ライフサイエンスコーパス: high-fat

1 overnight fast, before and after consuming a high-fat (64% energy), high-calorie (+47% kcal) diet for

2 Among other factors, diets with high fat and carbohydrate content have been implicated i

3 oxidative stress and adiposity in mice fed a high fat and fructose supplemented western diet (WD).

4 A Western diet (WD)-characterized by its high fat and simple sugar content-is thought to predispo

5 of Collaborative Cross (CC) mice were fed a high-fat and high-sucrose (HF/HS) diet or a control diet

6 re metabolically hyperactive and, when fed a high-fat and high-sugar diet, are resistant to obesity,

7 ith taste and reward processing to palatable high-fat- and high-fat/high-sugar food tastes.

8 owing breakfasts: 1) a very-low-carbohydrate high-fat breakfast (LCBF; <10% of energy from carbohydra

9 A very-low-carbohydrate high-fat breakfast lowers postbreakfast glucose excursio

10 , whereas high-fat dairy (total, butter, and high-fat cheese) consumption was positively associated [

11 An increase in full-fat milk, high-fat cheese, and total high-fat dairy was associated

12 inflammation and fibrosis after being fed a high-fat, -cholesterol, and -fructose (HFCF) diet.

13 ced human cells, as well as in mice fed with high-fat chow; mechanistically, inflammasome-activating

14 (-0.03 mmol/L; -0.05, -0.01 mmol/L), whereas high-fat dairy (total, butter, and high-fat cheese) cons

15 in full-fat milk, high-fat cheese, and total high-fat dairy was associated with greater increases in

16 increases in body weight and BMI [e.g., for high-fat dairy: beta = 0.13 (0.05, 0.21) kg and 0.04 (0.

17 ed [e.g., 0.04 (0.02, 0.06) mmol/L for total high-fat dairy].

18 ht, a second objective was to determine if a high fat diet (HF) would alter GWI outcomes.

19 een obesity and AD by feeding APP/PS1 mice a high fat diet (Hfd) and evaluating behavioral, physiolog

20 ve been implicated in the mechanism by which high fat diet (HFD) and saturated fatty acids (SFA) modu

21 In response to high fat diet (HFD) feeding for 6 or 18 weeks, WT and AI

22 We have previously shown that high fat diet (HFD) for 2 weeks increases airway hyperre

23 female mice were fed a normal chow (NC) or a high fat diet (HFD) for 5 weeks before mating, then also

24 ested the hypothesis that Nod2 protects from high fat diet (HFD)-dependent hepatic cancer.

25 a growing body of evidence illustrating that high fat diet (HFD)-induced maternal obesity can regulat

26 y changes occurring in response to a chronic high fat diet (HFD).

27 hondrial metabolic proteins in response to a high fat diet (HFD).

28 osis was induced in miR-144 knockout mice by high fat diet and vascular lesions were quantified by Oi

29 at CHRNA2 signaling is activated after acute high fat diet feeding and this effect is manifested thro

30 betic women and in a mouse model of maternal high fat diet feeding.

31 s in body weight or metabolic function after high fat diet feeding.

32 Mice were divided into control and high fat diet groups with or without exercise training.

33 sexes consuming more sucrose, sucralose and high fat diet if from obese mothers.

34 Unexpectedly, high fat diet induced extensive atherosclerosis in miR-1

35 g Rebaudioside A and sucralose on NASH using high fat diet induced obesity mouse model by substitutin

36 impact on weight gain and energy balance in high fat diet induced obesity.

37 diac-specific deletion of PKBalpha/beta or a high fat diet inhibits insulin-induced phosphorylation o

38 Triple transgenic dams were administered high fat diet or regular chow throughout 3 weeks gestati

39 Gestational high fat diet protects offspring against the development

40 rnal metformin treatment along with maternal high fat diet significantly increased mouse placental ab

41 t when female Ppp1r15a mutant mice are fed a high fat diet they gain less weight than wild type litte

42 ce (C57BL/6J mice, 33 weeks old), fed with a high fat diet which increases adipose tissue favouring o

43 were randomly assigned to receive chow diet, high fat diet with sugar in drinking water (Western diet

44 microvesicles are enhanced by exposure to a high fat diet, a known risk factor for atherosclerosis.

