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1 ess advantages of between-population mating (heterosis).
2 omponent underlying phenotypic variation and heterosis.
3 intensities enhanced both photosynthesis and heterosis.
4 in bioenergetic processes may contribute to heterosis.
5 allelic and/or isoform differences linked to heterosis.
6 fic proteins correlate with higher levels of heterosis.
7 development and may be a major component of heterosis.
8 regions may contribute disproportionally to heterosis.
9 ion in the hybrid and the molecular basis of heterosis.
10 o both parental strains, suggesting positive heterosis.
11 o speculate on mechanisms that might lead to heterosis.
12 cal dominant or overdominant explanations of heterosis.
13 of protein metabolism plays a role in growth heterosis.
14 ons of this work toward our understanding of heterosis.
15 isms, including overdominance, contribute to heterosis.
16 traits including egg-to-adult viability via heterosis.
17 s in the hybrid, which may have an impact on heterosis.
18 rtant role in the generation of the observed heterosis.
19 us showed a significant decrease in apparent heterosis.
20 ight at flowering was the only trait to show heterosis.
21 uantitative trait loci and investigations of heterosis.
22 etic changes are a cause or a consequence of heterosis.
23 n-level dominance plays an important role in heterosis.
24 na, according to the dominance hypothesis of heterosis.
25 re that overdominance does not contribute to heterosis.
26 ly related species, and the genetic basis of heterosis.
27 ozygosity can increase fitness and result in heterosis.
28 eties are tall, mainly due to a wider use of heterosis.
29 decreases only moderately in the presence of heterosis.
30 lings can quantitatively predict adult trait heterosis.
31 teractions can improve crop productivity via heterosis.
32 ution of parental methylation differences to heterosis.
33 some but not all of the observed patterns of heterosis.
34 procal recurrent selection scheme to exploit heterosis.
35 ntial role of SHELL in single-gene oil yield heterosis.
36 us than their parents, a phenomenon known as heterosis.
37 ibution of dosage to quantitative traits and heterosis.
38 opportunities for hybrid complementation in heterosis.
39 s line that could contribute to the observed heterosis.
40 ting a striking 332% average midparent-value heterosis.
41 mal productivity through the manipulation of heterosis.
42 lism, thus modulating biomass and leading to heterosis.
43 ay contribute to its high degree of observed heterosis.
44 he sizes of the ears are diagnostic of yield heterosis.
45 ses, and, in plants, may also play a role in heterosis.
46 le to exploit these relationships to predict heterosis?
47 at consistently exhibited either low or high heterosis across a variety of environments were examined
52 ognition and exploitation of hybrid vigor or heterosis among individual crosses of plants and animals
55 lluscs, related phenomena, marker-associated heterosis and distortion of marker segregation ratios, h
57 ting (over)dominance as the genetic cause of heterosis and estimating the (over)dominance coefficient
59 ad strongly supports the dominance theory of heterosis and inbreeding depression and establishes the
61 ed to explore the molecular genetic basis of heterosis and outbreeding depression, and how their magn
63 or maintenance of genetic diversity - social heterosis and social genomes - can similarly explain the
65 at an inhibitor of photosynthesis eliminated heterosis and that higher light intensities enhanced bot
70 among populations and compared mean fitness, heterosis, and inbreeding depression for eight large and
71 wer and repeatability of allozyme-associated heterosis, and that the allozyme-associated heterosis de
72 led that domestication, local adaptation and heterosis are all associated with QTN allele frequency c
74 put forward to explain the genetic basis of heterosis are the general dominance and the local overdo
75 mic and epigenetic perspectives suggest that heterosis arises from allelic interactions between paren
76 SP can help explain mysterious properties of heterosis as well as other effects of hybridization.
77 reproductive yield as cumulative outcomes of heterosis at different levels, tissues, and times of dev
78 isease with an earlier onset indicating that heterosis at Esr2 plays a significant role in regulating
83 he molecular mechanisms of hybrids vigor (or heterosis) between Dura, Pisifera and their hybrid proge
84 ene regulation, quantitative trait loci, and heterosis, but one that is not easily applied to sexuall
85 offspring under sterile conditions but that heterosis can be restored by inoculation with a simple c
86 herefore, the (over)dominance hypothesis for heterosis can be tested by estimating h, under either do
88 ing a robust hybridization platform in wheat.Heterosis can rapidly boost yield in crop species but de
89 cepticism that surrounds allozyme-associated heterosis comes from inconsistent and unreliable detecti
91 heterosis, and that the allozyme-associated heterosis detected in this study was the result of gener
92 Different mechanisms have been proposed for heterosis: dominance, overdominance, epistasis, epigenet
93 tern and epigenetic mechanisms contribute to heterosis during early flower development in allopolyplo
103 herited in autosomal fashion; the absence of heterosis for male fertility among the MA lines was ther
108 ransition is probably one critical stage for heterosis formation, in which epistatic QTLs are activat
109 nt an example of discovery and validation of heterosis generated by a combination of repulsion linkag
110 their combining ability (General/Specific), heterosis, genotypic and phenotypic correlation and path
115 However, previous theoretical studies on heterosis have been based on bi-parental segregating pop
117 parental expression inheritance which drives heterosis (HET) is significantly affected by environment
118 distance between hybridizing parents affects heterosis; however, the mechanisms for this remain uncle
119 essors are known to impact the expression of heterosis; however, the potential role of microbes in he
120 ple, the genetic and physiological causes of heterosis (hybrid vigor) have remained elusive for nearl
121 re consistent with the well-known effects of heterosis (hybrid vigour) described when outcrossing ani
123 Large increases in biomass and yield in high-heterosis hybrids suggest that alterations in bioenerget
125 es between natural populations can result in heterosis if recessive or nearly recessive deleterious m
126 ther, our data suggest that the magnitude of heterosis in A. suecica is environmentally regulated, ar
127 tal lines can directly or indirectly trigger heterosis in Arabidopsis hybrids independent of genetic
128 pite the importance and wide exploitation of heterosis in commercial crop breeding, the molecular mec
131 e of favorable genes to explain the observed heterosis in grain yield and other traits, although epis
133 s been shown to affect metabolic and biomass heterosis in interspecific hybrids or allotetraploids.
