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1 ion, Detection, Evaluation, and Treatment of High Blood Pressure).
2 ion, Detection, Evaluation, and Treatment of High Blood Pressure).
3 ion, Detection, Evaluation, and Treatment of High Blood Pressure.
4 products is associated with reduced risk of high blood pressure.
5 mm Hg or higher, or receiving treatment for high blood pressure.
6 a and Middle East, and central Europe it was high blood pressure.
7 ransgenic mice overexpressing sEng displayed high blood pressure.
8 d low, 2,887 (81.5%) normal, and 478 (13.5%) high blood pressure.
9 as a main therapeutic target in controlling high blood pressure.
10 use of antihypertensive medications to treat high blood pressure.
11 ility to lower either high glucose levels or high blood pressure.
12 its and harms of screening and treatment for high blood pressure.
13 was found for the benefits of screening for high blood pressure.
14 ovel therapeutic target for the treatment of high blood pressure.
15 factors for cardiovascular disease, such as high blood pressure.
16 ion, Detection, Evaluation, and Treatment of High Blood Pressure.
17 d structural adaptations are related to very high blood pressure.
18 d causes oxidative stress independent of the high blood pressure.
19 auses endothelial dysfunction independent of high blood pressure.
20 notherapy in preventing CVD complications of high blood pressure.
21 have a role in preventing the development of high blood pressure.
22 with statin treatment in patients with known high blood pressure.
23 ch have provided analyses of sub-groups with high blood pressure.
24 for disease such as elevated cholesterol and high blood pressure.
25 modification for prevention and treatment of high blood pressure.
26 dency to give birth to small babies and have high blood pressure.
27 , high blood cholesterol concentrations, and high blood pressure.
28 nsistent with an association between RCC and high blood pressure.
29 o increased bone-mineral loss in people with high blood pressure.
30 with macular degeneration is associated with high blood pressure.
31 ion, it is a potentially reversible cause of high blood pressure.
32 Fundus lesions suggest high blood pressure.
33 on Trial) on adverse events in patients with high blood pressure.
34 may play an important role in the genesis of high blood pressure.
35 aemia, abdominal obesity, dyslipidaemia, and high blood pressure.
36 quences, including overweight or obesity and high blood pressure.
37 ure development of overweight or obesity and high blood pressure.
38 ium intake is an established risk factor for high blood pressure.
39 on the inflammasome may be mediated through high blood pressure.
40 presenting with acute chest or back pain and high blood pressure.
41 between parity and elevated triglycerides or high blood pressure.
42 s of lower BMI reducing risk of diabetes and high blood pressure.
43 bclinical hypothyroidism was associated with high blood pressure (1.24; 1.04-1.48) and high serum tri
44 nosed diabetes, 6.38 (95% CI, 5.67-7.17) for high blood pressure, 1.88 (95% CI,1.67-2.13) for high ch
45 /low blood pressure, 1.87 (P=0.002); low CRP/high blood pressure, 2.54 (P<0.0001); and high CRP/high
47 , 9.5%), unsafe sex (92 million DALY, 6.3%), high blood pressure (64 million DALY, 4.4%), tobacco (59
49 risk factors for global disease burden were high blood pressure (7.0% [95% uncertainty interval 6.2-
51 e cardiovascular and metabolic outcomes (eg, high blood pressure, abnormal lipid levels, and insulin
52 ximately 9.7 million annual CVD deaths, with high blood pressure accounting for more CVD deaths than
53 on variables, namely, age, gender, diabetes, high blood pressure, acculturation score, income, and ed
54 tion has resulted in a reduced prevalence of high blood pressure and a concomitant decline in left ve
56 ction in the risk factors for heart disease (high blood pressure and cholesterol) but not on risk fac
57 ional studies suggest an association between high blood pressure and dementia, and in turn the use of
59 target organs have the potential to promote high blood pressure and end-organ damage, we show here t
61 ry reports were greater than 90%, except for high blood pressure and high cholesterol level (negative
63 lness, Mr R says at times he has ignored his high blood pressure and his physicians' recommendations.
64 thophysiological state that is manifested as high blood pressure and is a major risk factor for strok
65 asting blood glucose levels, the presence of high blood pressure and low levels of high-density lipop
66 elationship between excess sodium intake and high blood pressure and other adverse health outcomes.