45 Mice were fed a standard diet, a high fat diet, or these diets supplemented isocaloricall

46 Mice were fed a well-defined high fat diet, with (HFD/ATI) or without (HFD) nutrition

47 ption factor A (TFAM) and inhibited maternal high fat diet-impaired placental efficiency and glucose

48 ammation in both models, disturbed flow- and high fat diet-induced atherosclerosis, whereas Nck2 dele

49 Furthermore, established high fat diet-induced hepatic steatosis was effectively

50 mic overexpression of SH2B1 protects against high fat diet-induced obesity and metabolic syndromes.

51 lerated fat mass loss on a normal diet after high fat diet-induced obesity.

52 of streptozocin-induced type 1 diabetes and high fat diet-induced type 2 diabetes mouse models and l

53 burden in miR-144 knockout mice receiving a high fat diet.

54 improves metabolic parameters in mice fed a high fat diet.

55 uced weight gain and adiposity in mice fed a high fat diet.

56 alters the metabolic phenotype in mice fed a high fat diet.

57 ation in an obesity mouse model induced by a high fat diet.

58 emic blood from aged mice and upon feeding a high-fat diet (Apoe(-/-) mice).

59 ved choline-deficient, l-amino acid-defined, high-fat diet (CDAHFD) for 6 weeks or (d) 9 weeks (n = 8

60 r low-fat counterparts (LF mice), mice fed a high-fat diet (HF mice) had impairments in inflammatory

61 s demonstrate that in comparison with males, high-fat diet (HFD) allergic female mice exhibit a reduc

62 ed into two groups fed either a control or a high-fat diet (HFD) and then the mice on each diet were

63 red these with the effects of a prototypical high-fat diet (HFD) as well as cessation of exposure on

64 Mice fed a high-fat diet (HFD) become obese and develop osteoarthri

65 Previous studies showed that 12 weeks of high-fat diet (HFD) consumption caused not only prediabe

66 t et al. showed that feeding maternal mice a high-fat diet (HFD) during lactation attenuated the acti

67 ulin resistance in offspring of mothers on a high-fat diet (HFD) during pregnancy.

68 rate in a murine prostate cancer model, that high-fat diet (HFD) enhances the MYC transcriptional pro

69 s of a non-pharmacological intervention in a high-fat diet (HFD) fed mouse model, capable of recapitu

70 Here, we showed that maternal high-fat diet (HFD) feeding during lactation in mice eli

71 Control and ACC2 iKO mice were subjected to high-fat diet (HFD) feeding for 24 weeks to induce obesi

72 High-fat diet (HFD) feeding further exacerbates the K2KO

73 emia and glucose intolerance after long-term high-fat diet (HFD) feeding.

74 s-induced insulin resistance with short-term high-fat diet (HFD) in young mice.

75 As high-fat diet (HFD) is a potent inducer of gut dysbiosis

76 Consuming a high-fat diet (HFD) is a risk factor for obesity and dia

77 gene expression, we analyzed the impact of a high-fat diet (HFD) on Abcc8 knockout mice.

78 Here, we explored the effects of a high-fat diet (HFD) on energy balance, gut microbiota, a

79 The influence of maternal high-fat diet (HFD) on metabolic response to ozone was e

80 However, mice fed a high-fat diet (HFD) only develop fatty liver without sig

81 They were fed a high-fat diet (HFD) or regular chow for 4 weeks.

82 lobule membrane (MFGM-PL) supplementation to high-fat diet (HFD) rats during pregnancy and lactation

83 We demonstrate that a high-fat diet (HFD) reproducibly changes gut microbial c

84 Chronic high-fat diet (HFD) results in the accumulation of a mon

85 fed a control diet, plasmas from mice fed a high-fat diet (HFD) showed delayed PG and reduced PG vel

86 aluation paradigm, we found that exposure to high-fat diet (HFD) suppresses the intake of nutritional

87 b/ob (obese) or heterozygote (lean) mice fed high-fat diet (HFD) that received either 17beta-Estradio

88 -/-) .Leiden mice received 16 weeks either a high-fat diet (HFD) to induce obesity, or chow as refere

89 ntial metal distribution in adult mice fed a high-fat diet (HFD) were examined.

