183 fluorescent staining pattern, with unstained heterochromatic regions and a banded distribution along
184 brid incompatibilities in Drosophila involve heterochromatic regions and factors that interact with t
185 und that transcripts of many genes native to heterochromatic regions and TEs increased with age in fl
186                   Thus, associations between heterochromatic regions are a major component of the chr
187                                              Heterochromatic regions are associated with histone 3 ly
188             We find that attractions between heterochromatic regions are crucial for establishing bot
189  sites of transcription; however, as not all heterochromatic regions are equally active in transcript
190 d modelling suggest that attractions between heterochromatic regions are essential for the phase sepa
191               Non-coding RNAs generated from heterochromatic regions are processed into small RNAs by
192 and other chromosomes containing specialized heterochromatic regions called knobs.
193 genome assembly correspond to multi-megabase heterochromatic regions composed primarily of two relate
194 nded breaks and contributes to repair within heterochromatic regions during G2.
195 range interactions such as those among small heterochromatic regions embedded in euchromatic arms.
196 ed chromosome arms while it was inhibited in heterochromatic regions encompassing pericentromeric DNA
197 aled that sequences located within different heterochromatic regions have distinct properties, such a
198 enomic landscape of histone modifications in heterochromatic regions have revealed histone H3 lysine
199 st size of centromeres and their surrounding heterochromatic regions impede studies of genomic variat
200 lex incorporates the histone variant H3.3 at heterochromatic regions in a replication-independent man
201 cer regions in MBC-like WM and repressed and heterochromatic regions in PC-like WM.
202 ed (ORCA/LRWD1), preferentially localizes to heterochromatic regions in post-replicated cells.
203 l properties associated with euchromatic and heterochromatic regions in the genome.
204                                  To identify heterochromatic regions in the heavily studied model C.
205 Representation of the Y chromosome and other heterochromatic regions is particularly improved.
206 mulation of H2A.Z genome-wide, especially at heterochromatic regions normally H2A.Z-depleted in wild-
207 epeated DNAs, or satellites, are enriched in heterochromatic regions of eukaryotic genomes and contri
208                                              Heterochromatic regions of the genome are generally tran
209 gregation of active euchromatic and inactive heterochromatic regions of the genome(1,2).
210 ory elements to new locations, most often to heterochromatic regions of the genome.
211 stone H2A variant that is typically found in heterochromatic regions of the genome.
212  single-nucleotide alterations, primarily in heterochromatic regions of the genome.
213 hat loss of Vgl1 impairs Clr4 recruitment to heterochromatic regions of the genome.
214 1s are enriched at lamin-associated domains, heterochromatic regions of the nuclear periphery.
215 slow kinetics, including those localizing to heterochromatic regions or harboring additional lesions
216 ion is lost and is only restored in specific heterochromatic regions that have maintained competence
217      In this paper we show that tethering of heterochromatic regions to nuclear landmarks and random
218 f chromatin accessibility in euchromatic and heterochromatic regions under extended darkness in Arabi
219 n neurons and testes, often localized within heterochromatic regions, and important for viability.
220 he transcriptionally inert, late-replicating heterochromatic regions, including the constitutive hete
221 >0.10 was 95% and 91% in the euchromatic and heterochromatic regions, respectively.
222 tromeric regions, even including some of the heterochromatic regions, which are not present in gold s
223  uracil in the late replicating constitutive heterochromatic regions, while drug treatment induced a
224 sperm cells, 24-nt siRNAs were spread across heterochromatic regions, while in egg cells, 24-nt siRNA
225  de novo heterochromatin formation at native heterochromatic regions.
226 phisms (SNPs) from euchromatic and 1000 from heterochromatic regions.
227 fied the flanking sequence of breakpoints in heterochromatic regions.
228 oading factor (Nipped-B/NIPBL/Scc2) at dense heterochromatic regions.
229 atabase to further enable genomic studies of heterochromatic regions.
230 senting the endogenous centromeres and other heterochromatic regions.
231  lacking Ubp3 than in wild-type cells in all heterochromatic regions.
232 methylation and epigenetic gene silencing in heterochromatic regions.
233 H3K9me2, that mainly silences transposons in heterochromatic regions.
234 h transcript by associating with an intronic heterochromatic repeat element of IBM1.
235 criptional silencing of retrotransposons and heterochromatic repeats in plants.
236  significant differences in the stability of heterochromatic repression between various environmental
237 pression in human myeloid progenitors causes heterochromatic repression of NFI-A gene and channels gr
238       The ability to monitor fluctuations in heterochromatic repression uncovered previously unapprec
239 netic assay reveals that transient losses of heterochromatic repression, a heritable form of gene sil
240 show sporadic waves of silencing loss in the heterochromatic ribosomal DNA during the early phases of
241 and Keller et al. show distinct outcomes for heterochromatic RNAs that bind different chromodomain pr
242 ings reveal a new pathway for degradation of heterochromatic RNAs with essential roles in heterochrom
243  to heterochromatin, lead to accumulation of heterochromatic RNAs, and block spreading of H3K9me and
244 tion in differentiated cells, where it binds heterochromatic satellite repeats and chromocenters.