67 etarded growth in utero has been linked with high blood pressure and other risk factors for cardiovas
68 gnant corin- or ANP-deficient mice developed high blood pressure and proteinuria, characteristics of
70 ssociation between genetic predisposition to high blood pressure and risk of CVD in individuals with
71 ttributable to established CVD risk factors (high blood pressure and serum cholesterol, smoking, and
72 r spirometry levels; however, the effects of high blood pressure and taking beta-blockers diminished
73 2 of whom had preeclampsia (characterized by high blood pressure and the presence of protein in the u
74 ng Medical Subject Headings: hypertension or high blood pressure and trials and oldest old or very ol
75 ion, Detection, Evaluation, and Treatment of High Blood Pressure) and defined hypertension as a systo
76 ctors such as high serum cholesterol levels, high blood pressure, and cigarette smoking with the prev
77 n, exercise, lowering serum lipids, lowering high blood pressure, and daily antiplatelet therapy.
78 ported high cholesterol, high triglycerides, high blood pressure, and diagnosed type 2 diabetes melli
79 l conditions such as obesity, hyperglycemia, high blood pressure, and dyslipidemia that are major dri
80 c risk factors (obesity, insulin resistance, high blood pressure, and dyslipidemia) were associated w
83 ing and developed regions, alcohol, tobacco, high blood pressure, and high cholesterol were major cau
85 etS having a combination of central obesity, high blood pressure, and hyperglycemia had a 2.36-fold (
86 excessive alcohol consumption, uncontrolled high blood pressure, and hyperlipidaemia--that can be ef
87 dietary Na(+) restriction in the absence of high blood pressure, and its consequences for the kidney
88 high-density lipoprotein cholesterol levels, high blood pressure, and obesity, mainly central adiposi
91 lesterol level, overweight, current smoking, high blood pressure, and parental history of cardiovascu
92 elation between their use, the prevalence of high blood pressure, and the presence of electrocardiogr
93 ion, Detection, Evaluation, and Treatment of High Blood Pressure are also described; this report defi
95 implications for patients with premorbid low/high blood pressure are unknown.Objectives: To investiga
96 ludes glucose intolerance, dyslipidemia, and high blood pressure, as a strong predictor of the obesit
97 (allergies, arthritis, back pain, headaches, high blood pressure, asthma, and major depression) occur
99 mm Hg) SBP control in 9361 older adults with high blood pressure at increased risk of cardiovascular
100 ation of polycystin-2 to cilia could promote high blood pressure because of inability to synthesize N
101 age, sex, race, poverty, smoking, diabetes, high blood pressure, body mass index, and serum choleste
103 rvous system has been proposed as a cause of high blood pressure (BP) and may be related to diet and
104 methyltransferase (DNMT1) inhibitor, reduces high blood pressure (BP) by regulating aortic ECM remode
110 nt guidelines on diagnosis and management of high blood pressure (BP) include substantial changes and
116 While the pathogenesis of diabetes-induced high blood pressure (BP) is not entirely clear, current
117 sociated with iron deficiency in infancy and high blood pressure (BP) later in life.We investigated t
119 ing a 'J-curve' phenomenon such that low and high blood pressure (BP) levels are associated with incr
120 ttee panel recommendations for management of high blood pressure (BP) recommend a systolic BP thresho
124 dividuals, subjects with high triglycerides, high blood pressure (BP), and MetS were more likely to h
125 al artery stenosis (ARAS) is associated with high blood pressure (BP), decreased kidney function, ren
126 tiple potential risk factors for obesity and high blood pressure (BP), including chronic inflammation
129 a high sodium intake is causally related to high blood pressure, but debate over recommendations to
130 ation, insulin resistance, dyslipidemia, and high blood pressure, but their causal relation to these
131 ial sodium channel mutations appear to cause high blood pressure by increasing sodium reabsorption th
132 al VSMC physiology and support the idea that high blood pressure can arise from a primary abnormality
136 d age, smoking, peripheral arterial disease, high blood pressure, coronary artery disease, diabetes,
137 ion, Detection, Evaluation, and Treatment of High Blood Pressure criteria (<120/<80 mm Hg), there was
139 ovel mechanism involved in the prevention of high blood pressure development triggered by high-salt i
140 assic cardiovascular risk factors--including high blood pressure, diabetes and smoking--has a central
141 ined as at least 3 coexistent morbidities of high blood pressure, diabetes mellitus, osteoporosis, no
142 with each decade above 65 years; history of high blood pressure, diabetes mellitus, previous transie
145 if they self-reported a diagnosis of asthma, high blood pressure, diabetes, high cholesterol, heart p
146 lems, such as heart disease, kidney disease, high blood pressure, diabetes, stroke, obesity, and depr
