ライフサイエンスコーパス: high-salt

1 sin II, L-NG-nitroarginine methyl ester, and high salt).

2 alloprotease FtsH as well as the presence of high salt.

3 oes peeling exhibiting hysteresis at low and high salt.

4 tion of stable pre-RCs that are resistant to high salt.

5 ormotensive but become hypertensive when fed high salt.

6 us proteins are significantly less stable in high salt.

7 ule and lead to a surprising re-extension at high salt.

8 ing requires special digestion conditions in high salt (0.3 M KCl) or in Ni2+ buffer.

9 cyanobacterium Synechocystis sp. PCC 6803 in high-salt (0.7 m NaCl) stress but not in mild heat stres

10 of the C-rich RNA in low salt (10 mm KCl) or high salt (100 mm KCl) was typical of mixed sequence RNA

11 iet (percentage of lung: control = 44 +/- 6, high salt = 12 +/- 3, P < 0.05), without reducing primar

12 Fresh minced pork was exposed to either high salt (2 M KCl) only or high salt with lower pH to m

13 ter from 8% to 25% in all buffers except for high salt (250 mM NaCl).

14 processing temperatures (25-35 degrees C) or high salt (30%) concentration are needed, such as in fis

15 Under high salt, a significant proportion of the up-regulated

16 In vivo, in a high-salt administration experiment, male and female Dah

17 High salt also impairs Treg function by inducing IFNgamm

18 esponse of N. thermophilus to external pH at high salt and elevated temperature and identify mechanis

19 preserved sample morphology and removed the high salt and lipid content, which was present in the sa

20 ed with the known tolerance of B. villosa to high salt and the calcium-rich natural habitat of this w

21 The kinase in this fraction was resistant to high salt and Triton X-100 extraction at pH 6.5.

22 : high-salt, low-iron, or a combination of a high-salt and low-iron diet.

23 se environmental conditions, including cold, high salt, and drought.

24 ignificantly more susceptible to drought and high salt, and have increased rates of water loss.

25 < 90% for >12% of the night) were studied in high-salt balance pre- and post-CPAP therapy (>4 h CPAP

26 k contrast to Th17 cells and M1 macrophages, high salt blunted the alternative activation of BM-deriv

27 Exposure of these pellets to a high-salt buffer caused release of the vp13 to the super

28 zed not only by constant low temperature and high salt but also by low light during the austral summe

29 BA levels in Arabidopsis exposed to cold and high salt by differentially controlling NCED3 and NCED5

30 In line with these findings, a moderate high-salt challenge in a pilot study in humans reduced i

31 ditions of relatively high force (>2 pN) and high salt concentration (>0.5 M NaCl).

32 em of hydrophobically driven LLPS induced by high salt concentration (LLPS-HS), and compare it to ele

33 yl toxicity was enhanced when cultured under high salt concentration as a result of BPSS2242 overexpr

34 ationally designed aqueous electrolytes with high salt concentration can effectively resolve the inco

35 Given the relatively high salt concentration of urine, marine bacteria would

36 ic size, and tolerance to a wide pH range or high salt concentration over time.

37 chromatography (HIC) uses mobile phases with high salt concentration that are not compatible with mas

38 of GTP-FtsZ polymers previously observed at high salt concentration was maintained in all KCl concen

39 the microsized natural wood channels (with a high salt concentration) due to their different hydrauli

40 more, upon mutation of the salt bridge or at high salt concentration, an additional kinetic phase was

41 nd activity remain unchanged, or increase at high salt concentration, and that the L. quadripunctata

42 s, the relative populations of conformers at high salt concentration, and the inter-duplex angle (IDA

43 uence effects determine the conformations at high salt concentration.

44 fold when B. pseudomallei was cultured under high salt concentration.

45 sites is important for gene transcription at high salt concentration.

46 ustatory neurons led to the specific loss of high-salt concentration avoidance in larvae, whereas the

47 stability over a wide pH range (4-12) and at high salt concentrations (>100 mM Na(+) or Mg(2+)), brig

48 rticle crystals can be obtained at extremely high salt concentrations and in a divalent salt environm

49 lyplexes containing siRNA in the presence of high salt concentrations and serum proteins.

