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1 ur during long-term calorie restriction or a high protein diet.
2 t and decreased by starvation, high fat, and high protein diet.
3 reatic enzyme replacement and a high-energy, high-protein diet.
4 days, vs. almost 0% survival on the low-fat/high-protein diet.
5 increase in HDL seen in all diets except the high-protein diet.
6 activating TORC1 or by feeding the larvae a high-protein diet.
7 increased significantly with the normal and high protein diets.
8 ent years have seen strong tendencies toward high-protein diets.
9 consume relative to energy requirements from high-protein diets.
10 een considerable interest in the benefits of high-protein diets.
11 he calcitropic hormones with the medium- and high-protein diets.
12 higher hepatic GDH activity in mice fed the high-protein diets.
14 uirement of young women consuming a constant high-protein diet (1.55 g/kg body wt) and the effect of
16 n diet: 160 kcal/d [95% CI, 102-218 kcal/d]; high protein diet: 227 kcal/d [95% CI, 165-289 kcal/d])
17 ily energy) showed stronger effects than did high-protein diets (25% of daily energy) on reducing con
18 er after the low-protein diet than after the high-protein diet (253 +/- 70 compared with 225 +/- 63 g
19 rotein diet: 2.87 kg [95% CI, 2.11-3.62 kg]; high protein diet: 3.18 kg [95% CI, 2.37-3.98 kg]) incre
20 (P < 0.01) by 20% in subjects consuming the high protein diet (58.2 +/- 2.8 micromol kg-1 h-1, HP +
21 = 658), meal replacements (4 arms; n = 322), high-protein diets (6 arms; n = 865), dietary supplement
22 n diet; or a low-carbohydrate, high-fat, and high-protein diet (66%/18%/16% and 21%/43%/36% calories)
23 se of the large intestine linked to high fat-high protein diets, a dysbiotic microbiome, and a metabo
24 (liver FXR-knockout mice) were re-fed with a high-protein diet after 6 hours fasting and gavaged a (1
25 low-carbohydrate, low-GI, Mediterranean, and high-protein diets all led to a greater improvement in g
32 Low-carbohydrate, low-GI, Mediterranean, and high-protein diets are effective in improving various ma
33 were observed in rats fed ketogenic diets or high-protein diets, but AQP9 levels were elevated in liv
34 kg; median duration: 12 mo (10-26 mo)], and high-protein diets by 1.5 kg [95% CI: 0.8, 2.1 kg; media
35 several nutritional interventions, including high-protein diets, caloric supplementation, calcium and
38 in diet and increased significantly with the high-protein diet compared with the medium-protein diet
39 ffects of normal protein (control) diet with high protein diets containing whey, or its fractions lac
40 sponse of acid excretory pathways in mice to high-protein diets containing normal or low amounts of a
42 eased by 3.7 +/- 0.4 kg with the ad libitum, high-protein diet, despite a significantly decreased lep
44 a demonstrate that short-term consumption of high-protein diets does not disrupt calcium homeostasis
45 omeostasis and bone turnover are affected by high-protein diets during weight maintenance (WM) and ED
48 group (6.05 kg; 95% CI, 4.84-7.26 kg) or the high protein diet group (6.51 kg; 95% CI, 5.23-7.79 kg)
51 t the same time, health concerns relating to high-protein diets have been widespread in the literatur
53 andomized clinical trial demonstrated that a high protein diet (HPRO) combined with neuromuscular ele
54 sistent FoxO activation can be reversed by a high-protein diet in adulthood, through mTORC1 and GCN-2
57 ngs and appetite scores in participants with high-protein-diet intake (P = 0.027 and 0.047, respectiv
58 tention has been focused on low-carbohydrate-high-protein diets (LC-HP) and their potential impact on
59 ardiovascular effects of a low-carbohydrate, high-protein diet (LCHP) in the ApoE(-/-) mouse model of
61 resistance and beta cell function, whereas a high-protein diet may be more beneficial for white patie
62 ting that the much-maligned low-carbohydrate-high-protein diet may have a salutary effect on the epid
67 ced their colonic health, whereas a high fat-high protein diet negatively influenced colonic health,
69 The long-term safety of low-carbohydrate, high-protein diets on cardiovascular disease risk remain
71 ce receiving an isonitrogenic and isocaloric high-protein diet or the AIN-93M diet, and wild-type mic
74 tion and absorption by individuals consuming high-protein diets, particularly when the calcium conten
75 ; continuous feeding of a high carbohydrate, high protein diet, preferably by the enteral route; and
77 on was prevented when the rats were fed on a high-protein diet rich in glutamine, arginine, fish oil,
80 e maple syrup urine disease mice placed on a high protein diet that mimics the catabolic stress shown
84 rength (HGS) compared a standard high-energy high-protein diet to diet plus supplemental nocturnal NG
85 ormula diets have been shown to elevate, and high-protein diets to depress, the tryptophan-LNAA ratio
86 Anti-obesity drugs, meal replacements, and high-protein diets were associated with improved weight-
87 ition, and fat distribution in response to a high-protein diet, whereas an opposite genetic effect wa
90 We evaluated whether an energy-restricted high-protein diet with a low glycemic index and soluble
93 c index (GI), high-fiber, Mediterranean, and high-protein diets with control diets including low-fat,
95 of growth hormone (somatropin) as well as a high-protein diet would ameliorate the symptoms of the d