90 icantly increased in NT(+/+) mice fed with a high-fat diet (HFD) which were improved in NT-deficient

91 A previous study demonstrated that a high-fat diet (HFD), administered for one-three-days, in

92 Obesity was induced by high-fat diet (HFD), and blood pressure (BP) was measure

93 When challenged with high-fat diet (HFD), IRMOE mice are protected from diet-

94 Under an obesogenic high-fat diet (HFD), male offspring of exercised C57BL/6

95 ed mice fed a standard chow diet, short-term high-fat diet (HFD), or long-term HFD.

96 When mice were fed a high-fat diet (HFD), we found that fatty liver and dysli

97 r and cellular aspects of atherosclerosis in high-fat diet (HFD)-fed L13a KO and intact (control) mic

98 Furthermore, high-fat diet (HFD)-fed mice exhibit the downregulation

99 t in fatty livers in obese individuals or in high-fat diet (HFD)-fed mice.

100 ney (uninephrectomy [UniNx]) in mice reduced high-fat diet (HFD)-induced adipose tissue inflammation,

101 ate the role of neurovascular dysfunction in high-fat diet (HFD)-induced cognitive impairment.

102 Normal diet (ND) and high-fat diet (HFD)-induced DM mice were randomized into

103 ial transcription factor A (TFAM) attenuates high-fat diet (HFD)-induced fat gain and IR in mice in c

104 High-fat diet (HFD)-induced inflammation and steatosis o

105 transport and ATP synthesis, and aggravating high-fat diet (HFD)-induced NAFLD.

106 Here, we demonstrate that high-fat diet (HFD)-induced obesity impairs CD8(+) T cel

107 uromuscular dysfunctions in a mouse model of high-fat diet (HFD)-induced obesity.

108 ial vanilloid subfamily 1 (TRPV1) to counter high-fat diet (HFD)-induced obesity.

109 esity, hyperglycemia, and liver steatosis in high-fat diet (HFD)-treated male mice.

110 ly in AT and AT MDCs of wild-type mice fed a high-fat diet (HFD).

111 liver and further increases with fasting or high-fat diet (HFD).

112 ionship has been also found in rodents fed a high-fat diet (HFD).

113 tly increased in KCs of wild-type mice fed a high-fat diet (HFD).

114 ance and insulin sensitivity when exposed to high-fat diet (HFD).

115 Male Wistar rats were fed either a high-fat diet (HFD; 35% fat) or a standard diet (3.5% fa

116 normal diet, n=4; PCSK9-normal diet, n=6) or high-fat diet (HFD; WT-HFD, n=3; PCSK9-HFD, n=6).

117 aloric control diet and a 3-day hypercaloric high-fat diet (increase of 75% in energy, 81-83% energy

118 (tMCAO) in T2D/obese mice (after 7 months of high-fat diet [HFD]) and age-matched controls.

119 by challenging mice with a combination of a high-fat diet and angiotensin II.

120 icient to improve liver damage in mice fed a high-fat diet and in mice fed a methionine-choline-defic

121 Cancer risk factors, such as high-sugar or high-fat diet and inflammation, impact cell competition-

122 stress in mice-elicited by a combination of high-fat diet and inhibition of constitutive nitric oxid

123 esoid X receptor null (Fxr(Delta/E)) mice on high-fat diet as well as wild-type C57BL/6 and glucagon-

124 We show that high-fat diet attenuates the response of AgRP neurons to

125 tive of this study was to evaluate whether a high-fat diet can aggravate the liver disease caused by