50 rsion to a random coil structure; whereas at high salt concentrations both dissociation processes occ

51 n suppression often observed in samples with high salt concentrations can be overcome by preparing sa

52 substantial acidification of pI and require high salt concentrations for cooperative folding.

53 For example, high salt concentrations hamper disulfide bond reduction

54 Plant adaptation to high salt concentrations involves integrated functions,

55 s accomplished using alkaline solutions with high salt concentrations or deionized (DI) water.

56 High salt concentrations together with anaerobic conditi

57 ression of the genes encoding the pathway by high salt concentrations was established by transcriptom

58 High salt concentrations were used to establish the elec

59 ibited long-term stability in solutions with high salt concentrations without aggregation or silver e

60 showed attenuated antimicrobial activity at high salt concentrations, as well as lower membrane disr

61 variations in pH, high and low temperatures, high salt concentrations, or in biological media and cel

62 row pH operating range, limited tolerance to high salt concentrations, or/and high cost.

63 m soluble species in atmospheric waters with high salt concentrations, such as aerosols.

64 ese proteins on membranes are insensitive to high salt concentrations, suggesting a nonelectrostatic

65 At high salt concentrations, the AmPrbetaCD blockage of the

66 lized as adsorbed 3D-projected coils; (c) at high salt concentrations, the polymer coils reexpand and

67 However, at medium to high salt concentrations, this trend is reversed, and ne

68 attractive potential well at intermediate-to-high salt concentrations, which demonstrates that electr

69 I, a betaFP that forms beta-sheet fibrils at high salt concentrations.

70 often characterized by extremely low pHs and high salt concentrations.

71 that NCBD undergoes a charge reversal under high salt concentrations.

72 issolution and was facilitated in media with high salt concentrations.

73 hat glyoxal uptake is kinetically limited at high salt concentrations.

74 eatest increase in expression in response to high salt concentrations.

75 xternal stimulus set by exposure of cells to high salt concentrations.

76 t salting-out effect for these ions, even at high salt concentrations.

77 n solution but forms dimers and tetramers at high salt concentrations.

78 Target DNA is incubated with the plate in high salt concentrations.

79 ions provide an attractive stimulus, whereas high-salt concentrations are avoided.

80 d resists denaturation in strong detergents, high-salt concentrations, and ionic liquids.

81 equired for neuronal activity in response to high-salt concentrations.

82 larval gustatory organs for the detection of high-salt concentrations.

83 ion also eliminated the cellular response to high-salt concentrations.

84 is responsible for inhibiting egg-laying at high-salt concentrations.

85 Because DNA is strongly charged, high salt conditions are required to enable binding betw

86 Diverse stresses such as high salt conditions cause an increase in reactive oxyge

87 evealed transitions between two states under high salt conditions, but smFRET could not determine whe

88 nds and two crossover strands, stabilized by high salt conditions.

89 High-salt conditions activate the p38/MAPK pathway invol

90 s mutation conferred increased resistance to high-salt conditions and oxidative stress.

91 The TH17 cells generated under high-salt conditions display a highly pathogenic and sta

92 differences linked with adduct formation in high-salt conditions explain the molecular species obser

93 Saccharomyces cerevisiae self-splices under high-salt conditions in vitro, but requires the assistan

94 High-salt conditions lowered the chlorophyll and phycobi

95 nduction of many of these stress genes under high-salt conditions was significantly lower in flp-1 my

96 n of the choline pool inhibited growth under high-salt conditions with choline as the sole carbon sou

97 tant and acetonitrile phase separation under high-salt conditions.

98 holipase production and impacts growth under high-salt conditions.

99 respectively, than in the control strain in high-salt conditions.

100 DNA and that the MCM complex is stable under high-salt conditions.

101 nts in the presence of deoxyribonuclease and high-salt conditions.

102 A Western lifestyle with high salt consumption can lead to hypertension and cardi

103 Infected gerbils consuming diets with a high salt content developed gastric ulcers significantly

104 ufficiently robust to analyze samples with a high salt content.

