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1 tat1 (alpha-tubulin acetyltransferase 1) per higher organism.
2 chanical stresses in yeast and in cells from higher organisms.
3 Gamete formation is key to survival of higher organisms.
4 chromosome conformations in prokaryotes and higher organisms.
5 contributed to functional diversification in higher organisms.
6 c microorganisms pose severe problems to all higher organisms.
7 nctional precursor to the internal memory of higher organisms.
8 son capsids, including those domesticated in higher organisms.
9 ction of hydrogen sulfide, which is toxic to higher organisms.
10 ction on DNA in bacteria and in chromatin of higher organisms.
11 egions, with synaptic functions conserved in higher organisms.
12 peat structures found in the genomes of most higher organisms.
13 onal interaction has yet to be identified in higher organisms.
14 uggest a similar pathway may be conserved in higher organisms.
15 mic sequence, and they define cell status in higher organisms.
16 ted extensively to manipulate the genomes of higher organisms.
17 ughter cells in a process akin to mitosis in higher organisms.
18 d cooperation are not restricted to complex, higher organisms.
19 control of developmental gene expression in higher organisms.
20 deal with the complex functional demands of higher organisms.
21 cient secretion of all collagen molecules in higher organisms.
22 ids are regulators of body shape and size in higher organisms.
23 nism controlling protein localization in all higher organisms.
24 circuits controlling spatial orientation in higher organisms.
25 minant of tissue size in both Drosophila and higher organisms.
26 imal phyla suggests mechanistic relevance to higher organisms.
27 rm our understanding of germ cell biology in higher organisms.
28 ays previously known to be available only to higher organisms.
29 but significantly less than that observed in higher organisms.
30 functional genomics and network inference in higher organisms.
31 s critical for the proper development of all higher organisms.
32 of how miRNAs function in the development of higher organisms.
33 ein diversity in eukaryotes, particularly in higher organisms.
34 is essential for normal development in many higher organisms.
35 a signaling molecule and cytotoxic agent in higher organisms.
36 icardial-myocardial interactions relevant to higher organisms.
37 m to enhance cellular transport in yeast and higher organisms.
38 enorhabditis elegans, but is not possible in higher organisms.
39 t, metabolism, and aging in nematodes and in higher organisms.
40 ntrol of gene expression may have evolved in higher organisms.
41 important families of structural proteins in higher organisms.
42 lex gene expression patterns particularly in higher organisms.
43 important role for 3'-UTRs in the biology of higher organisms.
44 mpaction within defined chromatin domains in higher organisms.
45 ionally effective mimics of glycoproteins in higher organisms.
46 ure-function relations with P2X receptors of higher organisms.
47 on may serve as a regulatory modification in higher organisms.
48 yotes to their ability to elicit immunity in higher organisms.
49 ogenetic patterns that may have parallels in higher organisms.
50 hat distinguish it from the complex of other higher organisms.
51 and the impact mitochondria have on aging in higher organisms.
52 ered in the soil or defined as microbiota of higher organisms.
53 e a framework to study commensal biofilms in higher organisms.
54 nt can be found in developmental programs in higher organisms.
55 ommunication and other processes specific to higher organisms.
56 erve transmission and the immune response of higher organisms.
57 uggests that they may serve some function in higher organisms.
58 to regulate complex segmental body plans in higher organisms.
59 e RNase P holoenzyme from archaebacteria and higher organisms.
60 uggest a unique regulatory role for eIF3k in higher organisms.
61 of functions appears to be conserved in all higher organisms.
62 ts is essential for survival and behavior of higher organisms.
63 nding tissue-level electric field effects in higher organisms.
64 c changes in viruses, bacteria, and genes of higher organisms.
65 eton play an important role in cell shape in higher organisms.
66 ight regulate critical cellular processes in higher organisms.
67 haracteristic of all grasses and perhaps all higher organisms.
68 m for understanding dopaminergic function in higher organisms.
69 ing domains has so far been observed only in higher organisms.
70 er, little is known about PMR1 homologues in higher organisms.
71 molecules that carry out analogous roles in higher organisms.
72 stows upon bacteria some of the qualities of higher organisms.
73 tion of other symbiotic associations between higher organisms.
74 ome bacterial cytochromes c(1) but absent in higher organisms.
75 ed to other aspects of aging in yeast and in higher organisms.
76 mic and proteomic data, both in yeast and in higher organisms.
77 y tissue-specific physiological responses in higher organisms.
78 e more complex signaling pathways present in higher organisms.
79 This pathway appears to be unique to higher organisms.
80 re critical for successful interactions with higher organisms.
81 for the annotation of complete genomes from higher organisms.
82 or the regulation of phenotypic diversity of higher organisms.
83 ns is of great importance to the survival of higher organisms.
84 rb regulatory systems that control ageing in higher organisms.
85 olutionary forerunner of P2Y(1) receptors of higher organisms.
86 ue domains are widespread among bacteria and higher organisms.
87 ggests spatial organization of glycolysis in higher organisms.
88 their role as agents of natural selection in higher organisms.