126 demonstrate that both food restriction and a high-fat diet cause an endocannabinoid-dependent inhibit

127 al to subcutaneous fat (VAT/SAT) ratio after high-fat diet challenge, in comparison to their wild-typ

128 dults at moderate CVD risk, consumption of a high-fat diet containing SFA-reduced, MUFA-enriched dair

129 A high-fat diet did not alter arcuate NPY neuronal InsR ex

130 e previously determined that male mice fed a high-fat diet exhibit macrophage infiltration into the h

131 cise training protocol, in either low-fat or high-fat diet fed mice, did not require Bcl2-mediated au

132 r in vivo studies, ApoE(-/-) mice were fed a high-fat diet for 12 weeks.

133 Animals were fed a high-fat diet for 8 weeks and then sensitized and challe

134 rdiography in the Grb14-knockdown mice fed a high-fat diet for a period of four months.

135 DHEA in male iWAT and eWAT in response to a high-fat diet further strengthen the inference regarding

136 Modulating nutrient supply through high-fat diet improved survival, whereas high-glucose di

137 nating feeding mice with a low-fat diet or a high-fat diet in a 1-week switch protocol caused further

138 insulin resistance induced by a hypercaloric high-fat diet in humans.

139 echocardiograms to investigate the role of a high-fat diet in IAV-associated cardiac damage.

140 ed in obese patients and after 24 weeks of a high-fat diet in mice, accompanying signs of AT inflamma

141 ormin prevented weight gain in response to a high-fat diet in wild-type mice but not in mice lacking

142 in alveolar and interstitial macrophages in high-fat diet induced obese mice were lower than regular

143 on, we treated regular chow diet-fed mice or high-fat diet induced obese mice with lipopolysaccharide

144 increases paralleled profiles from long-term high-fat diet induced obesity in males.

145 and 530 significant changes were due to the high-fat diet intervention.

146 chromosome complement in combination with a high-fat diet led to enhanced weight gain in the presenc

147 these data provide the first evidence that a high-fat diet may be a risk factor for the development o

148 Using a high-fat diet model of obesity in mice and breast tissue

149 The low-fiber, high-fat diet of AN people and exposure to carcinogens d

150 fection negated the deleterious effects of a high-fat diet on cardiac function and remodeling, and ac

151 We have previously described that mice fed a high-fat diet rich in polyunsaturated fatty acids (HFD-P

152 f neutrophils in bone marrow than mice fed a high-fat diet rich in saturated fatty acids (HFD-S).

153 ROCK2(+/-) mice on a high-fat diet showed increased energy expenditure accomp

154 2(S587A) mice that were fed either a chow or high-fat diet showed similar weight gain as the wild-typ

155 High-fat diet stimulated lipid-specific mitochondrial ad

156 d and showed similarity to those observed in high-fat diet studies.

157 or 16-wks) on either a low-fat, high-fat, or high-fat diet supplemented with 1.5X branched chain amin

158 rsed by switching the mice from the SFA-rich high-fat diet to a MUFA-rich high-fat diet; nerve conduc

159 Mice were fed a high-fat diet to induce obesity and to study immunomodul

160 Using a high-fat diet to induce obesity, we examined preneoplast

161 I, Tan and Hang et al. report that feeding a high-fat diet to mice compromised the function of the or

162 hepatic transcriptional response in mice on high-fat diet treated with metformin was largely ablated

163 from mice who have undergone normal diet vs high-fat diet treatments.

164 The addition of cholesterol to a high-fat diet triggered hepatic pathology reminiscent of

165 ry of antibiotics in individuals consuming a high-fat diet was associated with the greatest risk for

166 For one group (n = 8), the high-fat diet was enriched with saturated long-chain FAs

167 nsgenerational inheritance of responses to a high-fat diet(9), thus raising the exciting possibility

168 ontrast, mice with comorbid diabetes (aging, high-fat diet, and streptozotocin-induced diabetes) had

169 ance and insulin resistance when raised on a high-fat diet, compared to wild-type (WT) mice.

