1 pid levels, and a neuromuscular abnormality (hind-limb clasping).

2  They also exhibited reduced body weight and hind limb clasping.

3 e absence of striatal pathology, begins with hind-limb clasping during tail suspension and tail stiff

4                                     Notably, hind limb clasping in Abcd1-/y mice was corrected throug

5 vere neurobehavioral abnormalities including hind limb clasping, overt seizures, motor impairment and

6 ge of phenotypes including ataxia, front and hind limb clasping, reduced brain size, and smaller neur

7 ral deficits on the accelerating rotorod and hind limb clasping tests in transgenic HD mice.

8 tionally, Kcne3(-/-) mice exhibited abnormal hind-limb clasping upon tail suspension (63% of Kcne3(-/

9 enic mouse (2D2(+) mouse) that presents with hind-limb clasping upon tail suspension and is associate

10 ce exhibited a relapsing-remitting course of hind-limb clasping with the development of progressive m