147 ily history, high triglycerides at baseline, high blood pressure, diuretics, pre-enrollment weight ch
149 forts to understand how risk factors such as high blood pressure, dysregulated blood lipids and diabe
150 advances since the inception of the National High Blood Pressure Education Program (NHBPEP) in 1972,
152 ren included in the database of the National High Blood Pressure Education Program Working Group on H
153 in Children and Adolescents by the National High Blood Pressure Education Program Working Group on H
156 eviously established AF risk factors, namely high blood pressure, elevated body mass index, diabetes
157 rs such as obesity, excess percent body fat, high blood pressure, elevated resting heart rate and sed
158 ion, Detection, Evaluation, and Treatment of High Blood Pressure emphasizes the importance of systoli
161 ea was inversely associated with the risk of high blood pressure, fasting blood sugar, and triglyceri
164 face of growing obesity, we sought to assess high blood pressure (HBP) secular trends in children and
165 east three co-existent morbidities of either high blood pressure (HBP), diabetes mellitus, osteoporos
166 diovascular risk factors (smoking, diabetes, high blood pressure, high cholesterol and angina) were q
167 th had high rates of coronary heart disease, high blood pressure, high cholesterol concentrations, an
168 work identified major risk factors for CHD (high blood pressure, high cholesterol levels and evidenc
169 were significantly associated with diabetes, high blood pressure, high cholesterol, asthma, arthritis
170 hort established the cardiac threat posed by high blood pressure, high cholesterol, smoking, obesity,
171 isks, tobacco smoking, high body mass index, high blood pressure, high fasting plasma glucose, physic
172 ysregulation (obesity, elevated blood sugar, high blood pressure, high levels of triglycerides and de
173 d height, weight and metabolic risk factors (high blood pressure, high triglycerides, low high-densit
174 However, CIH also causes morbidity (e.g., high blood pressure, hippocampal apoptosis), rendering i
175 at arterial stiffening precedes the onset of high blood pressure; however their molecular genetic rel
176 s between arsenic exposure and prevalence of high blood pressure; however, studies examining the rela
178 dolescence might substantially reduce excess high blood pressure/hypertension in this ethnic group.
180 es not play a crucial role in maintenance of high blood pressure in adult SHRs, and that the improvem
183 ion, Detection, Evaluation and Management of High Blood Pressure in Adults provides recommendations f
184 idence-Based Guideline for the Management of High Blood Pressure in Adults" has garnered much attenti
186 idence-based guideline for the management of high blood pressure in adults: Report from the panel mem
187 idence-based guideline for the management of high blood pressure in adults: Report from the panel mem
188 eatment of elevated lipids, and treatment of high blood pressure in all patients and of beta-blockers
189 mponents, the intervention decreased risk of high blood pressure in both sexes (relative risk, 0.83;
190 Pressure Education Program Working Group on High Blood Pressure in Children and Adolescents (the Ped
191 the Diagnosis, Evaluation, and Treatment of High Blood Pressure in Children and Adolescents by the N
192 as defined according to the Fourth Report on High Blood Pressure in Children and Adolescents from the
193 s that the evidence to support screening for high blood pressure in children and adolescents is insuf
194 the diagnosis, evaluation, and treatment of high blood pressure in children and adolescents' contain
200 lycemia and diabetes mellitus, together with high blood pressure in low-income countries, unless effe
202 with or without stenting will cure or reduce high blood pressure in more than half of all affected ch
203 the common clinical practice of maintaining high blood pressure in patients with intracranial stenos
204 nks the orexin system to the pathogenesis of high blood pressure in SHRs and suggests that modulation
206 We aimed to establish whether the effects of high blood pressure in the brain are evident as early as
208 ed in the pathophysiology and maintenance of high blood pressure in the spontaneously hypertensive ra
209 hibition of ACE and a long-term reduction in high blood pressure in the spontaneously hypertensive ra
211 Age, male sex, history of previous ASCVD, high blood pressure, increased body mass index, active s
218 data indicate that genetic predisposition to high blood pressure is associated with an increased risk
221 factors early in life for the development of high blood pressure is critical to the prevention of car
223 The identification of genes responsible for high blood pressure is of major importance, because it p
227 ion, Detection, Evaluation, and Treatment of High Blood Pressure (JNC 7 guidelines); individualized g
229 tion, Detection, Evaluation and Treatment of High Blood Pressure (JNC 7) guidelines (provider educati