170 abolism in mice fed either regular chow or a high-fat diet, demonstrating that these metabolic effect

171 Compared with Foxp3cre mice, after 13 wk of high-fat diet, Foxp3creInsrfl/fl mice exhibited improved

172 On a high-fat diet, HDAC6-deficient mice were depleted in rep

173 ght and body fat than did control mice fed a high-fat diet, resulting in ameliorated glucose toleranc

174 n Tgr5(-/-), but not Fxr(Delta/E) mice fed a high-fat diet, suggesting a role for intestinal Fxr.

175 In obese mice on a high-fat diet, the effects of metformin to reduce body w

176 tro from male and female mice fed control or high-fat diet, we demonstrated that macrophages derived

177 liver steatosis that developed in mice fed a high-fat diet, with or without combination with an inhib

178 High-fat diet-fed ApoE(-/-) mice displayed an increased

179 lEVs from high-fat diet-fed ApoE(-/-) mice, but not those from mic

180 containing H2Ab1 siRNA were administered to high-fat diet-fed C57BL/6 mice.

181 prostaglandin D(2) rescued myogenic tone in high-fat diet-fed control mice.

182 VSG or sham surgery was performed in high-fat diet-fed male hepatocyte-specific p53 wild-type

183 used endothelium-specific knockout mice and high-fat diet-fed mice to assess the role of endothelial

184 n completely ameliorate disease phenotype in high-fat diet-fed mice.

185 nd in vivo in human islets transplanted into high-fat diet-fed mice.

186 of neuropathy and restores nerve function in high-fat diet-fed murine models of peripheral neuropathy

187 verexpressing mice and in serum and PGWAT of high-fat diet-fed RBP4-overexpressing mice vs. wild-type

188 iated pMos and protected Ldlr(-/-) mice from high-fat diet-induced atherosclerosis.

189 ity and dyslipidemia, it protected mice from high-fat diet-induced glucose intolerance and insulin re

190 ptozotocin-induced beta-cell destruction and high-fat diet-induced glucose intolerance.

191 IRAK2 kinase inactivation also reduced high-fat diet-induced metabolic diseases.

192 Here we show that CSE knockout exacerbated high-fat diet-induced mouse obesity as well as its relat

193 with choline-deficient l-amino acid-defined high-fat diet-induced NASH.

194 hionyl-leucyl-phenylalanine, are elevated in high-fat diet-induced obese mice.

195 contrast, pADORA(1) signaling facilitates a high-fat diet-induced obesity (DIO).

196 mogenic genes in BAT, and are protected from high-fat diet-induced obesity and development of insulin

197 ic transfer of the EAT gene to mice prevents high-fat diet-induced obesity, insulin resistance and fa

198 ue-specific lymphangiogenesis during 16-week high-fat diet-induced obesity.

199 esulted in better glucose tolerance during a high-fat diet-induced regain phase (all, P < .05).

200 R deletion and were further increased by the high-fat diet.

201 xercise, mediate adaptations to exercise and high-fat diet.

202 ts showed the environment in vivo tends to a high-fat diet.

203 skeletal muscle adaptions to training during high-fat diet.

204 ocephalic arteries of ApoE-deficient mice on high-fat diet.

205 d diabetes phenotypes in mice fed a constant high-fat diet.

206 r compartments in fat and are activated by a high-fat diet.

207 hibit some, but not all, of the effects of a high-fat diet.

208 ion and peripheral glucose clearance after a high-fat diet.

209 lial hyperplasia, which is an indicator of a high-fat diet.

210 al glucose tolerance in mice made obese with high-fat diet.

211 ody weight and adiposity in obese mice fed a high-fat diet.

212 ic health of maternal mice challenged with a high-fat diet.

213 ior were also normal, even after exposure to high-fat diet.

214 pose and liver inflammation in response to a high-fat diet.