89 abolic rate and aging in yeast and, perhaps, higher organisms.
90 ed into much larger tRNA-binding proteins of higher organisms.
91 ant part of the energy homeostasis system of higher organisms.
92 -epimerase has been isolated previously from higher organisms.
93 gest that similar results may be obtained in higher organisms.
94 iption factors involved in such responses in higher organisms.
95 ction(s) related to embryonic development in higher organisms.
96 ion dominate the mRNA stability landscape in higher organisms.
97 t for the wingless/wnt signaling pathways in higher organisms.
98 hromatin structure studies in both yeast and higher organisms.
99 undancy seems to be widespread in genomes of higher organisms.
100 hat could be used toward similar purposes in higher organisms.
101 action system itself-toward the evolution of higher organisms.
102 ancient and ubiquitous defense mechanism in higher organisms.
103 he extracellular matrix adhesion proteins of higher organisms.
104 onfer extracellular and nuclear functions in higher organisms.
105 lutionary conservation of the Gcm cascade in higher organisms.
106 l wave formation in the respiratory cilia of higher organisms.
107 an reveal novel components and mechanisms in higher organisms.
108 gnition motifs within replication origins of higher organisms.
109 complexes are highly conserved from yeast to higher organisms.
110 th several mechanistic aspects distinct from higher organisms.
111 and transfer through the aquatic food-web to higher organisms.
112 g cellular toxicities and ribosomopathies in higher organisms.
113 provides an essential source of N atoms for higher organisms.
114 ble to morphogenesis processes in tissues in higher organisms.
115 ptimizing and widening their applications in higher organisms.
116 NAs in other bacteria and regulatory RNAs in higher organisms.
117 ulation, the sulphur cycle and signalling to higher organisms.
118 ental process underlying the behavior of all higher organisms.
119 ereby providing essential nitrogen atoms for higher organisms.
120 s a cold and pain sensor in humans and other higher organisms.
121 te highly important communication systems in higher organisms.
122 oli may help to predict toxicity patterns in higher organisms.
123 cular ageing factors conserved from yeast to higher organisms.
124 networks of chromatin-based interactions in higher organisms.
125 heir targets across bacterial species and in higher organisms.
126 anism regulating histone deubiquitination in higher organisms.
127 ortant epigenetic modification in the DNA of higher organisms.
128 ce, a role for P4Hs that may be conserved in higher organisms.
129 involved in sensing touch and sound waves in higher organisms.
130 t account for the helicase domain present in higher organisms.
131 d regulation of distinct subsets of genes in higher organisms.
132 n is structurally homologous to hnRNP-C from higher organisms.
133 riety of oxidation reactions in microbes and higher organisms.
134 thylation is an epigenetic control factor in higher organisms.
135 questions about how these results relate to higher organisms.
136 ty to suggested mineralization mechanisms in higher organisms.
138 ent from the specificity factors employed in higher organisms, a comparison of the sigma/RNA polymera
140 ins to species richness are partly caused by higher organism abundance and are common in plants and b
141 ranzymes) play a critical role in protecting higher organisms against intracellular infections and ce
144 igh-pressure-inducible phenomena observed in higher organisms, anaesthetics antagonize high-pressure
145 n important tool for genetic manipulation of higher organisms and a model for site-specific DNA-recom
146 nated Sin3A and Sin3B have been described in higher organisms and although functional differences bet
147 stability and expression within the cells of higher organisms and can exhibit specific antigen recogn
148 is widely expressed in nearly all tissues of higher organisms and couples cellular energy status to m
151 system with features common to those used by higher organisms and outlines a novel mechanism for deli
152 provide an example of trans-kingdom RNAi in higher organisms and suggest the potential of bacteria-m
154 se LIN proteins have also been identified in higher organisms, and here we analyze the MALS/Veli (mam
155 core set of regulatory factors conserved in higher organisms, and the complex pattern of EB1 targeti
156 tant consideration given that the genomes of higher organisms are riddled with partial tRNA sequences
158 ondrial receptors for DRP-1-like proteins in higher organisms as well and that BH3-only proteins play
160 mbination and repair in bacteria, and in all higher organisms as well, due to the functional and stru
162 Homologous enzymes may be widespread in higher organisms, as sulfide-oxidizing activities have b
165 -deficient mice to succumb more quickly with higher organism burden, increased lung pathology, and de
167 are often targets of natural selection among higher organisms, but quantifying the effects of such se
168 utes to the complexity of gene expression in higher organisms, but the regulation of the splice site
170 odulation of functional amyloid formation in higher organisms can be accomplished through alternative
174 in genetics textbooks, gene transcription in higher organisms cannot be fully understood by analysing
176 Casein kinase CK2 is an essential enzyme in higher organisms, catalyzing the transfer of the gamma p
178 hough they share similar folds, the FEN1s of higher organisms contain a 3'-extrahelical nucleotide (3
179 nase activity than is present in the PKGs of higher organisms containing only two allosteric sites.