134 echanisms that may cause allozyme-associated heterosis in natural populations has proven difficult.
137 eveloping hybrids with greater expression of heterosis in productivity and concentrations of provitam
142 aving repulsion linkage between two inbreds, heterosis in the hybrid can appear as a single locus wit
143 e gene-expression patterns underlying growth heterosis in the Pacific oyster (Crassostrea gigas) in t
145 It may be that this example of molecular heterosis in vitro provides the basis for maintenance of
146 are not the only example of hybrid vigour or heterosis in yeasts, but the full breadth of interspecie
147 Arabidopsis thaliana shows hybrid vigor (heterosis) in progeny of crosses between Columbia-0 and
150 Various models have been posited to explain heterosis, including dominance, overdominance, and pseud
151 oil steaming increased rather than decreased heterosis, indicating that the direction of the effect d
154 this study suggest that a major component of heterosis is a mechanism that is modulated by dosage-sen
166 effect was an increase in apparent allozyme heterosis later in ontogeny coinciding with a series of
167 ds included in this study exhibit a range of heterosis levels; however, we did not observe difference
169 gulation, indicating that expression GxE and heterosis may result from the evolution of transcription
170 ctions corresponding to the evolutionary and heterosis mechanisms, asking whether any effects of gene
172 ovides a straightforward way to evaluate the heterosis of crossbreeds and the breeding values of pure
173 agriculture due to its potential to preserve heterosis of F(1) hybrids through subsequent generations
174 nalysis of the congenic lines argues against heterosis of outbred backgrounds contributing to Egfrtm1
176 crosatellite markers and immediate recovery (heterosis) of egg viability and flight metabolic rate in
177 insights into associations between sRNAs and heterosis, often using a single hybrid genotype or tissu
179 ng depression and the converse phenomenon of heterosis or hybrid vigor remain poorly understood despi
180 echanisms underlying this phenomenon, called heterosis or hybrid vigor, are not well understood despi
183 ynthesis, we describe a case of single-locus heterosis, or overdominance, where the heterozygote disp
184 es identifying a specific genetic example of heterosis, our research indicated that integrated molecu
185 addition to genetic factors contributing to heterosis, our results strongly suggest that epigenetic
186 enotype, revealing effects of strain dosage, heterosis, parent of origin, epistasis, and sex-specific
195 hybrids differed in the level of high-parent heterosis relative to the derived triploid inbreds.
198 lating level of heterozygosity and degree of heterosis should take into account this nonuniform distr
199 e correlations are viewed as a phenomenon of heterosis, so that it cannot possibly occur under within
200 As such, modes of inheritance that drive heterosis, such as dominance or overdominance, may be co
201 approximately 350 candidate genes for growth heterosis that exhibit concordant nonadditive expression
202 nd Hardy-Weinberg analysis suggested partial heterosis, that is, an increased risk for heterozygotes,
204 inance vs overdominance as an explanation of heterosis; the classical vs balance hypothesis for genet
205 nes directly contribute to, or merely mimic, heterosis, they may aid generation of more vigorous and
207 :1) additive loci, dominant contributions to heterosis to outnumber overdominant, and extensive pleio
208 ing the early developmental manifestation of heterosis under fluctuating environmental conditions in
209 ssed the possible epigenetic contribution to heterosis using epigenetic inbred lines (epiRILs) with v
210 effect; 2) the ranking of factors affecting heterosis was dominance > dominance-by-dominance > over-
211 mplementation hypothesis in a direct manner, heterosis was examined in diploid inbreds and reciprocal
212 fitness was 68% lower in small populations; heterosis was significantly greater for small (mean = 70
213 ular components that may contribute to trait heterosis, we analyzed paired proteomic and transcriptom
214 relevance of expression differences to trait heterosis, we compared seedling leaf protein levels to t
215 To comprehensively decipher the genetics of heterosis, we present a new design of multiple linked F1
216 ensive model to explain the phenomenology of heterosis, we provide the details of what needs to be ex
217 sign is used to dissect the genetic basis of heterosis which accelerates maize molecular design breed
218 lant size and cell number are reminiscent of heterosis, which also increases plant size primarily thr
219 Muller CD (neo-X excess), while males showed heterosis with excessive (neo-X, D. nasuta Muller CD) ge
220 ci showed a significant increase in apparent heterosis with ontogeny, while one locus showed a signif
221 A wide range of microbiome features display heterosis within individual crosses, consistent with pat