215 rteries of endothelial Fto-deficient mice on high-fat diet; conversely, direct addition of prostaglan

216 om the SFA-rich high-fat diet to a MUFA-rich high-fat diet; nerve conduction velocities and intraepid

217 oma, and metabolic derangements induced by a high-fat diet; therefore, elucidating the intracellular

218 We find that short-term high-fat-diet (HFD) feeding of mice activates prepronoci

219 e report that cardiac dysfunction induced by high-fat-diet (HFD) persists for two subsequent generati

220 Our previous reports showed that high-fat-diet (HFD)-fed mice with liver-specific knockou

221 KO mice), we showed that AKT1 is involved in high-fat-diet (HFD)-induced growth and survival of beta

222 found that when chimeric animals were fed a high-fat-diet, animals with low levels of chimerism show

223 pecific and whole-body insulin resistance in high-fat-diet-induced obese mice.

224 eased energy expenditure and amelioration of high-fat-diet-induced obesity and markedly improved gluc

225 e-specific MyD88 or IRAK2 deficiency reduced high-fat-diet-induced weight gain, increased energy expe

226 idation when glycolysis is sustained after a high-fat dietary challenge.

227 al bacteria to offset the adverse effects of high fat diets, C57BL/6J mice were fed control/low fat (

228 use gut microbiota in a manner distinct from high-fat diets (HFDs).

229 e are concerns about the association between high-fat diets and cognitive decline, this study aimed t

230 umulate in adipose and muscle tissues during high-fat diets and contribute to a state of local inflam

231 cantly less body weight and fat mass when on high-fat diets compared with littermate controls and wer

232 ism by feeding Sirt5 knockout mice (Sirt5KO) high-fat diets containing either C(8)/C(10) fatty acids

233 spheres as well as tumorigenesis induced by high-fat diets in an in vivo mouse model.

234 protection from weight gain on standard and high-fat diets, and an adiposity-dependent improvement i

235 abetic mouse models: streptozotocin-treated, high-fat fed, and Ins2Akita.

236 atory markers in regular chow-fed but not in high fat-fed mice.

237 tion and by decreased de novo lipogenesis in high fat-fed mice.

238 When administered in high-fat-fed mice, Hld-producing S. epidermidis signific

239 e normal GI tract and levels increase during high-fat feeding and gut infection and inflammation.

240 Upon high-fat feeding or reduced physical activity, phenotypi

241 m, thereby contributing to the prevention of high-fat feeding-induced skeletal muscle impairment.

242 rendered obese and insulin resistant through high-fat feeding.

243 leads to similar metabolic effects caused by high-fat feeding.

244 thus the only experience offspring had with high fat food was via maternal exposure.

245 nses (motor impulsivity) and binge intake of high-fat food (HFF) seen in binge eating disorder.

246 05) while, maximum reduction was observed in high-fat goshtaba (HFC).

247 of prolonged exposure to a high cholesterol high fat (HCHF) diet on LTL in a baboon model of atheros

248 ic disease (obesity/prediabetes) via chronic high-fat (HF) diet and modeled VCID via unilateral commo

249 Mice were fed high-fat (HF) diet and orally given arctigenin at 50 mg/

250 s established by feeding C57BL/6 mice with a high-fat (HF) diet before or during pregnancy.

251 We have shown that high-fat (HF) feeding during pregnancy significantly red

252 rporated >=3 servings/d of LF dairy, and the high-fat (HF) group incorporated >=3 servings/d of HF da

253 overnutrition was tested using chow-fed and high-fat (HF)-fed SkM-specific AMPKalpha1alpha2 knockout

254 7BL/6J mice were fed control/low fat (CD) or high fat (HFD) diets each supplemented with or without 2

255 ctive rhythmic metabolism is associated with high-fat high-caloric diet (HFD) feeding, ageing and obe

256 e tolerance at baseline and after 4 weeks of high-fat high-fructose (HFHF) feeding.