230 ion, Detection, Evaluation, and Treatment of High Blood Pressure (JNC VI) as one of the first-choice
231 ion, Detection, Evaluation, and Treatment of High Blood Pressure (JNC VI) of less than 140 mm Hg (sys
232 e on Detection, Evaluation, and Treatment of High Blood Pressure (JNC) reports, have recognized the h
233 ion, Detection, Evaluation, and Treatment of High Blood Pressure (JNC-7) classifies BP as normal, pre
236 s defined as syndrome X (high blood glucose, high blood pressure, low high density lipoprotein (HDL)
237 risk factors out of high visceral fat mass, high blood pressure, low high-density-lipoprotein choles
238 ion with cultural framings of stress causing high blood pressure, many patients turn to stress manage
239 anguage to the more patient-centered term of high blood pressure may help patients better understand
240 hildren is associated with increased risk of high blood pressure, metabolic syndrome, and future card
241 ndrome--a combination of disorders including high blood pressure, obesity, high triglyceride, and ins
242 etic determinants of essential hypertension, high blood pressure of unknown cause, are expected to be
244 isks for developing hypertension and stage 1 high blood pressure or higher (greater-than-or-equal to
245 rast, the residual lifetime risk for stage 2 high blood pressure or higher (greater-than-or-equal to
246 ugh the decline in lifetime risk for stage 2 high blood pressure or higher represents a major achieve
247 benefits, even when used in patients without high blood pressure or left ventricular dysfunction (the
248 ertensive subjects) were individuals who had high blood pressure or were taking antihypertensive medi
249 actors for both fetal growth restriction and high blood pressure or whether there is a genetic tenden
251 olesterol, high triglycerides, high glucose, high blood pressure, or high body mass index) previously
254 unger participants (age 25 to 44 years), and high blood pressure predominated in older participants (
256 re critically involved in the origins of the high blood pressure prevalent among a majority of adult
257 entails maternal vascular alterations (e.g., high blood pressure, proteinuria, and edema) and, in som
258 ion, Detection, Evaluation, and Treatment of High Blood Pressure provides a practical, evidence-based
259 ion, Detection, Evaluation, and Treatment of High Blood Pressure" provides a new guideline for hypert
260 ading to uncontrolled nerve activity causing high blood pressure, rapid heart rate, and abnormal hear
261 ned over 10 000 participants with documented high blood pressure requiring drug therapy and they both
265 alence of chronic conditions (heart disease, high blood pressure, stroke, emphysema, high cholesterol
266 empted to reduce modifiable risk factors for high blood pressure, such as excess sodium intake or hig
268 ve health outcomes as cancer, heart trouble, high blood pressure, suicidal ideation, and changes in e
269 h total cholesterol (> or =5.17 mmol/liter), high blood pressure (systolic blood pressure: > or =140
272 disorder is less effective in patients with high blood pressure than it is in normotensive individua
273 ion, Detection, Evaluation, and Treatment of High Blood Pressure, the multivariate-adjusted hazard ra
274 the morbidity and mortality of patients with high blood pressure, the precise cellular mechanisms inv
277 ion, Detection, Evaluation, and Treatment of High Blood Pressure, this interpretation of ALLHAT broad
278 sible interventions: scaling up treatment of high blood pressure to 70%, reducing sodium intake by 30
279 obesity, diabetes, hypertriglyceridemia, and high blood pressure to assign them to metabolic risk cat
281 pulation, ethnicity, current smoking status, high blood pressure, total cholesterol, body mass index,
282 sing the majority of infectious diseases and high blood pressure treatment beginning to affect the pr
284 e drinking has been linked to heart disease, high blood pressure, type 2 diabetes, and the developmen
285 dings provide the first direct evidence that high blood pressure upregulates the Ca(L) channel alpha1
288 t LDL cholesterol reduction in patients with high blood pressure was convincingly demonstrated in the
291 general outpatient population of adults with high blood pressure, we found a small reduction in systo
292 2.53); however, associations with new-onset high blood pressure were attenuated (OR: 1.14; 95% CI: 0
293 jects with normal blood pressure, those with high blood pressure were not at increased risk of incide
294 rtaken in South Africa, patients treated for high blood pressure were randomly allocated in a 1:1:1 r
295 diac hypertrophy and heart failure caused by high blood pressure were studied in single myocytes take
296 flect greater calcium losses associated with high blood pressure, which may contribute to the risk of
297 tion, Detection, Evaluation and Treatment of High Blood Pressure will agree with the American Diabete
299 and glucose metabolism, central obesity, and high blood pressure, with an increased risk of type 2 di
300 ers of people affected and the prevalence of high blood pressure worldwide are expected to increase o