105 In vitro, high salt decreased the ability of M(IL-4+IL-13) macroph

106 Exposure to combinations of lower pHs and high salt decreased water-holding and increased surface

107 On high salt diet (HS), BPs of hAS(+/-) mice were significa

108 xamined murine models of colitis on either a high salt diet (HSD) or a low salt diet.

109 In Sprague-Dawley rats, high salt diet activated c-Src and induced redistributio

110 greatly reduced in Pkd1 knockout mice fed a high salt diet compared with controls.

111 r in VP neurons from animals that were fed a high salt diet compared with controls.

112 Moreover, mice fed a high salt diet exhibited reduced M2 activation following

113 rane potential observed in VP neurons in the high salt diet group.

114 itro and ex vivo results, Efnb1 KO mice on a high salt diet showed a statistically significant height

115 Specifically, in Dahl R rats, high salt diet significantly stimulated phosphorylation

116 m by which an environmental factor such as a high salt diet triggers TH17 development and promotes ti

117 l salt-sensitive (Dahl-S) rats were fed with high salt diet with or without 0.1% caffeine in drinking

118 -regulated in Dahl salt-sensitive rats fed a high salt diet.

119 Rats treated with high-salt diet (5-9 weeks after I/R) manifested progress

120 Dahl salt-sensitive rats were fed a high-salt diet (8% NaCl) from 7 weeks of age to induce H

121 Dahl salt-sensitive rats were fed a high-salt diet (8% NaCl) from 7 weeks of age to induce H

122 pecifically, we investigated the effect of a high-salt diet (a known risk factor for gastric adenocar

123 llenged with deoxycorticosterone acetate and high-salt diet (DOCA-salt).

124 O(3) -induced metabolic alkalosis (MAlk) and high-salt diet (HSD) also increase expression of NBCn1 a

125 e MPS cells prevents the VEGFC response to a high-salt diet (HSD) and increases blood pressure.

126 A high-salt diet (HSD) in humans is linked to a number of

127 development of hypertension in SS rats on a high-salt diet (n = 7-8, p < 0.05).

128 e (n=15) were fed normal chow or a high-fat, high-salt diet (WD).

129 A high-salt diet also impaired human Treg function and was

130 In a separate experiment, a high-salt diet and subcutaneous angiotensin II was admin

131 bacter pylori infection and consumption of a high-salt diet are each associated with an increased ris

132 were detected in the H. pylori-infection and high-salt diet combined group compared with the other gr

133 cagA transcription in vivo in animals fed a high-salt diet compared to those on a regular diet.

134 Moreover, mice fed with a high-salt diet develop a more severe form of EAE, in lin

135 In C57BL6/J mice, exposure to a high-salt diet exacerbated disease in both sexes, while

136 genotype (AG vs. AA) and fed them a low- or high-salt diet for 1 week, after which they were challen

137 ificantly increased when rats had been fed a high-salt diet for 7 days (n = 6 or 9, p < 0.01).

138 als infected with the WT strain, those fed a high-salt diet had more severe gastric inflammation, hig

139 ate that cortical EGF levels decrease with a high-salt diet in salt-sensitive rats, promoting ENaC-me

140 model combined with H. pylori infection and high-salt diet is useful for gene expression profiling i

141 We propose a model in which a high-salt diet leads to high levels of gastric inflammat

142 Feeding these salt-sensitive rats a high-salt diet led to lower levels of EGF in the kidney

143 A high-salt diet markedly increased lupus features in MRL/

144 isolated from Mongolian gerbils fed either a high-salt diet or a regular diet for 4 months by proteom

145 e model was also reduced after exposure to a high-salt diet or induced CKD.

146 ference comparable to the changes induced by high-salt diet or saline infusion.

