ライフサイエンスコーパス: high BP

1 nly 12% of patients with hypertension with a high BP measurement during an ambulatory visit received

2 Although high BP is a major risk factor for CAD, the genetic inte

3 ere periodontitis, hypertension history, and high BP.

4 tension, poorly controlled hypertension, and high BP levels are associated with a decline in pCBF.

5 with increased events rates at both low and high BP values, both unadjusted and after adjustment for

6 important in reducing adulthood obesity and high-BP risk.

7 se-response effect for the length of time at high BP levels.

8 A causal effect exists between high BP and a reduced late-life risk of AD.

9 ge from 1999-2000 to 2011-2012 in borderline high BP (7.6% [95% CI, 5.8-9.8] vs 9.4% [95% CI, 7.2-11.

10 11.9]; P = .90) or either high or borderline high BP (10.6% [8.4-13.1] vs 11.0% [95% CI, 8.8-13.4]; P

11 and 2011-2012, but either high or borderline high BP remained stable.

12 The presence of both high BP and diabetes was associated with fourfold odds o

13 tabolic syndrome and its two key components, high BP and diabetes were associated with age-related ca

14 he individual metabolic syndrome components, high BP was associated with all three cataract types; di

15 In contrast, high BP is not associated with higher risk of mortality

16 eceived educational brochures on controlling high BP.

17 known to have limited accuracy in detecting high BP, while the utility of standing BP in diagnosing

18 omes were annual BP screening documentation, high BP follow-up documentation at 6 months and 1-year,

19 In either case, overperfusion during high BP may cause oxidative injury to the outer retina,

20 An EMR high BP advisory combined with team training, audit, and

21 This quality improvement study of an EMR high BP advisory intervention found significantly improv

22 .42-2.40) for diabetes, 1.92 (1.47-2.52) for high BP, and 1.27 (1.04-1.55) for metabolic syndrome.

23 We constructed a genetic risk score for high BP by using 314 published BP loci in 277 005 indivi

24 Screening for high BP in school settings appears to be feasible and co

25 In both age groups, high BP coexisted with other cardiovascular disease risk

26 7 American Academy of Pediatrics guidelines, high BP was defined as stage 1 and 2 hypertension, with

27 ffspring with high BP whose parents also had high BP showed an unexpected rise in plasma epinephrine

28 n in offspring with low BP whose parents had high BP or in offspring with high BP whose parents had l

29 ents with high BP in one site tended to have high BP in another site.

30 to screen and identify adolescents who have high BP and initiate interventions to control the burden

31 Irrespective of family history, high BP is associated with increased body weight and hyp

32 ence estimate of masked asleep hypertension (high BP while sleeping but without high BP measured in t

33 Although the groups presented with identical high BP, endothelial-specific Epas1 gene deletion accent

34 There was a decrease in high BP between 1999-2000 (3.0% [95% CI, 2.0-4.3]) and 2

35 ation of the optimally rapid brain kinetics, high BP and brain uptake, and favorable metabolic profil

36 r and metabolic diseases, including obesity, high BP, diabetes, CKD, myocardial infarction, and strok

37 was to determine whether this attenuation of high BP is associated with prevention of other pathophys

38 ense (AT1R-AS) attenuates the development of high BP in the spontaneously hypertensive (SH) rat model

39 rged showing that the detrimental effects of high BP can be demonstrated at BP levels considered norm

40 e responsible for the increased frequency of high BP and kidney disease in African Americans, with pa

41 These results document the importance of high BP as a modifiable risk factor for ESRD in China.

42 -based recommendations for the management of high BP and should meet the clinical needs of most patie

43 blic health guidelines for the management of high BP contain numerous dietary recommendations, of whi

44 pertinent to the diagnosis and management of high BP in adults with CKD, excluding those receiving ki

45 ss to dental care improved the prediction of high BP by 2%.

46 The prevalence of high BP in younger children with stunting was high at 40

47 the prevalence of high BP was 35.1% (95% CI, 31.5%-38.9%) in children aged

48 his cross-sectional study, the prevalence of high BP, along with cardiovascular risk factors, was sub

49 or in fiber is associated with prevalence of high BP.

50 may be added to treatment and prevention of high BP.

51 besity were associated with a higher risk of high BP in both younger (prevalence ratio, 1.17; 95% CI,

52 tein cholesterol levels had a higher risk of high BP.

53 novel SNPs in MDM4 and HRH1 with sequelae of high BP including coronary artery disease (CAD), left ve

54 ficant decrease in the trend of follow-up of high BP measurement at 6 months (1265 of 4941 patients w

55 al HTN guidelines; however, the follow-up of high BP was still suboptimal.

56 AD is associated with central obesity and/or high BP.

57 e than 1 in 10 had either borderline high or high BP.

58 Persistent high BP, or hypertension, is a complex trait with both g

59 patient's private physician for persistently high BP.

60 HSD < 9439.5 um(2) predicted high BP and arterial stiffness (95% CI in all participan

61 e added value of dental visits in predicting high BP over the variables included in the Framingham Hy

62 he predominant causes of ESRD are reportedly high BP and diabetes mellitus.

63 we aimed to estimate prevalence of sustained high BP in 3 public secondary schools using the American

64 Participants with sustained high BP underwent 24-h ambulatory BP monitoring (step 4)

65 In the high-BP group, BF(ONH) had no significant change during

66 ages of disclosing genetic predisposition to high BP for risk stratification needs careful evaluation

67 e role of these factors in predisposition to high BP, we studied 100 young adults with high or low BP

68 personal BP and a familial predisposition to high BP.

69 pared with those without high triglycerides, high BP, and MetS after adjusting for potential confound

70 Untreated high BP, or hypertension (HTN), is associated with incre

71 A very high BP (systolic BP >180 mm Hg) was observed 11 637 tim

72 high BP; the estimated probability of a very high BP was greater in the low-engagement group (1.42%;

73 am can support long-term BP control and very high BP detection.

74 ement was associated with lower risk of very high BP; the estimated probability of a very high BP was

75 Severe periodontitis was associated with high BP, with OR of 2.93 (95% CI: 1.25 to 6.84), after a

76 ing the best methods to screen children with high BP measurements and manage their care.

77 P-CATCH trial found that among children with high BP measurements, racial and ethnic disparities in r

78 Offspring with high BP whose parents also had high BP showed an unexpec

79 ose parents had high BP or in offspring with high BP whose parents had low BP.

80 Approximately one-third of participants with high BP on screening and ambulatory BP monitoring diagno

81 values correlated to show that patients with high BP in one site tended to have high BP in another si

82 entified the first 17 eligible patients with high BP measurements each month.

83 with up-to-date management of patients with high BP.

84 f reaching medically underserved people with high BP cared for at a safety-net Emergency Departments,

85 cose, hemoglobin A1c, and lipid profile with high BP were examined using log binomial regression.

86 Subjects with high BP, irrespective of parental BP, were heavier (P=.0

87 eart Association BP guideline and those with high BP (120 to 159/<100 mm Hg) were further stratified

88 rtension (high BP while sleeping but without high BP measured in the clinic [clinic BP]) for the Unit