180 t an intermediate degree of sensitivity, and higher organisms (containing cholesterol) are largely re
181 ese results we propose that GST evolution in higher organisms could be linked to the defense against
187 ultiple molecules and elements reflects that higher organism develops a complex translation regulator
189 t value to the study of related processes in higher organisms due to the growing evidence for the cro
191 triction and oxidative stress, features that higher organisms exploit in defending themselves against
193 h may prove useful to study gene function in higher organisms for which transgenic technology is avai
195 ation events occurring within the genomes of higher organisms, for example, detecting alternative RNA
196 der to construct gene regulatory networks of higher organisms from gene expression and promoter seque
202 wever, the complexity of circadian clocks in higher organisms has prevented a clear understanding of
204 t opening of the heme crevice, suggests that higher organisms have evolved to inhibit peroxidase acti
205 ed to test for dsRNA interference effects in higher organisms in which it is feasible to construct tr
208 likely to elucidate circadian timekeeping in higher organisms, including how transcription and transl
215 te that degradation of proteins by MPCs from higher organisms involves a nonprocessive mechanism comp
222 his overview is that the unit of function in higher organisms is neither the genome nor the cell alon
225 g evidence indicates that gene expression in higher organisms is regulated by RNA polymerase II stall
230 which is essential for normal development in higher organisms, is one such macromolecular machine.
231 nal circuitry that takes years to develop in higher organisms, it also poses a major obstacle for CNS
232 es by plants suggested toxicological risk to higher organisms known to utilize macrophytes as a food
233 ortant for the interactions of bacteria with higher organisms - leading to rhizosphere colonization a
234 e presence of highly conserved paralogues in higher organisms led us to assess whether compensation b
235 ng in both lower organisms like bacteria and higher organisms like yeast, which allows them to prefer
238 fe-span extension by CR and suggest that, in higher organisms, multiple members of the Sir2 family ma
242 one's own body, is a fundamental ability of higher organisms, playing a central role in many percept
245 ained and propagated within somatic cells of higher organisms, recent in vitro and in vivo evidence d
246 E-B unstructured C-terminal domain unique to higher organisms regulates DUE-B intermolecular binding.
248 the circadian clock in Drosophila and other higher organisms relies on the quantification of rhythmi
250 de novo pathway in bacteria and plants, most higher organisms rely on a salvage pathway that phosphor
251 t molecular mechanisms, both prokaryotes and higher organisms rely on programmed genetic variation to
257 uent quantitative analysis of total RNA from higher organisms revealed varying levels and TET-indepen
258 r based on host defense peptides (HDPs) from higher organisms, show promising activity against human
259 ew the relevant literature on tRF biology in higher organisms, single cell eukaryotes, and prokaryote
266 tingly, R(442) is conserved in most STATs in higher organisms, suggesting conservation of function.
267 he amino acid sequences of p67 from lower to higher organisms suggests that there is a progressive ad
268 e closely related to 2-Cys peroxiredoxins of higher organisms than to most other eubacterial AhpC pro
269 peptides are important defense compounds of higher organisms that can be used as therapeutic agents
270 tion is a post-translational modification of higher organisms that deiminates arginines in proteins a
271 Redundancy is common among phenotypes of higher organisms that experience low mutation rates and
273 network of apoptotic pathways has evolved in higher organisms that possess homologs within each set o
274 a class of small cationic peptides found in higher organisms that serve as both antimicrobial and ce
275 tances, directly influence interactions with higher organisms, the broader physiological significance
277 disadvantages like notable toxicity against higher organisms, the high price, and low abundance of s
279 of TyrRS directly controlled by tRNA(Tyr) in higher organisms, the NLS of lower eukaryotes was abando
281 genetic pathways and pathway components with higher organisms, the study of its interaction with bact
282 s an integral role in calcium homeostasis in higher organisms through its actions in the intestine, k
284 nt a new class of genes that have evolved in higher organisms to govern the synthesis of highly speci
285 tic life cycle, as well as traits that allow higher organisms to survive rare environmental disasters
287 ein degradative process that is activated in higher organisms under conditions of prolonged starvatio
288 llular apoptosis: why do the mitochondria of higher organisms, unlike their bacterial ancestors, use
289 espite what is known about apocarotenoids in higher organisms, very little is known about apocaroteno
290 hese postulates are valid for the MPC from a higher organism, we examined the size distributions of p
291 d feature prediction tools in the genomes of higher organisms, we evaluated their performance on a la
292 , inspired by scaffold-directed signaling in higher organisms, we modularize prokaryotic signal trans
293 t composite TFBS elements, commonly found in higher organisms where two or more TFBSs form functional
294 n of replicating entities (molecules, cells, higher organisms), where evolutionary principles prevail
296 biosynthesis requires 10 enzymatic steps in higher organisms, while prokaryotes require an additiona
297 ted at the beginning of the fossil record of higher organisms, while the differences between the anci
299 head: understanding the molecular biology of higher organisms will require revealing all proteins (Pr
300 teractions between vibrios, (micro)algae and higher organisms, with major ecological and practical im