147 These results indicate that a high-salt diet potentiates the carcinogenic effects of c

148 diet, the output strains from animals fed a high-salt diet produced higher levels of proteins involv

149 After 7 weeks of a high-salt diet, 31 of 38 rats showed diastolic dysfuncti

150 In Dahl salt-sensitive rats that were fed a high-salt diet, a model for hypertension-induced congest

151 On a high-salt diet, all mice with D(5)(-/-) kidneys excreted

152 To assess whether a high-salt diet, as measured by urinary sodium concentrat

153 Within 10 days on a high-salt diet, BP increased similarly in ES and SS allo

154 When mice were fed with a high-salt diet, knockdown of miR-192 blunted the adaptat

155 During the first week on a high-salt diet, SS rats and SS rats with only one functi

156 Additional studies showed that, on a high-salt diet, Tmem27(Y/-) mice had lower renal blood f

157 To investigate the effects of a high-salt diet, we infected Mongolian gerbils with a wil

158 On a high-salt diet, WT mice with Tmem27(Y/-) kidneys had the

159 e versus 32.0 mmHg in WT mice (P<0.01) fed a high-salt diet.

160 xpression in the macula densa increases on a high-salt diet.

161 ce (P<0.01) but not NOS1flox/flox mice fed a high-salt diet.

162 transgenic mice, an effect accentuated by a high-salt diet.

163 difference (PDamil) than control mice fed a high-salt diet.

164 insensitive, congenic SS.13(BN26) rats fed a high-salt diet.

165 oup were inoculated with H. pylori and fed a high-salt diet.

166 ed by combination of H. pylori infection and high-salt diet.

167 aintained the animals on a regular diet or a high-salt diet.

168 model combined with H. pylori infection and high-salt diet.

169 ly, developed hypertension when exposed to a high-salt diet.

170 ood pressure at baseline or in response to a high-salt diet.

171 t develop elevated blood pressure when fed a high-salt diet.

172 noma was detected in 100% of the WT-infected/high-salt-diet animals, 58% of WT-infected/regular-diet

173 enesis and causes organismal lethality under high-salt dietary stress.

174 Whilst high salt diets have been shown to worsen autoimmune dis

175 ction of proinflammatory Th17 cells and that high-salt diets exacerbate experimental models of autoim

176 and potassium did not change with regular or high-salt diets or potassium loading in control or Scnn1

177 The high-salt DNA extraction procedure utilizes green solven

178 is due to electroosmotic flow separation, a high-salt electrokinetic effect.

179 of UWO 241 to its unique low-temperature and high-salt environment favors the phosphorylation of a PS

180 when trying to analyze organic species in a high-salt environment.

181 d Sea in 1975, Haloferax volcanii thrives in high salt environments and has emerged as an important a

182 tures, metabolic pathways, and adaptation to high salt environments.

183 ish are freshwater fish that cannot tolerate high-salt environments and would therefore benefit from

184 n has not been well characterized in natural high-salt environments.

185 ope-dilution measured lower lesion levels in high-salt extracts than in phenol extracts.

186 ow salt favoring the closed conformation and high salt favoring the open conformation in the absence

187 uninephrectomized, aldosterone-infused, and high salt-fed (ALDO) systemic GC-A KO mice with enhanced

188 reased after subtotal nephrectomy and during high-salt feeding, raising the question of whether colle

189 combinations, and possibly processed and/or high salt foods.

190 scenarios were developed: 1) substitution of high-salt foods with low-salt foods, 2) a reduction in t

191 Pro, and Suc) accumulated to high levels in high salt-grown UWO 241 cultures.

192 High salt-grown UWO 241 exhibited increased thylakoid pr

193 Adding a high-salt, high-fat diet accelerates endothelial senesce

194 High salt, however, compromises prospects for label-free

195 e initially fed with either low salt (LS) or high salt (HS) diet for a period of 6 weeks, followed by

196 wn that some of the deleterious effects of a high-salt (HS) diet are independent of elevated blood pr

197 study aimed to assess the effect of a 1-week high-salt (HS) diet on the role of cyclo-oxygenases (COX

198 in Dahl salt-sensitive (SS) rat attenuated a high-salt (HS)-induced increase in epithelial Na(+) chan

199 L-NAME/high salt increased macrophage and dendritic cell surfac

200 the grik1-2 grik2-1 mutant was sensitive to high salt, indicating that GRIKs are also involved in sa

201 ot only explain the epigenetic mechanisms of high-salt induced autoimmunity but also provide an attra

202 We further identified a high salt-induced reduction in glycolysis and mitochondr

203 ally, in rats treated with an ODN to prevent high salt-induced up-regulation of brain Galphai(2) prot

204 protected renal function from IR, attenuated high-salt-induced AKI-to-CKD progression in rats, and de

205 The angiotensin/high-salt-induced increase in systolic blood pressure, p

206 ion of p38/MAPK, NFAT5 or SGK1 abrogates the high-salt-induced TH17 cell development.