257 d-type littermates to a fast food-mimicking, high-fat high-sucrose diet and profiled the metabolic ph

258 d patch-clamp electrophysiology, how chronic high-fat high-sugar (HFHS) diet changes the physiology o

259 mice were placed on either control (P-CD) or high fat, high sucrose diet (P-HFHS) 1 week prior to and

260 Consumption of high fat, high sugar (western) diets is a major contribu

261 We have recently shown that a high-fat, high-calorie (HFHC) diet decreases whole body

262 A high-fat, high-calorie (HFHC) diet reduces whole body gl

263 L lactis subsp cremoris in female mice on a high-fat, high-carbohydrate (Western-style) diet caused

264 Female C57BL/6 mice were fed either a high-fat, high-carbohydrate (Western-style) diet that co

265 , and insulin resistance in female mice on a high-fat, high-carbohydrate diet.

266 o the protein composition resulting from the high-fat, high-cholesterol diet in this model.

267 PCSK9 transgenic Yucatan minipigs were fed a high-fat, high-cholesterol diet to induce atherosclerosi

268 In a high-fat, high-cholesterol diet-induced rodent model of

269 ucts (milk, cheese, and butter) to achieve a high-fat, high-dairy isoenergetic daily dietary exchange

270 essels were clamped for 1 hour in rats fed a high-fat, high-fructose (HFHF) diet for 5, 10, or 15 wee

271 h palmitate and in hearts of mice fed with a high-fat, high-sucrose (HF-HS) diet to develop obesity a

272 In C57BL/6JJcl mice fed with high fat-high sucrose chow (HFS), multifunctionality of

273 We investigated high-fat/high-carbohydrate (HF/HC) diet-induced nonalcoh

274 The mice were maintained on a high-fat/high-cholesterol diet for 16 weeks.

275 Purely dietary models, such as high-fat/high-cholesterol, Western diet, and choline-def

276 ing standard (normal) low-fat chow (NC) or a high-fat/high-sucrose Western diet (WD) in the absence (

277 In mice fed a Western (high-fat/high-sucrose) diet for 16 weeks, GLP-1 secretio

278 reward processing to palatable high-fat- and high-fat/high-sugar food tastes.

279 lation of SIRT1 is associated with aging and high-fat-induced obesity.

280 Notably, although a high-fat ketogenic diet elevates ISC function and post-i

281 Very low-carbohydrate, high-fat ketogenic diets (KDs) induce a pronounced shift

282 chemical ER stress inducer tunicamycin or by high-fat, low-methionine, and choline-deficient (HFLMCD)

283 and inferior frontal gyrus in response to a high-fat/low-sugar compared with low-fat/low-sugar milks

284 8% with fasting, and increased by 49% with a high-fat meal.

285 ion of endothelial postprandial responses to high-fat meals (HFMs) preserves metabolic health in chro

286 This study assessed the impact of 2 high-fat meals and endotoxemia on attention.

287 with a major propensity for defection (e.g. high-fat meals).

288 diet or Western-type diet (characterized by high fat, no fiber, and decreased minerals and vitamins)

289 The physical and oxidative stability of high-fat omega-3 delivery systems such as fish oil-in-wa

290 were kept (for 16-wks) on either a low-fat, high-fat, or high-fat diet supplemented with 1.5X branch

291 rfeeding (STOF) (50% carbohydrate, 30% fat), high-fat overfeeding (HFOF) (60% fat, 20% carbohydrate),

292 ective effects on glucose homeostasis during high-fat overfeeding.

293 The high-fat + periodontitis group presented an increase in

294 e identified with factor analyses: meats and high-fats; prudent diets; sugar, refined grains, and pro

295 sleep pressure, have low ATP levels despite high-fat stores, indicating a defective response to cell

296 insulin resistance and hepatic steatosis in high-fat-sucrose diet (HFS) induced obese mice.

297 for SAM regardless of HIV status involves a high-fat therapeutic diet.

298 Finally, switching from a high-fat to a low-fat diet, attenuates the MYC transcrip

299 eceived chow diet and normal water (CDNW) or high fat western diet and ad lib sugar water (WDSW).

300 f the unfolded protein response, linking the high-fat Western diet to systemic inflammatory disease.