245 lication-where replication of constitutively heterochromatic satellite sequences is delayed-extends S
246 evance, including the preferred locations of heterochromatic satellites of different chromosomes, and
247  adults, suggesting that desilencing of many heterochromatic segments of the genome contributes to st
248  not sufficient to induce the same events in heterochromatic segments.
249 to position unmapped Drosophila melanogaster heterochromatic sequence to specific chromosomal regions
250                                 We find that heterochromatic sequences preferentially require DDM1 fo
251 e also identified and mapped 110 kb of novel heterochromatic sequences.
252 ork confirms that this important but elusive heterochromatic sex chromosome is evolving extremely rap
253 leotide microRNAs (miRNAs) and 24-nucleotide heterochromatic short interfering RNAs (siRNAs).
254                                      How the heterochromatic silenced loci are transcribed remains la
255 ained both by transposons to evade long-term heterochromatic silencing and by their hosts for genome
256 , intergenic transcriptional enhancers evade heterochromatic silencing and converge into interchromos
257 tiotemporal transcription factor coordinates heterochromatic silencing and male germline maturation.
258 TRX, are required for H3.3 incorporation and heterochromatic silencing at telomeres, pericentromeres,
259  that Spt6 is required for multiple steps in heterochromatic silencing by controlling chromatin, tran
260 iquitinase (DUB) Ubp10 is thought to promote heterochromatic silencing by maintaining low H2B-Ub at s
261 ts in changes of H3K9 methylation levels and heterochromatic silencing defects.
262  a challenge to SIR complex-mediated de novo heterochromatic silencing due to the presence of antagon
263  ARID1 in the generative cell causes reduced heterochromatic silencing in both bicellular and mature
264 e male germline, resulting in the release of heterochromatic silencing in the male germline.
265 itosis, mediates siRNA movement to reinforce heterochromatic silencing in the male germline.
266 regions and variant gene expression based on heterochromatic silencing is observed only in some genes
267                                Budding yeast heterochromatic silencing is strictly dependent on the s
268                          A prevalent view of heterochromatic silencing is that these and associated f
269 of Dos1 identifies key residues required for heterochromatic silencing, consistent with Dos1's role a
270  Surprisingly, even though qrf is needed for heterochromatic silencing, induction of qrf initially pr
271 mere length homeostasis of ESCs by mediating heterochromatic silencing.
272 nfigurations of nucleosome arrays facilitate heterochromatic silencing.
273 tion and histone H3 Lys-9 dimethylation, but heterochromatic siRNA levels were not affected.
274 ted nucleotides of 23 nt siRNAs arising from heterochromatic siRNA loci.
275 ay result from modifications added to longer heterochromatic siRNA precursors.
276 As (miRNAs), trans-acting siRNAs (tasiRNAs), heterochromatic siRNAs (hc-siRNAs) populations and their
277                In plants, 24 nucleotide long heterochromatic siRNAs (het-siRNAs) transcriptionally re
278                                              Heterochromatic siRNAs are derived from repetitive regio
279  However, non-templated nucleotides in plant heterochromatic siRNAs have not been deeply studied, esp
280                  The existence and extent of heterochromatic siRNAs in other land plant lineages has
281                             We conclude that heterochromatic siRNAs, and their biogenesis pathways, a
282 leaching measurements, which showed that, at heterochromatic sites, wild-type HELLS is very dynamic,
283  that the majority of the genome exists in a heterochromatic state defined by inaccessible chromatin
284 gradation to initiate destabilization of the heterochromatic state of endogenous silenced loci.
285                      We show that the normal heterochromatic state of the donors does not impair dono
286 NA pathway components are abundant, with the heterochromatic state subsequently propagated by chromat
287 gh level of DNA polymorphisms and from their heterochromatic state, notably their dense DNA methylati
288 er of repeat copies can nucleate a transient heterochromatic state, which, on longer repeat arrays, m
289 ions and independent pathways to arrive at a heterochromatic state.
290 s and promoting a switch from euchromatic to heterochromatic states.
291 t function of H3.3 is to support chromosomal heterochromatic structures, thus maintaining genome inte
292                  Here we show that about 100 heterochromatic TEs are activated in VCs, mostly by DEME
293       In ddm1 mutants, stress hyperactivates heterochromatic transcription and transcription persists
294 e release of transposable elements and other heterochromatic transcripts.
295 ly transcribed regions, cooperate to silence heterochromatic transcripts.
296 RNAs derive from dual-strand piRNA clusters, heterochromatic transposon graveyards that are transcrib
297 ve cells, likely reflecting transcription of heterochromatic transposons in this cell type.
298 sposons and repeats, as well as silencing of heterochromatic transposons.
299                        In P. falciparum, the heterochromatic virulence gene cluster had a strong repr
300                     HP1D2 accumulates on the heterochromatic Y chromosome in male germ cells, strongl

 
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