207 Finally, chronic high-salt ingestion produces endothelial dysfunction, ev

208 Here we show that high salt intake affects the gut microbiome in mice, par

209 ypertension produced by the combination of a high salt intake and administration of angiotensin II, t

210 rt the unexpected observation that long-term high salt intake did not increase water consumption in h

211 Here we show that chronic high salt intake impairs baroreceptor inhibition of rat

212 We show that high salt intake increases the spontaneous firing rate o

213 costerone-acetate (DOCA) in combination with high salt intake induced arterial hypertension of simila

214 onstrate that osmotic balance in response to high salt intake involves a complex regulatory process t

215 High salt intake is a major risk factor for hypertension

216 aintenance of osmotic balance in response to high salt intake is a passive process that is mediated l

217 High salt intake is a top dietary risk factor.

218 High salt intake leads to high blood pressure, even when

219 Our results connect high salt intake to the gut-immune axis and highlight th

220 include Helicobacter pylori infection, age, high salt intake, and diets low in fruit and vegetables.

221 In conditions of high salt intake, defective uromodulin processing predis

222 , thereby influencing BP under conditions of high salt intake.

223 between the knockout and control mice during high salt intake.

224 grally involved in the vascular responses to high salt intake.

225 were increased in Pkd1 knockout mice during high salt intake; administration of NS-398, a selective

226 e exhibited reduced arterial pressure during high salt intake; this associated with an increased natr

227 mice were challenged to a normal- or chronic high-salt intake (1% NaCl).

228 o levels that may occur in human blood after high-salt intake can potentiate, in serum-free culture c

229 high blood pressure development triggered by high-salt intake through the modulation of the contracti

230 After 4 weeks of high-salt intake, ES rats still showed a lower mean seru

231 During high-salt intake, the sik1(+/+) mice exhibited an increa

232 nd DeltanW, the dominant folded structure at high salt is most likely the antiparallel stacked-X stru

233 High salt levels interfere with alternative activation o

234 tible with mass spectrometry (MS) due to the high salt levels, it is laborious to identify the consti

235 eased amounts of reactive oxygen species and high salt levels.

236 Furthermore, Pax8 expression is induced by high-salt levels in collecting duct cells and activates

237 ial pressure and enhanced natriuresis during high salt loading in Pkd1 knockout mice were associated

238 ility toward environmental stressors such as high salt, low pH, reactive oxygen radicals, and cell wa

239 sociated with increased H. pylori virulence: high-salt, low-iron, or a combination of a high-salt and

240 opuABCD mutant strains are more resistant to high-salt, low-pH and -hydrogen peroxide, conditions tha

241 levels were in infected gerbils consuming a high-salt/low-iron diet.

242 High salt may additionally drive autoimmunity by inducin

243 d, in cultured dendritic cells we found that high salt media potentiates cytokine expression downstre

244 Efficient locomotion in urine samples and in high-salt media is illustrated.

245 ed molar concentrations of KCl when grown in high salt medium as detected by x-ray microanalysis and

246 decorated this residue in cells grown in the high-salt medium.

247 effect has been demonstrated using heat, pH, high salt mediums, and high energy ionising radiation.

248 In the L-NAME/high-salt model, memory T cells of the kidney were predo

249 this work, we present a facile method termed high-salt molecular rheotaxis (HiSMRT) to concentrate an

250 Exposure to high salt only increased water-holding of myofibrils and

251 of the E1841K mutation in mice subjected to high salt or angiotensin II (Ang II) as models of hypert

252 arginine methyl ester hydrochloride (L-NAME)/high salt or repeated angiotensin II stimulation in mice

253 s and did not develop hypertension to either high salt or the second angiotensin II challenge and wer

254 ers to solid surfaces is severely limited by high salt, pH, and hydration, yet these conditions have

255 tic digestion of the C. elegans cuticle with high-salt phase-separation of DNA has been developed and

256 We show that high salt recruits the two primary aversive taste pathwa

257 In contrast, hypoxia, the dauer state, and high salt reduce touch sensitivity by preventing the rel

258 lectively, this study provides evidence that high salt reduces noninflammatory innate immune cell act

259 e interactions of glass nanopipettes in this high-salt regime with a variety of surfaces and propose

260 logy applications, but their scalability and high salt rejection when in a strong cross flow for long

261 emerging water treatment technology that has high salt rejection; however, its commercialization pote

262 have a maximum allowable charge capacity, a high salt removal rate in flow-electrode capacitive deio

263 ing upgraded FCDI units exhibiting extremely high salt removal rates (>100 mg m(-2) s(-1)), good cycl

264 Consistent ZLD performance of high salt removals and product water quality was maintai

265 Formation of stable complexes, resistant to high salt, requires ATP hydrolysis.

266 UWO241 grown under its natural condition of high salt resulted in swelling of the thylakoid lumen.

267 In high salt, rHb (betaE6V/alphaH20R) is the only mutant th

268 In high salt, S-layer glycoprotein Asn-13 and Asn-83 are mo

269 The multivariable-adjusted odds ratio of high salt sensitivity of systolic BP was 0.66 (95% CI: 0

270 High salt sensitivity was defined as a decrease in mean

271 under challenging physiological conditions (high salts, serum, and acidic pH).

272 mildly increased albuminuria in response to high salt; severe albuminuria, nephrinuria, FSGS, and po

273 In this study, high salt (sodium chloride, NaCl), under physiological c

274 to an ATP-sepharose matrix and washed with a high salt solution followed by nicotinamide adenine dinu

275 ts on fluid balance following ingestion of a high-salt solution-rats produced significantly more urin

276 ects the nanoparticles from aggregating in a high-salt solution.

277 ong-term stability in biological buffers and high-salt solutions.

278 ATII-LCL mercuric reductase is functional in high salt, stable at high temperatures, resistant to hig

279 ptidergic signaling potentiates responses to high salt stimuli, which may promote ion homeostasis.

280 Exposure to a high salt stress (150 mM NaCl) triggered a rapid repress

281 creased plant sensitivity toward osmotic and high-salt stress, indicating that NatB is required for t

282 However, it remains unclear how low- and high-salt taste perceptions are differentially encoded.

283 inhibition by flooding and anoxia, drought, high salt, the presence of fungal and bacterial pathogen

284 4 tail deletion suppresses the attraction at high salts to a larger extent than H3 tail deletion.

285 ting factor for this food association is its high salt tolerance allowing this organism to survive co

286 ytes and provided good reproducibility and a high salt tolerance, underscoring the potential applicat

287 Skin macrophages from high-salt-treated mice with either genetic or pharmacolo

288 nd that macrophages isolated from kidneys of high-salt-treated WT mice have increased levels of COX-2

289 Kidneys from high-salt-treated WT mice transplanted with Cox2-/- BM h

290 ith low-salt treatment 6.6 mg/m(3) (n = 14), high-salt treatment 10.8 mg/m(3) (n = 15) or placebo 0.3

291 -associated intermediate that is stable upon high-salt treatment and other MHR mutants arrested as la

292 In the high-salt treatment group, it was 12 192 mug/l vs 11 803

293 Further, coupling this with post-binding high-salt washes and a brief, low-percentage formaldehyd

294 formaldehyde cross-linking step prior to the high-salt washes provided the optimal balance between re

295 ge binding capacity for U sequestration from high salt water (HSW) simulant (54 mg U/g sorbent).

296 the opposing behavioral responses to low and high salt were determined largely by an elegant bimodal

297 At high salt, wild-type SSB and a variant without the IDL,

298 e pool, inhibited growth under conditions of high salt with glucose as the primary carbon source.

299 xposed to either high salt (2 M KCl) only or high salt with lower pH to mimic conditions in freezing.

300 caffeine attenuates hypertension induced by high salt without affecting sympathetic nerve activity i